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1 chromosomes in abnormal nuclear structures (micronuclei).
2 es damaged DNA out of the nucleus by forming micronuclei.
3 er chromatid exchanges (SCEs) and numbers of micronuclei.
4 olate status was an independent predictor of micronuclei.
5 H1 by shRNA suppressed induction of H2AX and micronuclei.
6 ster chromatid exchanges, and show increased micronuclei.
7 aneous and radiation- or mitomycin C-induced micronuclei.
8 lymphoproliferative activity, and lymphocyte micronuclei.
9 re homozygous, rad51 nulls in their germline micronuclei.
10 re not associated with a higher frequency of micronuclei.
11 tions and are extruded into nuclear blebs or micronuclei.
12 ed the frequency of prevalence of cells with micronuclei.
13 res become elongated in both macronuclei and micronuclei.
14 ar convolution and the formation of multiple micronuclei.
15 inesis resulting in daughter cells with many micronuclei.
16 Brca2(Tr2014/Tr2014) MEFs frequently develop micronuclei.
17 ch we found associated with the formation of micronuclei.
18 and have increased numbers of macrocytes and micronuclei.
19 ells containing mitochondria surrounding the micronuclei.
20 of these DNA molecules in radiation-induced micronuclei.
21 sing altogether from transcriptionally inert micronuclei.
22 on of linker histone in mitotically dividing micronuclei.
23 n establishing transcriptional competence in micronuclei.
24 formation of ultra-fine anaphase bridges and micronuclei.
25 DNA from aberrant nuclear structures called micronuclei.
26 oss chromosomal aberrations, 53BP1 foci, and micronuclei.
27 neven chromosome segregations that resembles micronuclei.
28 frequency of acentric-bearing, lamin-coated micronuclei.
29 osomes in aberrant nuclear structures called micronuclei.
30 efore the appearance of anaphase bridges and micronuclei.
31 rophe, irregularly shaped nuclei or multiple micronuclei.
32 reaks via the formation of structures called micronuclei.
33 e fusion, resulting in formation of multiple micronuclei.
34 uent lagging of chromosomes and formation of micronuclei.
35 om enwrapping individual chromosomes to form micronuclei.
36 and accumulate cells with higher ploidy and micronuclei.
38 ersus 12% of controls) had >or=4 spontaneous micronuclei, 66% of cases (and no controls) had >or=4 sp
39 us 6% of the controls) had >or=5 NNK-induced micronuclei, 89% of cases (and no controls) had >or=6 in
42 o a striking increase in hepatocytes bearing micronuclei, a marker of genomic instability, which is s
43 with p21Waf1 expression, gamma-H2AX foci and micronuclei, adding SOD and catalase has no effect on th
44 tial inhibition of Plk1 was the formation of micronuclei, an increase in tetraploid progeny, and sene
45 resulting in an increase in cells harboring micronuclei, an indication of defects in chromosome segr
47 f spontaneous chromosome instability such as micronuclei and 53BP1 nuclear bodies, known consequences
48 iciency is associated with the appearance of micronuclei and aberrant mitoses that are a by-product o
50 was sufficient to increase the prevalence of micronuclei and binucleated cells in parallel with aberr
51 ss, incomplete DNA replication, formation of micronuclei and chromatin bridges and eventually cell de
54 ase to the medium decreases the formation of micronuclei and induction of p21Waf1 and gamma-H2AX foci
56 h a high spontaneous frequency of cells with micronuclei and LST and specific alterations in DNA repa
58 lls exposed to eleutherobin contain multiple micronuclei and microtubule bundles, and they arrest in
59 chromosomal instability and the formation of micronuclei and multinucleation through its interaction
65 s normally restricted to unrearranged DNA of micronuclei and/or developing nuclei, but germline-limit
67 plex was essential for the induction of DSB, micronuclei, and apoptosis in human cells by chromate.
68 tion-dependent formation of gamma-H2AX foci, micronuclei, and chromosomal aberrations (chromatid brea
71 ases (P < .05) in centrosomal amplification, micronuclei, and micronuclei containing whole chromosome
72 xicity, including centrosomal amplification, micronuclei, and micronuclei containing whole chromosome
76 unequal chromosome segregation, formation of micronuclei, and/or failure to complete cell division ar
78 ronucleus, and elimination of one of the new micronuclei are blocked if actinomycin D is added at lea
84 ent of DNA damage using the comet assay, the micronuclei assay, and the gamma-H2AX immunostaining ass
87 nexin-V/propidium iodide binding, comet, and micronuclei assays, showed that the reduction in viabili
91 in the peripheral blood for the presence of micronuclei before these cells are removed by the spleen
92 p1p is missing from transcriptionally silent micronuclei but is enriched in heterochromatin-like chro
94 V600E) expression increased the frequency of micronuclei by both clastogenic and aneugenic events.
95 s also activate this pathway, recognition of micronuclei by cGAS may act as a cell-intrinsic immune s
96 dy, chromosome breakage and the formation of micronuclei by targeting cellular ligases through a sT d
98 PCS), lagging chromosomes, anaphase bridges, micronuclei, centrosome amplification, multinucleated ce
100 macronuclei, but not transcriptionally inert micronuclei, contain a robust histone methyltransferase
102 centrosomal amplification, micronuclei, and micronuclei containing whole chromosomes were found in m
104 nes from tumor cells via their entrapment in micronuclei contributes to the improved therapeutic resp
105 creased the formation of centromere-negative micronuclei, demonstrating that induced DSB were ineffic
106 DNA damage) and normochromatic erythrocytes (micronuclei) during chemical or immune-mediated colitis.
108 Both activities were lost from germ line micronuclei following the programmed arrest of micronucl
110 ome instabilities including chromosome loss, micronuclei formation and chromosome breakage that are f
111 ed frequencies of chromosomal aberration and micronuclei formation and exhibit an enhanced proliferat
112 omosomal instability manifested by increased micronuclei formation and numerical chromosomal aberrati
113 M, or of BRCA1 and FANCM, leads to increased micronuclei formation and synthetic lethality in ALT cel
114 matin, and consistent with a mitotic origin, micronuclei formation and the proinflammatory response f
116 plification, sister chromatid exchanges, and micronuclei formation in cells exposed to genotoxic agen
117 ies by Harding et al. demonstrate a role for micronuclei formation in DNA damage-induced immune activ
118 derate degree of chromosomal instability and micronuclei formation in short-term cultures established
122 cantly (P < .05 to .001) higher frequency of micronuclei formation was observed in mice that received
123 ncreased gene mutations, gene amplification, micronuclei formation, and chromosomal instability.
124 mbers, formed multipolar spindles, displayed micronuclei formation, and elevated nuclear DNA content.
125 and RNA-Seq, GOF p53-induced origin firing, micronuclei formation, and fork protection were traced t
127 e Gravin rescued chromosome misalignment and micronuclei formation, but a mutant Gravin that cannot b
128 , stabilizes replication forks, and promotes micronuclei formation, thus facilitating the proliferati
133 monstrate that chromosomes mis-segregated to micronuclei frequently undergo chromothripsis-like rearr
135 eased occurrence (more than 2-fold) of extra micronuclei, implying faulty control of micronuclear div
136 elated with mitotic and meiotic divisions of micronuclei in a fashion that closely coincides with chr
137 rg et al. have described a method to measure micronuclei in an enriched reticulocyte population using
138 plies single-laser flow-cytometry to measure micronuclei in an enriched transferrin-positive reticulo
140 cent protein (GFP)-tagged Cna1p localizes in micronuclei in dots whose number and behavior during mit
144 the MAPK pathway in mediating v-ras-induced micronuclei in NIH 3T3 cells was examined by inhibiting
146 tic nuclei, anaphase bridges during mitosis, micronuclei in pollen tetrads, and 30% seed abortion.
147 oth the apoptotic wave and the appearance of micronuclei in red blood cells are likely cellular conse
148 se in both CREST negative and CREST positive micronuclei, indicating that both clastogenic and aneuge
149 addition, Tax-1 PBM was responsible for the micronuclei induction activity of Tax-1 relative to that
152 t that LT, dependent on Bub1 binding, causes micronuclei, lagging chromatin, and anaphase bridges, wh
153 and play an unexpected role in the germ line micronuclei late in conjugation that is unrelated to tra
154 us DNA damage by alkaline comet assay, basal micronuclei levels, the number of large-scale chromosoma
155 ions were restricted to mitotically dividing micronuclei; macronuclei, which are amitotic, showed no
156 hput method for quantifying the formation of micronuclei, markers of genome instability, in mouse ery
160 rosis) and occurrence of chromosomal damage (micronuclei MN, nculeoplasmic bridge NPBs and nuclear bu
163 ions from a particle microbeam, the yield of micronuclei (MN) in the nontargeted cells was increased,
164 tely 380 keV/microm), a 1.4-fold increase of micronuclei (MN) was detected demonstrating a bystander
167 in high genomic instability characterized by micronuclei, multiple nuclei, and a wide distribution of
168 predict cancer risk based on the numbers of micronuclei, nucleoplasmic bridges, and nuclear buds def
169 analyzed on a continuous scale, spontaneous micronuclei, nucleoplasmic bridges, and nuclear buds wer
170 of NNK-induced chromosomal damage endpoints (micronuclei, nucleoplasmic bridges, and nuclear buds) pe
171 h percentiles of spontaneous and NNK-induced micronuclei, nucleoplasmic bridges, and nuclear buds, re
176 In growing cells, H2A.Y is incorporated into micronuclei only during S phase, which occurs immediatel
177 d, along with interphase cells that harbored micronuclei or multiple nuclei, consistent with unbalanc
181 ient cells showed chromosome rearrangements, micronuclei, sensitivity to replication stress and DNA d
182 ased mitotic defects, chromatin bridges, and micronuclei, suggesting HDAC1/2 are necessary for accura
184 veloped a general and rapid method to purify micronuclei that are known to entrap extrachromosomal el
185 phosphorylation can be detected in germline micronuclei that divide mitotically but not in somatic m
186 ibution from chromosome-containing fragments/micronuclei that frequently emerge and may persist or be
187 hese mitotic errors generate an abundance of micronuclei that predispose chromosomes to subsequent ca
190 inger et al. further improved the scoring of micronuclei to enable the use of bench-top instruments i
193 cluding apoptotic cells and cells expressing micronuclei, was determined as a function of time in cul
195 rrant mitotic segregation, since most of the micronuclei were CREST-positive, reflective of lost chro
196 ed, suggesting a failure of cytokinesis, and micronuclei were formed, indicative of genomic instabili
197 gation, and the protein localizes to meiotic micronuclei when bidirectional germ line transcription o
198 rete dots in those nuclei destined to become micronuclei, whereas it remains diffuse and is gradually
200 tion is also associated with the presence of micronuclei, which are shown to contain unstable chromos
201 tranded DNA breaks leads to the formation of micronuclei, which precede activation of inflammatory si
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