ライフサイエンスコーパス: microspike

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します

1 g Rac activation and formation of filopodia (microspikes).

2 (F-actin), in this work called filopodia or microspikes.

3 sulting in the formation of peripheral actin microspikes.

4 ote the formation of Cdc42-dependent F-actin microspikes.

5 ormation but had a reduced ability to induce microspikes.

6 oskeleton and have numerous F-actin-enriched microspikes.

7 n the ability of cells to form stable fascin microspikes.

8 ttached to thrombospondin-1 but did not form microspikes.

9 in from stress fibers, membrane ruffles, and microspikes.

10 lamellopodium, with long parallel bundles at microspikes and filopodia.

11 orrelates with decreased formation of fascin microspikes and increased assembly of focal contacts by

12 n, an actin-bundling component of filopodia, microspikes and lamellipodial ribs, and protein kinase C

13 eptor Abl tyrosine kinase stimulates F-actin microspikes and membrane ruffles in response to adhesion

14 anced formation of dendritic-like processes, microspikes and other dynamic actin based structures.

15 in the formation of microfilament bundles of microspikes and stress fibers in cultured cells.

16 g protein that is found in membrane ruffles, microspikes, and stress fibers.

17 ssembly of actin filament bundles present in microspikes as well as in membrane ruffles and stress fi

18 of CLL cells by controlling the dynamics of microspike-containing protrusions and cell steering.

19 ephrin-B substratum induces the formation of microspikes filled with F-actin.

20 ization, including the formation of membrane microspikes, filopodia, and lamilliopodia.

21 lity of the activated Cdc42 mutant to induce microspikes/filopodia in NIH 3T3 cells, whereas they eli

22 WIPa-overexpressing cells exhibit multiple microspike formation and defects in chemotactic efficien

23 ss 3T3 fibroblast cultures, ZCL278 abolished microspike formation and disrupted GM130-docked Golgi st

24 which lacks Galphao binding region, promoted microspike formation in Swiss 3T3 cells or neurite exten

25 rane dynamics, including membrane ruffle and microspike formation, fusion or fission of intracellular

26 s cell growth, spreading, and filopodia-like microspike formation.

27 tivation specifically promoted Vav3-mediated microspike formation.

28 mulates fibroblasts to form filopodia, actin microspikes formed upon the stimulation of Cdc42.

29 tially extended lamellipodia, filopodia, and microspikes from their corners.

30 that c-Abl stimulated the formation of actin microspikes in fibroblasts spreading on fibronectin.

31 Vav3 induced marked membrane ruffles and microspikes in NIH 3T3 cells, while the N-terminal trunc

32 in detached fibroblasts, stimulated F-actin microspikes independent of cell attachment.

33 The Abl-dependent F-actin microspikes occurred under conditions where the Rho-fami

34 SP transfectants exhibit extensive MCSP-rich microspikes on adherent cells, where it also colocalizes

35 endogenous fascin remained competent to form microspikes on thrombospondin-1, and cells that expresse

36 g activity could retain the peripheral actin microspike (PAM)-inducing activity of Cdc42, interaction

37 d to the rear of the lamellipodium and along microspikes radiating through the lamellipodium.

38 ls induces the formation of peripheral actin microspikes, similar to that previously observed when ce

39 triggers the sustained formation of F-actin microspikes that contain the actin-bundling protein fasc

40 clic-AMP, they stereotypically produce first microspikes, then blebs and pseudopods only later.

41 l to regain their ability to ruffle and form microspikes, unlike cells rescued with wild-type WAVE2.

42 To further understand the function of fascin microspikes, we examined whether their assembly is regul

WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。