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1 iotics and alterations in fecal microbiota ("microflora").
2  make up the microbiota (previously known as microflora).
3 ted by an immune response to bacteria in the microflora.
4 infections that do not belong to the vaginal microflora.
5  in the mutant mice required the presence of microflora.
6  broad spectrum of antibiotics to reduce gut microflora.
7 btained from volunteers with healthy vaginal microflora.
8 ct is in intimate contact with the commensal microflora.
9 ignal transducer for TLRs in response to the microflora.
10 s and inflammation triggered by the resident microflora.
11 n part by an aberrant response to intestinal microflora.
12 s evolved in the presence of diverse enteric microflora.
13 to excessive immunologic responses to normal microflora.
14 ate immune responses of the colon to enteric microflora.
15 een the host immune system and its commensal microflora.
16 growth of selected members of the intestinal microflora.
17  vitamins that are synthesized by the normal microflora.
18 ffering microbiologic effects on the vaginal microflora.
19 esponsive to food antigens and the commensal microflora.
20 s evolved in the presence of diverse enteric microflora.
21 may contribute to the maintenance of vaginal microflora.
22 al, a function dependent on licensing by gut microflora.
23 the appendix to sequester it from intestinal microflora.
24 rom interactions between GALT and intestinal microflora.
25 t is commonly found as a member of the human microflora.
26 ated and perpetuated by the intestinal tract microflora.
27 nic inflammation in response to inflammatory microflora.
28 rmal role as a benign inhabitant of the oral microflora.
29 d by higher eukaryotes from the diet and gut microflora.
30 g upon probing, and reducing the subgingival microflora.
31 n human colon xenografts that lack a luminal microflora.
32 phabeta TCR(+) IELs even in the absence of a microflora.
33 ditis, despite being part of the normal oral microflora.
34 f periodontal disease and to the periodontal microflora.
35 in the establishment and ecology of the oral microflora.
36 sence of CpsB cross-reactive antigens in the microflora.
37 ent of the normal mouse and human intestinal microflora.
38 labeled cells of strain 299R from other leaf microflora.
39 athogenic bacteria without disturbing normal microflora.
40 rabbits and thereby acquired a different gut microflora.
41 ges in antibiotic susceptibility of the host microflora.
42 esponsiveness toward the lumenal prokaryotic microflora.
43 ased production of ethanol by the intestinal microflora.
44 ions of bacterial RNA derived from symbiotic microflora.
45 ll-intestinal epithelium requires the normal microflora.
46 isease in individuals with a compromised gut microflora.
47  cleanliness of the mouse colony and the gut microflora.
48 produced by the epithelium or H2S-generating microflora.
49 , ion concentrations, nutrient quantity, and microflora.
50 gy and mortality compared to mice deplete of microflora.
51  to environmental microbes and the commensal microflora.
52 with non-pathogenic members of the commensal microflora.
53 elieved to be under the influence of the gut microflora.
54 iols by beta-lyases that originate from oral microflora.
55 ic acids) were biodegraded by the endogenous microflora.
56 osedly induced by beta -lyases from the oral microflora.
57 each trial were fermented only by indigenous microflora.
58 se to gain a metabolic advantage over intact microflora.
59 , appears to reflect microaspiration of oral microflora.
60 etween the immune system of the host and gut microflora.
61 ese factors may include subsets of commensal microflora.
62 h might impact the intestinal homeostasis of microflora.
63 ar frequency of breakthrough growth of stool microflora (25% versus 31%, respectively).
64 s demonstrate a transition in the intestinal microflora accompanied by a dynamic change of its abilit
65 a isoflavone concentrations and modified gut microflora activities [beta-glucoside hydrolysis and equ
66                               The intestinal microflora also impacts immunity but its role in enteric
67 Here we report that the intestinal bacterial microflora and a functional Toll-like receptor 4 (TLR4),
68 s pathway involves synergy between commensal microflora and a sex-dependent liver enzyme, flavin-cont
69  new evidence for a possible role of altered microflora and altered host microbial interactions may p
70  the large intestine of mice is dependent on microflora and coincident with modulation of the host im
71 m affects both composition of the intestinal microflora and colonization of the gastrointestinal trac
72 S. aureus is a common inhabitant of the skin microflora and colonizes the nares and other human mucos
73  completely dependent on the presence of gut microflora and could be established by colonization with
74 t leads to the establishment of a cariogenic microflora and demineralization of the tooth.
75 ice showed no significant decline in the gut microflora and developed EAE similar to untreated mice,
76 stinal mucosa is exposed to a diverse normal microflora and dietary Ags and is a common site of entry
77                                Commensal gut microflora and dietary fiber protect against colonic inf
78 r of transgenes from GM plants to intestinal microflora and enterocytes.
79 we characterized the Drosophila melanogaster microflora and examined the occurrence of enterococci in
80                                     Mammals, microflora and gut-dwelling macrofauna have co-evolved o
81 o protect themselves from the nasopharyngeal microflora and host immune response.
82  for eukaryotes in the maintenance of normal microflora and in protection from pathogenic bacteria.
83  contributed colitis-predisposing intestinal microflora and increased intestinal ATP, whereas Nod2 de
84  the upper respiratory tract, where resident microflora and inhaled environmental microbes may contin
85 ave profound effects on the intestinal tract microflora and intestinal function.
86 lex and dynamic interaction between the oral microflora and its host, which may lead, ultimately, to
87                                    The total microflora and lactic acid bacteria counts increased rap
88  species are normal members of the human gut microflora and most are regarded as safe when administer
89  the host genetic susceptibility, intestinal microflora and mucosal immune responses through the patt
90  alter the interaction of the host with both microflora and pathogens, promoting prolonged production
91 llin-associated alteration of the indigenous microflora and prevented overgrowth of pathogens.
92 so harboring the highest burden of commensal microflora and representing a major portal of pathogen e
93 ates the composition of intestinal commensal microflora and that it suppresses bacterial infection an
94 the early onset of CD by altering intestinal microflora and the gut mucosal barrier.
95 ve dialogue between members of the commensal microflora and the host mucosal immune system is rapidly
96 ss of the symbiotic relationship between the microflora and the host.
97  inter-kingdom signaling between the enteric microflora and the immune system to promote commensalism
98                    Thus nutrition, commensal microflora and the mucosal immune system are all intimat
99 teractions between components of the mucosal microflora and the mucosal immune system can involve eit
100 distinctive in their selective effect on the microflora and their propensity to produce flatulence.
101 to normally harmless antigens in the mucosal microflora and therefore responses to antigens that by t
102 gests that changes in the composition of gut microflora and/or deranged epithelial barrier function e
103      Critical interactions between bacteria (microflora) and parasites (macrofauna) introduced a new
104 elates with alterations in fecal microbiota (microflora) and widespread use of antibiotics (the "hygi
105 ptosis of intestinal epithelium, changed gut microflora, and elevated ATP.
106 s form associations with neighboring plants, microflora, and microfauna, while humans maintain symbio
107 te the relationship between GALT, intestinal microflora, and modulation of the antibody repertoire.
108  of mucosal immune dysregulation, colonizing microflora, and wound-healing mechanisms.
109 y considered a benign inhabitant of the oral microflora, and yet it is a primary etiological agent in
110 wn in adults, the composition of infant skin microflora appears to be site specific.
111  bacteria normally present in the intestinal microflora are able to trigger redistribution of the cys
112                   In rabbits, the intestinal microflora are also required for developing the preimmun
113                  Genetic factors and enteric microflora are driving forces regulating mucosal immune
114                        Indigenous intestinal microflora are known to afford protection against coloni
115  specific, currently unidentified intestinal microflora are required for Ab repertoire diversificatio
116 re undiversified, indicating that intestinal microflora are required for somatically diversifying the
117 rrant immune response and changes in the gut microflora are the main causes of inflammatory bowel dis
118 a daidzein metabolite produced by intestinal microflora) are antioxidants in vitro; equol is a partic
119 soy protein are biotransformed by intestinal microflora, are absorbed, undergo enterohepatic recyclin
120                These data implicate the oral microflora as a source of antigen-stimulating anti-PC re
121 ent on both IFN-gamma and a normal commensal microflora, as indicated by experiments in IFN-gamma-kno
122  bid, exerted an antimicrobial effect on the microflora associated with adult periodontitis.
123 lecular analysis has revealed a more diverse microflora associated with endodontic infections than th
124 we have characterized GPX-DKO mice that have microflora-associated intestinal cancers, which are corr
125 aluate the variation in the small intestinal microflora at repeated sampling.
126 d components and drugs, binding of commensal microflora, attachment and initiation of defense mechani
127 or was strongly limited by the environmental microflora because of the lack of oxygen, limiting the u
128 e relationship between humans and their oral microflora begins shortly after birth and lasts a lifeti
129 dy is to compare the submucosal peri-implant microflora between FES and PES.
130 e performed comparing differences in vaginal microflora between the two treatment arms and between vi
131    Because of the complexity of the host and microflora biology and the associated chemistry, it is d
132 kely due to effects mediated through the gut microflora, bowel transit, and enhancement of gastrointe
133  disease, we will need to study not only the microflora but also the host-immune response.
134 ndida albicans is a normal part of the human microflora, but it is also an opportunistic fungal patho
135 rt, on associations with disease-suppressive microflora, but little is known about the role of plant
136 OD2 regulates innate responses to intestinal microflora by downregulating multiple TLR responses and
137  resolving CDI symptoms, preservation of the microflora by fidaxomicin is associated with a lower lik
138  bacteria is avoided due to sequestration of microflora by surface epithelia.
139 ecently described that alteration of the gut microflora can affect a population of Foxp3(+)T(reg) cel
140 ty of at least one species of the indigenous microflora can induce expression of surface glycoconjuga
141 mal inflammation after wounding and that the microflora can modulate specific cutaneous inflammatory
142     Alterations in the autochthonous vaginal microflora can predispose women to recurring attacks of
143 ogens, genetic susceptibility, and commensal microflora, can lead to intestinal pathology.
144 d by an inner mucus layer that the commensal microflora cannot penetrate.
145 ded to determine if acid suppression-related microflora changes predict clinical infection risk; thes
146  environmental samples such as the human gut microflora, combined with the sustained exponential grow
147 ent and with the presence of a different gut microflora compared to mice maintained in SPF facilities
148 ons resulted in positive effects on both the microflora composition and the immune response.
149 mixture (B-GOS) on immune function and fecal microflora composition in healthy elderly subjects.
150                                          The microflora comprised staphylococci, enterococci (2.2 log
151                               The intestinal microflora consists of a heterogeneous population of mic
152 ion feedbacks involving landscapes and their microflora could contribute to appraising the impact tha
153  F11r(-/-)Rag1(-/-) mice exhibited increased microflora-dependent colitis.
154 fers resistance to colonocyte apoptosis in a microflora-dependent manner.
155 As this low level of epsps in the intestinal microflora did not increase after consumption of the mea
156  The current evidence suggests that the oral microflora differs between individuals who are fully ede
157                  The intestinal but not oral microflora elicited age- and cell type-specific immunost
158 riasis, and rosacea with an imbalance of the microflora even in the absence of classical infection.
159 onauts, increase microbial proliferation and microflora exchange, alter virulence and decrease antibi
160 nd participate in the development of the gut microflora for infants.
161 s and must compete with other members of the microflora for the same niche.
162 eriodontal disease and more specifically the microflora found around the gingival margins.
163         We isolated the predominant aciduric microflora from root-caries lesions (n = 14) and sound r
164 obiome was observed, suggesting that the gut microflora has a direct impact on the drug metabolism ca
165 arious bacteria that are part of the vaginal microflora has been reported.
166   The indigenous gastrointestinal (GI) tract microflora has profound effects on the anatomical, physi
167 various anti-gingivitis treatments on plaque microflora, here a double blinded, randomized controlled
168 segments (as a source of colonic enzymes and microflora) hydrolyzed Cer-beta-glucuronide to release C
169 FMT is able to restore the wide diversity of microflora, improve C. difficile-related symptoms and pr
170 een used previously to define normal vaginal microflora in a predictive model.
171 hanges antibiotic susceptibility of the oral microflora in adults with periodontitis.
172 and molecular analyses were performed on the microflora in aspirate samples collected from 5 infected
173                    Suppression of intestinal microflora in atherosclerosis-prone mice inhibited dieta
174 d an unbiased metagenomic approach to survey microflora in CCD hives, normal hives, and imported roya
175 sue, as well as bacteria growing among mixed microflora in enrichment cultures and in pure culture on
176 ed mucosal immune response to the intestinal microflora in genetically predisposed hosts.
177 onses driven by apparently normal intestinal microflora in genetically susceptible hosts.
178                          Introduction of gut microflora in GF mice rapidly initiated maturation of gl
179 e composition of the submucosal peri-implant microflora in healthy and peri-implant mucositis conditi
180 10s have explored the role of the intestinal microflora in human health.
181 ignificantly affect the growth of select gut microflora in humans, which suggests the potential prebi
182 ppreciation of the importance of the enteric microflora in IBD has led to a considerable interest in
183                                 It is normal microflora in marine environments, where seawater and mo
184 s currently available on the role of the gut microflora in modulating isoflavone bioavailability or o
185                              The subgingival microflora in patients presenting concurrently with peri
186 ctive in reducing the subgingival cultivable microflora in shallow periodontal pockets compared to cu
187 to be generated from flavonoid glycosides by microflora in the lower gastrointestinal tract.
188 itional investigation revealed commensal gut microflora in the mesenteric lymph nodes and elevated LP
189  suggesting that AI-2 produced by indigenous microflora in the murine oral cavity may complement the
190 tem and its interactions with the intestinal microflora in the pathogenesis of inflammatory bowel dis
191 o the role of antibiotics and the intestinal microflora in the rising obesity epidemic.
192 ecies and the nature of predominant aciduric microflora in the root caries process.
193 for interactions between GALT and intestinal microflora in the selective expansion of V(H)a B cells.
194 valuating critical components of the vaginal microflora in women with signs and symptoms of vaginitis
195 trobenzene sulfonic acid, colitis induced by microflora (in gnotobiotic interleukin-10(-/-)), and col
196 strated in intestinal epithelia, the colonic microflora includes strains of uropathogenic E. coli.
197 be modulated according to composition of gut microflora, including ingested probiotics.
198 ses to distinct components of the intestinal microflora induce intestinal inflammation, we characteri
199 f oncogene-environment interaction, in which microflora-induced TLR activation regulates oncogene exp
200 the intention of identifying how the mucosal microflora influences specific functions of the mucosal
201 is known about how members of the indigenous microflora interact with their mammalian hosts to establ
202 nfluence of the rich source of nutrients and microflora introduced with organic compost amendments.
203                                     The oral microflora is a likely source of antigen inducing antiCl
204              The human endogenous intestinal microflora is an essential "organ" in providing nourishm
205  However, the contribution of the intestinal microflora is beyond simple microbial translocation as a
206                                     The oral microflora is composed of both health-promoting as well
207 we find that activation of TLRs by commensal microflora is critical for the protection against gut in
208       Following antimicrobial treatment, the microflora is disrupted, and C. difficile spores germina
209                           A rich residential microflora is harboured by the distal outer root sheath
210                                The human gut microflora is important in regulating host inflammatory
211                        The mucosa-associated microflora is increasingly considered to play a pivotal
212 negative staphylococci (CoNS) and other skin microflora is reported.
213 ies have indicated that a subset of the oral microflora is responsible for endodontic infections.
214 bolic cluster present in the normal, colonic microflora is responsible for preventing C. difficile in
215 nctive taxa-a century later, the Torridonian microflora is still being characterized as primarily non
216 sponsiveness' towards the resident commensal microflora is thought to permit their successful colonis
217 I) was 0.83 (CI, 0.42-0.99) for conjunctival microflora isolates, 0.80 (CI, 0.54-0.94) for ocular inf
218                                   Intestinal microflora keeps C. difficile in the spore state and pre
219 rinking water affects the composition of gut microflora, leading to an altered autoimmune response an
220 l generation in conjunction with the colonic microflora, leading to the production of carcinogens or
221 elative to naive GC-C+/+ mice, the commensal microflora load in uninfected GC-C-/- mice was decreased
222 n a balance in the composition of intestinal microflora; long-lived macrofauna have also been shown t
223  flow and the composition of the subgingival microflora may serve as predictors for reinfection in th
224 th amino acid, energy, purine, lipid and gut microflora metabolisms.
225  Concentrations of isoflavones and their gut microflora metabolites in the plasma, urine, and feces w
226 nto the bioactive natural products and human microflora metabolites of dietary ellagic acid derivativ
227                    Relationships between gut microflora, morphological and metabolic traits were unco
228                            Here we show that microflora-MyD88-ERK signaling in intestinal epithelial
229 molecular techniques for analysis of the gut microflora, new manufacturing biotechnologies, and incre
230                                    The yeast microflora of all three host groups had similar species
231 llus spp./ BV-related species in the vaginal microflora of baboons (Papio spp.).
232 me article about the submucosal peri-implant microflora of FES and PES were selected.
233 gher abundance of dominant genera in the gut microflora of group JD.
234 investigate peri-implant and intraconnection microflora of healthy implants restored with cemented an
235 taiotaomicron, a component of the intestinal microflora of mice and humans, uses a repressor, FucR, a
236 s fragilis is a member of the normal colonic microflora of most mammals and is the most commonly isol
237 ctively, these findings suggest that the gut microflora of the honey bee harbours bacterial members w
238  the vitamins that are synthesized by normal microflora of the large intestine.
239                     The resident prokaryotic microflora of the mammalian intestine influences diverse
240 imately 4.0-6.5logcfu/g were recorded in the microflora of the salad, whereas the Pseudomonas spp. po
241                                   The normal microflora of the skin includes staphylococcal species t
242 -frequency gene transfer from GM soya to the microflora of the small bowel before their involvement i
243 les indicate that dogs have a highly diverse microflora of the small intestine, with marked differenc
244       Quantitative changes in the intestinal microflora of these animals were assessed first using co
245 idermidis, a major constituent of the normal microflora on healthy human skin, acts as a barrier agai
246 anded our ability to study the impact of the microflora on human health and disease.
247 f the resident non-pathogenic or 'commensal' microflora on mucosal immune function and gut health has
248 ance role in maintaining the steady state of microflora on mucosal surfaces.
249 have a profound effect on the impact of this microflora on the regulation of nematode populations by
250  by CD4 T cells is induced by the intestinal microflora, oral delivery of specific Ag, and type I IFN
251 e under tissue and developmental control and microflora play a major role in their specific ontogeny
252 ecognized that innate immunity to intestinal microflora plays a significant role in mediating immune
253                             The microbiota ("microflora") plays a crucial role in the development of
254 unity-based mechanism for protecting the gut microflora, preserving its functional robustness during
255 rvations indicate that the normal human skin microflora protects skin by various modes of action, a c
256 electively fermented by the gastrointestinal microflora, resulting in benefits to human health.
257 iotaomicron, a predominant member of the gut microflora, revealing a mechanism whereby intestinal com
258 ansition was associated with a change from a microflora rich in TLR4-stimulatory proteobacteria to on
259                      Assessment of the cecal microflora showed a rise in numbers of Escherichia coli
260                                          The microflora-sparing properties of fidaxomicin were examin
261                               The indigenous microflora stimulates the host immune system to respond
262    However, it is unknown how the indigenous microflora stimulates the immune system and how this res
263 ns in shaping the composition of the enteric microflora, such polymorphisms may influence outcomes in
264 r a potentially more pathogenic peri-implant microflora than FES.
265 s a member of the mammalian gastrointestinal microflora that has become a leading cause of nosocomial
266 an body is colonized with a diverse resident microflora that includes viruses.
267 bound on the host response to the indigenous microflora that is consistent with current biological vi
268 teractions between a host and its intestinal microflora that lead to commensalism are unclear.
269  In particular, the capacity of autochthonic microflora that live on natural organic matter as the so
270            The alterations in the intestinal microflora that occur after B16 infection remain unknown
271 late the composition of the small intestinal microflora, that development of crypt organoid culture s
272 ons, such as circulating glucose levels, gut microflora, time of year, and even diurnal rhythm, which
273 c explanation for the capacity of intestinal microflora to control liver inflammation.
274 oid tissues (GALTs) interact with intestinal microflora to drive GALT development and diversify the p
275 neered commensal bacteria within the vaginal microflora to inhibit heterosexual transmission of HIV.
276 s muris was recently demonstrated to utilise microflora to initiate its life cycle.
277 ms and the relationship with the normal skin microflora to maintain healthy skin.
278 ized here the ability of intestinal and oral microflora to stimulate individual pattern recognition r
279 s and may also relate age-related changes in microflora to susceptibility to enteropathogens.
280                               The intestinal microflora, typically equated with bacteria, influences
281 nerally exists in harmony with the commensal microflora, under certain conditions, these organisms ma
282                            Reduction of host microflora using antibiotics, neutralization of serum LP
283 immunostimulatory activity of the intestinal microflora varied among individual mice but was largely
284 major stimulatory activity of the intestinal microflora was still intact in NOD1-, NOD2-, TLR2-, TLR4
285 rstand how selenium regulates the intestinal microflora, we used high-throughput sequencing to examin
286        None of the changes or differences in microflora were considered to be clinically significant.
287 samples (sourdough breads prepared with wild microflora) were spoiled approximately at the 7th day.
288 er this leads to differences in peri-implant microflora when implants are installed.
289 errestrial organisms, as well as belowground microflora, whether and how soil symbionts regulate phot
290 HIV infection leads to changes in basal lung microflora, which may contribute to chronic pulmonary co
291 cosal surface is colonized by the indigenous microflora, which normally maintains an ecological balan
292 ease, resulting in changes in the colonizing microflora with unknown future consequences.
293 gulating the innate inflammatory response to microflora within the lower bowel, likely through its ab
294 ter colonizing germ-free mice with commensal microflora without any known pathogens (SPF), <9% of GPX
295 s them to interact with and contain the oral microflora without eliciting a marked inflammatory respo
296 myces lactis occur as part of Stilton cheese microflora yet are not controlled during production.

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