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1 d at the nuclear periphery shortly after the midblastula transition.
2 ately 150 genes are transcribed prior to the midblastula transition.
3 widespread pausing occurs de novo during the midblastula transition.
4 d RNA polymerase II C-terminal domain at the midblastula transition.
5 d myosin cages continued to divide until the midblastula transition.
6 arly embryogenesis, and then falls after the midblastula transition.
7 ic ribonucleoprotein (RNP) bodies during the midblastula transition.
8 L dynamically regulate RNA metabolism at the midblastula transition.
9 endent G(2) cell cycle arrest just after the midblastula transition.
10 to zygotic genetic control occurs called the midblastula transition.
11 mbryo, Xcdc6A becomes undetectable following midblastula transition.
12 but arrest in G2 phase immediately after the midblastula transition.
13 it is anchored in the cytoplasm prior to the midblastula transition.
14 nt in embryos exposed to gamma-IR before the midblastula transition.
15 h cycloheximide (100 microg/ml) prior to the midblastula transition.
16 This developmental change is termed the midblastula transition.
17 branes at different time points prior to the midblastula transition.
18 edly, in the rapid cell cycles preceding the midblastula transition, a defined timing program was pre
19 affects the onset of cellularization at the midblastula transition and found that nuclear cell-cycle
20 hancers are de-methylated shortly before the midblastula transition and reside in a unique epigenetic
22 on starts 6 hours after fertilization at the midblastula transition and therefore the first steps in
23 interacts through cleavage stages until the midblastula transition, and a second protein binds from
24 hout the animal hemisphere shortly after the midblastula transition, and becomes restricted to prospe
26 tivate Smad2 phosphorylation until after the midblastula transition, and the onset of responsiveness
27 microg/ml) delayed development prior to the midblastula transition but resumed DNA synthesis, initia
28 absent during cycles 2-12, reappeared at the midblastula transition coincident with the disappearance
29 nt, zygotic gene expression initiated at the midblastula transition converts maternal information on
30 tions that cause developmental arrest at the midblastula transition, defects in cell viability, embry
31 ted with the same dose of gamma-IR after the midblastula transition developed normally and exhibited
33 e cortical regions of blastomeres; after the midblastula transition exogenous plakoglobin accumulates
35 n of a homeobox gene, vega1, is activated at midblastula transition in all blastomeres, but is down-r
36 odeling of the cell cycle that occurs at the midblastula transition in early Xenopus laevis embryos.
38 upport a model for cell-cycle control at the midblastula transition in which titration of a maternal
40 t of transcriptional quiescence prior to the midblastula transition in Xenopus, dorsal specification
41 in genes controlling processes prior to the midblastula transition, including egg development, blast
42 Additionally, cyclin E1 is degraded at the midblastula transition independently of protein synthesi
43 s of gamma-irradiation (gamma-IR) before the midblastula transition induced apoptotic cell death and
44 xes accumulate in the nucleus only after the midblastula transition, irrespective of the stage at whi
45 h to zygotic control of embryogenesis at the midblastula transition is accompanied by significant inc
47 ggest that a developmental checkpoint at the midblastula transition is maternally regulated and can t
48 l and biochemical changes that accompany the midblastula transition, lead to a continuation of the ma
52 to zygotic regulation of development at the midblastula transition (MBT) follows mitosis 13, when th
55 a division cycles and fail to undergo timely midblastula transition (MBT) or arrest following ionizin
56 many genes whose onset of expression at the midblastula transition (MBT) requires Smicl and is corre
57 in eggs, and after fertilization, until the midblastula transition (MBT) when levels of cyclin E1 pr
60 phase and accumulates continuously until the midblastula transition (MBT), after which it is degraded
61 avage phase of development terminates at the midblastula transition (MBT), at which point the cell cy
62 anscription of beta-catenin starts after the midblastula transition (MBT), but does not rescue dorsal
65 of many metazoan embryos are followed by the midblastula transition (MBT), during which the cell cycl
66 ion of ten DNA replication factors after the midblastula transition (MBT), including a marked decline
67 ome in many organisms is quiescent until the midblastula transition (MBT), when large-scale transcrip
69 first major developmental transition is the midblastula transition (MBT), when zygotic transcription
85 moothing of the enveloping cell layer at the midblastula transition occurred normally and expression
86 eus to activate the transcription, after the midblastula transition, of target genes such as Xbra and
88 se that VegT activation of Sox17alpha at the midblastula transition prevents mesodermal gene expressi
89 aster embryos approach cell cycle 14 and the midblastula transition, rapid embryonic cell cycles slow
90 alize Xenopus embryos if expressed after the midblastula transition, strengthening the idea that zygo
92 signaling is thought to function first after midblastula transition to regulate axial patterning via
93 r, the onset of Sox17alpha expression at the midblastula transition was dependent on VegT, but not on
94 c accumulation of zygotic transcripts at the midblastula transition, we observe the formation of gian
95 cription begins in many organisms during the midblastula transition when the cell cycle of the dividi
96 n the blastocoel, suggesting that before the midblastula transition Xenopus embryos use apoptosis to
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