ライフサイエンスコーパス: microvascular endothelial cell

1 ndothelial cells and primary human pulmonary microvascular endothelial cells.

2 tumor microenvironment with cancer cells and microvascular endothelial cells.

3 rier disruption in LPS-challenged human lung microvascular endothelial cells.

4 ctive macropinocytic entry into human dermal microvascular endothelial cells.

5 ages and the tubule forming of mouse retinal microvascular endothelial cells.

6 nt, which profoundly alters the phenotype of microvascular endothelial cells.

7 es STIM1, thus inhibiting SOCE in human lung microvascular endothelial cells.

8 ion release but not entry into primary human microvascular endothelial cells.

9 ch as Andes virus (ANDV), which targets lung microvascular endothelial cells.

10 wing HPAI virus infection of human pulmonary microvascular endothelial cells.

11 nvestigated the role of P-Rex1 in human lung microvascular endothelial cells.

12 surface of IEs binding to human receptors on microvascular endothelial cells.

13 rvival, and proliferation of human pulmonary microvascular endothelial cells.

14 tyrosine nitration in cultured human cardiac microvascular endothelial cells.

15 obtained using an in vitro model with brain microvascular endothelial cells.

16 -mediated induction of PDGF-B in human brain microvascular endothelial cells.

17 l cells, alveolar macrophages, and pulmonary microvascular endothelial cells.

18 nstrictor endothelin-1 (ET-1) from pulmonary microvascular endothelial cells.

19 erties of monocyte subsets using human brain microvascular endothelial cells.

20 and survival of BMCs or mature human cardiac microvascular endothelial cells.

21 l NanA had increased invasion of human brain microvascular endothelial cells.

22 ion at threonine 495 levels in human cardiac microvascular endothelial cells.

23 e, and in supernatants of primary human lung microvascular endothelial cells.

24 engineered HeLa cells and Nox2(-/-) coronary microvascular endothelial cells.

25 Similar effects were observed in human brain microvascular endothelial cells.

26 avirus (PUUV)-infected patients and in human microvascular endothelial cells.

27 haracteristics different from those of other microvascular endothelial cells.

28 reted MFAP5 promotes tube formation of human microvascular endothelial cells.

29 tions and prevent virus transit across brain microvascular endothelial cells.

30 insulin and how this occurs in the relevant microvascular endothelial cells.

31 sing NTR1 (NCM460-NTR1) and human intestinal microvascular-endothelial cells.

32 esis study was evaluated in vitro with human microvascular endothelial cells-1 and in vivo with the M

33 anner in which they interact with macro- and microvascular endothelial cells, a key enabling componen

34 PCR-array screening of human brain microvascular endothelial cells after ABCD1 silencing re

35 BDNF protein were quantified in human brain microvascular endothelial cells after exposure to advanc

36 RNA silencing studies in microvascular endothelial cells, along with knockout and

37 uman serum disrupted the barrier function of microvascular endothelial cells, an effect fully neutral

38 ukotriene formation, in both human pulmonary microvascular endothelial cells and a transformed human

39 ion across monolayers of primary human brain microvascular endothelial cells and diminished BBB damag

40 Human microvascular endothelial cells and glomerular endotheli

41 ole of NO production by human adult cerebral microvascular endothelial cells and human fetal astrocyt

42 ion at threonine 495 levels in human cardiac microvascular endothelial cells and improved BMC angioge

43 s in the barrier function of human pulmonary microvascular endothelial cells and in neutrophil traffi

44 Y4 nucleotide receptor, expressed on cardiac microvascular endothelial cells and involved in postnata

45 GATA5 is expressed in microvascular endothelial cells and its genetic inactiva

46 deleterious effects induced by IL2 on brain microvascular endothelial cells and may inform the devel

47 crosslink HLA I on human aortic, venous, and microvascular endothelial cells and measured the binding

48 PAI-1 expression in primary human pulmonary microvascular endothelial cells and monocytes through ac

49 tained from db/db mice in vivo and in kidney microvascular endothelial cells and podocytes treated wi

50 fering with circulating PCa cell adhesion to microvascular endothelial cells and potentially reducing

51 xes between the central nervous system (CNS) microvascular endothelial cells and the choroid plexus e

52 ions, GD RBCs adhered more strongly to human microvascular endothelial cells and to laminin than CTR.

53 Here we show that Irx3 is expressed in human microvascular endothelial cells, and expression is eleva

54 n their surface, including HUVEC, human lung microvascular endothelial cells, and human coronary arte

55 ures, primary lung fibroblasts, primary lung microvascular endothelial cells, and primary alveolar ty

56 e, hantaviruses induced tPA but not PAI-1 in microvascular endothelial cells, and the induction was d

57 nd that VEGF activates PLD1 in human retinal microvascular endothelial cells, and this event is depen

58 c (~2-fold vs. ~1.6-fold increase) for human microvascular endothelial cells, and Y2/Y5 receptor anta

59 an lung, human alveolar epithelial cells and microvascular endothelial cells are cultured in the micr

60 an enhanced ability to adhere to human brain microvascular endothelial cells as compared with monocyt

61 WNV-MAD78 replicated in and traversed brain microvascular endothelial cells as efficiently as WNV-NY

62 anistic studies in primary brain and retinal microvascular endothelial cells, as well as occluded rat

63 also inhibits the migration of human dermal microvascular endothelial cells at similar concentration

64 Cultured human umbilical vein and microvascular endothelial cells avidly engulf BODIPY (4,

65 We focused our study on microvascular endothelial cells because they play a key

66 hosphatidylinositol-anchored glycoprotein of microvascular endothelial cells, binds lipoprotein lipas

67 arrier (BBB), which mainly consists of brain microvascular endothelial cells (BMEC).

68 brin-Matrigel mixed gel by coculturing brain microvascular endothelial cells (BMECs) and human mesenc

69 Acute Krit1 gene inactivation in mouse brain microvascular endothelial cells (BMECs) changes expressi

70 ation and tube formation properties of brain microvascular endothelial cells (BMECs) were analyzed as

71 Primary mouse brain microvascular endothelial cells (BMECs) were cultured an

72 a human in vitro BBB model comprising brain microvascular endothelial cells (BMECs), pericytes, astr

73 Thrombin reduced brain microvascular endothelial cell (BMVEC) and perivascular

74 in several BBB models of rat and human brain microvascular endothelial cells (BMVEC) using a recyclab

75 mediators and leukocyte engagement of brain microvascular endothelial cells (BMVECs) contribute to b

76 ected to sham or stroke surgery and in brain microvascular endothelial cells (BMVECs) from Wistar and

77 usly that GSK3beta inhibition in human brain microvascular endothelial cells (BMVECs) reduced monocyt

78 Exposure of primary human brain microvascular endothelial cells (BMVECs) to CD40L upregu

79 human brain tissues and primary human brain microvascular endothelial cells (BMVECs), we demonstrate

80 adherin mediated by miRNA-101 in human brain microvascular endothelial cells (BMVECs).

81 High levels of virus replication occur in microvascular endothelial cells but without a virus-indu

82 events, inhibit VEGF-induced angiogenesis in microvascular endothelial cells by both (a) cleavage and

83 ration, and migration of cultured intestinal microvascular endothelial cells by phosphorylating Akt,

84 ed individuals, as controls) and human brain microvascular endothelial cells by using quantitative po

85 ned from patients during remission, to human microvascular endothelial cells caused vascular endothel

86 Silencing of ABCD1 in human brain microvascular endothelial cells causes accumulation of v

87 transporter 1 (GLUT-1) levels in human brain microvascular endothelial cells, causing disruption of b

88 -angiogenic effect on human brain and dermal microvascular endothelial cells co-cultured with establi

89 protects cultured HT22 neuronal and primary microvascular endothelial cells co-cultured with primary

90 a set of differentially expressed miRNAs in microvascular endothelial cells co-cultured with tumour

91 man bronchial epithelial cells and pulmonary microvascular endothelial cells, compared with the abili

92 involved in lipid metabolism, in a cerebral microvascular endothelial cell culture system (hCMEC/D3)

93 ombinant proteinase-3 applied to human brain microvascular endothelial cells degraded both the tight

94 In cytokine-treated human lung microvascular endothelial cells, disruption of B1R-CPM h

95 respectively, in 10-day KSHV-infected dermal microvascular endothelial cells (DMVEC).

96 ure formation, and migration of db/db dermal microvascular endothelial cells (DMVECs), as well as rem

97 lood vessel growth primarily from host organ microvascular endothelial cells (EC), and microvasculatu

98 ating leukocytes with IL-1- or TNF-activated microvascular endothelial cells (ECs) and pericytes (PCs

99 Microvascular endothelial cells (ECs) are increasingly r

100 Microvascular endothelial cells (ECs) display a high deg

101 We demonstrate that microvascular endothelial cells (ECs) from Anxa2(-/-) mi

102 ed neurotrophic factor (BDNF) is secreted by microvascular endothelial cells (ECs) in the brain, func

103 Dermal microvascular endothelial cells (ECs) isolated from this

104 Knockdown of ZNF24 by siRNA in human primary microvascular endothelial cells (ECs) led to significant

105 TG microvascular endothelial cells (ECs) treated with AngII

106 Microvascular endothelial cells (ECs) within different t

107 y, we describe their effects on human dermal microvascular endothelial cells (ECs), a natural target

108 he disassembly of junctional proteins within microvascular endothelial cells (ECs).

109 ns cells (LCs) to T cells through actions on microvascular endothelial cells (ECs).

110 romotes the in vitro tube formation of human microvascular endothelial cells, ex vivo vessel outgrowt

111 We found that both human and murine brain microvascular endothelial cells express constituents of

112 Human lung microvascular endothelial cells expressed the TJ protein

113 We showed that acute stimulation of murine microvascular endothelial cells expressing the tumor nec

114 PAH pericytes seeded with healthy pulmonary microvascular endothelial cells failed to associate with

115 ue ABCD2, is highly expressed in human brain microvascular endothelial cells, far exceeding its expre

116 viously determined that stimulation of human microvascular endothelial cells from lung (HMVEC-L) resu

117 lls and angiogenic tubule formation of human microvascular endothelial cells from lungs (HMEC-Ls).

118 vitro, angiotensin-(1-7) protected pulmonary microvascular endothelial cells from thrombin-induced ba

119 In human pulmonary microvascular endothelial cells, G was 20.4 +/- 12 Pa an

120 ) and chondroitin sulfate (CS) to glomerular microvascular endothelial cell (GEnC) glycocalyx and exa

121 ulin]) and AP components in human glomerular microvascular endothelial cells (GMVECs) and in HUVECs,

122 sis across individual, primary human adipose microvascular endothelial cells (HAMECs), involving insu

123 our recently described role for human brain microvascular endothelial cells (HBEC) in modulating imm

124 of high therapeutic dosage on a human brain microvascular endothelial cell (HBMEC) model of the BBB.

125 mutant resulted in disruption of human brain microvascular endothelial cell (hBMEC) monolayer integri

126 We used human brain microvascular endothelial cells (HBMEC) as the in vitro

127 218 activates Rac1 (GTP-Rac1) of human brain microvascular endothelial cells (HBMEC) in a time-depend

128 m for inflammatory activation of human brain microvascular endothelial cells (HBMEC) in response to i

129 tococcus binding and invasion of human brain microvascular endothelial cells (HBMEC) is a prerequisit

130 l in serum, bacterial entry into human brain microvascular endothelial cells (HBMEC) is governed by L

131 Coculturing human brain microvascular endothelial cells (hBMEC) or NOTCH ligand

132 Human brain microvascular endothelial cells (HBMEC) produced abundan

133 am and interact with specialized human brain microvascular endothelial cells (hBMEC), which constitut

134 utes to type III GBS invasion of human brain microvascular endothelial cells (HBMEC), which constitut

135 n to promote E. coli invasion of human brain microvascular endothelial cells (HBMEC), which constitut

136 ntributes to E. coli invasion of human brain microvascular endothelial cells (HBMEC), which constitut

137 Escherichia coli K1 infection of human brain microvascular endothelial cells (HBMECs) induces the exp

138 xidase (GlpO), was cytotoxic for human brain microvascular endothelial cells (HBMECs) via generation

139 Here, we found that infection of human brain microvascular endothelial cells (hBMECs) with GBS and ot

140 f neonatal meningitis, can enter human brain microvascular endothelial cells (hBMECs), but the host r

141 strates that CnMVs can fuse with human brain microvascular endothelial cells (HBMECs), the constituen

142 to adhere, invade, and penetrate human brain microvascular endothelial cells (hBMECs), the single-cel

143 hibitor of E. coli invasion into human brain microvascular endothelial cells (HBMECs).

144 acterized for binding to primary human brain microvascular endothelial cells (HBMECs).

145 er-endothelial cell junctions in human brain microvascular endothelial cells (HBMECs).

146 Human brain microvascular endothelial cells (hBMVECs) that constitut

147 r generating primary cultures of human brain microvascular endothelial cells (HBMVECs).

148 using Heme-treated and untreated human brain microvascular endothelial cells (HBVEC), and determined

149 adhere to and migrate through human cerebral microvascular endothelial cells (HCMEC/D3), in a manner

150 tigated the mechanisms by which human dermal microvascular endothelial cells (HDMECs) perceive mechan

151 In human dermal microvascular endothelial cells (HDMECs), exposure to hy

152 d Rac1 in a sustained manner in human dermal microvascular endothelial cells (HDMVECs).

153 y in a time-dependent manner in human dermal microvascular endothelial cells (HDMVECs).

154 n in a time-dependent manner in human dermal microvascular endothelial cells (HDMVECs).

155 Microvascular endothelial cell heterogeneity and its rel

156 Human intestinal microvascular endothelial cells (HIMEC) and human intest

157 We investigated whether human intestinal microvascular endothelial cells (HIMEC) undergo EndoMT a

158 e induces EndoMT in primary human intestinal microvascular endothelial cells (HIMECs) and whether End

159 ucosal biopsies and primary human intestinal microvascular endothelial cells (HIMECs) isolated from s

160 , and characterized human kidney peritubular microvascular endothelial cells (HKMECs) and reconstitut

161 ), as well as for GHRH itself, in human lung microvascular endothelial cells (HL-MVEC).

162 In normal human lung microvascular endothelial cells (HLMVEC), bradykinin (BK

163 that iNOS in cytokine-stimulated human lung microvascular endothelial cells (HLMVECs) is highly regu

164 lipodia formation and motility of human lung microvascular endothelial cells (HLMVECs) via PI3K/Akt s

165 pre-requisite for migration using human lung microvascular endothelial cells (HLMVECs).

166 n Tg(fli1:EGFP) zebrafish and inhibits human microvascular endothelial cell (HMEC-1) proliferation, t

167 Guided by cDNA microarray analysis of human microvascular endothelial cells (HMEC-1 line) subjected

168 y studying capillary tube formation in human microvascular endothelial cells (HMEC-1) on growth facto

169 Human microvascular endothelial cells (HMEC-1) stimulated with

170 its than control serum on unstimulated human microvascular endothelial cells (HMEC-1).

171 Induction of miR-199a-5p in human dermal microvascular endothelial cells (HMECs) blocked angiogen

172 cape of R. conorii during infection of Human Microvascular Endothelial Cells (HMECs) by strand-specif

173 Delivery of the miR-200b mimic in human microvascular endothelial cells (HMECs) suppressed the a

174 on protein (STAT) signaling pathway in human microvascular endothelial cells (HMECs), the most releva

175 Using human microvascular endothelial cells (HMECs), we examined inf

176 present in the RA joint, induces human lung microvascular endothelial cell (HMVEC) migration mediate

177 tions present in the RA joint, induced human microvascular endothelial cell (HMVEC) migration that wa

178 Human microvascular endothelial cells (HMVEC) and human period

179 P-EA exerted antiangiogenic effects in human microvascular endothelial cells (HMVEC) and vasodilatory

180 d associated signaling to enter human dermal microvascular endothelial cells (HMVEC-d), an in vivo ta

181 iated herpesvirus (KSHV) enters human dermal microvascular endothelial cells (HMVEC-d), its naturalin

182 During de novo infection of human dermal microvascular endothelial cells (HMVEC-d), Kaposi's sarc

183 ociated herpesvirus (KSHV) into human dermal microvascular endothelial cells (HMVEC-d), natural in vi

184 ngiogenic potential of neonatal dermal human microvascular endothelial cells (HMVEC-dNeo).

185 formation and chemotaxis assays using human microvascular endothelial cells (HMVECs) transfected wit

186 s, RA synovial tissue fibroblasts, and human microvascular endothelial cells (HMVECs) were determined

187 PS mediate Ang2 signaling in human pulmonary microvascular endothelial cells (HPMECs) remain understu

188 To this end, human retinal microvascular endothelial cells (HRMEC) transfected with

189 In vitro studies using human retinal microvascular endothelial cells (HRMECs) showed increase

190 subtypes expressed on primary human retinal microvascular endothelial cells (HRMECs).

191 ive agonist, beta-LGND2, using human retinal microvascular endothelial cell (HRMVEC) cultures and a m

192 s Pyk2 activation in mediating human retinal microvascular endothelial cell (HRMVEC) migration, sprou

193 Western blots of human retinal microvascular endothelial cells (hRMVECs) stimulated wit

194 induced Kdr phosphorylation in human retinal microvascular endothelial cells (HRMVECs).

195 in a time-dependent manner in human retinal microvascular endothelial cells (HRMVECs).

196 In human primary retinal microvascular endothelial cells, hypoxia induces the exp

197 2) stimulated migration and tubulogenesis of microvascular endothelial cells, implicating a proangiog

198 mononuclear cells and in primary human lung microvascular endothelial cells in a concentration- and

199 otypic adhesion were further confirmed using microvascular endothelial cells in a static condition.

200 n imaging reveal that the plasma membrane of microvascular endothelial cells in caveolin 1(-/-) mice

201 ion of human cerebral, dermal, and pulmonary microvascular endothelial cells in comparison with pulmo

202 igrated and proliferated less than wild-type microvascular endothelial cells in response to vascular

203 all, these data show for the first time that microvascular endothelial cells in the bone marrow and s

204 ced adherence to and invasion of human brain microvascular endothelial cells in vitro, demonstrating

205 lated gremlin secretion from human pulmonary microvascular endothelial cells in vitro, which inhibite

206 were abundantly expressed in cultured mouse microvascular endothelial cells, including NLRP3, apopto

207 of glutathione peroxidase-1 (GPx-1) in human microvascular endothelial cells increases CD14 gene expr

208 d caspase-1 activity and IL-1beta release in microvascular endothelial cells, indicating an activatio

209 ost response mechanisms in macrovascular and microvascular endothelial cells infected with R. rickett

210 CSE expression was also increased in cardiac microvascular endothelial cells, isolated from endotheli

211 4var09) cytoadhere in vitro to a human brain microvascular endothelial cell line (HBEC-5i).

212 Experiments in a human microvascular endothelial cell line demonstrated that TN

213 ABSTRACT: The human cerebral microvascular endothelial cell line hCMEC/D3 was used to

214 onolayers of the immortalized human cerebral microvascular endothelial cell line hCMEC/D3.

215 h human embryonic kidney cells and the human microvascular endothelial cell line, HMEC-1.

216 Using the human microvascular endothelial cell line, human mast cell lin

217 ther rat alveolar macrophages (AMs) and lung microvascular endothelial cells (LMVECs) support Seoul v

218 Upon KSHV infection, primary dermal microvascular endothelial cells lost expression of endot

219 Here we employ LEC, microvascular endothelial cells (MEC), and human umbilic

220 d the lipoma-preferred partner (LPP) gene in microvascular endothelial cells (MECs) and that LPP expr

221 The relevance of tissue specificity of microvascular endothelial cells (MECs) in the response t

222 We utilised isogenic keratinocytes, microvascular endothelial cells, melanocytes and fibrobl

223 EphrinB2 DeltaV primary kidney microvascular endothelial cells migrated and proliferate

224 cantly attenuated VEGF-induced human retinal microvascular endothelial cell migration, proliferation,

225 The studies conducted in polarized human microvascular endothelial cell monolayers (hCMEC/D3) in

226 hibited leukocyte adherence to human retinal microvascular endothelial cell monolayers and leukostasi

227 In these studies, rat brain microvascular endothelial cell monolayers exposed to cal

228 Permeability of human brain microvascular endothelial cell monolayers was increased

229 bilical vein endothelial cell and human lung microvascular endothelial cell monolayers were treated w

230 bilical vein endothelial cell and human lung microvascular endothelial cell monolayers.

231 induce changes in the permeability of brain microvascular endothelial cell monolayers.

232 demonstrated with lysates of mouse pulmonary microvascular endothelial cells (MPMVECs) that were stim

233 Microvascular endothelial cells (MVEC) were developed fr

234 th factor signaling at the receptor level in microvascular endothelial cells (MVEC), and CD36 has bee

235 We found that the CLL stroma included microvascular endothelial cells (MVECs) expressing BAFF

236 g, migration, and sprouting of primary brain microvascular endothelial cells (MVECs) in a dose-depend

237 Microvascular endothelial cells (MVECs) injury is a crit

238 ction in all forms of PAH and tested whether microvascular endothelial cells (MVECs) or pulmonary art

239 down-regulation of their cognate receptor on microvascular endothelial cells (MVECs), CD36.

240 36, a cell surface glycoprotein expressed on microvascular endothelial cells (MVECs), for it to elici

241 d functions of ICAM-1 in cerebral and dermal microvascular endothelial cells (MVECs).

242 /-BQ788) was given to cultured rat pulmonary microvascular endothelial cells overexpressing ETB recep

243 GF, IL8, and CXCL12 leading to chemotaxis of microvascular endothelial cells, phosphorylation of VE-c

244 ved by the disease process, with luminal and microvascular endothelial cells playing a critical role

245 activated protein kinase (AMPK) in pulmonary microvascular endothelial cell (PMVEC) repair.

246 agonist-induced NOX2 activation in pulmonary microvascular endothelial cells (PMVEC) and that the eff

247 ermeability of monolayers of human pulmonary microvascular endothelial cells (PMVECs) in vitro and lu

248 of NADPH oxidase type 2 (NOX2) in pulmonary microvascular endothelial cells (PMVECs), alveolar macro

249 ro transmigration model with human pulmonary microvascular endothelial cells (PMVECs).

250 lood-brain barrier, mainly composed of brain microvascular endothelial cells, poses an obstacle to dr

251 endothelial cell adhesion molecules in brain microvascular endothelial cell proliferation and apoptos

252 monary veins associated with foci of intense microvascular endothelial-cell proliferation of the capi

253 rent human blood monocytes and in human lung microvascular endothelial cells, providing a mechanism f

254 eractions between pathogenic rickettsiae and microvascular endothelial cells remain poorly understood

255 Isolated cardiomyocytes and microvascular endothelial cells responded to AngII with

256 Graft microvascular endothelial cells responded to DSA in vivo

257 RNA-mediated knockdown of SRSF2 in pulmonary microvascular endothelial cells resulted in elevated lev

258 3 gain and loss of function studies in human microvascular endothelial cells resulted in the modulati

259 T bone marrow-derived macrophages with renal microvascular endothelial cells results in increased lev

260 Our in vitro studies using pulmonary microvascular endothelial cells revealed that HIMF stimu

261 We isolated primary renal microvascular endothelial cells (RMEC) and aortic endoth

262 formed antibody reactivity against DKO renal microvascular endothelial cells (RMEC) in vitro.

263 We show that keratinocytes and microvascular endothelial cells show greatest NO release

264 Cell-binding studies using microvascular endothelial cells showed receptor-specific

265 Gene transfection of human pulmonary microvascular endothelial cells showed that C242T p22(ph

266 In vitro experiments using human lung microvascular endothelial cells showed that exogenous S1

267 macrovascular endothelial cells and retinal microvascular endothelial cells that C-1-P induces invas

268 ximum wall shear stress, whereas low-density microvascular endothelial cells that lack cell-cell cont

269 major entry pathway of KSHV in human dermal microvascular endothelial cells, the natural target cell

270 In brain microvascular endothelial cells, there was no change in

271 flammatory mediators by primary human dermal microvascular endothelial cells through a signaling path

272 mediated induction of PDGF-BB in human brain microvascular endothelial cells through the binding to i

273 e-mediated induction of ALCAM in human brain microvascular endothelial cells through the translocatio

274 Exposure of bEnd3 microvascular endothelial cells to elevated extracellula

275 we used human brain microvascular endothelial cells to examine the role of N

276 this device, we investigated the response of microvascular endothelial cells to shear-stress gradient

277 In vitro exposure of human microvascular endothelial cells to Shiga toxin recapitul

278 hese exosomes alone can activate human brain microvascular endothelial cells to stimulate adhesion mo

279 In TNFalpha-stimulated primary human retinal microvascular endothelial cells, total levels of epoxyei

280 Human microvascular endothelial cells transfected with hHO-1 d

281 S, we performed microarray analysis of human microvascular endothelial cells treated with TNF-alpha i

282 In retinal microvascular endothelial cells, TRPV4 channels regulate

283 In human central nervous system microvascular endothelial cells, VEGFA and the TYMP prod

284 in modulating proliferation and apoptosis of microvascular endothelial cells via its modulation of CD

285 ithelial cell wound healing, maintained lung microvascular endothelial cell viability, and proliferat

286 ial electrical resistance of human pulmonary microvascular endothelial cells was analyzed.

287 of human bronchial epithelial cells and lung microvascular endothelial cells was exposed to immunosup

288 VEGF, transendothelial migration through CNS microvascular endothelial cells was regulated by VEGF.

289 ibitory activity of TSP1 in large vessel and microvascular endothelial cells was replicated by a reco

290 A unique feature of all microvascular endothelial cells was the lack of induced

291 ICAM-1(null)/ICAM-2(-/-) primary mouse brain microvascular endothelial cells, we demonstrate that neu

292 In human microvascular endothelial cells, we found that GPx-1 def

293 horylation and barrier disruption, pulmonary microvascular endothelial cells were engineered for the

294 , motility and polarization toward pulmonary microvascular endothelial cells were reduced, whereas wi

295 lical vein endothelial cells or adult dermal microvascular endothelial cells were transduced with the

296 ger annexin A1 (ANXA1) is expressed in brain microvascular endothelial cells, where it regulates BBB

297 (WNV-NY) strains in neurons, astrocytes, and microvascular endothelial cells, which comprise the neur

298 nificantly induced CX3CL1 production in lung microvascular endothelial cells, which was blocked by in

299 Treatment of brain microvascular endothelial cells with AGE caused a simila

300 Transcriptomic analysis of human microvascular endothelial cells with GATA5 knockdown rev