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3 e period (1985-1988) of a prospective study, midcycle and luteal-phase estrogens and progestins were
4 nt's cycle and pooled to create follicular-, midcycle-, and luteal-phase samples, respectively, for a
7 activity in amygdala, OFC, and aCING during midcycle but not in early follicular, suggesting less co
8 ezing was higher on cycle Days 10-22, with a midcycle dip near the time of putative ovulation (approx
10 proposed as a global mechanism to upregulate midcycle genes, but the effects on early genes are not k
16 ma cells of preovulatory follicles after the midcycle gonadotropin surge, suggesting important local
22 ences in any of the hormones measured either midcycle or during the luteal phase, despite good statis
24 ponsiveness to supercoiling shown by the two midcycle promoters and the increased level of negative s
26 found during early follicular compared with midcycle timing in central amygdala, paraventricular and
27 al cycle phase compared with late follicular/midcycle, using negative valence/high arousal versus neu
28 increased chlamydial supercoiling levels in midcycle via their supercoiling-responsive promoters in
29 in young women around the time of ovulation (midcycle) were significantly higher than those measured
30 algorithms based on cycle length, such as a midcycle window or a window determined by assuming a fix
31 l plasmid was most negatively supercoiled at midcycle, with an approximate superhelical density of -0
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