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1 nd is not dependent on the central regulator microphthalmia-associated transcription factor.
2 AMP response element-binding protein and the microphthalmia-associated transcription factor.
3 in copper status affected the expression of microphthalmia-associated transcription factor, a transc
4 through beta-catenin-mediated regulation of microphthalmia-associated transcription factor activity,
7 ss the protein and mRNA expression levels of microphthalmia-associated transcription factor and tyros
8 c acid, lipoic acid, and resveratrol reduced microphthalmia-associated transcription factor and tyros
10 ompanied by increased transcription of MITF (microphthalmia-associated transcription factor) and tyro
11 in and immunohistochemical markers (melan-A, microphthalmia-associated transcription factor, and SRY-
12 e supporting the concept that the effects on microphthalmia-associated transcription factor are depen
13 tified the melanocyte master regulator MITF (microphthalmia-associated transcription factor) as the t
14 Human MITF is, by convention, called the "microphthalmia-associated transcription factor" because
15 ult and neonatal melanocytes, SOX9 regulates microphthalmia-associated transcription factor, dopachro
16 ression of the melanocyte determining factor microphthalmia-associated transcription factor, elevated
18 Our findings suggest that modulation of microphthalmia-associated transcription factor expressio
19 ETV1 overexpression elevated microphthalmia-associated transcription factor expressio
20 functions by initially stimulating levels of microphthalmia-associated transcription factor expressio
21 Changes in endogenous microphthalmia-associated transcription factor expressio
23 cultures, cAMP-induced transcription of the microphthalmia-associated transcription factor gene (Mit
24 in traditional Chinese medicine, upregulated microphthalmia-associated transcription factor gene expr
25 tion and further confirm the central role of microphthalmia-associated transcription factor in melano
28 noma and inversely correlates with FOXO3 and microphthalmia-associated transcription factor levels.
29 ytes via activation of melanocyte-restricted microphthalmia-associated transcription factor (M-MITF)
30 igration and survival by directly repressing microphthalmia-associated transcription factor-M and FOX
31 FOXO3, whereas enhanced expression of either microphthalmia-associated transcription factor-M or FOXO
32 duced by a DGK inhibitor, but tyrosinase and microphthalmia-associated transcription factor messenger
34 ced tanning response, we show that while the microphthalmia-associated transcription factor Mitf regu
35 h tooth shape; one region contained the gene microphthalmia-associated transcription factor Mitf that
36 irection to the nucleus to interact with the microphthalmia associated transcription factor (MITF).
39 ility to potently downregulate expression of microphthalmia-associated transcription factor (MITF) an
40 xpression in the RPE of transgenic mice, and microphthalmia-associated transcription factor (MITF) an
42 gulation of two important signaling factors, microphthalmia-associated transcription factor (MITF) an
43 ific expression in the eye, and we suggested microphthalmia-associated transcription factor (MITF) as
45 expression of the lineage survival oncogene microphthalmia-associated transcription factor (MITF) co
46 g mediator of cell death (BIM) induction and microphthalmia-associated transcription factor (MITF) do
48 ntly a candidate approach was used to select microphthalmia-associated transcription factor (MITF) fo
49 e Type 2 is caused by mutations in the human Microphthalmia-associated transcription factor (MITF) ge
50 otic gene 3 (PAX3) is a key regulator of the microphthalmia-associated transcription factor (Mitf) in
65 , we show that the lineage survival oncogene microphthalmia-associated transcription factor (MITF) is
70 ), developmental and oncogenic roles for the microphthalmia-associated transcription factor (MITF) pa
73 reveals that the melanocyte master regulator microphthalmia-associated transcription factor (MITF) pr
76 s overexpressing the teneurin-1 ICD, several microphthalmia-associated transcription factor (MITF) ta
77 PROM1); ribosomal protein L13A (RPL13A); and microphthalmia-associated transcription factor (MITF) we
78 tfa gene encodes a zebrafish ortholog of the microphthalmia-associated transcription factor (Mitf) wh
79 the nucleus, thereby reducing expression of microphthalmia-associated transcription factor (MITF), a
80 at results in a decrease in beta-catenin and microphthalmia-associated transcription factor (MITF), a
81 fenib resistance correlated with the loss of microphthalmia-associated transcription factor (MITF), a
82 munohistochemical stains for MART-1, HMB-45, microphthalmia-associated transcription factor (MiTF), a
83 ding a key regulator of RPE gene expression, microphthalmia-associated transcription factor (MITF), c
86 phosphorylation of p38 MAPK, which activates microphthalmia-associated transcription factor (MITF), k
87 n is associated with increased expression of microphthalmia-associated transcription factor (Mitf), w
88 -211, a known target of the master regulator microphthalmia-associated transcription factor (MITF).
89 nin formation in melanocytes by inducing the microphthalmia-associated transcription factor (MITF).
90 ssion of mast cell-specifying genes Hes1 and microphthalmia-associated transcription factor (Mitf).
91 6) in the melanoma-lineage-specific oncogene microphthalmia-associated transcription factor (MITF).
92 sed invasion and, often, decreased levels of microphthalmia-associated transcription factor (MITF).
93 tive stress results in reduced expression of microphthalmia-associated transcription factor (MITF).
94 reas via the suppression of beta-catenin and microphthalmia-associated transcription factor (MITF).
95 oximately 6 microm) with their substrate the microphthalmia-associated transcription factor (MITF).
96 for the expression of the RPE key regulator microphthalmia-associated transcription factor (Mitf); h
97 blue) and immunohistochemical probes (S-100, microphthalmia-associated transcription factor [MiTF], H
98 In vivo Brn-2 represses expression of the microphthalmia-associated transcription factor, MITF, to
99 virus (SFFV) proviral integration 1 (PU.1), microphthalmia-associated transcription factor, NF-kappa
100 promoter, we identified agents that modulate microphthalmia-associated transcription factor promoter
101 luciferase reporter construct driven by the microphthalmia-associated transcription factor promoter,
103 sphodiesterase 4D3 inhibitors, T-oligos, and microphthalmia-associated transcription factor regulator
104 ag GTPases bound and regulated activation of microphthalmia-associated transcription factor, suggesti
105 on of the master regulator of melanogenesis, microphthalmia-associated transcription factor, thus sti
107 he essential osteoclast transcription factor microphthalmia-associated transcription factor were incr
108 0, controls the expression of another, MITF (microphthalmia-associated transcription factor), which i
109 se that co-expressed SOX2 and either CK20 or microphthalmia-associated transcription factor, which ar
110 pment from the neural crest, SOX10 regulates microphthalmia-associated transcription factor, which co
111 se element-binding protein and expression of microphthalmia-associated transcription factor, which en
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