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1 ressed miRNAs in human cancers obtained from microarray data.
2 as validated by using independent, published microarray data.
3 ical management of cancer in the presence of microarray data.
4 f false positives and false negatives in the microarray data.
5 heir magnitude, the first such model for SNP microarray data.
6 m gene selection to infer such networks from microarray data.
7 plicitly models the possible outliers in the microarray data.
8 t germ cell-specific expression from gonadal microarray data.
9  in an independent set of publicly available microarray data.
10 o counteract the presence of outliers in the microarray data.
11 lobal test in both simulation and a diabetes microarray data.
12 een developed to identify bimodal genes from microarray data.
13 ictive clinical tool using published patient microarray data.
14 oth CCNE2 and CDC6 were downregulated in the microarray data.
15 el algorithm, VIPR, for analyzing diagnostic microarray data.
16 essment of model predictions against patient microarray data.
17 reus, Saccharomyces cerevisiae and in silico microarray data.
18 performance using alternative sequencing and microarray data.
19 ultiple etiologies by exploring high quality microarray data.
20 f known relevant genes in large compendia of microarray data.
21 uency) copy-number variants (CNVs) using SNP microarray data.
22 t pair-wise synergy in simulation and cancer microarray data.
23 can also be applied to stabilize variance in microarray data.
24 designed for archiving and analyzing protein microarray data.
25 ene regulatory networks from gene expression microarray data.
26 d apply it to the integration of RNA-seq and microarray data.
27  performed for statistical assessment of the microarray data.
28 prehensive analysis report for their protein microarray data.
29 ine part of the analysis of high-dimensional microarray data.
30 fective method to estimate purities from the microarray data.
31 se when combining batches of gene expression microarray data.
32 omes reduced to low-coverage sequence and HD microarray data.
33  space, rather than in the original space of microarray data.
34 identified from the analyses of leafy spurge microarray data; (2) 3 orthologs of Arabidopsis "general
35 e, we have integrated four public expression microarray data (320 samples) from the Gene Expression O
36                       We screened C. elegans microarray data [5, 6] to identify male germline-enriche
37 ere constructed from all currently available microarray data, 90% phenotype prediction accuracy, or t
38  and standardize Meta-Analysis of Affymetrix Microarray Data analysis (MAAMD) in Kepler.
39 own to reduce overfitting noises involved in microarray data analysis and discover functional gene se
40 e have also created a pathway module for the microarray data analysis portal ArrayAnalysis.org that c
41                                              Microarray data analysis revealed striking correlations
42                                              Microarray data analysis showed a pleiotropic effect of
43 munohistochemistry (IHC) staining and public microarray data analysis showing that DACH1 was higher i
44   We developed an R package for resequencing microarray data analysis that integrates a novel statist
45 alysis is an invaluable tool for exploratory microarray data analysis, as it organizes the data into
46 lthough FDR control is frequently applied to microarray data analysis, gene expression is usually cor
47 ntified important proteins were confirmed by microarray data analysis.
48  and salt stresses, as indicated by previous microarray data analysis.
49                                  It uses the microarray data and a 28-trait image array to evaluate e
50  optimize the strength of interactions using microarray data and an artificial neural network fitness
51 logy will facilitate re-analysis of archived microarray data and broaden the utility of the vast quan
52 chromatin immunoprecipitation sequencing and microarray data and DNase I hypersensitive site sequenci
53 al-inverse-Wishart priors based on discarded microarray data and examine the performance on synthetic
54 r from the inclusion of excessive 'noise' in microarray data and false-positives in binding data, esp
55 anscriptional responses to TDB/TDM, we mined microarray data and identified early growth response (Eg
56 nthesis in Arabidopsis thaliana, we screened microarray data and identified transcriptional upregulat
57               QRT-PCR analysis confirmed the microarray data and revealed expression patterns of some
58 pL, and bcrA-like were carried out to verify microarray data and showed the same level of up- or down
59  of antibody epitope prediction from peptide microarray data and shows utility in analyzing phage dis
60                      Through analyses of our microarray data and the published Arabidopsis (Arabidops
61 tal cancer (CRC) metastasis was suggested by microarray data and validated by the survey of 120 patie
62 numerous transfer RNAs (tRNAs) dominated the microarray data and were validated on RNA gel blots.
63                 Additionally, R objects, for microarray data, and binary alignment format files, for
64 tion model used for background correction of microarray data, and modified it to formulate an error c
65  low-coverage sequence and high-density (HD) microarray data, and remained high even with a read erro
66 ds developed for bulk RNA sequencing or even microarray data, and the suitability of these methods fo
67 dicting the survival of cancer patients from microarray data, and to classification of obese and lean
68      With Arabidopsis (Arabidopsis thaliana) microarray data annotated to the PathoPlant database, 73
69 the methods for GSA previously developed for microarray data are based on the assumption that genes w
70 ream analysis tools previously restricted to microarray data are now available for RNA-seq as well.
71                                              Microarray data are used to determine which genes are ac
72 s from multiple tissues when log-transformed microarray data are used; (ii) estimation of both tumor
73 ficity are similar to those calculated using microarray data as a reference.
74 ological perspective, we used a set of yeast microarray data as a working example, to evaluate the fu
75 ely estimate absolute expression levels from microarray data, at both gene and transcript level, whic
76  Finally, reassessing previous C. pneumoniae microarray data based on codon content, we found that up
77 r 12 candidate transcripts selected from the microarray data based upon fold change and biological re
78 component analysis and k-means clustering of microarray data, because our traditional cardiac and ser
79 athway analysis strategy comparing miRNA and microarray data between three mouse models and human don
80 f our approach on real world gene expression microarray data by applying it to existing data from amy
81 istical methods that have been developed for microarray data can be applied to RNA-Seq data, they are
82 t feature selection approaches developed for microarray data cannot handle multivariate temporal data
83 h was developed in ecology and sociology, to microarray data (CCA on Microarray data, CCAM).
84 gy and sociology, to microarray data (CCA on Microarray data, CCAM).
85                                              Microarray data clustering revealed a striking pattern o
86 entification of differential exon use in the microarray data, clustering of exon inclusion/exclusion
87                                   Using gene microarray data collected in a large scale serially samp
88 ignificantly differentially expressed in the microarray data collected under the differing conditions
89 thods that are applied in biology to analyze microarray data, concerns regarding the compatibility of
90                           Publicly available microarray data confirmed differential expression of all
91                 RNA-Seq and qRT-PCR, but not microarray data, confirmed the expected reversal of SPAR
92               Similarly, human breast cancer microarray data demonstrated that high LOX/low GATA3 exp
93                                  Analysis of microarray data demonstrated that the transcriptome of C
94 ed to identify over-represented processes in microarray data derived from various disease states.
95  serum assays, including 2-way comparison of microarray data, did not lead to the identification of a
96 hidden within multilayered immunosignaturing microarray data due to its fundamental mathematical prop
97 elation coefficients between the RNA-Seq and microarray data each exceeded 0.80, with 66%~68% overlap
98 e regulatory network from publicly available microarray data, employing steps to enrich for physiolog
99                                  Genome-wide microarray data enable unbiased documentation of alterat
100                 Many cleaning approaches for microarray data exist, however these methods are aimed t
101                              Analysis of raw microarray data files (e.g. Affymetrix CEL files) can be
102                     Mandatory deposit of raw microarray data files for public access, prior to study
103 pplied association mining on a set of glycan microarray data for 211 influenza viruses from five host
104                                              Microarray data for 24-week-old BKS db/db and db/+ mouse
105              We conducted a meta-analysis of microarray data for 240 NFE2L2-mediated genes that were
106 quencing data and Affymetrix gene expression microarray data for 30 breast cancer cell lines represen
107 ve seed-specific expression, as indicated by microarray data for Arabidopsis.
108 the RT-qPCR validation were in line with the microarray data for both miRNAs, and statistically signi
109                 Furthermore, using available microarray data for gene expression, we show that in bot
110 = 84), ELISAs of blood/BM sera (n = 41), and microarray data for mRNA levels (n = 261) were performed
111 port detailed structural analysis and glycan microarray data for recombinant hemagglutinins from A(H6
112                                    Given the microarray data for the alterations in gene expression,
113 n RBPs and RIP-ChIP (RNP immunoprecipitation-microarray) data for 69 yeast RBPs to construct a networ
114 proteins involved in lamination, we utilized microarray data from 13 subtypes to identify differentia
115 pse-free survival (RFS) were evaluated using microarray data from 148 patients with stage I lung aden
116         Here, we analyzed publicly available microarray data from 16 diverse skin conditions in order
117                                        Using microarray data from 16 ferret samples reflecting cystic
118                                              Microarray data from 207 AML patients confirmed a greate
119                  This prompted us to analyze microarray data from 261 patients from a third cohort.
120                     Using publicly available microarray data from 46 primary human melanomas (GSE1560
121 tern blot, and immunofluorescence), analyzed microarray data from 99 patients with IPF and 43 control
122 iochemical interactions, our method utilizes microarray data from a group of mutants for its construc
123  demonstrate how mining publically available microarray data from a range of skin disorders can eluci
124 To this end, we performed a meta-analysis of microarray data from a variety of cytokinin-treated samp
125 dules and schizophrenia was replicated using microarray data from an independent tissue collection.
126                                              Microarray data from both cell types showed significant
127                                              Microarray data from cell lines of Non-Small Cell Lung C
128  outcome prediction in patient cohorts using microarray data from diagnosis specimens.
129                      Comparative analysis of microarray data from females after mating and after 20E
130                                  Analysis of microarray data from healthy human intestine further rev
131                                    Mining of microarray data from human tumor data sets revealed that
132 cell-specific patterns of gene expression in microarray data from mammalian gonads, specifically duri
133                              By interpreting microarray data from MATa cells, MATa/alpha cells, a sta
134                                        Using microarray data from orbitofrontal cortex of control sub
135 istent with these findings, meta-analysis of microarray data from over 4,000 breast cancer patients r
136  deletions and uniparental disomy) using SNP microarray data from over 50,000 subjects recruited for
137 g glucose through the use of gene expression microarray data from peripheral blood samples of partici
138 ighted gene coexpression network analysis on microarray data from singing zebra finches to discover g
139                                              Microarray data from the Cftr-deficient colon and the sm
140                   By re-analysis of existing microarray data from the FGF8, Lim1 and Wnt4 knockouts,
141                                   Expression microarray data from the kidney cortex and medulla, live
142                         Pathways analysis of microarray data from the mouse brain revealed gene netwo
143  to generate Illumina RNA-seq and Affymetrix microarray data from the same liver samples of rats expo
144 oinformatic analysis was performed using DNA microarray data from two experimental formats: (1) ventr
145 ChIP-sequencing results with gene expression microarray data from unloaded muscle to map several dire
146                  We analysed gene expression microarray data from whole blood samples from 228 multip
147 is system in four cohorts and extracted from microarray data (GeneChip) in the other two.
148  comprehensively analyzed RNA sequencing and microarray data generated by the Immunological Genome Pr
149 ful analytical tool using publicly available microarray data generated exclusively from Arabidopsis t
150 s to compare parallel paired-end RNA-Seq and microarray data generated on 5-azadeoxy-cytidine (5-Aza)
151 ed cytosine (5C), 5mC and 5hmC from Infinium microarray data given the signal intensities from the ox
152                                       The HS microarray data guided the selection of compounds that c
153         The recent advent of high-throughput microarray data has enabled the global analysis of the t
154 he existing methods for analyzing diagnostic microarray data has the capacity to specifically identif
155                                       Custom microarray data have been generated using RNA isolated f
156                              However, recent microarray data have indicated that nuc is under the con
157                     Through meta-analysis of microarray data, here we nominate nephroblastoma overexp
158 n silico analysis exploiting mNK cell subset microarray data, highlighting various genes and microRNA
159    Additionally, FungiDB contains cell cycle microarray data, hyphal growth RNA-sequence data and yea
160                                          The microarray data identified 454 candidate genes with expr
161                        Analysis of published microarray data identified a network of genes up-regulat
162 chain reaction, and motifADE analysis of the microarray data identified potential FK506-mediated path
163                   Re-analysis of genome-wide microarray data in 9 patient blood and 10 skin samples r
164                    Bioinformatic analyses of microarray data in ceh1 plants established the overrepre
165 a in various primary tumors, gene expression microarray data in over 1000 cancer cell lines and prote
166                        The interpretation of microarray data in the context of coexpression network a
167 a diverse collection of PDAC gene expression microarray data, including data from primary tumor, meta
168                             Analysis of rice microarray data indicated higher expression levels for O
169                                 Our previous microarray data indicated sphingosine 1-phosphate (S1P)
170          Pathway analyses of mRNA expression microarray data indicated that cells exposed to C4BP and
171           Analyses of breast cancer clinical microarray data indicated that high expression of SND1 i
172                             Biolog Phenotype MicroArray data indicated that mmp deletion increased su
173 s was determined by FACS analysis.Affymetrix microarray data indicated that NuMA was overexpressed in
174             Analysis of previously published microarray data indicated unscheduled transcription of a
175          Comparison between the ChIP-seq and microarray data indicates that FHY3 quickly regulates th
176 antly impacted in a given condition based on microarray data is a crucial step in current life scienc
177                    However, finding relevant microarray data is complicated by the large number of av
178 he majority of the valuable original protein microarray data is still not publically accessible.
179               Removing systematic noise from microarray data is therefore crucial.
180 der to reduce the impact of batch effects on microarray data, Johnson, Rabinovic, and Li developed Co
181 is of Microarrays (SAM) or Linear Models for Microarray Data (LIMMA) for processing cDNA microarrays,
182                However, the information from microarray data may not be fully deciphered through the
183                                  We generate microarray data measuring time-dependent gene-expression
184 results also supported by Oncomine analyses, microarray data (n=2878) and genomic data from paired tu
185               Gene ontology analysis of cDNA microarray data obtained after BMP9 treatment of primary
186 ounding effects of ITH using gene expression microarray data obtained from multiple tumour regions of
187                            Using time-series microarray data of Arabidopsis thaliana infected with Ps
188           Using GMine we reanalyzed proteome microarray data of host antibody response against Plasmo
189                                           On microarray data of hundreds of deletion mutants in two,
190  also compared with the previously published microarray data of Li1 ovule tissues.
191  involved in SA-induced folate accumulation, microarray data of responsive genes in Arabidopsis were
192                                  Analysis of microarray data on gene expression and methylation allow
193 -small cell lung cancer (NSCLC), we analyzed microarray data on gene expression and methylation.
194 etwork construction, we generate independent microarray data on selected deletion mutants to prospect
195 s and heteroskedasticity across 19 groups of microarray data on the sign and magnitude of gene-to-gen
196                                      We used microarray data on whole blood from two independent case
197 2 pathways by RNA-Seq data only, and none by microarray data only.
198 ditionally, criteria such as comparison with microarray data or a number of known polymorphic sites h
199        Functional enrichment analysis of the microarray data predicted that multiple biological funct
200 isease susceptibility, while gene expression microarray data provide genome-wide transcriptional prof
201 lity of our approach with both simulated and microarray data; random graphs and weighted (partial) co
202 glucuronidase) assays and publicly available microarray data revealed a differential spatio-temporal
203                                          Our microarray data revealed a distinct gene expression prof
204           Integration analysis of mRNA-miRNA microarray data revealed a potential role of 51 dysregul
205          Evaluation of neuroblastoma patient microarray data revealed an association between TGFBR3,
206                                              Microarray data revealed changes in expression of 504 ge
207                                   In humans, microarray data revealed declines in E2F3 and IGF2 expre
208                                  Analysis of microarray data revealed that adenine stimulation led to
209                                Mining public microarray data revealed that NCOR1-targeted genes were
210                  A thorough analysis of this microarray data revealed unique patterns of gene express
211 d and Consensus Clustering Analysis Tool for Microarray Data (SC(2)ATmd) is a MATLAB-implemented appl
212 ost cells validates the previously published microarray data set demonstrating feed-forward control o
213                              Second, a large microarray data set of prostate cancer was used to asses
214 scription factor analysis was performed in a microarray data set profiled in four different brain reg
215     Thus a clinical classifier weighted with microarray data set results in significantly improved di
216  paper, we reanalyzed a zebrafish (D. rerio) microarray data set using GeneSpring and different diffe
217           We demonstrate that for a specific microarray data set using the Human HG_U133A Affymetrix
218                    We therefore analyzed our microarray data set, cellular proteomes of separated lyt
219                               Using a public microarray data set, we identified via TEAK linear sphin
220  data set, and a combined publicly available microarray data set.
221 licly available glioblastoma gene expression microarray data set.
222 cuity in a publicly available small molecule microarray data set.
223         Integrative analysis of ChIP-seq and microarray data sets also reveals a consistent role of N
224 tperforms classical algorithms developed for microarray data sets as well as recent approaches design
225 project uses the abundant publicly available microarray data sets combined with a suite of single-arr
226 lly expressed at significant levels in the 5 microarray data sets compared, providing new insights in
227 pport of our in vitro data, analysis of mRNA microarray data sets demonstrated that high levels of FK
228 med in silico analysis of publicly available microarray data sets from prostate or breast carcinomas.
229 including CCND2, hTERT, and GCLC Analysis of microarray data sets further demonstrated that MUC1 leve
230                           Using whole-genome microarray data sets of the Immunological Genome Project
231 ed TEAK with experimental studies to analyze microarray data sets profiling stress responses in the m
232                    Meta-analyses of relevant microarray data sets revealed the hematopoietic stem cel
233       Finally, an analysis of paired RNA-seq/microarray data sets suggests that no or modest trimming
234                        We interrogated known microarray data sets to define NAMPT knockdown-influence
235                                  We analyzed microarray data sets to identify a subset of genes whose
236  functional relationships between genes from microarray data sets using rule-based machine learning.
237                           In two independent microarray data sets, 77% to 100% of tumors had substant
238 ces, such as the Allen Mouse Brain Atlas and microarray data sets, by providing quantitative expressi
239             By a meta-analysis of DNA stress microarray data sets, three family members of the SIAMES
240 e performed against publicly available human microarray data sets.
241 IGF2BP family members was first evaluated by microarray data sets.
242                              Two independent microarray data-sets from human renal allograft biopsies
243      Transcriptome in silico analysis of the microarray data showed major metabolomic variations in k
244                                              Microarray data showed that 324 genes were up-regulated
245                        Analysis of published microarray data showed that AK4 was upregulated in lung
246                               In this study, microarray data showed that either addition of oxygen or
247              Analysis of ovarian cancer gene microarray data showed that higher expression of Nectin-
248                                  Analysis of microarray data showed that iron deficiency in utero res
249 of the diffuse large B-cell lymphoma patient microarray data showed that miR-155 expression is invers
250                                Comparison of microarray data showed that NF-kappaB was among the tran
251      Systematic analysis of tissue-profiling microarray data showed that the zinc transporter ZIP12 (
252             Our in silico analysis of public microarray data shows that auxin and glutathione redox s
253 ated clinical literature and gene expression microarray data stored in large international repositori
254            Accurate differential analysis of microarray data strongly depends on effective treatment
255                                              Microarray data suffers from several normalization and s
256                                              Microarray data suggest that SDSE degraded host tissue p
257 ymidine kinase genes (AtTK1a and AtTK1b) and microarray data suggest they might have redundant roles.
258                          Pathway analysis of microarray data suggested activation of the p53 and reti
259                    Gene ontology analysis of microarray data suggested that the beta-catenin-independ
260    Thus repeat annotation of gene expression microarray data suggests that a complex interplay betwee
261                      Further analysis of the microarray data suggests that B. thailandensis, when exp
262 Lastly, semi-quantitative model analysis via microarray data superimposed onto the model with a score
263                                          Our microarray data support our histological findings and re
264              We performed a review of public microarray data that revealed a significant down-regulat
265 ustom 'Ae. aegypti detox chip' and validated microarray data through RT-PCR comparing susceptible and
266                                 We then used microarray data to develop classifiers that assigned ant
267 and an R function which uses DNA methylation microarray data to infer tumor subtypes with the conside
268               In this study we have explored microarray data to investigate the expression pattern of
269 n genetic data from GWAS and gene expression microarray data to reposition drugs for PD.
270 constructed gene co-expression networks from microarray data to study large-scale transcriptional pat
271 nomes pathway enrichment cluster analyses of microarray data using wild-type and c-Jun-deleted macrop
272                                              Microarray data, validated against direct RNA sequencing
273                        Arabidopsis seed coat microarray data was analysed for genes expressed in the
274        Functional annotation analysis of DNA microarray data was consistent with depressed innate imm
275 t format and specificity which correlated to microarray data was demonstrated.
276 ng independent normal data and one involving microarray data), we show that the proposed method, when
277                            Using time-series microarray data, we analyzed the temporal behavior of mR
278                               Using existing microarray data, we defined 24 circadian time phase grou
279 Arabidopsis thaliana) homologs and available microarray data, we identified 60 candidate genes for co
280                             Using RNAseq and microarray data, we identified a set of genes that are h
281  correlation of chemical data and phylogenic microarray data, we identified several bacteria that cou
282                          In this work, using microarray data, we investigate the feasibility and effe
283                                              Microarray data were analyzed using efficiency analysis
284  gene expression analysis was performed, and microarray data were assessed by Ingenuity Pathways Anal
285                                              Microarray data were further validated by immunoblotting
286 n the present study, the whole transcriptome microarray data were generated from peripheral blood mon
287 signed for DEG identification in RNA-Seq and microarray data, were applied to compare the cross-platf
288  methods for the analysis of DNA methylation microarray data, which account for tumor purity.
289  study the rewiring of gene networks through microarray data, which is becoming an important compleme
290 r mechanisms, we analyzed publicly available microarray data, which revealed a developmentally coordi
291        Furthermore, coexpression analysis of microarray data, which reveals the dynamics of host resp
292  from this work and the predictions based on microarray data will help explore novel metabolic proces
293 network-type analyses along with time series microarray data will lead to advancements in our underst
294 e algorithms that reconcile case-control DNA microarray data with a molecular interaction network by
295 amined the behaviors of different methods to microarray data with different properties, and whether t
296                       Through integration of microarray data with genome-wide histone modification Ch
297                    StRAP houses multi-cancer microarray data with major emphasis on radiotherapy stud
298      We investigate the impact of augmenting microarray data with semantic relations automatically ex
299                      By correlating clinical microarray data with the patients' outcome, a link betwe
300 ties allow users to analyse both RNA-seq and microarray data with very similar pipelines.

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