ライフサイエンスコーパス: microbial pathogen

1 d black polymer insects generate to seal off microbial pathogens).

2 ta-caryophyllene in floral defense against a microbial pathogen.

3 ione-driven transnitrosylation of an enteric microbial pathogen.

4 ted monoclonal antibody and used to target a microbial pathogen.

5 on the fitness and virulence of a eukaryotic microbial pathogen.

6 0 and CCL2/MCP-1 production in response to a microbial pathogen.

7 to vision loss when initiated by a virulent microbial pathogen.

8 to design new and effective vaccines against microbial pathogens.

9 t can promote Th17-mediated immunity against microbial pathogens.

10 nous and modified host-derived molecules and microbial pathogens.

11 g the clinical routine for identification of microbial pathogens.

12 ntal history modulates the plant response to microbial pathogens.

13 ut activating immune defenses in response to microbial pathogens.

14 tool for typing and controlling outbreaks of microbial pathogens.

15 ent of human infections caused by eukaryotic microbial pathogens.

16 fective protection against a wide variety of microbial pathogens.

17 Swiss army knife' in innate immunity against microbial pathogens.

18 ally antagonize host protective responses to microbial pathogens.

19 kinetics, and biofilm formation of potential microbial pathogens.

20 l for the elimination of an immense array of microbial pathogens.

21 e skin and mucosae are frequently exposed to microbial pathogens.

22 nitiation of innate immune responses to some microbial pathogens.

23 nced antimicrobial defense against exogenous microbial pathogens.

24 for further design of specific inhibitors of microbial pathogens.

25 s by demonstrating a role in the response to microbial pathogens.

26 are important players in the defense against microbial pathogens.

27 that plays a front line role in eliminating microbial pathogens.

28 rs that recognize PAMPs from a wide range of microbial pathogens.

29 nd detrimental roles in host defense against microbial pathogens.

30 ostasis, which modulates immune responses to microbial pathogens.

31 ognition receptors to defend themselves from microbial pathogens.

32 master transcription factors in response to microbial pathogens.

33 llular factors that support cell invasion by microbial pathogens.

34 application of studying AVG inactivation in microbial pathogens.

35 ction is fundamental to host defense against microbial pathogens.

36 ogical reaction to noxious stimuli including microbial pathogens.

37 resource between higher organisms and their microbial pathogens.

38 imary role in adaptive immunity to cytosolic microbial pathogens.

39 may undergo adaptive changes in response to microbial pathogens.

40 itical for host defense against a variety of microbial pathogens.

41 rtant part of innate immunity to flagellated microbial pathogens.

42 rucial for virulence and stress responses in microbial pathogens.

43 uman immune system and an important trait in microbial pathogens.

44 ective lung mucosal immune responses against microbial pathogens.

45 in the adherence properties and virulence of microbial pathogens.

46 tion is essential for efficient clearance of microbial pathogens.

47 rt of the gut mucosal innate defense against microbial pathogens.

48 okines, which are tailored to combat various microbial pathogens.

49 lex immune mechanisms for protection against microbial pathogens.

50 nt role in host defense against a variety of microbial pathogens.

51 shown to initiate innate immune responses to microbial pathogens.

52 e innate immune mechanisms to defend against microbial pathogens.

53 daptable for quantitative detection of other microbial pathogens.

54 se (MAPK) pathway in mediating resistance to microbial pathogens.

55 le in the vertebrate immune response against microbial pathogens.

56 lasses and others for an optimal response to microbial pathogens.

57 apted for the inoculation and study of other microbial pathogens.

58 is essential for defense against a range of microbial pathogens.

59 tion and is crucial for the rapid removal of microbial pathogens.

60 thus a receptor for patterns associated with microbial pathogens.

61 be important in the virulence of a number of microbial pathogens.

62 s in regulating the inflammatory response to microbial pathogens.

63 ble inflammatory cells to recognize invading microbial pathogens.

64 architecture of the CD4 response to complex microbial pathogens.

65 ontribute directly to immune defense against microbial pathogens.

66 ously implicated in the host defense against microbial pathogens.

67 ponent of the plant defense response against microbial pathogens.

68 hat coordinate cellular responses induced by microbial pathogens.

69 signaling pathways against various types of microbial pathogens.

70 or the activation of innate immunity against microbial pathogens.

71 llose response is required for resistance to microbial pathogens.

72 is essential for the elimination of invading microbial pathogens.

73 ation of innate immune responses to invading microbial pathogens.

74 f conserved microbial structures of invading microbial pathogens.

75 regulators of innate immune response against microbial pathogens.

76 rget to augment host defense against diverse microbial pathogens.

77 are essential in the clearance of infectious microbial pathogens.

78 and increased susceptibility to infection by microbial pathogens.

79 ing proteins mediating the adhesion of other microbial pathogens.

80 ialized mechanism for innate Ab responses to microbial pathogens.

81 nitiation of innate immune responses to some microbial pathogens.

82 effects of LNG use on human host response to microbial pathogens.

83 ater-cooperator polymorphism and polymorphic microbial pathogens.

84 ell-autonomous immunity to a wide variety of microbial pathogens.

85 tects immunoglobulin abnormalities caused by microbial pathogens.

86 plants in a manner similar to perception of microbial pathogens.

87 r control of infection are often targeted by microbial pathogens.

88 ring infection, supports the defense against microbial pathogens.

89 n-mediated suppression of innate immunity by microbial pathogens.

90 at (NLR) superfamily to detect many types of microbial pathogens.

91 nt cell walls are important barriers against microbial pathogens.

92 gnaling in in vivo CD8(+) T cell immunity to microbial pathogens.

93 host defense against aerosol infection with microbial pathogens.

94 ed class of sirtuins, found predominantly in microbial pathogens.

95 be a potent tool for indirectly controlling microbial pathogens.

96 of the application of population genomics to microbial pathogens.

97 ar result from infectious diseases caused by microbial pathogens.

98 ragment receptors (Ig FcRs), and aggregating microbial pathogens.

99 ry response syndrome often in the absence of microbial pathogens.

100 ing a robust immune system effective against microbial pathogens.

101 king innate and adaptive immune responses to microbial pathogens.

102 at a controlled rate that in turn disinfect microbial pathogens.

103 mediators of host defense to a wide range of microbial pathogens.

104 in vaccine development against encapsulated microbial pathogens.

105 Although effective against microbial pathogens, a growing body of literature now su

106 Microbial pathogens adapt to the stress of infection by

107 ls against environmental challenges, such as microbial pathogens, allergens and stress.

108 Many microbial pathogens alter expression and/or posttranslat

109 itially activated Ag-specific CD4 cells by a microbial pathogen and document a novel strategy for bac

110 otic stresses caused by insects and numerous microbial pathogens and abiotic stresses caused by adver

111 Both recognize microbial pathogens and activate the classical complemen

112 nse to these ligands that are shared by many microbial pathogens and affect the cells lining the peri

113 tors (TLRs) contribute to host resistance to microbial pathogens and can drive the evolution of virul

114 ther hand, AHAs may protect against invasive microbial pathogens and cancer.

115 Recognition of microbial pathogens and dead cells and their phagocytic

116 ocytosis is crucial for host defense against microbial pathogens and for obtaining nutrients in Dicty

117 central for the emergence or re-emergence of microbial pathogens and for their adaptation to a specif

118 In the absence of microbial pathogens and given the strong similarities to

119 gands, such as sugar structures displayed by microbial pathogens and glycans on the surface of phagoc

120 s an important hormone in plant responses to microbial pathogens and herbivorous insects, and in the

121 e discuss the challenges posed by eukaryotic microbial pathogens and how these are similar to, or dif

122 The ancient biological 'arms race' between microbial pathogens and humans has shaped genetic variat

123 ed immune response can be protective against microbial pathogens and in cancer immunotherapy.

124 rved molecular signatures of a wide range of microbial pathogens and initiate innate immune responses

125 g plant responses to herbivorous insects and microbial pathogens and is an important regulator of pla

126 entral feature of innate immune responses to microbial pathogens and is mediated via Toll-like recept

127 es that often exhibit broad activity against microbial pathogens and mammalian tumor cells.

128 While inflammatory phagocytosis of microbial pathogens and non-inflammatory phagocytosis of

129 ticated immune system to cope with different microbial pathogens and other invaders.

130 plays critical roles in the phagocytosis of microbial pathogens and phagosome maturation.

131 c response that is essential for eliminating microbial pathogens and repairing tissue after injury.

132 ceptors that play crucial roles in detecting microbial pathogens and subsequent development of immune

133 al and dissemination of at least a subset of microbial pathogens and suggest that common variation in

134 infection highlights the rapid detection of microbial pathogens and suggests an important role for t

135 he innate immune system of plants recognizes microbial pathogens and terminates their growth.

136 r the identification of virulence factors in microbial pathogens and the development of potential new

137 Studies of microbial pathogens and the toxins they produce are impo

138 that aid in the clearance of a wide range of microbial pathogens and their products.

139 complex called the inflammasome that senses microbial pathogens and then activates the proinflammato

140 are believed to mediate host defense against microbial pathogens and tissue homeostasis within the in

141 ls that can contribute to protection against microbial pathogens and to the development of harmful au

142 Immunoglobulins recognize and clear microbial pathogens and toxins through the coupling of v

143 TLRs recognize microbial pathogens and trigger an immune response, but

144 eneration of effective antibody responses to microbial pathogens and tumoral cells.

145 s the first line of defense against invading microbial pathogens, and as such is the primary suspect

146 by nonribosomal peptide synthetases against microbial pathogens, and discovered an antibiotic for wh

147 ponses to foreign antigens, such as those of microbial pathogens, and self-antigens, such as those ta

148 Outer membrane proteins (OMPs) of microbial pathogens are critical components that mediate

149 Microbial pathogens are exposed to damaging reactive oxy

150 Eukaryotic microbial pathogens are major contributors to illness an

151 have found that immunoglobulins disrupted by microbial pathogens are specifically detected by leukocy

152 n of a broad range of molecules expressed by microbial pathogens as well as host-derived danger signa

153 ation and maintenance of immune responses to microbial pathogens as well as to allergens, but the exa

154 ctor genes which may recognize and eliminate microbial pathogens as well as uncharacterized gene clas

155 eton to the plant's defense response against microbial pathogens, as well as the mechanisms used by p

156 By analogy with microbial "pathogen-associated molecular patterns" (PAMP

157 E can also act as an innate immune sensor of microbial pathogen-associated molecular pattern molecule

158 iated molecular patterns (DAMPs), but not to microbial pathogen-associated molecular patterns (PAMPs)

159 Microbial pathogen-associated molecular patterns (PAMPs)

160 Despite the absence of exogenous microbial pathogen-associated molecular patterns, the Tf

161 ployed by the innate immune system to detect microbial pathogens based on conserved microbial pathoge

162 technology in the typing and epidemiology of microbial pathogens, but the increase in genomic informa

163 ve foreign pathogens, such as those found on microbial pathogens, but when persistent or maladaptive,

164 TLRs initiate the host immune response to microbial pathogens by activating cells of the innate im

165 otal role in immune evasion by certain other microbial pathogens by driving the differentiation of re

166 CWFs remove/deactivate microbial pathogens by employing two mechanisms: metalli

167 l to address many unresolved questions about microbial pathogens by facilitating the identification o

168 play important roles in immune responses to microbial pathogens by monitoring prenyl pyrophosphate i

169 However, whereas microbial pathogens can be recognized as nonself by immu

170 d renewed interest with the recognition that microbial pathogens can be responsible for the chronic i

171 Some microbial pathogens can induce pathogenic antibodies cro

172 Several microbial pathogens can modulate the host apoptotic resp

173 and accurate detection and identification of microbial pathogens causing urinary tract infections all

174 Many microbial pathogens deliver effector proteins via the ty

175 on of type I interferon (IFN) in response to microbial pathogens depends on a conserved cGAS-STING si

176 ruses and bacterial pathogens indicates that microbial pathogens deploy deamidases or enzyme-deficien

177 ple preparation for detecting and genotyping microbial pathogens directly from clinical specimens; th

178 Most human microbial pathogens do not infect animals and the metabo

179 ge-associated molecular patterns rather than microbial pathogens elicit the response.

180 Microbial pathogens employ sophisticated virulence strat

181 Conversely, microbial pathogens employ their own strategies in order

182 y be an evolutionary strategy for eukaryotic microbial pathogens, enabling de novo genotypic and phen

183 Accordingly, microbial pathogens evolved to subvert these organ-speci

184 Several microbial pathogens exploit cellular actin polymerizatio

185 lls, but also as defensive responses against microbial pathogens externally or the ill effects of dam

186 high levels of fecal indicator bacteria and microbial pathogens, generating concern about long-term

187 Naive CD8(+) T lymphocytes responding to microbial pathogens give rise to effector T cells that p

188 Our understanding of the evolution of microbial pathogens has been advanced by the discovery o

189 lar pattern-triggered immunity (PTI) against microbial pathogens has been recently demonstrated.

190 ility to sequester nutrient Zn(II) ions from microbial pathogens has been recognized for over two dec

191 yD88 in the regulation of innate immunity to microbial pathogens has been well demonstrated.

192 antibiotic resistance (AR) among infectious microbial pathogens has questioned the future utility of

193 Although genome sequencing of microbial pathogens has shed light on the evolution of v

194 Microbial pathogens have evolved sophisticated mechanism

195 Microbial pathogens have evolved virulence factors that

196 geous for switching ecological niches, as in microbial pathogen host switch events, has not been expl

197 ariety of DNA lesions in a broad spectrum of microbial pathogens; however, levels of the DNA deaminat

198 plasminogen activators are commonplace among microbial pathogens, implying a central role of host pla

199 ted T cells, we posited DC-HIL can recognize microbial pathogens in a similar manner.

200 ich normally functions to control particular microbial pathogens in a T-independent manner, to contri

201 ation, and reporting of infectious viral and microbial pathogens in a wide variety of point-of-care a

202 al mechanism of containment and clearance of microbial pathogens in advance of the development of acq

203 involved in plant defense machinery against microbial pathogens in Arabidopsis thaliana.

204 The oral occurrence of putative microbial pathogens in humans has been documented in hea

205 hat opsonizes and permeabilizes membranes of microbial pathogens in mammalian lungs.

206 The accurate and rapid identification of the microbial pathogens in patients with pulmonary infection

207 elmintic treatment can enhance the spread of microbial pathogens in some real-world situations.

208 rism attack, (ii) horizontal transmission of microbial pathogens in the community, and (iii) persiste

209 of stresses, including those encountered by microbial pathogens in the host.

210 However, the NLRs that respond to microbial pathogens in vivo are poorly defined.

211 mice are more susceptible to infections with microbial pathogens, including the bacterial pathogen Sa

212 With the emergence of microbial pathogens increasingly resistant against commo

213 Many microbial pathogens infecting immunocompromised hosts, f

214 sponse is essential for host defense against microbial pathogen infections and is mediated by pattern

215 essential for host defenses against primary microbial pathogen infections, yet their involvement in

216 helium environment in the protection against microbial pathogen infections.

217 es in a host of responses to a wide range of microbial pathogens, inflammatory diseases, cancer, and

218 Ps), molecular moieties produced by invading microbial pathogens, initiate innate immune responses by

219 cell biologists to incorporate the study of microbial pathogens into their research programs.

220 Following microbial pathogen invasion, one of the main challenges

221 c signals orchestrate plant defenses against microbial pathogen invasion.

222 Protection against microbial pathogens involves the activation of cellular

223 The detection of microbial pathogens involves the recognition of conserve

224 an effective innate immune strategy against microbial pathogens involves triggering local cell death

225 Understanding the evolutionary history of microbial pathogens is critical for mitigating the impac

226 Immunological protection against microbial pathogens is dependent on robust generation of

227 Plant defense to microbial pathogens is often accompanied by significant

228 A challenge for microbial pathogens is to assure that their translocated

229 goal of the analysis of sequenced genomes of microbial pathogens is to improve the therapy of infecti

230 initial encounters between plants and their microbial pathogens, is composed of a complex mixture of

231 We report that many microbial pathogens lacking an identifiable intercellula

232 Because encounter with microbial pathogens leads to the simultaneous ligation o

233 Specialized microbial pathogens may also exhibit gene loss relative

234 enetic architecture of plant defense against microbial pathogens may be influenced by pathogen lifest

235 etect microbial pathogens based on conserved microbial pathogen molecules.

236 L-PGS and A-PGS inhibitors that could render microbial pathogens more susceptible to antimicrobial co

237 To initiate infection, a microbial pathogen must be able to evade innate immunity

238 ociation in our current understanding of the microbial pathogen, Mycobacterium tuberculosis.

239 relationship between the lung microbiota and microbial-pathogen niches.

240 The glycoproteins of selected microbial pathogens often include highly modified carboh

241 ly unrecognized capacity of SIgA to "disarm" microbial pathogens on mucosal surfaces and prevent colo

242 on molecules whose major function is to bind microbial pathogens or cellular debris during infection

243 no identity with those of other proteins of microbial pathogens or the human alpha-actinin 1 (HuACTN

244 howed no change in resistance to a number of microbial pathogens, or in the progression of leaf senes

245 Candida species are microbial pathogens originally thought to be asexual, bu

246 ungs is key in the response against invading microbial pathogens, other sentinels, such as alveolar m

247 olecules (such as flagellin) associated with microbial pathogens (pathogen-associated molecular patte

248 Surface proteins of mucosal microbial pathogens play multiple and essential roles in

249 the "keystone-pathogen" where low-abundance microbial pathogens (Porphyromonas gingivalis) can orche

250 To subvert host defenses, some microbial pathogens produce proteins that interact with

251 The evolution of drug resistance in microbial pathogens provides a paradigm for investigatin

252 system must recognize and rapidly respond to microbial pathogens, providing a first line of host defe

253 ell that contributes to the early control of microbial pathogen re-infections.

254 peptides is essential for protection against microbial pathogens, recognition of self-peptides by T c

255 mediated by interactions with integrins and microbial pathogens, respectively.

256 Stimulation of cells with cytokines and microbial pathogens results in the activation of TAK1, w

257 olipids and regulate actin filament dynamics.Microbial pathogens secrete effector proteins into host

258 d gene disruptions in obligate intracellular microbial pathogens seriously hampers the identification

259 The prolonged presence of microbial pathogens stimulates inflammation of the local

260 Many microbial pathogens subvert cell surface heparan sulfate

261 h and represent a novel mechanism by which a microbial pathogen subverts host cell signaling and tran

262 Microbial pathogens succeed in acquiring essential metal

263 s of UV disinfection include reduced risk of microbial pathogens such as Cryptosporidium and reduced

264 similar to those that drive the movement of microbial pathogens such as vaccinia virus.

265 immunity to extracellular and intracellular microbial pathogens, such as Candida and Salmonella.

266 in response to CpG-Dotap and stimulation by microbial pathogens, such as Leishmania major, Escherich

267 Toll-like receptor (TLR)4 recognizes microbial pathogens, such as lipopolysaccharide, and med

268 pathway is absent in humans but essential in microbial pathogens, suggesting that it provides potenti

269 ay be an adaptive response to selection by a microbial pathogen, supporting the influence of microbia

270 wledge, this is the first demonstration of a microbial pathogen suppressing IL-17-mediated inflammati

271 owever, analogous to classical activation by microbial pathogens, Th2 cells are required for maintena

272 n this region is much less likely to contain microbial pathogens than surface water but often contain

273 ions are also the main interaction sites for microbial pathogens that bind host FH to evade complemen

274 s (IECs) are a first line of defense against microbial pathogens that enter the host through the inte

275 of new vaccines are being developed against microbial pathogens that might be used as bioweapons.

276 particularly important for organisms such as microbial pathogens that utilise genome plasticity as a

277 lve the evolutionary history of an important microbial pathogen, the chytrid Batrachochytrium dendrob

278 ern recognition receptors detect invasion by microbial pathogens, the field of immunology has witness

279 For a wide variety of microbial pathogens, the outcome of the infection is ind

280 By identifying and characterizing emerging microbial pathogens, these developments provided signifi

281 Plants defend themselves against microbial pathogens through a range of highly sophistica

282 ceptors suppresses inflammatory responses to microbial pathogens through cAMP-dependent signaling cas

283 demonstrates that in vivo transformation of microbial pathogens to a tissue destroying phenotype may

284 The capacity of microbial pathogens to alter their host tropism leading

285 Molecular factors enabling microbial pathogens to cause plant diseases have been so

286 Antigenic variation allows microbial pathogens to evade immune clearance and establ

287 grins are recognized by such a wide range of microbial pathogens to invade host cells.

288 The ability of microbial pathogens to target specific cell types is a k

289 from the perspective of Candida albicans, a microbial pathogen uniquely adapted to its human host.

290 Microbial pathogens use adhesive surface proteins to bin

291 A variety of microbial pathogens use antigenic variation, an immune e

292 Microbial pathogens use environmental cues to trigger th

293 to recognize distinct ligands from a single microbial pathogen via multiple pattern recognition rece

294 The innate immune system recognizes microbial pathogens via pattern recognition receptors.

295 's virtually inevitable exposure to external microbial pathogens warrants efficient tissue-specialize

296 Multicellular eukaryotes coevolve with microbial pathogens, which exert strong selective pressu

297 nth infections inhibit host immunity against microbial pathogens, which has largely been attributed t

298 ulated to ensure adequate protection against microbial pathogens while minimizing damage to host tiss

299 Lethal mutagenesis, the killing of a microbial pathogen with a chemical mutagen, is a potenti

300 system is critical for host defense against microbial pathogens, yet many pathogens express virulenc