1 he target population) were tested for L. loa microfilaremia.

2 r role in both the presence and intensity of microfilaremia.

3 r the detection and quantification of L. loa microfilaremia.

4  microfilaremic, whereas 18 had cleared both microfilaremia and antigenemia.

5                           The prevalences of microfilaremia and clinical morbidity were lowest in per

6 ecific microfilarial rate, mean intensity of microfilaremia, and prevalence of leg edema.

7 assessed for both W. bancrofti infection and microfilaremia by controlling for individual risk factor

8 Patent infections with long-term, high-grade microfilaremia do not develop in nonendemic individuals.

9 R was equally sensitive for the detection of microfilaremia due to Wuchereria bancrofti (2 of 46 samp

10 o infection with Wuchereria bancrofti and to microfilaremia in a village of the Republic of Congo.

11    Antibody reactivity was detectable before microfilaremia in experimentally infected rhesus monkeys

12                            The prevalence of microfilaremia in the entire study population was 66%.

13 ly constant for the subjects with persistent microfilaremia (Mf(+/+)), in contrast to sharp decreases

14 r both EN subjects and subjects with cleared microfilaremia (Mf(+/-)).

15 d mice, particularly those that did not have microfilaremia (Mf(-)), had more severe anemia and loss

16 ug treatment regimens capable of suppressing microfilaremia to very low levels, along with improvemen

17 5) were associated with antigenemia, whereas microfilaremia was associated with significantly decreas