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1 developed hepatic steatosis characterized by microvesicular and macrovesicular lipid accumulation and
3 rtal mononuclear infiltrates, hepatocellular microvesicular changes, cytoplasmic lipid droplets, and
4 ne COS7 and found that a cellugyrin-positive microvesicular compartment was present in all cell types
6 g almost exclusively in the third trimester; microvesicular fatty infiltration of hepatocytes causes
7 isomal beta-oxidation, development of severe microvesicular fatty liver, peroxisome assembly, cell de
9 s, including reduced liver weight, transient microvesicular fatty metamorphosis, prolonged extramedul
11 NI1/hSNF5 is completely absent from purified microvesicular fractions, it is specifically incorporate
12 a short-term treatment (5 days) that induces microvesicular hepatic steatosis and marked hypercholest
13 We found that VLCAD-deficient hearts have microvesicular lipid accumulation, marked mitochondrial
15 mal beta-oxidation system, exhibit extensive microvesicular steatohepatitis, leading to hepatocellula
17 al liver histology consisting of significant microvesicular steatosis and fatty Kupffer cells but no
18 CV, there was an inverse correlation between microvesicular steatosis and level of autophagy (r = -0.
19 nodeficiency virus infection, which includes microvesicular steatosis and more severe hepatic injury
20 cholestasis and cirrhosis in the former and microvesicular steatosis and oncocytic transformation (m
21 tical information including the detection of microvesicular steatosis and quantitation of liver lipid
23 at-free diet and converted macrovesicular to microvesicular steatosis in B6.V-Lep(ob) obese mice as d
24 id screening of mitochondrial toxins-induced microvesicular steatosis in primary hepatocyte cultures.
29 acts/delays rapid progression of the hepatic microvesicular steatosis to the characteristic macrovesi
31 ectron-microscopic examination showed severe microvesicular steatosis with severe mitochondrial injur
32 reated bcl-2 (-/+) mice displayed only early microvesicular steatosis without progression to extensiv
33 this virus developed diffuse hepatocellular microvesicular steatosis, an abnormal accumulation of in
34 higher degrees of necrosis, fibrosis stage, microvesicular steatosis, and ductular reaction among ot
35 rmal liver histology with macrovesicular and microvesicular steatosis, fatty Kupffer cells, extensive
36 itis with changes in hepatic tissues such as microvesicular steatosis, likely caused by an increase i
49 mers in microvesicles/exosomes and show that microvesicular TDP-43 is preferentially taken up by reci
50 n of tubular injury in all biopsy specimens: microvesicular tubular epithelial cytoplasmatic vacuoliz
51 ique, we provide evidence for preferentially microvesicular uptake as well as both soma-to-soma "hori
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