ライフサイエンスコーパス: microbiota

1 treat dietary allergies by modulation of the microbiota.

2 umoniae infections result from patients' own microbiota.

3 on is accompanied by exposure to a healthier microbiota.

4 organisms and is also colonized by a diverse microbiota.

5 y invasive tool for sampling the oesophageal microbiota.

6 thetaiotaomicron, a member of the human gut microbiota.

7 ealth and performance through modulating gut microbiota.

8 s revealed hundreds of species in almost all microbiota.

9 ated with lower alpha diversity of the stool microbiota.

10 e major nutrients available to the human gut microbiota.

11 manipulate the animal's associated external microbiota.

12 companying diversification of the intestinal microbiota.

13 tensively metabolized, mainly by the colonic microbiota.

14 abundances of various bacterial taxa in gut microbiota.

15 d of other organisms, that is, the commensal microbiota.

16 drives microbial diversity in the human gut microbiota.

17 onally complete diet in the absence of a gut microbiota.

18 ting the structural configuration of the gut microbiota.

19 molecules in human blood are products of the microbiota.

20 is a widespread adaptation within the human microbiota.

21 y of new strategies targeting the intestinal microbiota.

22 tions between immune cells and the commensal microbiota.

23 en Clostridiales were absent in the neonatal microbiota.

24 interactions between diet and the intestinal microbiota affect development of mucosal inflammation or

25 his nutrition source also contains a diverse microbiota affecting the development and health of the n

26 ritis, and that modulation of the intestinal microbiota after the onset of arthritis may offer therap

27 ncing we characterised the mucosa-associated microbiota along the length of the intestine of piglets,

28 on against diseases later in life.Early-life microbiota alterations can affect infection susceptibili

29 Fecal microbiota analysis of 3 successful FT recipients demons

30 isease removed harmful bacteria from the gut microbiota and attenuated SLE-like disease in lupus-pron

31 by HPDs, but was associated with a specific microbiota and bacterial metabolite profile.

32 on between the composition of the intestinal microbiota and clinical features of irritable bowel synd

33 to demonstrate associations between the gut microbiota and cognition in human infants.

34 s a critical factor that is regulated by gut microbiota and determines thrombus growth in Tlr2(-/-) m

35 the host for diet-induced changes of the gut microbiota and energy metabolism.

36 -life regulation of the immune system by the microbiota and how it can be related to allergy developm

37 Accordingly, both the gut microbiota and immune system are implicated in the etiop

38 re based on the early modulation of the host microbiota and immunity.

39 Nopal modified gut microbiota and increased intestinal occludin-1 in the HF

40 The microbiota and its larger host represent a metaorganism

41 lishing a strong association between the gut microbiota and obesity in humans, a causal relationship

42 We examined the impact of IS drugs on gut microbiota and on the expression of ileal antimicrobial

43 there is niche-specificity to the placental microbiota and placental microbiome studies should consi

44 roborate the association between the vaginal microbiota and PTB, demonstrate the benefits of high-res

45 hus, diabetes-enhanced IL-17 alters the oral microbiota and renders it more pathogenic.

46 Communication pathways between gut microbiota and the central nervous system could include

47 ediate the interaction between the diet, the microbiota and the host.

48 Gut microbiota and the immune system interact to maintain ti

49 nteractions occurring between the intestinal microbiota and the immune system, and we will discuss ho

50 The enteric microbiota and the innate and adaptive immune system rep

51 mechanisms that mediate interactions between microbiota and the intestinal epithelium are not fully u

52 these mice generated animals that had hybrid microbiotas and displayed intermediate susceptibility to

53 onal imbalances, metabolic disturbances, gut microbiota, and cancer.

54 Because HMOs, gut microbiota, and infant health are interrelated, the rela

55 the primary source of the river-transported microbiota, and that cell export from the GrIS is depend

56 Indeed, mixed anaerobic microbiota are associated with a majority of HS lesions.

57 trointestinal tract (GIT) and its associated microbiota are essential for survival and milk productio

58 aphic and cultural differences in intestinal microbiota are necessary to define applicability of new

59 Interactions between the host and its microbiota are of mutual benefit and promote health.

60 ed that Clostridia added to mouse infant gut microbiota are sufficient to limit colonization of patho

61 The stool microbiota around the time of neutrophil recovery post-H

62 colorectal cancer, thereby establishing the microbiota as a potential therapeutic target.

63 uperorganism networks that consider host and microbiota as a whole-may uncover their code, clarifying

64 g women with abnormal versus healthy vaginal microbiota as defined by Nugent score.

65 conclusion, drug discovery targeting the gut microbiota as well as the characterization of microbiota

66 ss the pro- and anticarcinogenic role of the microbiota, as well as highlighting the therapeutic pote

67 cells are abundant in the gut and recognize microbiota-associated metabolites, we assessed their pot

68 ged environment keeping intestinal commensal microbiota at bay and defending against invading enteric

69 We aimed to profile the microbiota at different stages of Barrett's carcinogenes

70 us, and mycophenolate mofetil modified fecal microbiota at the family level in each experimental repl

71 Designing successful microbiota-based therapies requires in-depth understandi

72 ions in the composition of mucosa-associated microbiota, between small and large intestine, concordan

73 terohepatic circulation, as well as host and microbiota bile acid metabolism, favor bile acid accumul

74 to a strong association between sex and gut microbiota, bile acids (BAs), and gastrointestinal cance

75 Alterations in nasal microbiota but not of throat microbiota were associated

76 at phyllosphere microbiota, like rhizosphere microbiota, can potentially mediate plant species coexis

77 had evidence for intestinal dysbiosis of the microbiota, characterized by expansion of Escherichia co

78 ishmania braziliensis develop dysbiotic skin microbiota, characterized by increases in the abundance

79 a with specific prebiotics modulated the gut microbiota closer to that of breast-fed infants.

80 l barrier function, host metabolism and host-microbiota co-metabolism were further modified by Bifido

81 e intestinal epithelial cells disclosed that microbiota colonization leads to activation or inactivat

82 The microbiota colonizing dental implants has been said to b

83 e of fat content in the diet in altering gut microbiota community by shifting phylotype composition a

84 Microbiota community instability was associated with fas

85 ristics, alpha-diversity, or overall vaginal microbiota community state type (CST).

86 tions in the neonatal (immediately at birth) microbiota community structure were associated with the

87 Prednisolone disrupted fecal microbiota community structure, decreased Bacteroidetes,

88 dataset, individuality was a major driver of microbiota composition (P = 0.002) and was more pronounc

89 We hypothesized that the respiratory microbiota composition and development in infancy is aff

90 splaying despair behavior, we found that the microbiota composition and the metabolic signature drama

91 al consortia to individuals with compromised microbiota composition may reduce inter-patient transmis

92 Differences in microbiota composition might influence the response to a

93 Fecal and rectal biopsy-associated microbiota composition was analyzed by 16S ribosomal DNA

94 Microbiota composition was heterogeneous, but there was

95 us, ultrafine particles ingestion alters gut microbiota composition, accompanied by increased atherog

96 i et al. (2017) show that RegIIIbeta impacts microbiota composition, decreasing vitamin B6 production

97 Many (58%) did not report an analysis of microbiota composition.

98 oth the intestine and plasma via altered gut microbiota composition.

99 Analysis revealed significant segregation of microbiota compositions which was validated by Beta dive

100 about the interrelationship between consumer microbiota configurations and biotransformation of food

101 Their microbiotas consistently contained abundant bacteria tha

102 To dissect how the microbiota contributes to tumor distribution, we generat

103 Vaginal microbiota CST and alpha-diversity are not related to GB

104 e in IgA(-/-) mice after CT immunization was microbiota dependent and was associated with increased s

105 tion, and antimicrobial gene expression in a microbiota-dependent manner, as assessed by antibiotic a

106 trimethylamine N-oxide (TMAO) levels, a gut microbiota-dependent metabolite associated with coronary

107 the coevolved relationship between host and microbiota, depleting bacterial populations critical for

108 ted mouse abolished the protective effect of microbiota depletion upon renal I/R injury.

109 itis in critically ill patients signified by microbiota depletion, and reestablishment of a physiolog

110 -tetrachlorodibenzo-p-dioxin (TCDD) and also microbiota-derived AhR ligands tryptamine, indole and 1,

111 Hesperetin-3'-O-glucuronide and the colonic microbiota-derived catabolite 3-(3'-hydroxy-4'-methoxyph

112 sibility of developing therapeutics based on microbiota-derived indole or its derivatives to extend h

113 ening the inflammatory response by targeting microbiota-derived mediators might be a promising therap

114 icrobiota as well as the characterization of microbiota-derived metabolites can represent innovative

115 ilize a variety of carbon sources, including microbiota-derived succinate.

116 Antibiotic-mediated microbiota destruction and the consequent loss of coloni

117 a correlation between NET complexes and both microbiota diversity (P = .009) and dominance of Haemoph

118 models have shown that perturbations of the microbiota during early life can cause immune effects th

119 An aberrant IgA responsiveness to the gut microbiota during infancy precedes asthma and allergy de

120 ut significant differences in the intestinal microbiotas during a critical early window of ontogeny,

121 wild-type mice disappeared after intestinal microbiota equalization.

122 In this setting, fecal microbiota, evaluated by 16S rRNA gene amplicon sequenci

123 research has shown that the gastrointestinal microbiota exhibits circadian rhythms and that the timin

124 The human gut microbiota ferments dietary non-digestible carbohydrates

125 supporting therapeutic targeting of the gut microbiota for brain-gut axis disorders, opening new ave

126 m, allowing the bacteria to compete with the microbiota for colonization.

127 tics and synthetic biology to mine the human microbiota for N-acyl amides that interact with G-protei

128 High-throughput DNA sequencing of microbiota from a diverse collection of fecal samples ob

129 ther the transfer of an antibiotic-perturbed microbiota from mothers to their children could affect t

130 In this study, fecal microbiota from patients with constipation and healthy c

131 commensal bacteria and that manipulation of microbiota genes encoding metabolites that elicit host c

132 ted studies in understanding the role of gut microbiota (GM) in viral-associated diarrhea.

133 The human microbiota greatly affects physiology and disease; howev

134 S-resident and peripheral immune pathways in microbiota-gut-brain communication during health and neu

135 ) ;Il10(-/-) mice conventionalized by an SPF microbiota had significantly more colon tumors compared

136 Gut microbiota has a proven role in regulating multiple neur

137 We report the commensal microbiota has anti-anabolic effects suppressing osteobl

138 CKGROUND & AIMS: Dysbiosis of the intestinal microbiota has been associated with development of aller

139 The intestinal microbiota has been identified as an environmental facto

140 The study of the intestinal microbiota has begun to shift from cataloging individual

141 composition and functions of the intestinal microbiota have been implicated in multiple disease proc

142 The gut microbiota have been linked with the development of obes

143 6.55% [95% CI 43.09 to 90.37]), intermediate microbiota (HIV incidence 1.8 per 100 person-years in th

144 interest in recent years has focused on gut microbiota-host interaction because accumulating evidenc

145 Diabetes has a significant impact on the gut microbiota; however, studies in the oral cavity have bee

146 rental mouse strains and in mice with hybrid microbiotas identified the bacterial family Lachnospirac

147 ard monitoring pathobiont-induced changes in microbiota immune targeting as a new concept in IBD diag

148 anisms for reciprocal interactions among the microbiota, immunity, gut function, and behavior.

149 stinal epithelium and that miRNAs respond to microbiota in a highly cell type-specific manner.

150 For each, we discuss the role of microbiota in biogeochemical cycling and outline ecologi

151 ity of prebiotics to specifically modify gut microbiota in children with overweight/obesity or reduce

152 changes in the composition of the intestinal microbiota in Crohn's disease, but its role on skin micr

153 We investigated the role of skin microbiota in cutaneous leishmaniasis and found that hum

154 The impact of the microbiota in health and disease is in part a function o

155 A growing body of data supports the role of microbiota in health and in disease.

156 Development of the gut microbiota in infancy is important in maturation of the

157 Involvement of gut microbiota in lung diseases by the gut-lung axis has bee

158 utational techniques to test the role of the microbiota in mediating despair behavior.

159 e to understand the relationship between the microbiota in mouse gut and diet type.

160 gical variable in studies on the role of gut microbiota in obesity-related mood disorders.

161 We aimed to characterize the airway microbiota in patients with symptomatic stable asthma an

162 The evidence on the role of gut microbiota in post-infectious irritable bowel syndrome (

163 hogen, highlighting a potential role for the microbiota in promotion or inhibition of GBS colonizatio

164 nce has revealed the pivotal role of the gut microbiota in shaping the immune system.

165 ween dietary ingredients, nutrients, and the microbiota in specific pathogen-free (SPF) and germ-free

166 ogical activities, and the importance of gut microbiota in supplying micronutrients to animals.

167 little information about resistance of skin microbiota in the biofilm form to antimicrobial decontam

168 ced evidence for a causal role of intestinal microbiota in the etiology of obesity and insulin resist

169 beneficial allergy-protective members of the microbiota in the regulation of tolerance to food has ex

170 Here, we examined the milk microbiota in water deer (Hydropotes inermis, the most p

171 iffer significantly among women with healthy microbiota (incidence 0.6 per 100 person years in PrEP g

172 Microbiota-induced PPAR-gamma signaling also limits the

173 results clearly prove that modulation of the microbiota induces positive effects on neuronal pathways

174 -precursors in vitro, indicate the commensal microbiota induces sustained changes in RANKL-mediated o

175 mammalian gastrointestinal tract (i.e., the microbiota) influence numerous aspects of host physiolog

176 Transfer of antibiotic-modified gut microbiota inhibits CS, but this response can be restore

177 These results reveal that the microbiota-inosine-A2A receptor axis might represent a p

178 Grasping the principles of host-microbiota interactions (HMIs) at the molecular level is

179 To understand the contribution of plant-microbiota interactions, we studied the root-associated

180 ence of metabolic diversification of enteric microbiota involved in the degradation of algal biomass

181 The gut microbiota is implicated in numerous aspects of health a

182 The microbiota is increasingly recognized for its ability to

183 The microbiota is known to influence the generation of hemat

184 al and adult mice, we show that the neonatal microbiota is unable to prevent colonization by two bact

185 ota in Crohn's disease, but its role on skin microbiota is unknown.

186 gut colonization model in which the natural microbiota is unperturbed, the disruption of glpG but no

187 d Staphylococcus epidermidis) and intestinal microbiota (Lactobacillus reuteri, Enterococcus faecalis

188 lity, environmental factors, and altered gut microbiota, leading to dysregulated innate and adaptive

189 Our results suggest that phyllosphere microbiota, like rhizosphere microbiota, can potentially

190 Enriching HFD with inulin restored microbiota loads, interleukin-22 (IL-22) production, ent

191 The human gut microbiota makes key contributions to the metabolism of

192 e times more worms than adults without their microbiota manipulated as tadpoles.

193 (GOS), is an appealing but underinvestigated microbiota manipulation.

194 Gut microbiota may be altered in patients with cirrhosis, an

195 VD as well as other related diseases.The gut microbiota may play a role in cardiovascular diseases.

196 es associated with non-Lactobacillus vaginal microbiota may trigger immune responses as well as degra

197 nt developmental trajectories of respiratory microbiota members were mode of delivery, infant feeding

198 Distinct changes in specific microbiota members were seen after fluticasone treatment

199 rther, some of the predicted pomegranate gut microbiota metabolites modulated (14)C-D-glucose and (14

200 ablishment of a physiologic gastrointestinal microbiota might be beneficial for this condition.

201 ing early-life disruption of host-associated microbiota might confer protection against diseases late

202 The intestinal microbiota might contribute to enteropathy associated wi

203 We investigated whether vaginal microbiota modulated tenofovir gel microbicide efficacy

204 The ability of microbiota-neuronal interactions to modulate afferent si

205 The cervicovaginal microbiota of 51 participants were composed of community

206 Methods The intestinal microbiota of 541 patients admitted for allo-HCT was pro

207 Different factors may modulate the gut microbiota of animals.

208 Differences have been observed in the microbiota of children with and without allergies, but t

209 by transfer to germ-free mice, that the oral microbiota of diabetic mice is more pathogenic.

210 This bacterium is part of the normal microbiota of estuarine waters and occurs in high number

211 The intestinal microbiota of finishing pigs, fed with 16%, 13% and 10%

212 syringae pv aesculi, and the bark-associated microbiota of horse chestnut (Aesculus hippocastanum) tr

213 in different locations may influence the gut microbiota of infants.

214 In females, stress caused the gut microbiota of lean mice to more closely resemble that of

215 The vaginal microbiota of reproductive-aged women is largely made up

216 s with constitutive immunity defined the gut microbiota of their cohabitants.

217 immune system and the not-yet-stabilized gut microbiota of young children to facilitate its persisten

218 We further discuss potential effects of the microbiota on ASD-associated symptoms, drawing on signal

219 on have drawn attention to the impact of the microbiota on bee health.

220 ic effects of dietary macronutrients and the microbiota on intestinal health and development of colit

221 odels were used to investigate shifts in the microbiota over time and their associations with healing

222 harbour large communities of microorganisms (microbiota), particularly in the gut, and at least 20% o

223 ution correlates with alterations in the gut microbiota, particularly enrichment of Propionibacterium

224 Our observations suggest that intestinal microbiota perturbations precede arthritis, and that mod

225 lating evidence has revealed that intestinal microbiota play an important role in human health and di

226 The symbiotic gut microbiota play pivotal roles in host physiology and the

227 showed that both inoculum and genotype shape microbiota populations in the offspring.

228 Finally, the gut microbiota produces molecules that act on enteric neuron

229 were no significant associations between the microbiota profiles and these severity outcomes (all P>/

230 The groups had similar fecal microbiota profiles, serum markers of inflammation, and

231 sis suggested interactions between HNF4A and microbiota promote gene expression patterns associated w

232 We found that gut microbiota promotes the development of chemotherapy-indu

233 The intestinal microbiota provides colonization resistance against many

234 systemic antibiotics destabilized the wound microbiota, rather than altering overall diversity or re

235 ns, and antibiotic-mediated depletion of the microbiota reduces host resistance to infection.

236 mechanistic insight into how the intestinal microbiota regulates body composition and establish NFIL

237 Despite knowledge the gut microbiota regulates bone mass, mechanisms governing the

238 ence, Wang et al. (2017) reveal that the gut microbiota regulates the expression of circadian-clock g

239 ntation, although composition of the in situ microbiota remained unchanged.

240 s local antimicrobial peptides to facilitate microbiota remodelling.

241 e select for a cariogenic or periopathogenic microbiota, respectively, in a chain of self-reinforcing

242 versity in the gut ensures robustness of the microbiota's ability to generate a consistent immunomodu

243 fic-pathogen-free mice suggest the commensal microbiota's anti-osteoblastic actions are mediated via

244 one Axis intriguingly implies the normal gut microbiota's osteoimmunomodulatory actions are partly me

245 ne mass, mechanisms governing the normal gut microbiota's osteoimmunomodulatory effects on skeletal r

246 model, we show that a constituent of the gut microbiota, segmented filamentous bacteria (SFB), distan

247 uminococcus and Dorea were identified as gut microbiota signatures of NAFL onset and NAFL-NASH progre

248 Depletion of the microbiota significantly attenuated renal damage, dysfun

249 family Bovidae), to determine if common milk microbiota species were present across all three ruminan

250 tion and development in infancy is affecting microbiota stability and thereby resistance against resp

251 For infants at 6 weeks of age, the microbiota structure and function had expanded and diver

252 ve method for reducing the noise inherent in microbiota studies and enabling identification of causal

253 A correlation between host skin peptides and microbiota suggests a mechanism of host-directed symbios

254 The aim of the present study is to identify microbiota surrounding exposed dental implants in patien

255 lf statistical tools to be safely applied to microbiota surveys.

256 saccharides play extensive roles in host-gut microbiota symbiosis beyond dietary polysaccharide diges

257 Social bees harbor a simple and specialized microbiota that is spatially organized into different gu

258 diet high in fiber led to changes in the gut microbiota that played a protective role in the developm

259 Here, we identify members of the microbiota that protect against respiratory infection by

260 ation, and properties of bacteria within the microbiota that regulate lung immunity, and delineate th

261 eding results in increased levels of FFA and microbiota that, even in absence of hyperglycaemia or ov

262 identifying intestinal factors, such as the microbiota, that alter enteric viral replication, we sou

263 IL3 as an essential molecular link among the microbiota, the circadian clock, and host metabolism.

264 Within the composition of the microbiota, the presence of 3 distinct bacterial taxa wa

265 on of one of the main metabolites of the gut microbiota, the short-chain fatty acid acetate.

266 lls (pAPCs) recognize and respond to the gut microbiota through multiple pattern-recognition receptor

267 d to confidently assess the contributions of microbiota to human health.

268 lements such as copper and zinc, altered gut microbiota to more pathogenic bacteria, increased inflam

269 es; the contribution by the gastrointestinal microbiota to this balance has received little attention

270 ution of resident gut microorganisms-the gut microbiota-to human health has surged.

271 ow initial community structure may drive the microbiota trajectory across host development or influen

272 t Clostridium difficile infection with fecal microbiota transplantation (FMT) at a tertiary referral

273 Fecal microbiota transplantation (FMT) from cancer patients wh

274 Fecal microbiota transplantation (FMT) may improve dysbiosis;

275 elopment of cardiovascular diseases, but the microbiota-triggered pattern recognition signaling mecha

276 pecies (ROS) in bacterial cultures and fecal microbiota using 2',7'-dichlorofluorescein diacetate and

277 The human gut microbiota utilizes complex carbohydrates as major nutri

278 Evidence suggests the vaginal microbiota (VM) may influence risk of persistent Human P

279 llected from mice and the composition of the microbiota was analyzed by 16S ribosomal RNA sequencing.

280 mbers of the four major phyla of the poultry microbiota was assembled, including bacterial strains th

281 The composition of gut microbiota was assessed by sequencing the 16S rRNA gene.

282 hese effects were driven by depletion of the microbiota, we performed fecal transplants in antibiotic

283 Fecal samples were collected and microbiota were analyzed using Gut Low-Density Array qua

284 ations in nasal microbiota but not of throat microbiota were associated with asthma.

285 These rare taxa of normal skinfold microbiota were associated with lesions independently of

286 Diversity and composition of fecal microbiota were determined by 16S ribosomal RNA gene amp

287 Diabetic foot ulcer microbiota were found to exist in one of four community

288 Adjacent microbiota were isolated from biopsies and analyzed by 1

289 ividual species levels in subgingival plaque microbiota were not detectable; however, a small portion

290 ed an important role in the structure of the microbiota where flies with constitutive immunity define

291 ituted the bulk of the large-intestinal core microbiota where topologically distinct co-occurrence ne

292 antimicrobial defenses and disrupts the gut microbiota, which leads to overgrowth of indigenous E. c

293 The human microbiota, which plays an important role in health and

294 Food deprivation also challenges the gut microbiota, which relies heavily on host diet for metabo

295 es such as those from dietary intake and the microbiota with cardiometabolic traits.

296 study, we examined the relationship of oral microbiota with EAC and ESCC risk in a prospective study

297 ght be possible to manipulate the intestinal microbiota with prebiotics or other agents to prevent or

298 l visits had a stable lactobacilli dominated microbiota with prevailing Lactobacillus iners.

299 ions occurring between parasites and the gut microbiota, with a profound impact on both host immunity

300 asma cells to coat and contain the commensal microbiota, yet the specificity of these antibodies rema