ライフサイエンスコーパス: microfilariae

1 ively but a third correlated negatively with microfilariae.

2 sons with high levels of circulating Loa loa microfilariae.

3 e, the majority of the mice remained free of microfilariae.

4 intracorneal binding complex or live Brugia microfilariae.

5 ost of whom (67.5%) had no detectable L. loa microfilariae.

6 st antigens from adult female worms and skin microfilariae.

7 examined them by microscopy for W bancrofti microfilariae.

8 eatment, we examined blood samples again for microfilariae.

9 Parasitic extracts prepared from B. malayi microfilariae and adult worms were incubated with recomb

10 ycle stages that live in the mammalian host (microfilariae and adult worms).

11 Brugia malayi microfilariae and adults were exposed in vitro to 0.75 t

12 al level, between infection with O. volvulus microfilariae and bilateral blindness was examined, by u

13 C57BL/6 mice immunized with killed B. malayi microfilariae and challenged intravenously with live mic

14 itability estimates suggested that levels of microfilariae and circulating adult worm antigen, as wel

15 nfection status as determined by blood-borne microfilariae and filarial antigenemia.

16 from doxycycline-treated patients contained microfilariae, and 97% of those patients were without mi

17 male, adult female, blood-borne and uterine microfilariae, and infective L3 larvae.

18 extended to mosquitoes that were exposed to microfilariae but did not support larval development.

19 iced leader sequence SL1 and is expressed in microfilariae but not in third-stage larvae or adult wor

20 t dependent on the appearance of circulating microfilariae, but may be due to initial low levels of p

21 y response is associated with the release of microfilariae by female worms.

22 A B. malayi microfilariae cDNA library was immunoscreened with antis

23 divided into an early phase with killing of microfilariae, clinical symptomatology, increases in pla

24 ssion and/or pathology (directed toward skin microfilariae) could provide the necessary 'final punch'

25 ariae and challenged intravenously with live microfilariae exhibit many of the characteristics of hum

26 he parasite, with high levels of circulating microfilariae, few clinical symptoms, and diminished fil

27 Birds with microfilariae had elevated heterophil to lymphocyte rati

28 Microfilariae harboring Wolbachia organisms were injecte

29 ich are used mostly in combination to reduce microfilariae in blood (lymphatic filariasis) and skin (

30 -friendly diagnostic tool to quantify L. loa microfilariae in peripheral blood, enables rapid, point-

31 ce the concentration of Wuchereria bancrofti microfilariae in the blood for months to years.

32 Children with high concentrations of microfilariae in the blood were admitted to hospital and

33 chia in O. volvulus is effective in clearing microfilariae in the skin of onchocerciasis patients wit

34 dium spp.) and parasitic filarial nematodes (microfilariae) in wild birds (New Caledonian Zosterops s

35 ee main stages of the B. pahangi life cycle: microfilariae, infective larvae and adults.

36 s suppressed basophils in vitro, transfer of microfilariae into mice did not result in basophil suppr

37 h of the cDNA libraries when compared to the microfilariae, L2, and both adult stages of the parasite

38 For microfilariae, median inhibitory concentrations (IC50 va

39 , led to the clearance of Brugia malayi (Bm) microfilariae (mf) from infected jirds.

40 in which an allergic response is induced to microfilariae (Mf) in the lungs.

41 ing live infective-stage larvae (L3) or live microfilariae (Mf) of Brugia malayi, a causative agent o

42 on of human monocytes, cells were exposed to microfilariae (mf) of Brugia malayi, and their phenotypi

43 individuals, we examined the effect of live microfilariae (mf) on expression and function of TLRs in

44 l measures where onchocerciasis was endemic, microfilariae (MF) prevalence rates were 82.8% and 65.1%

45 ght patients who had skin snips positive for microfilariae (Mf) were studied 120 days after receiving

46 nting cell function and is most specific for microfilariae (MF), the parasite stage found in large nu

47 om elutriated monocytes were exposed to live microfilariae (mf), the parasite stage that circulates i

48 indicate that Bm-spn-2 is expressed only by microfilariae (Mf), which are the long-lived blood-dwell

49 for point-of-care quantification of Loa loa microfilariae (mf).

50 d macrophages, were exposed in vitro to live microfilariae (mf).

51 filarial antigens (CFAs) and 44 who also had microfilariae (MF).

52 tin, suggesting that the loss of circulating microfilariae, not the reduction of Wolbachia bacteria,

53 Among all five villages with a prevalence of microfilariae of 2 to 38%, the probability of transmissi

54 Finally, live microfilariae of B. malayi were able to downregulate cel

55 We have shown that live microfilariae of Brugia malayi induce caspase-dependent

56 n response to live infective-stage larvae or microfilariae of Brugia malayi, we found significant imp

57 Although excretory/secretory products from microfilariae of L. sigmodontis suppressed basophils in

58 Microfilariae of some species exhibit temporal separatio

59 ive-stage larvae of Brugia malayi but not to microfilariae of this parasite.

60 oa microfilarial density greater than 20,000 microfilariae per milliliter of blood, who were consider

61 manifestations of human onchocercal disease, microfilariae-positive Ghanaian subjects with inflammato

62 vity of the test with serum samples from 106 microfilariae-positive subjects was 90.6%.

63 flammatory ocular disease were compared with microfilariae-positive subjects without ocular disease.

64 ete symbionts, block embryogenesis, and stop microfilariae production.

65 Blood-borne Brugia malayi microfilariae survived for significantly longer time per

66 19) had significantly higher levels of skin microfilariae than children of uninfected mothers (n = 1

67 sis (river blindness), including the larvae (microfilariae) that migrate into the cornea.

68 was isolated from the sheath of W. bancrofti microfilariae through ultrafiltration, followed by chrom

69 Importantly, co-occurrence patterns with microfilariae varied in direction among avian malaria sp

70 osquitoes rapidly destroy invading B. malayi microfilariae via a defense response known as melanotic

71 roduction of TNF-alpha after chemotherapy of microfilariae was also only detected in LPS-responsive C

72 ortion of children who remained positive for microfilariae was significantly lower in the ivermectin

73 i adult males, adult females, L3 larvae, and microfilariae were assessed using a wide dose range (0-1

74 Microfilariae were more sensitive to the action of CaGC

75 Ivermectin has controlled transmission of microfilariae, with an African Program elimination targe

76 ctor control and drug treatment to eliminate microfilariae, with no means available to prevent infect