ライフサイエンスコーパス: microtubular

1 dium difficile toxin B significantly reduced microtubular acetylation and the delivery of viral DNA t

2 acellular transport in plant cells occurs on microtubular and actin arrays.

3 We hypothesized that the microtubular and actin cytoskeletons influence the expre

4 w insights into interaction between both the microtubular and microfilament cytoskeleton and cellular

5 cl-2 phosphorylation following disruption of microtubular architecture, serving a role similar to p53

6 relationship of motile cilia with the 9 + 2 microtubular arrangement have helped explain some of the

7 sts, mitochondria were observed close to the microtubular array and displayed both short- and long-ra

8 The mitotic spindle is a microtubular assembly required for chromosome segregatio

9 hic inversion on 17q21, sometimes called the microtubular associated protein tau (MAPT) inversion, is

10 ion of Beclin 1, and increased conversion of microtubular-associated protein 1 light chain 3-I to -II

11 y distributed throughout the cytosol without microtubular association, C19ORF5C specifically accumula

12 is an essential ciliary protein required for microtubular attachment of ODAs in the axoneme.

13 y cilia are sensory organelles composed of a microtubular axoneme and a surrounding membrane sheath t

14 lar (interchangeable terms) membrane and the microtubular axoneme of motile and sensory cilia.

15 In addition to a microtubular axoneme, the flagellum contains a crystalli

16 Tau proteins are microtubular binding proteins localized in the axonal co

17 ealization that targeting various aspects of microtubular biology with small molecules might offer ne

18 hieves its effects by acting on the neuronal microtubular content, which is involved with growth, sta

19 d to detect the presence of alpha-tubulin, a microtubular cytoskeletal component, in isolated nuclear

20 t C19ORF5 mediates communication between the microtubular cytoskeleton and mitochondria in control of

21 dles at the leading margin direct the distal microtubular cytoskeleton as growth cones turn to avoid

22 lexes and immunofluorescence analyses of the microtubular cytoskeleton of mitotic cells using wild-ty

23 demonstrated that iso-2 associates with the microtubular cytoskeleton that underlies the cell body m

24 Disruption of the microtubular cytoskeleton with colchicine did not affect

25 y, and an intact actin cytoskeleton, but not microtubular cytoskeleton, are required for disruption o

26 TubA1 resulted in an incorporation into the microtubular cytoskeleton, demonstrating the effectivene

27 bacterial motility structures and became the microtubular cytoskeleton, including the mitotic apparat

28 ogenesis, and components of the cortical and microtubular cytoskeleton.

29 (ts) alp4 and alp6 mutants show two types of microtubular defects.

30 ways are involved with cellular detection of microtubular disarray and subsequent activation of JNK/S

31 with IDA and central apparatus defects with microtubular disorganization (IDA/CA/MTD; n = 40).

32 Thus, microtubular disruption provides a noninvasive method fo

33 To test the hypothesis that microtubular disruption should promote transgene persist

34 ry stages of infection such as modulation of microtubular dynamics, movement of virus in the cytoplas

35 gnaling cascades in apoptosis resulting from microtubular dysfunction induced by paclitaxel, we have

36 unrelated phenotypes have now been linked to microtubular dysfunction, especially in systems dependen

37 We show that it involved an interaction with microtubular elements, required activation of the kinase

38 on characteristics and optimization of these microtubular engines are described, along with their eff

39 These mass-produced microtubular engines are only 8 mum long, are self-prope

40 MIP-based catalytic microtubular engines are prepared by electropolymerizati

41 Highly efficient catalytic microtubular engines are synthesized rapidly and inexpen

42 ential for efficient propulsion of catalytic microtubular engines.

43 ustion yields freestanding CNT or reduced GO microtubular fibers.

44 the production of carbon nanomaterial-based microtubular fibers.

45 ed perinuclear positioning of the convergent microtubular framework.

46 ing shared pathogenic mechanisms in terms of microtubular function and interaction with microtubule-a

47 eroallergen exposure, implicating epithelial microtubular functions in the pathogenesis of Th2-mediat

48 alization is an aggregation of extracellular microtubular-like structures found within the sclerad re

49 ific differences in the hypertrophy of these microtubular-like structures may be related to inherent

50 ith the motor protein is responsible for the microtubular localization of PP5 in vivo.

51 underlying axonal neurofilament lattice and microtubular loss.

52 cyte microparasol, composed of a perinuclear microtubular/melano-phagolysosomal complex, protects the

53 polarity proteins with microtubules and the microtubular motor KIF3/Kinesin-II.

54 ytoplasmic factor that does not seem to be a microtubular motor or a kinase/phosphatase.

55 nding sites for cellular proteins needed for microtubular movement and actin tail formation.

56 To study the possible involvement of the microtubular network in the alpha-synuclein-dependent tr

57 4 amino acids from the C-terminal, reveals a microtubular network localization by confocal microscopy

58 esulted in almost complete disruption of the microtubular network, abolished the adaptive increases i

59 ns help tether incoming viral capsids to the microtubular network, thus promoting cytoplasmic traffic

60 f a Stau-bcd mRNA complex through a nonpolar microtubular network, which confines the bcd mRNA to the

61 uorescent confocal microscopy to disrupt the microtubular network.

62 ed continued protein synthesis and an intact microtubular network.

63 ining endosomes to the autophagosome via the microtubular network.

64 es EspG and EspG2, known to disrupt the host microtubular network.

65 ctivity, by tethering the transporter to the microtubular network.

66 MV MP, which, instead, was redirected to the microtubular network.

67 t CD155-containing endocytic vesicles to the microtubular network.

68 with decreased free tubulin or a diminished microtubular network.

69 a-tubulin, and the intermediate filament and microtubular networks of the transfected cells appeared

70 on microscopy revealed that the manchette, a microtubular organelle essential for sperm head and flag

71 e primary cilium is a ubiquitous, non-motile microtubular organelle lacking the central pair of micro

72 withdrawal of leading processes, changes in microtubular organization and, in some instances, to det

73 active Nercc has important functions at the microtubular organizing center during cell division.

74 consanguineous families with PCD and central-microtubular-pair abnormalities.

75 oma, are transduced by the primary cilium, a microtubular projection found on many cells.

76 Excitatory amino acids may promote microtubular proteolysis observed in ischemic neuronal d

77 link between excitotoxic neurotransmission, microtubular proteolysis, and neuronal degeneration in f

78 ulin binding sites spaced 8 nm apart along a microtubular protofilament.

79 Such acid-driven microtubular rockets offer considerable potential for di

80 r compartment and anchored into the axonemal microtubular scaffold via the ODA docking complex (ODA-D

81 cyte aging and improves the integrity of the microtubular spindle apparatus in young and old oocytes.

82 We tested the effects of a microtubular stabilizer (Taxol) in liver cell preservati

83 of isolated tubulin in vitro, disrupted the microtubular structure in MCF-7 cells as visualized by c

84 We hypothesize that this novel microtubular structure is involved in transporting mater

85 using this nuclear shaping is generated by a microtubular structure termed the manchette, which attac

86 igher concentrations of colchicine disrupted microtubular structure, but also caused increased actin

87 and FKBP1b-mediated restoration of neuronal microtubular structure.

88 We propose that these microtubular structures contribute to a checkpoint contr

89 ound to be required to generate late-mitotic microtubular structures located at the division plane, a

90 When these microtubular structures were disrupted, the actin ring m

91 centrations <1.0 microM caused disruption of microtubular structures, but had little effect on either

92 that taurocholate-mediated changes involve a microtubular system.

93 ient tubulin for assembly and maintenance of microtubular systems.

94 known to be transported to the nucleus along microtubular tracks by cytoplasmic dynein.

95 the latest stage of organelle traffic along microtubular tracks in the proplatelet shafts as shown b

96 nein, a molecular motor that processes along microtubular tracks to the nucleus.

97 g cellular transport of specific cargo along microtubular tracks via kinesin motor proteins.

98 ate of mass transfer is optimally related to microtubular transport and clustering properties of vesi

99 At the same time, microtubular transport and vesicle clustering were model

100 stand the relationship of mass transfer with microtubular transport and vesicle clustering, we varied

101 he best representation of diffusion, whereas microtubular transport is accurately modeled with fracti

102 mation of a zone around the centrosome where microtubular transport of lysosomes is suppressed, resul

103 Any perturbation to either microtubular transport or vesicle aggregation led to red

104 Disruption of cell microtubular transport system by colchicine blocked the

105 Disruption of cell microtubular transport system by colchicine inhibited th

106 om viral factories at speeds consistent with microtubular transport to the peripheries of ATIs, where

107 a-lumicolchicine, which does not affect cell microtubular transport, did not inhibit the stimulatory

108 tional double-membrane envelope that enables microtubular transport, exocytosis, and actin polymeriza

109 articles with a double membrane that enables microtubular transport, exocytosis, and actin polymeriza