ライフサイエンスコーパス: microvascular permeability

1 s is preceded and/or accompanied by enhanced microvascular permeability.

2 did not alter pancreatic edema or pulmonary microvascular permeability.

3 more potent than histamine as an inducer of microvascular permeability.

4 ated arachidonic acid metabolism and altered microvascular permeability.

5 OS)-derived NO is a key factor in regulating microvascular permeability.

6 endothelial cell proliferation and increased microvascular permeability.

7 vessels was associated with increased tumor microvascular permeability.

8 pression of proinflammatory transcripts, and microvascular permeability.

9 , showing the impact of CXCR7 on stabilizing microvascular permeability.

10 rated optical intensity (IOI) as an index of microvascular permeability.

11 for intrinsic Cat-S/PAR2 in ischemia-induced microvascular permeability.

12 adhesions in the physiological regulation of microvascular permeability.

13 ance of endothelial junctional integrity and microvascular permeability.

14 paracellular pathway, resulting in increased microvascular permeability.

15 mouse LECs is a key determinant of increased microvascular permeability.

16 efemoral lymph fistulas in order to estimate microvascular permeability.

17 teraction in the physiological regulation of microvascular permeability.

18 atherosclerotic endothelium is dependent on microvascular permeability.

19 ularity, cutaneous vascular architecture, or microvascular permeability.

20 lung myeloperoxidase activity and pulmonary microvascular permeability.

21 han those stored in EC, indicating decreased microvascular permeability.

22 confirmed small increases in muscle and skin microvascular permeability after ANP.

23 rowth factor (VEGF), is a potent enhancer of microvascular permeability and a selective endothelial c

24 to high tidal volume VILI, with increases in microvascular permeability and bronchoalveolar lavage le

25 AM-1 blockade prevented the increase in lung microvascular permeability and edema formation at all ti

26 tion and prevented the increase in pulmonary microvascular permeability and edema formation in mice a

27 uli, and has profound influences on systemic microvascular permeability and haemodynamics.

28 enhanced inflammation, along with increased microvascular permeability and hemorrhage.

29 t assessed in a murine model of LPS-mediated microvascular permeability and inflammation with marked

30 oefficient (Kf,c), a sensitive index of lung microvascular permeability and injury, was made at basel

31 c endothelial cells is pivotal in regulating microvascular permeability and leukocyte recruitment.

32 tion of the IgE/antigen-mediated increase in microvascular permeability and of bleomycin-induced pulm

33 emission tomography and K(trans) (reflecting microvascular permeability and perfusion) by magnetic re

34 orous inflammatory response characterized by microvascular permeability and polymorphonuclear neutrop

35 result of marked increases in both pulmonary microvascular permeability and pressure.

36 saccharide and thrombin increased mouse lung microvascular permeability and resulted in a delayed act

37 with reduced parenchymal myeloperoxidase and microvascular permeability, and altered airspace and ser

38 siological processes including inflammation, microvascular permeability, and endothelial mechanotrans

39 increased angiogenesis, microvessel density, microvascular permeability, and growth of malignant tumo

40 tly determined also to promote inflammation, microvascular permeability, and mucus secretion.

41 Data showed that lung edema formation, lung microvascular permeability, and neutrophil infiltration

42 Subsequently, pulmonary vascular resistance, microvascular permeability, and thromboxane were measure

43 Angiogenesis and enhanced microvascular permeability are hallmarks of a large numb

44 we also examined whether these increases in microvascular permeability are NO-dependent.

45 phonuclear leukocyte; PMN) sequestration and microvascular permeability are not well understood.

46 The LPS-induced increase in lung microvascular permeability as measured by Evans blue ext

47 Microvascular permeability, as estimated with the endoth

48 The hallmark of severe dengue is increased microvascular permeability, but alterations in the micro

49 NEP has important roles in regulating basal microvascular permeability by degrading SP and BK, and m

50 ulates endothelial cell growth and increases microvascular permeability by interacting with two endot

51 essed histologically and by determining lung microvascular permeability by measuring accumulated 125I

52 nd neutrophil sequestration, and a decreased microvascular permeability by more than twofold.

53 ly visualize tissue oxygen concentration and microvascular permeability by using a hyperpolarized (1)

54 mice showed reduced neutrophil migration and microvascular permeability changes.

55 Quantitative microvascular permeability characteristics estimated fro

56 used canine lung, we identified increases in microvascular permeability coefficients in response to t

57 05) lower respiratory compliance and greater microvascular permeability compared with sham animals.

58 flammatory cytokine concentrations, and lung microvascular permeability compared with transfusion of

59 C) concentrations in the airspaces, and lung microvascular permeability compared with transfusion of

60 l growth factor (VEGF) chronically increases microvascular permeability, compliance and vessel diamet

61 /0 (but not 45/10), there was an increase in microvascular permeability, cyclical abolition of preloa

62 mural cells of microvessels, which regulate microvascular permeability, development, and maturation

63 (VEGF) is the principal agent that increases microvascular permeability during physiological and path

64 nd apparently also by declining the enhanced microvascular permeability during the late phase of endo

65 helial growth factor (VEGF) greatly enhances microvascular permeability; however, the molecular mecha

66 plays a dual regulatory role in controlling microvascular permeability: (i) as a structural protein

67 ole in the changes in systemic and pulmonary microvascular permeability in combined smoke inhalation/

68 establish eNOS as an important regulator of microvascular permeability in inflammation.

69 not pancreatic edema or increased pulmonary microvascular permeability in mild, secretagogue-induced

70 bserved that E. coli failed to increase lung microvascular permeability in p47(phox-/-) and gp91(phox

71 ovascular reserve, and for the assessment of microvascular permeability in patients with intracranial

72 leads to a major increase in epithelial and microvascular permeability in the lungs.

73 l retention) and the LPS-induced increase in microvascular permeability in these organs.

74 r endothelial growth factor (VEGF) increases microvascular permeability in vivo and has been hypothes

75 ese data suggest that VEGF acutely increases microvascular permeability in vivo through a mechanism t

76 d (4) caveolin-1 inhibits eNOS regulation of microvascular permeability in vivo.

77 le of deltaPKC, which is thought to regulate microvascular permeability, in the development of hypert

78 of Cx43 completely blocked thrombin-induced microvascular permeability increases.

79 in on myocardial perfusion (MP) and coronary microvascular permeability index (PI) at baseline and du

80 The increased pulmonary microvascular permeability induced by E. coli is solely

81 lungs, heart, and liver; and c) increase in microvascular permeability induced by LPS in these organ

82 itment of PMNs in lungs and increase in lung microvascular permeability induced by TNF-alpha.

83 It increases microvascular permeability, induces endothelial cell mig

84 ions drawn from such studies in the areas of microvascular permeability, inflammation, mechanotransdu

85 molecular basis of focal adhesion-dependent microvascular permeability is currently under investigat

86 Understanding the regulation of microvascular permeability is essential for the identifi

87 Microvascular permeability is mediated by (i) the caveol

88 etic macular edema, resulting from increased microvascular permeability, is the most prevalent cause

89 sequestration (approximately 16-fold), lung microvascular permeability K(f,c) (approximately 5.7-fol

90 tional model yielded MR imaging estimates of microvascular permeability (K(PS)) and fractional plasma

91 ies, and because VPF/VEGF promotes increased microvascular permeability leading to activation of the

92 d VPF/VEGF(164) induced an early increase in microvascular permeability, leading within 24 hours to e

93 fference between the two study groups in the microvascular permeability of burned tissue.

94 est this hypothesis directly, we studied the microvascular permeability of Cav-1 null mice using a va

95 ation levels, local oxygen concentration and microvascular permeability of OX63 can be simultaneously

96 An effect on albumin microvascular permeability of the synthetic colloids dex

97 F/VEGF and did not affect other mediators of microvascular permeability or endothelial-cell prolifera

98 l) (EPA+GLA), and antioxidants improves lung microvascular permeability, oxygenation, and cardiopulmo

99 l nitric oxide synthase (eNOS) regulation of microvascular permeability remain unresolved.

100 lycocalyx to one critical vascular function, microvascular permeability, remains unclear.

101 nthase (iNOS) plays a role in the changes in microvascular permeability seen with this injury.

102 enes contract airway smooth muscle, increase microvascular permeability, stimulate mucus secretion, d

103 gene expression and VPF/VEGF enhancement of microvascular permeability, suggesting that these distin

104 al ischemia/reperfusion-mediated increase in microvascular permeability than those treated with CMV o

105 Dynamic MR imaging revealed microvascular permeability to a high-molecular-weight co

106 However, red blood cell velocities and microvascular permeability to albumin were higher in the

107 ar diameters, red blood cell velocities, and microvascular permeability to albumin.

108 al surface layer in parallel with defects in microvascular permeability to both water and albumin, in

109 helial glycocalyx are critical regulators of microvascular permeability to both water and albumin.

110 th U and the previously described changes in microvascular permeability to K(+) with U, suggest that

111 ivo role of RhoGDI-1 in regulating pulmonary microvascular permeability using RhoGDI-1(-/-) mice.

112 ts injected locally with agents that enhance microvascular permeability: vascular permeability factor

113 h, serum alanine aminotransferase, and liver microvascular permeability vs. KIM6+-infected animals (p

114 f the lung was determined by flow cytometry, microvascular permeability was assessed by the extravasa

115 The sepsis-induced increase in peripheral microvascular permeability was associated with significa

116 kappaB) in TNF-induced increases in cerebral microvascular permeability was evaluated both in vitro,

117 ient (Kf), a sensitive measure of changes in microvascular permeability, was determined.

118 ation of neutrophils and increased pulmonary microvascular permeability, was reduced in NK1R-/- anima

119 Changes in pulmonary microvascular permeability were significantly suppressed