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1 nduction, iron deficiency and a hypochromic, microcytic anemia.
2 ssive loss of body (but not facial) hair and microcytic anemia.
3   Heterozygous gammabeta(0) mice suffer from microcytic anemia.
4 ion by red blood cells leads to hypochromic, microcytic anemia.
5 -/- mice represent a new paradigm of genetic microcytic anemia.
6 hianti (cia) mutant manifests a hypochromic, microcytic anemia after the onset of embryonic circulati
7                 Mice lacking Pcbp1 exhibited microcytic anemia and activation of compensatory erythro
8 ns or deletions may be a cause of refractory microcytic anemia and bone marrow iron depletion in pati
9                       It is characterized by microcytic anemia and by iron loading, and can be treate
10 entified in a female with severe hypochromic microcytic anemia and iron overload.
11                  Mice that lack IRP2 develop microcytic anemia and neurodegeneration associated with
12                     bdh2 null mice developed microcytic anemia and tissue iron overload, especially i
13 yndrome (joint contractures, muscle atrophy, microcytic anemia, and panniculitis-induced childhood-on
14 rized by joint contractures, muscle atrophy, microcytic anemia, and panniculitis-induced lipodystroph
15  (cdy), a zebrafish mutant with hypochromic, microcytic anemia, and positioned the mutant gene on lin
16                                 Hypochromic, microcytic anemias are typically the result of inadequat
17   The mutation, zinfandel, has a hypochromic microcytic anemia as an embryo, but later recovers in ad
18 ted with higher odds of anemia, particularly microcytic anemia (asthma: 1.61; 1.09-2.38; P = .02; ecz
19 deficiency anemia patients, who present with microcytic anemia caused by hyperhepcidinemia, and of qu
20            These animals exhibit hypochromic microcytic anemia due to impaired intestinal iron absorp
21 plinary guidelines on the management of rare microcytic anemias due to genetic disorders of iron meta
22 ome akin to typhoid fever with splenomegaly, microcytic anemia, extramedullary erythropoiesis, and in
23 derstanding of the pathogenesis of inherited microcytic anemias has gained from the identification of
24 9 nmol/L]), leukocytosis (13 800/mm(3)), and microcytic anemia (hemoglobin level, 7.2 g/dL).
25 cythemia in heterozygotes and a hypochromic, microcytic anemia in homozygotes.
26 yses reveal a mild, congenital, hypochromic, microcytic anemia intrinsic to the hematopoietic system
27                                              Microcytic anemia (mk) mice and Belgrade (b) rats have s
28 notype are similar to those reported for the microcytic anemia (mk) mutation in the mouse.
29 athogenesis of a new recessive, hypochromic, microcytic anemia mouse mutant, nm1054.
30 a genetic locus not previously implicated in microcytic anemia or iron phenotypes.
31 nhibition of mTORC1 results in macrocytic or microcytic anemia, respectively.
32 e normal recipients of HBD marrow obtained a microcytic anemia similar to the donor.
33  and alpha2-globulin and more frequently had microcytic anemia than those without such deposits (P =
34 ene symbol hbd) is characterized by a severe microcytic anemia that is inherited in an autosomal-rece
35          These Tf-deficient mice have severe microcytic anemia, tissue iron overload, and hepcidin de
36                               A hypochromic, microcytic anemia was present from birth, and platelet c
37 d-specific ALA synthase 2 (ALAS2) gene cause microcytic anemia, whereas mitochondrial DNA deletions a
38          Chronic turpentine treatment led to microcytic anemia, which was prevented by concurrent adm
39       The Rac1(-/-);Rac2(-/-) mice developed microcytic anemia with a hemoglobin drop of about 20% an
40 Homozygous sublytic mice develop hypochromic microcytic anemia with reduced osmotic fragility of RBCs
41 ice alters actin assembly in RBCs and causes microcytic anemia with reticulocytosis, implicating Rac
42  moderately severe, congenital, hypochromic, microcytic anemia, with an elevated red cell zinc protop

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