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1 influenced by an interaction with the TNFa6 microsatellite polymorphism.
2 C polymorphism, and 13% had the short-allele microsatellite polymorphism.
3 of these linkage groups contain at least one microsatellite polymorphism.
4 carcinoma in each patient was compared using microsatellite polymorphisms.
5 tern Spain are associated with different TNF microsatellite polymorphisms.
6 iomyomas by polymerase chain reaction for 26 microsatellite polymorphisms.
7 ge groups (2n = 80) from the analysis of 288 microsatellite polymorphisms, 13 allozyme markers, and p
11 losses affecting the gene were evaluated by microsatellite polymorphism analysis and genomic alterat
12 ations were observed between any single TNFa microsatellite polymorphism and disease severity, althou
13 /2J, C57BL/6J, and BALB/cJ, the frequency of microsatellite polymorphisms appears to be strain specif
14 e to tobacco and that gene amplification and microsatellite polymorphism are evolutionary drivers in
15 siblings using both the ACE D/I marker and a microsatellite polymorphism at the neighboring locus for
16 t allele (designated allele 86) of the Ctla4 microsatellite polymorphism (AT)n located in the 3'-untr
17 Polymerase chain reaction (PCR) analysis of microsatellite polymorphisms corresponding to four loci
19 ; GenBank no. L34155), and on three flanking microsatellite polymorphism (D18S45, D18S478, and D18S48
22 llelic loss of chromosome 9p, as assessed by microsatellite polymorphisms flanking the CDKN2/p16 regi
25 ventories from 2,082 worldwide languages and microsatellite polymorphisms from 246 worldwide populati
26 on studies, and the level of simple-sequence microsatellite polymorphisms has important implications
30 ngle nucleotide polymorphisms (SNPs) and one microsatellite polymorphism in MMP genes MMP-1, MMP-2, M
32 BD by linkage analysis of highly informative microsatellite polymorphisms in 43 families with multipl
33 hat are distinguished by point mutations and microsatellite polymorphisms in and near the CQR transpo
34 y to the p-1562 and (CA) dinucleotide repeat microsatellite polymorphisms in the promoter region of t
35 n tested shared a variant allele at 2 nearby microsatellite polymorphisms, indicating a common ancest
36 28 unrelated Europeans were used to estimate microsatellite polymorphism (number of alleles, heterozy
37 siblings, by using both ACE D/I and a nearby microsatellite polymorphism of the human growth hormone
38 Therefore, we evaluated LD between 21 common microsatellite polymorphisms on chromosome 18q21 in 2 ge
40 describe here the identification of 11 novel microsatellite polymorphisms on human chromosome 19.
42 e association of severity measures with TNFa microsatellite polymorphisms stratified by SE status, an
44 Alleles of the IL-10 (-)1064 promoter region microsatellite polymorphism that possess greater numbers
45 ariable number of tandem repeats (VNTRs), or microsatellite polymorphisms, this SNP-based method prov
46 kage analyses were performed with the use of microsatellite polymorphisms to determine a locus for th
47 h whole genome amplification and analysis of microsatellite polymorphisms to determine the frequency
54 s no association of either the TNFd or IL-10 microsatellite polymorphisms with mortality (P =.43 and.
55 enotyped for an interferon-gamma (IFN-gamma) microsatellite polymorphism, within intron 1, for which
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