ライフサイエンスコーパス: microcirculation

1 side-stream dark-field imaging of sublingual microcirculation.

2 is known about the role of S1P in the renal microcirculation.

3 ivated mesenteric venules and inflamed brain microcirculation.

4 g blood cells to vascular endothelium of the microcirculation.

5 ence of MVO indicates a highly dysfunctional microcirculation.

6 ransduce nitric oxide bioactivity within the microcirculation.

7 agonists, and cause microthrombosis in liver microcirculation.

8 owing vascular injury in both the macro- and microcirculation.

9 auses vasoconstriction in the human coronary microcirculation.

10 tion improves renal, hepatic, and intestinal microcirculation.

11 are mediated by a modulation of the coronary microcirculation.

12 cific focus on systemic hemodynamics and the microcirculation.

13 ented the lowest IMR, suggesting a preserved microcirculation.

14 ell understood, particularly in the cerebral microcirculation.

15 s sac was determined by studies of the local microcirculation.

16 Pa, much smaller than typically found in the microcirculation.

17 viding tight control of myogenic tone in the microcirculation.

18 ls from in vivo recordings of the sublingual microcirculation.

19 interactions and albumin permeability in the microcirculation.

20 ation in liver function by compromise of the microcirculation.

21 Vasoconstriction was evident in the microcirculation.

22 have been hypothesized to regulate cortical microcirculation.

23 etween neutrophils and platelets in the lung microcirculation.

24 ay a key role in the development of cerebral microcirculation.

25 ed the effect of GIT1 gene deletion on brain microcirculation.

26 gest that this may not apply to the cerebral microcirculation.

27 able experimental model of the human retinal microcirculation.

28 sed the parietal cortex for visualization of microcirculation.

29 fat and potentiated YFP cell adhesion in the microcirculation.

30 ough diffuse vasodilatation of the pulmonary microcirculation.

31 n on the function of the coronary macro- and microcirculation.

32 f SK(Ca) and IK(Ca) channels in the coronary microcirculation.

33 s to maintain vascular integrity in inflamed microcirculation.

34 laser-induced injury in the cremaster muscle microcirculation.

35 exists between 2 EDHFs in the human coronary microcirculation.

36 5-fold increase in leukocyte adhesion in the microcirculation.

37 uid shear stress by the flowing blood in the microcirculation.

38 rizing factors (EDHFs) in the human coronary microcirculation.

39 eserve resides in remodeling of the coronary microcirculation.

40 ring PPCI can be used to assess the coronary microcirculation.

41 that otherwise restricts blood flow into the microcirculation.

42 ess of leukocyte recruitment to the inflamed microcirculation.

43 or later permanent structural changes to the microcirculation.

44 e markedly to the concomitant changes in the microcirculation.

45 ow pulsatility transmitted into the cerebral microcirculation.

46 flammation, and significantly improved graft microcirculation.

47 renal resistive index (P < 0.001) and kidney microcirculation.

48 hing therapies that strategically target the microcirculation.

49 even for semidilute volume fractions of the microcirculation.

50 is extremely important for maintaining renal microcirculation.

51 thout affecting myeloid cell adhesion in the microcirculation.

52 nduced vasodilatation in the human cutaneous microcirculation.

53 on neurons, without considering the cerebral microcirculation.

54 logy exists between the retinal and cerebral microcirculations; a hypothesized association between di

55 tem/progenitor cells (HSPCs) on the cerebral microcirculation after ischemia-reperfusion injury (I/RI

56 oxin A4 (15-epi-lipoxin A4), on the cerebral microcirculation after ischemia/reperfusion injury.

57 played accelerated thrombus formation in the microcirculation after local trauma.

58 henotype transformation in the regulation of microcirculation after SAH.

59 ular damage and improves graft viability and microcirculation after transplantation.

60 further decreased HAP which impaired hepatic microcirculation, aggravated parenchymal damage, deceler

61 hepatic sinusoids, perturbations in hepatic microcirculation along with changes in various liver cel

62 show that RESA influences ring-stage Pf-RBCs microcirculation, an effect that is fever-enhanced.

63 zed at a shear stress typical of that in the microcirculation and after treatment with a myosin-II in

64 w-energy shockwave therapy would restore the microcirculation and alleviate renal dysfunction in reno

65 measure of the vasodilatory capacity of the microcirculation and calculated using the equation: base

66 The microcirculation and contractility of the subtended myoc

67 accentuate acidosis accompanying changes in microcirculation and contribute to enhanced muscle fatig

68 option for the correction of impaired renal microcirculation and endothelial dysfunction.

69 Diabetes impairs the microcirculation and function of various vital tissues t

70 potential to repair and regenerate the lung microcirculation and have shown promise in preclinical e

71 c hemoglobin-derived products, reductions in microcirculation and hypoperfusion.

72 mia is a blood disorder, known to affect the microcirculation and is characterized by painful vaso-oc

73 moving mice, and its application to imaging microcirculation and neuronal Ca(2+) dynamics.

74 lude enhanced (protein) metabolism, improved microcirculation and organ function, effects on immune f

75 VAI that anti-angiogenic therapy can improve microcirculation and oxygen saturation and reduce vessel

76 ole in normal erythrocyte deformation in the microcirculation and participates in maintenance of eryt

77 characterized by abnormalities in sublingual microcirculation and peripheral tissue perfusion, which

78 The graft microcirculation and portal venous flow were increased b

79 ation regarding the function of the coronary microcirculation and provides a quantitative surrogate e

80 can break the cycle of injury and enable the microcirculation and renal function to recover.

81 % revealed devastated hepatic and intestinal microcirculation and severe side effects in CLP induced

82 HMP and HR similarly improved cortical microcirculation and significantly reduced maximal serum

83 infection/inflammation requires exiting the microcirculation and subsequent crossing of epithelial b

84 implicate astrocytes in the control of local microcirculation and suggest that one of their physiolog

85 ta2-deficient mice showed unaltered cerebral microcirculation and the absence of ischemia after PbA i

86 ascular permeability, but alterations in the microcirculation and their evolution over the course of

87 an be used to analyse the functioning of the microcirculation and to establish quantitative relations

88 large part to failure to rapidly access the microcirculation and to gain access to this highly antic

89 Insulin delivery to muscle microcirculation and transendothelial transport are 2 di

90 gested the presence of disorders in cerebral microcirculation and/or changes in cerebral oxygenation.

91 and kidneys and determining renal function, microcirculation, and accumulation of tubular 4-hydroxyn

92 avior of human CLL cells within the mouse LN microcirculation, and discovered that CLL cells bind to

93 pericyte alpha-SMA phenotype transformation, microcirculation, and neurological function in SAH rats.

94 gulated, provide new insight into control of microcirculation, and provide a framework to explain its

95 atelet consumption, fibrin deposition in the microcirculation, and tissue damage.

96 opy was used to visualize the mouse cerebral microcirculation: AnxA1 null mice exhibited more white b

97 lary networks, and fluid handling across the microcirculation are compelling avenues for future inves

98 Arterioles in the peripheral microcirculation are exquisitely sensitive to changes in

99 Abnormalities in the coronary microcirculation are increasingly recognized as an eleme

100 Decreases in buccal microcirculation are indicative of the severity of hemor

101 tained or even increased, alterations in the microcirculation are likely.

102 ease; namely, that cells deoxygenated in the microcirculation are not merely undeformable, but will a

103 at regulate passage of leukocytes out of the microcirculation are now understood.

104 o examine the role of dietary factors in the microcirculation are required.

105 CD8(+) T cells in the liver microcirculation are visualized making intimate contact

106 Using mucosal microcirculation as an in situ assay, cheek pouch tissue

107 otic debris plugging the downstream coronary microcirculation as well as ischemia/reperfusion injury

108 of CO-saline increased flow maximally in the microcirculation at 30 mins after infusion (207% in arte

109 n PPCI-treated patients with STEMI, coronary microcirculation begins to recover within 24 h and recov

110 Differences in microcirculation between the hemispheres in eyes with gl

111 n therapies is to promote integration of the microcirculation between the implanted cells and the hos

112 Endothelial function in macro- and microcirculation, blood pressure, anthropometric measure

113 changes in local blood flow in the cortical microcirculation but also to augmented metabolism.

114 HES improved liver microcirculation, but exhibited significantly increased

115 debris and prevent its embolization into the microcirculation, but their effect on clinical outcome h

116 The conjunctival microcirculation can be non-invasively imaged and thus e

117 HES improved liver microcirculation, cardiac index and DO(2)-I, but signifi

118 oncept, endothelial dysfunction of the renal microcirculation causes albuminuria by increasing glomer

119 wo parallel pathways: (1) reduction in brain microcirculation causing diminished brain capillary perf

120 totic pressure at which the flow through the microcirculation ceases, the reservoir pressure related

121 or donor-specific antibodies (DSA), C4d, and microcirculation changes (MC).

122 l transplantation-induced changes in hepatic microcirculation contributed to transplanted cell cleara

123 We tested whether the chaotic tumor microcirculation could be reconfigured by the mesenchyme

124 To determine whether the regenerated microcirculation could regulate flow, the muscle was sub

125 Buccal microcirculation decreased after bleeding but was restor

126 The microcirculation-derived backward expansion wave is a ne

127 ial cell dysfunction and ameliorated hepatic microcirculation disorder.

128 RA showed no intestinal microcirculation disturbance compared to sham, but signi

129 organ integrity, systemic inflammation, and microcirculation during hemolysis.

130 Impaired cerebral microcirculation during sepsis is associated with progre

131 macrocirculation to the buccal and cerebral microcirculations during hemorrhage and after fluid resu

132 d to increased compression of the intramural microcirculation, especially at the subendocardium.

133 hysiological endothelial cell barrier of the microcirculation, even in the absence of increased overt

134 The microcirculation exemplifies the mesoscale in physiologi

135 speculated that erythrocyte-pausing in both microcirculations facilitates metabolic exchange across

136 Despite robust neovascularization, the microcirculation formed by regenerative angiogenesis in

137 Transfection of the microcirculation guided by a targeted, acoustically-acti

138 Thrombus formation in the cerebral microcirculation has been proposed to contribute to seco

139 mechanism of IL2-induced disruption of brain microcirculation has not been determined previously.

140 ormation on dynamic changes in the cutaneous microcirculation; however, these only weakly correspond

141 is a frequent condition that involves renal microcirculation impairment, infiltration of inflammator

142 Natural killer (NK) cells localize in the microcirculation in antibody-mediated rejection (AMR) an

143 ital microscopy of inflamed cremaster muscle microcirculation in bone marrow chimera mice.

144 lenishment with EPCs would protect the renal microcirculation in chronic experimental renovascular di

145 A healthy, functional microcirculation in combination with nonobstructed epica

146 Our goal was to characterize the sublingual microcirculation in healthy volunteers and patients with

147 the effect of RBC transfusion on sublingual microcirculation in hemorrhagic shock patients.

148 The precise manipulation of microcirculation in mice can facilitate mechanistic stud

149 Here, by directly imaging the lung microcirculation in mice, we show that a large number of

150 estigate the effect of CKD on the myocardial microcirculation in patients referred for clinical (82)R

151 ss the vasodilatory capacity of the coronary microcirculation in patients with NSTEMI when compared w

152 mization, inhaled nitric oxide would improve microcirculation in patients with sepsis and that improv

153 The main characteristics of sublingual microcirculation in patients with septic shock are hypop

154 solutions on liver and intestine injury and microcirculation in septic rodents.

155 chitecture and function of the reconstructed microcirculation in skeletal muscle.

156 o demonstrated an increase in the density of microcirculation in the ischemic hind limb, suggesting t

157 ysm, but how leaked hemoglobin regulates the microcirculation in the pathophysiology of early brain i

158 ty and 2) indicate the important role of the microcirculation in the regenerative capacity of a muscl

159 s currently available to assess the coronary microcirculation in the setting of ST-segment-elevation

160 was to assess the time course of sublingual microcirculation in traumatic hemorrhagic shock during t

161 Alterations of microcirculation in traumatic hemorrhagic shock patients

162 ro as well as targeted apoptosis of the lung microcirculation in vivo.

163 ng novel in vitro microfluidic models of the microcirculation, including 1 capable of deoxygenating t

164 clusion, lower capillary density of the skin microcirculation independently associated with albuminur

165 RBC transfusion improves sublingual microcirculation independently of macrocirculation and t

166 Microcirculation inflammation (MCI = g + ptc score) was

167 c antibodies (dnDSA) we studied the value of microcirculation inflammation (MI; defined by the additi

168 of natural killer markers were correlated to microcirculation inflammation and graft outcomes to a gr

169 because both groups had a similar degree of microcirculation inflammation and peritubular capillary

170 4d deposition, transplant glomerulopathy, or microcirculation inflammation had a 100.0% graft surviva

171 as observed between patients with or without microcirculation inflammation in contrast to previous re

172 Microcirculation inflammation was prevalent, with glomer

173 rterial pressure, transplant glomerulopathy, microcirculation inflammation, and de novo/recurrent glo

174 renal transplant patients with de novo DSA, microcirculation inflammation, defined by g + ptc, assoc

175 There was, however, no change in microcirculation inflammation, gene expression patterns,

176 Microcirculation inflammation, particularly glomerulitis

177 Microcirculation inflammation, particularly glomerulitis

178 ced C4d staining in peritubular capillaries, microcirculation inflammation, splenic plasma cells, cir

179 ) recurrent biopsies showing the presence of microcirculation inflammation.

180 orter time to rejection (P<0.001), increased microcirculation injury (P=0.002), and C4d capillary dep

181 pecificity of prediction of TG compared with microcirculation injury score alone.

182 as well as MSCs, are able to egress from the microcirculation into the parenchymal space, forming pro

183 nteraction of B. turicatae with the cerebral microcirculation involves both binding and crossing brai

184 estration of parasitized erythrocytes in the microcirculation is considered the central pathophysiolo

185 6C(low)) patrolling along the vessels of the microcirculation is critical for endothelial homeostasis

186 wever, the role of this pathway in the brain microcirculation is not known.

187 The vasodilatory capacity of the microcirculation is preserved in selected patients with

188 The microcirculation is responsible for orchestrating adjust

189 se network attributes exist in a regenerated microcirculation is unknown, and methodologies for answe

190 lates with vasoconstrictive responses in the microcirculation is unknown.

191 elium-to-blood ratios are much higher in the microcirculation, it is likely that stasis contributes t

192 excessive pulsatile energy into the cerebral microcirculation, leading to microvascular damage and im

193 ss and excessive flow pulsatility damage the microcirculation, leading to quantifiable tissue damage

194 The scores correlated with the presence of microcirculation lesions and donor-specific antibody.

195 psies from 315 patients, based on histology (microcirculation lesions) and donor-specific HLA antibod

196 flammatory activities in the murine inflamed microcirculation (leukocyte adhesion being the readout)

197 PAH, which would account for skeletal muscle microcirculation loss and exercise intolerance.

198 ce in PAH is associated with skeletal muscle microcirculation loss and impaired angiogenesis secondar

199 cise capacity ([Formula: see text]o2max) and microcirculation loss on quadriceps muscle biopsy (in CD

200 We therefore hypothesized that the cerebral microcirculation may be preserved in hemorrhagic shock i

201 trance of bioactive oxidized lipids into the microcirculation may mediate adverse clinical outcomes d

202 e indicates that alterations of the cerebral microcirculation may play a role in Alzheimer disease, t

203 Alterations in the macrocirculation and microcirculation may represent early clinical manifestat

204 ime points and included visualization of the microcirculation, measurement of tissue oxygenation, and

205 crofluidic optical stretcher, a microfluidic microcirculation mimetic, and real-time deformability cy

206 ecisely organized and exquisitely responsive microcirculation must form.

207 neous circulation improved postresuscitation microcirculation, myocardial and cerebral functions, and

208 ignificantly better postresuscitation tissue microcirculation, myocardial ejection fraction, and neur

209 tized and intestinal (n = 6/group) and liver microcirculation (n = 6/group) were obtained using intra

210 estration of parasitized erythrocytes in the microcirculation, not from hypovolemia.

211 formation, i.e. conditions that exist in the microcirculation of active skeletal muscle.

212 o assess the effects on macrocirculation and microcirculation of adding an intra-aortic balloon pump

213 of the isolated retina and in the cremaster microcirculation of anesthetized mice.

214 d after ischemia-reperfusion in the coronary microcirculation of angptl4-deficient mice.

215 The specialized tightly controlled microcirculation of craniofacial neurosensory organs is

216 These findings demonstrate that the microcirculation of healthy aged skin can further dilate

217 The authors demonstrate a tool to study the microcirculation of healthy subjects noninvasively.

218 The microcirculation of septic patients has been characteriz

219 xercise, vasodilatation initiated within the microcirculation of skeletal muscle ascends the resistan

220 w the sympathetic nervous system affects the microcirculation of skeletal muscle with ageing or wheth

221 MS adhesion in the microcirculation of the anterior and posterior chamber w

222 clinical studies demonstrated that impaired microcirculation of the gastric conduit almost recovers

223 , red blood cell zinc protoporphyrin, in the microcirculation of the lower lip.

224 way, much has been learned about the unique microcirculation of the lung, including immune cell migr

225 ry signal provided by MC3R in the mesenteric microcirculation of the mouse, acting to down-regulate c

226 Understanding the differences in vascular microcirculation of the peripapillary retinal nerve fibe

227 To investigate the changes in the microcirculation of the peripapillary RNFL of eyes with

228 Albumin permeability in the intact microcirculation of the Zucker diabetic fatty (ZDF) rat

229 The cremasteric microcirculation of wild-type (C57BL/6), Gal-3(-/-), and

230 Extravasation of albumin in the microcirculation of ZDF rats was significantly increased

231 of hemorrhagic shock, including decreases in microcirculations of buccal mucosa, cerebral microvascul

232 Evaluation of microcirculation perfusion is critical for optimization

233 e (NO) depletion by plasma hemoglobin in the microcirculation plays a central role in the pathogenesi

234 data suggest that the status of the coronary microcirculation plays a role in determining susceptibil

235 nctional and structural abnormalities of the microcirculation plays an important role in systemic scl

236 dark-field videomicroscopy of the sublingual microcirculation prior to and 2 hours after study drug i

237 Hepatic damage, microcirculation, regeneration, and vascular remodeling

238 How APOE genotype affects brain microcirculation remains elusive.

239 ular benefits, but its action in the retinal microcirculation remains unknown.

240 Skin microcirculation responded to ANP similarly.

241 albuminuria, measurements of skin and muscle microcirculation, retinal arteriolar and venular diamete

242 dependent relation after adjusting for total microcirculation scores.

243 al microscopy studies of the mouse cremaster microcirculation showed that tumor necrosis factor-alpha

244 TEMI when compared with a model of preserved microcirculation (stable angina [SA] cohort: culprit and

245 magnetic carriers in blood vessels and tumor microcirculation still remains unclear.

246 me subsumes myocardial dysfunction, impaired microcirculation, systemic inflammatory response, and ne

247 as of incomplete clearance of blood from the microcirculation that did not clear after a further atte

248 y plays an important role in maintaining the microcirculation that is essential for the movement of w

249 wever, there are unique aspects of the human microcirculation that serve as the focus of this review.

250 (Dhb) hemoglobin specifically in the mucosal microcirculation through polarization gating.

251 insulin-induced vasoreactivity in the muscle microcirculation through secretion of adiponectin and su

252 Microcirculation through the newly formed vascular anast

253 edance are thought to sensitize the cerebral microcirculation to harmful effects of excessive pressur

254 ntravital microscopy of rat cremaster muscle microcirculation to track intraarterially delivered MSCs

255 um-b-value regimens as a surrogate marker of microcirculation, to study its correlation with dynamic

256 (H2O2) regulates vascular tone in the human microcirculation under physiological and pathophysiologi

257 pts the blood-brain barrier and alters brain microcirculation, underlying vascular leak syndrome that

258 servational study to evaluate the sublingual microcirculation using side-stream dark-field imaging in

259 e simulate deformable red blood cells in the microcirculation using the immersed boundary method with

260 ulmonary artery pressures but did not affect microcirculation variables in cardiogenic shock patients

261 d that the SVF cells would form a functional microcirculation via vascular assembly and inosculation

262 Thus, the curcumin vasomotor activity on microcirculation was alpha-Ad and beta-Ad receptor-depen

263 Pulmonary microcirculation was analyzed at 0 hr and 2 hrs using in

264 usive reactive hyperaemia (PORH) in the skin microcirculation was assessed by laser Doppler flowmetry

265 The sublingual microcirculation was assessed with a Sidestream Dark Fie

266 The buccal microcirculation was concurrently reduced.

267 Reduced RNFL microcirculation was detected in the normal hemisphere o

268 However, the cerebral microcirculation was essentially unaffected by hemorrhag

269 The sublingual microcirculation was estimated at the study inclusion af

270 The sublingual microcirculation was evaluated by means of sidestream da

271 ndard hemodynamic assessment, the sublingual microcirculation was evaluated using sidestream dark-fie

272 Compared with normal eyes, reduced RNFL microcirculation was found in the normal hemisphere of e

273 Sublingual microcirculation was impaired for 72 hours despite resto

274 tion of the macrocirculation, the sublingual microcirculation was impaired for at least 72 hours.

275 The RNFL microcirculation was measured within an annulus region c

276 Microcirculation was not altered by the increased number

277 In the control group, no microcirculation was observed after 6 hours of cold stor

278 In addition, the sublingual microcirculation was observed using sidestream dark-fiel

279 DCT) values were determined; contribution of microcirculation was quantified in perfusion fraction (F

280 ts with severe sepsis in whom the sublingual microcirculation was visualized using orthogonal polariz

281 Since inflammation is known to modulate the microcirculation, we investigated the hypothesis that ch

282 complement-mediated injury to the allograft microcirculation, we transplanted C3-deficient and C3-in

283 servational study where macrocirculation and microcirculation were assessed with clinical-, Doppler e

284 rization spectral imaging, recordings of the microcirculation were taken at baseline and hourly inter

285 d side-stream dark-field-assessed sublingual microcirculation were unchanged by stopping and restarti

286 The cerebral and buccal microcirculations were visualized concurrently with the

287 RA exhibited no derangements in liver microcirculation when compared to sham and HES.

288 usion revealed best results in mortality and microcirculation, when compared with colloid infusion.

289 , megakaryocytes extend projections into the microcirculation, where shear facilitates fragmentation

290 y with IVFs affects sRBC biomechanics in the microcirculation, where vaso-occlusion takes place.

291 to the nucleus is not dependent on the lens microcirculation, which is believed to be responsible fo

292 s to the endothelium mediated by PMNs in the microcirculation, which provides a rationale and mechani

293 pacity to reconcile barrier phenotype of the microcirculation with an angiogenic cascade is not known

294 significant reduction of liver and intestine microcirculation with severe side effects on coagulation

295 In addition, treatment of the cerebral microcirculation with tempol restored impaired nNOS-depe

296 col describes an in vitro model of the human microcirculation with the potential to recapitulate disc

297 , noninvasive techniques for measuring human microcirculation, with a focus on methods, interpretatio

298 ional structure of the fiber cell tissue and microcirculation within it, as required to maintain tran

299 esized that the vasodilatory response of the microcirculation would be preserved in NSTEMI.

300 on in patients with sepsis and that improved microcirculation would improve lactate clearance and mul