ライフサイエンスコーパス: microangiopathic

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1 ischemic-hemorrhagic transformation and/or a microangiopathic basis for the association of ischemic l

2 As part of the ancillary Genetics of Microangiopathic Brain Injury (GMBI) study, a neurocogni

3 We investigated microangiopathic changes in the cerebellum of 16 genetic

4 e classified as confirmed (diarrhea, anemia, microangiopathic changes, low platelet count, and acute

5 ing and atherogenesis in diabetes and to the microangiopathic complications of the disease.

6 at causes hemolytic uremic syndrome (HUS), a microangiopathic disease characterized by hemolytic anem

7 rombotic thrombocytopenic purpura (TTP) is a microangiopathic disorder diagnosed by thrombocytopenia

8 ated in the pathogenesis of the catastrophic microangiopathic disorder, thrombotic thrombocytopenic p

9 will be made on the prototypes of thrombotic microangiopathic disorders, resulting from complement or

10 insights into the pathogenesis of thrombotic microangiopathic disorders.

11 asis for future classification of thrombotic microangiopathic disorders.

12 The failing grafts exhibited thrombotic microangiopathic glomerulopathy with multiple platelet-f

13 ient graft dysfunction with reversible, mild microangiopathic glomerulopathy, probably associated wit

14 ssociated with the development of thrombotic microangiopathic glomerulopathy.

15 ndrome characterized by thrombocytopenia and microangiopathic haemolytic anaemia, was almost universa

16 ity, which was followed by resolution of the microangiopathic hemolysis and improvement of the graft

17 gnificant protection from the development of microangiopathic hemolysis and renal insufficiency.

18 rovasculature resulting in thrombocytopenia, microangiopathic hemolysis, and organ dysfunction.

19 are both characterized by thrombocytopenia, microangiopathic hemolysis, and organ dysfunction.

20 TP) is characterized by thrombocytopenia and microangiopathic hemolytic anemia (MAHA) without an obvi

21 pathophysiological pathways that can lead to microangiopathic hemolytic anemia and a procoagulant sta

22 Profound thrombocytopenia and microangiopathic hemolytic anemia characterize thromboti

23 ated with hemolytic uremic syndrome (HUS), a microangiopathic hemolytic anemia characterized by throm

24 The infant displayed severe microangiopathic hemolytic anemia with renal involvement

25 drome (HUS), a disorder of thrombocytopenia, microangiopathic hemolytic anemia, and acute renal failu

26 with the clinical features of hypertension, microangiopathic hemolytic anemia, and acute renal failu

27 ndrome characterized by acute renal failure, microangiopathic hemolytic anemia, and thrombocytopenia

28 disorder characterized by thrombocytopenia, microangiopathic hemolytic anemia, mental status changes

29 ay also be associated with thrombocytopenia, microangiopathic hemolytic anemia, neurologic symptoms,

30 cell injury of various origins, resulting in microangiopathic hemolytic anemia, platelet consumption,

31 thrombotic microangiopathy characterized by microangiopathic hemolytic anemia, severe thrombocytopen

32 odes of small-vessel thrombosis resulting in microangiopathic hemolytic anemia, thrombocytopenia and

33 Microangiopathic hemolytic anemia, thrombocytopenia, and

34 Hemolytic uremic syndrome is a triad of microangiopathic hemolytic anemia, thrombocytopenia, and

35 emolytic uremic syndrome (HUS) is a triad of microangiopathic hemolytic anemia, thrombocytopenia, and

36 tic microangiopathy that is characterized by microangiopathic hemolytic anemia, thrombocytopenia, and

37 me (aHUS), a rare condition characterized by microangiopathic hemolytic anemia, thrombocytopenia, and

38 ment at the cell surface level that leads to microangiopathic hemolytic anemia, thrombocytopenia, and

39 ura (TTP) is the common name for adults with microangiopathic hemolytic anemia, thrombocytopenia, wit

40 the common features of thrombocytopenia and microangiopathic hemolytic anemia.

41 unction deteriorates, even in the absence of microangiopathic hemolytic anemia.

42 ects might have an impact on the severity of microangiopathic lesions after exposure to Stx-producing

43 This inflammatory microangiopathic process is independently associated wit

44 linked to HIV infection includes thrombotic microangiopathic renal diseases, immune-mediated glomeru

45 of a thrombotic thrombocytopenic (TTP)-like microangiopathic state, that persisted longer when PCTx

46 er transplantation is idiopathic, thrombotic microangiopathic syndromes and nephrotic syndrome have b

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