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1 nduction, iron deficiency and a hypochromic, microcytic anemia.
2 ssive loss of body (but not facial) hair and microcytic anemia.
3 Heterozygous gammabeta(0) mice suffer from microcytic anemia.
4 ion by red blood cells leads to hypochromic, microcytic anemia.
5 -/- mice represent a new paradigm of genetic microcytic anemia.
6 hianti (cia) mutant manifests a hypochromic, microcytic anemia after the onset of embryonic circulati
8 ns or deletions may be a cause of refractory microcytic anemia and bone marrow iron depletion in pati
13 yndrome (joint contractures, muscle atrophy, microcytic anemia, and panniculitis-induced childhood-on
14 rized by joint contractures, muscle atrophy, microcytic anemia, and panniculitis-induced lipodystroph
15 (cdy), a zebrafish mutant with hypochromic, microcytic anemia, and positioned the mutant gene on lin
17 The mutation, zinfandel, has a hypochromic microcytic anemia as an embryo, but later recovers in ad
18 ted with higher odds of anemia, particularly microcytic anemia (asthma: 1.61; 1.09-2.38; P = .02; ecz
19 deficiency anemia patients, who present with microcytic anemia caused by hyperhepcidinemia, and of qu
21 plinary guidelines on the management of rare microcytic anemias due to genetic disorders of iron meta
22 ome akin to typhoid fever with splenomegaly, microcytic anemia, extramedullary erythropoiesis, and in
23 derstanding of the pathogenesis of inherited microcytic anemias has gained from the identification of
26 yses reveal a mild, congenital, hypochromic, microcytic anemia intrinsic to the hematopoietic system
33 and alpha2-globulin and more frequently had microcytic anemia than those without such deposits (P =
34 ene symbol hbd) is characterized by a severe microcytic anemia that is inherited in an autosomal-rece
37 d-specific ALA synthase 2 (ALAS2) gene cause microcytic anemia, whereas mitochondrial DNA deletions a
40 Homozygous sublytic mice develop hypochromic microcytic anemia with reduced osmotic fragility of RBCs
41 ice alters actin assembly in RBCs and causes microcytic anemia with reticulocytosis, implicating Rac
42 moderately severe, congenital, hypochromic, microcytic anemia, with an elevated red cell zinc protop
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