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1 l damage in all 2-VO rats and slight loss of microtubule associated protein-2.
2 protein kinase Cgamma and phosphorylation of microtubule-associated protein 2.
3 le labeling for the dendrite-specific marker microtubule-associated protein 2.
4 lig2 was coexpressed with a neuronal marker, microtubule-associated protein 2.
5 immunoreactivity for the dendritic protein, microtubule-associated protein 2.
6 against nonphosphorylated neurofilaments and microtubule-associated protein 2.
7 in of Fyn binds to a conserved PXXP motif on microtubule-associated protein-2.
8 of neurites containing the dendritic marker microtubule-associated protein-2.
9 -43 as well as the dendrite-specific marker, microtubule-associated protein-2.
10 as positive for fibronectin and negative for microtubule-associated protein 2 (a neuronal marker) and
12 o worsened hAPP/Abeta-dependent depletion of microtubule-associated protein 2, a marker of neuronal d
13 otic cytochrome c release and degradation of microtubule-associated protein-2, a marker of neuronal s
14 sociated with differentiated neurons such as microtubule associated protein 2 and neurofilament 200.
15 n of neuronal processes and disappearance of microtubule-associated protein 2 and neurofilament immun
16 tribution of the three neuronal populations: microtubule-associated protein 2 and nonphosphorylated n
18 nd immunoreactivity to neuronal derived Ags (microtubule-associated protein-2 and N-methyl d-aspartat
19 s coexpressed neuronal markers: NeuN, HuC/D, microtubule-associated protein 2, and a dextran placed i
20 the activity-regulated cytoskeletal protein, microtubule-associated protein 2, and alpha-CaM Kinase I
21 calmodulin-dependent kinase II, dendrin, the microtubule-associated protein 2, and neurogranin (RC3)
22 izing-hormone receptor (LHR), inhibin-alpha, microtubule-associated protein 2D, and the PKA type IIbe
23 ecific markers for neuronal nuclear antigen, microtubule-associated protein-2, and calbindin D28k, in
24 t expresses both the neuron-specific marker, microtubule-associated protein-2, and the neurokinin-1 r
26 ots, we show that alterations in hippocampal microtubule-associated protein-2 appear to be highly cor
27 ific microtubule-associated proteins tau and microtubule-associated protein-2 are substrates for the
28 s a reflection of total synaptic density and microtubule-associated protein 2 as a dendritic structur
29 splayed multipotency by differentiating into microtubule-associated protein 2, beta-III tubulin, and
30 PR-ir colocalizes with the neuronal marker, microtubule associated protein-2, but not with the glial
31 proteins, calbindin, calretinin, synapsin I, microtubule-associated protein-2, calcitonin gene-relate
33 s identified with calbindin, calretinin, and microtubule-associated protein-2 did not express CB(1) r
34 amount of dendritic simplification based on microtubule-associated protein 2 immunohistochemical sta
35 thod for the determination of the density of microtubule-associated protein 2-immunolabeled neurons i
36 red a widespread network of precocious MAP2 (microtubule-associated protein 2)-immunoreactive cells,
37 d labeling (TUNEL), and decreased numbers of microtubule-associated protein-2-immunoreactive neurons
38 cidic protein immunoreactivity and increased microtubule-associated protein-2 immunoreactivity in the
40 d (E)GFP+ cells expressed betaIII-tubulin or microtubule-associated protein-2; many incorporated brom
43 etal injury with preserved immunolabeling of microtubule-associated protein 2 (MAP 2) at 6 and 24 h a
45 duction of significant neurite outgrowth and microtubule-associated protein 2 (MAP-2) and neuron-spec
46 5days in culture), when proteins such as the microtubule-associated protein 2 (MAP-2) and the glutama
50 ls of immunostaining for tubulin, actin, and microtubule-associated protein 2 (MAP-2), and levels of
51 describe induction of a juvenile isoform of microtubule-associated protein 2 (MAP-2c) in cultured me
52 as neurons by using neuron-specific markers, microtubule-associated protein-2 (MAP-2) and anti-neuron
53 cal microscopy of sections immunolabeled for microtubule-associated protein-2 (MAP-2) and synaptophys
54 s of the CA1 region of the hippocampus using microtubule-associated protein-2 (MAP-2) histochemistry
55 identify degenerating neurons) and decreased microtubule-associated protein-2 (MAP-2) immunoreactivit
59 e (FITC)-dextran, GFAP immunoreactivity, and microtubule associated protein-2 (MAP2) immunoreactivity
61 ajority of M1 and M2 ipRGCs expressed Isl-1, microtubule associated protein-2 (MAP2), gamma-synuclein
62 localized with neuronal and survival markers microtubule-associated protein 2 (MAP2) and Bcl-2 using
65 strated that Tat causes rapid degradation of microtubule-associated protein 2 (MAP2) and the collapse
66 -SPION-rIgPxcIgY carries chick polyclonal to microtubule-associated protein 2 (MAP2) as Ran-SPION-rIg
69 -labeling with a monoclonal antibody against microtubule-associated protein 2 (MAP2) in hatchlings sh
73 indicates that the phosphorylation state of microtubule-associated protein 2 (MAP2) may play a key r
75 serves as a marker of dendritic spines, and microtubule-associated protein 2 (MAP2), an overall dend
76 alyze three neuronal markers: synaptophysin, microtubule-associated protein 2 (MAP2), and calbindin.
77 y diminished neuronal damage, as assessed by microtubule-associated protein 2 (MAP2), class III beta-
78 ble immunolabelling for poly(ADP-ribose) and microtubule-associated protein 2 (MAP2), glial fibrillar
80 tection against severing is also afforded by microtubule-associated protein 2 (MAP2), which has a tau
81 feration (+16%), decreased neurogenesis into microtubule-associated protein 2 (MAP2)-positive neurons
84 to tyrosine hydroxylase (TH), an antibody to microtubule-associated protein 2 (MAP2, a dendritic mark
85 ciated with activity/anxiety behaviours, and microtubule-associated protein 2 (Map2, rs13475902) was
90 brillary acidic protein (astrocytic marker), microtubule-associated protein 2 (neuronal marker), or 2
91 l cultures derived from two brains generated microtubule-associated protein-2+ neurons, which incorpo
92 enerated cells expressed the neuronal marker microtubule-associated protein-2, or neuron-specific tub
93 e v-abl construct (CA-Abl) show an exuberant microtubule-associated protein 2-positive (MAP2-positive
94 d by a twofold increase in the percentage of microtubule-associated protein 2-positive cells even in
95 ptophysin-positive presynaptic terminals and microtubule-associated protein 2-positive neuronal dendr
96 Oct3/4-positive, undifferentiated status to microtubule-associated protein 2-positive neurons and gl
97 EC24C is disrupted, remained confined to the microtubule-associated protein 2-positive somatodendriti
98 ontrast, Li restored HIVE-associated loss of microtubule-associated protein-2-positive neurites and s
99 tin-positive neuroblasts (-28%), and mature, microtubule-associated protein-2-positive neurons (-36%)
100 ptors were also expressed by postmigrational microtubule-associated protein-2-positive neurons of the
101 onth before training altered the hippocampal microtubule-associated protein-2 response; in these anim
102 tibodies to growth-associated protein-43 and microtubule-associated protein-2 revealed segregation of
103 division, 64 kDa, class III beta-tubulin, or microtubule-associated protein-2 showed that the vast ma
104 synaptotoxicity (decreased synaptophysin and microtubule-associated protein-2 staining) and reactive
105 ncreases in immunohistochemical staining for microtubule-associated protein-2 suggested that there wa
107 as well as the endogenous protein substrates microtubule-associated protein 2, tau and spectrin, was
108 igh molecular weight neurofilament proteins, microtubule-associated protein 2, tau, and synaptophysin
111 In SIVE, staining for postsynaptic protein microtubule-associated protein-2 was significantly decre
112 f class III neuron-specific beta-tubulin and microtubule-associated protein 2 were significantly incr
113 with reductions in the immunoreactivity for microtubule-associated protein 2, which affects cytoskel
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