ライフサイエンスコーパス: middle cerebral artery

1 ociated with changes in flow velocity in the middle cerebral artery.

2 n adult Sprague Dawley rats by occluding the middle cerebral artery.

3 underwent 90 min transient occlusions of the middle cerebral artery.

4 ombosis in the murine carotid artery and the middle cerebral artery.

5 s in layer 2/3 located just posterior to the middle cerebral artery.

6 ops span the entire territory sourced by the middle cerebral artery.

7 0, 30, 60 or 90min filament occlusion of the middle cerebral artery.

8 sion at any level of the internal carotid or middle cerebral artery.

9 tonin-evoked constriction of pressurized rat middle cerebral arteries.

10 rial lesions were frequently detected in the middle cerebral artery (23%), internal carotid artery (1

11 ral artery (39 vs 109 cm3; P = .004), and M2 middle cerebral artery (33 vs 59 cm3; P = .04) occlusion

12 Most aneurysms were in the middle cerebral arteries (36%) and the internal carotid

13 rtery terminus (75 vs 190 cm3; P < .001), M1 middle cerebral artery (39 vs 109 cm3; P = .004), and M2

14 set of vasospasm was 4 days (+/- 2 d) in the middle cerebral arteries and 5 days (+/- 2.5 d) in the b

15 Changes of middle cerebral arteries and basilar arteries were extre

16 nts had occlusion of the internal carotid or middle cerebral artery and evidence of salvageable brain

17 iation between neurovascular coupling in the middle cerebral artery and gait speed in elderly individ

18 hed infarcts in the anterior cerebral artery-middle cerebral artery and middle cerebral artery-poster

19 rminal internal carotid artery [ICA] with M1 middle cerebral artery and/or A1 anterior cerebral arter

20 d spectroscopy, blood flow velocities of the middle cerebral artery, and cardiac output at baseline,

21 two 1 h TCD recordings from the ipsilateral middle cerebral artery at baseline and one 1 h recording

22 using near-infrared spectroscopy along with middle cerebral artery blood flow measured using transcr

23 In the middle cerebral artery blood flow velocities and vasomot

24 Beat-by-beat measurements of middle cerebral artery blood flow velocity (MCAv; transc

25 Mean middle cerebral artery blood velocity (MCA V(mean)), mea

26 al, prefrontal cortex tissue oxygenation and middle cerebral artery blood velocity (MCAV) were assess

27 ding cortical areas of hyperintensity in the middle cerebral artery borderzone regions-was classified

28 ovascular coupling was assessed in bilateral middle cerebral arteries by measuring cerebral blood flo

29 s of superficial temporal artery branch to a middle cerebral artery cortical branch for the surgical

30 Patients with significant carotid or middle-cerebral artery disease or impaired vasoreactivit

31 s induced by transient (1h) occlusion of the middle cerebral artery, during which mean arterial blood

32 Bilateral transcranial middle cerebral artery flow velocities using Doppler and

33 between cerebral perfusion pressure and mean middle cerebral artery flow velocity (intracranial press

34 al Doppler ultrasonography to calculate mean middle cerebral artery flow velocity after traumatic bra

35 velocity but four patients (9%) had low mean middle cerebral artery flow velocity and five children (

36 Most children (n = 33; 79%) had normal mean middle cerebral artery flow velocity but four patients (

37 locity and five children (12%) had high mean middle cerebral artery flow velocity despite cerebral pe

38 wer hematocrit was associated with high mean middle cerebral artery flow velocity.

39 usion pressure and low, high, or normal mean middle cerebral artery flow velocity.

40 and 1100 msec to consecutive segments of the middle cerebral artery from distal to the circle of Will

41 that the raw recordings from left and right middle cerebral arteries had higher content of mutual in

42 ces of ET-1-mediated vasoconstriction of the middle cerebral artery in a rat model.

43 The relatively low mean flow velocity in the middle cerebral artery in combination with normal jugula

44 The angio-CT revealed an aneurysm of the middle cerebral artery, in its distal branch.

45 d ADC maps obtained less than 12 hours after middle cerebral artery infarct.

46 ntal and parietal cortex is common following middle cerebral artery infarction, leading to upper extr

47 n, 70 years; range, 61 to 82) with malignant middle-cerebral-artery infarction to either conservative

48 ts 61 years of age or older with a malignant middle-cerebral-artery infarction.

49 er with complete or subtotal space-occupying middle-cerebral-artery infarction.

50 Survival after malignant middle cerebral artery infarcts is dismal.

51 Consecutive patients with M1 segment middle cerebral artery +/- intracranial internal carotid

52 t of middle cerebral artery vs M2 segment of middle cerebral artery), intravenous alteplase (yes vs n

53 10 patients, 18 to 80 years old, with acute middle cerebral artery ischemic stroke.

54 al and stroke volume after femoral artery or middle cerebral artery ligation, respectively.

55 (intracranial internal carotid artery and/or middle cerebral artery M1 and/or M2) on computed tomogra

56 h after stroke induction by occlusion of the middle cerebral artery markedly reduced infarct size, an

57 .3 hyperpolarization was investigated in rat middle cerebral arteries (MCA).

58 ain parenchyma, hydrocephalus, and so-called middle cerebral artery (MCA) "pseudofeeders" were correl

59 t mice was induced by ligations of the right middle cerebral artery (MCA) and both common carotid art

60 The risk of seizures after malignant middle cerebral artery (MCA) infarction with decompressi

61 stoperatively, by recording 1-hour bilateral middle cerebral artery (MCA) microembolic signals (MES).

62 ars) with acute ischemic stroke (AIS) due to middle cerebral artery (MCA) occlusion were enrolled; 75

63 generated occlusive VWF-rich thrombi in the middle cerebral artery (MCA) of mice.

64 in patients with internal carotid artery or middle cerebral artery (MCA) stroke and to evaluate the

65 am, n = 10), rats were sacrificed for either middle cerebral artery (MCA) structure and function asse

66 of the GPIIb/IIIa antagonist tirofiban, in a middle cerebral artery (MCA) thrombosis model in guinea

67 e blood clots or macroscopic emboli into the middle cerebral artery (MCA) via an indwelling carotid a

68 Occlusion of the middle cerebral artery (MCA) with an endovascular filame

69 were subjected to permanent occlusion of the middle cerebral artery (MCA).

70 ertebral artery (VA) and CBF velocity at the middle cerebral artery (MCA).

71 nduced following photochemical injury to the middle cerebral artery (MCA).

72 arterial transcranial Doppler (aTCD) on the middle cerebral artery (MCA): MCA pulsatility index (PIa

73 l carotid artery (ICA), basilar artery (BA), middle cerebral artery (MCA)], the submandibular gland (

74 lower in Eln(+/-) than Eln(+/+) mice in the middle cerebral artery (MCA, P < 0.001), but was similar

75 tile range, 5-17]), of the M1 segment of the middle cerebral artery (MCA; 52 patients: median NIHSS s

76 re were compared in isolated and pressurized middle cerebral arteries (MCAs) and PAs taken from the s

77 +/- 5 mm Hg vs. 41 +/- 2 mm Hg; p < .05) and middle cerebral artery mean flow velocity (37 +/- 9 cm.s

78 < .05), and higher mean arterial pressure-to-middle cerebral artery mean flow velocity phase differen

79 ation, endotoxemia was associated with lower middle cerebral artery mean flow velocity variability (1

80 Systemic hemodynamics, middle cerebral artery mean flow velocity, and dynamic c

81 We included 67 patients with malignant middle cerebral artery [MMCA] stroke who underwent decom

82 with both vehicle-treated and BQ788-treated middle cerebral artery occluded groups.

83 to be useful in infarct volume reduction in middle cerebral artery occluded rat brain.

84 Vehicle-treated middle cerebral artery occluded rats demonstrated high l

85 urs was calculated in patients with proximal middle cerebral artery occlusion (derivation cohort) wit

86 h, 1, 3, 5 and 7 days after permanent distal middle cerebral artery occlusion (dMCAO) in mice compare

87 ult male rats received a 90-min right distal middle cerebral artery occlusion (dMCAo).

88 or were initiated at 48 h after mouse distal middle cerebral artery occlusion (dMCAO).

89 and wild-type mice were subjected to 1 h of middle cerebral artery occlusion (MCAO) and 24-72 h of r

90 HDAC6 inhibitor, in a rat model of transient middle cerebral artery occlusion (MCAO) and an in vitro

91 equent severe ischemic injury, as induced by middle cerebral artery occlusion (MCAo) and if this prot

92 eeks, the animals were subjected to a 2-hour middle cerebral artery occlusion (MCAO) and sacrificed a

93 nimal model encompasses a combination of the middle cerebral artery occlusion (MCAO) and spatial rest

94 dels of acute brain injury: stroke caused by middle cerebral artery occlusion (MCAO) and traumatic br

95 nopause (reproductive senescence) shows that middle cerebral artery occlusion (MCAo) causes a larger

96 al model of permanent and transient (45 min) middle cerebral artery occlusion (MCAO) during the hyper

97 homing to the ischemic site after transient middle cerebral artery occlusion (MCAO) followed by repe

98 ic (non-Tg) littermates underwent reversible middle cerebral artery occlusion (MCAO) for 1 hour follo

99 present study, the model of reversible right middle cerebral artery occlusion (MCAO) for 2 h was used

100 Adult male C57BL/6 mice were subjected to middle cerebral artery occlusion (MCAO) for stroke induc

101 BF data for the 30-min, 60-min and permanent middle cerebral artery occlusion (MCAO) group, respectiv

102 We also tested H(2) in middle cerebral artery occlusion (MCAO) in adult rats (M

103 Following transient middle cerebral artery occlusion (MCAO) in adult rats, e

104 in ischemia and 2) infarct volumes 24h after Middle Cerebral Artery Occlusion (MCAO) in all 3 types o

105 We evaluated various outcomes following middle cerebral artery occlusion (MCAO) in ovariectomise

106 In addition, the functional recovery to middle cerebral artery occlusion (MCAO) in rats and hMCT

107 and improved behavioral outcome after distal middle cerebral artery occlusion (MCAO) in rats.

108 chemia, followed by continuous decline after middle cerebral artery occlusion (MCAO) in the mouse bra

109 Ischemia induced either by embolic middle cerebral artery occlusion (MCAO) in vivo or by ox

110 Experimental middle cerebral artery occlusion (MCAO) increases tPA ac

111 Middle cerebral artery occlusion (MCAO) is a popular mod

112 and improve sensorimotor functions in a rat middle cerebral artery occlusion (MCAO) model after a si

113 V injection were investigated in a permanent middle cerebral artery occlusion (MCAO) model in the adu

114 he vehicle-treated groups in a 12h permanent middle cerebral artery occlusion (MCAO) model of focal i

115 a single intravenous (IV) injection in a rat middle cerebral artery occlusion (MCAO) model of ischemi

116 itis (EAE) model of multiple sclerosis and a middle cerebral artery occlusion (MCAO) model of stroke,

117 Using an in vivo middle cerebral artery occlusion (MCAO) model only the 5

118 ed brain injury in mouse photothrombotic and middle cerebral artery occlusion (MCAo) models.

119 ral ischemia was induced by permanent distal middle cerebral artery occlusion (MCAO) on day 14 of veh

120 Male rats were subjected to a two-hour middle cerebral artery occlusion (MCAO) procedure.

121 ale Sprague-Dawley rats undergoing permanent middle cerebral artery occlusion (MCAO) received three i

122 this study, rats were sacrificed 24 h after middle cerebral artery occlusion (MCAO) stroke and gene

123 rating immune cell populations in mice after middle cerebral artery occlusion (MCAO) strongly implica

124 Infarct sizes 72 h after 60 min middle cerebral artery occlusion (MCAo) were on average

125 PGRN(+/-) and PGRN(-/-) mice underwent middle cerebral artery occlusion (MCAO) with monitoring

126 cit despite smaller infarcts after transient middle cerebral artery occlusion (MCAO) with the suture

127 In a mouse model of middle cerebral artery occlusion (MCAO), p38 MAPK activa

128 ischemia induced by permanent and transient middle cerebral artery occlusion (MCAO), we observed an

129 Rats in the model groups underwent middle cerebral artery occlusion (MCAO).

130 ischemia was induced by a transient (90 min) middle cerebral artery occlusion (MCAO).

131 n to rats with experimental stroke caused by middle cerebral artery occlusion (MCAO).

132 fusion proteins, following a 1-h reversible middle cerebral artery occlusion (MCAO).

133 type (WT) mice were subjected to a transient middle cerebral artery occlusion (MCAO).

134 (WT) littermate were subjected to 45 min of middle cerebral artery occlusion (MCAO).

135 subjected to permanent, 60-min and 30-min of middle cerebral artery occlusion (MCAO).

136 llowing a focal stroke caused by a transient middle cerebral artery occlusion (MCAO).

137 ebral ischemia produced in rats by permanent middle cerebral artery occlusion (MCAO).

138 se model of focal ischemic stroke induced by middle cerebral artery occlusion (MCAO).

139 ts of rats infused with HUCB cells 48h after middle cerebral artery occlusion (MCAO).

140 ularly with ATX or vehicle prior to a 60-min middle cerebral artery occlusion (MCAo).

141 5 minutes) was induced by intraluminal right middle cerebral artery occlusion (MCAO).

142 Male 129/SV mice were subjected to 30-min middle cerebral artery occlusion (MCAo)/reperfusion and

143 within 1 and in most cases 2 h of permanent middle cerebral artery occlusion (pMCAO), mild sensory s

144 e and excitatory neurotoxicity in reversible middle cerebral artery occlusion (rMCAO) model in vivo.

145 erebral infarction using a stroke model with middle cerebral artery occlusion (see figure).

146 further evaluated in vivo using a transient middle cerebral artery occlusion (t-MCAO) model of strok

147 Furthermore, by transient middle cerebral artery occlusion (tMCAO) in rats, the tr

148 A transient middle cerebral artery occlusion (tMCAO) model was used

149 e in chronic diaschisis by using a transient middle cerebral artery occlusion (tMCAO) rat model.

150 nt arterial thrombosis models: the transient middle cerebral artery occlusion (tMCAO) stroke model an

151 c brain injury in a mouse model of transient middle cerebral artery occlusion (tMCAO).

152 ng, and corresponding controls, to transient middle cerebral artery occlusion (tMCAO).

153 r CR2-fH have improved outcomes after 60-min middle cerebral artery occlusion and 24-h reperfusion.

154 We used rodent models of middle cerebral artery occlusion and cell-culture models

155 mice were subjected to 60 min of reversible middle cerebral artery occlusion and evaluated for infar

156 a mouse model of focal cerebral ischemia by middle cerebral artery occlusion and reperfusion (I/R) i

157 In vivo, transient middle cerebral artery occlusion and reperfusion in kina

158 Ab-deficient Rag1(-/-) mice after 60 min of middle cerebral artery occlusion and reperfusion.

159 control or energy-restricted diets prior to middle cerebral artery occlusion and reperfusion.

160 l deficits and poststroke inflammation after middle cerebral artery occlusion by preventing microglia

161 ke Scale scores at those times, and proximal middle cerebral artery occlusion demonstrated prior to t

162 -) mice of either sex subjected to transient middle cerebral artery occlusion developed dramatically

163 mice of either sex challenged with transient middle cerebral artery occlusion developed significantly

164 nfarct volume of mice subjected to transient middle cerebral artery occlusion even up to 3 to 5 hours

165 Male Swiss Webster mice underwent middle cerebral artery occlusion for 1 h followed by rep

166 We performed right middle cerebral artery occlusion for 3 hours, administer

167 Mice were subjected to middle cerebral artery occlusion for 40 min, followed by

168 ent cerebral ischemia was induced in mice by middle cerebral artery occlusion for 60 minutes and s-NS

169 and endothelin receptors following permanent middle cerebral artery occlusion for 7 days.

170 Male Sprague-Dawley rats were subjected to middle cerebral artery occlusion for 70 min followed by

171 Animals were subjected to transient middle cerebral artery occlusion for 90 mins.

172 To define the role of AhR in stroke, we used middle cerebral artery occlusion in mice and oxygen-gluc

173 creased the severity of stroke in a model of middle cerebral artery occlusion in mice.

174 g-term spontaneous functional recovery after middle cerebral artery occlusion in mice.

175 c injury, and cerebral edema formation after middle cerebral artery occlusion in mice.

176 ome-encapsulated clodronate before transient middle cerebral artery occlusion in postnatal day 7 rats

177 ic penumbra when administered six hours post middle cerebral artery occlusion in rats.

178 ting the ET(B) receptors following permanent middle cerebral artery occlusion in rats.

179 t volume, and clinical outcome in a model of middle cerebral artery occlusion in rats.

180 onset and until completion of 15 min distal middle cerebral artery occlusion in spontaneously hypert

181 Middle cerebral artery occlusion in the rat brain result

182 After transient middle cerebral artery occlusion in the rat brain, eleva

183 human umbilical cord blood cells 48 h after middle cerebral artery occlusion increased Akt phosphory

184 Infarct volume following middle cerebral artery occlusion is dramatically diminis

185 eased aggression; (3) larger infarcts in the middle cerebral artery occlusion ischemic stroke model;

186 ental cerebral ischemia induced by transient middle cerebral artery occlusion it selectively dilates

187 stasis in the ischemic brain after transient middle cerebral artery occlusion leading to increased in

188 n protein were investigated with a permanent middle cerebral artery occlusion model in the rat.

189 A transient middle cerebral artery occlusion model was used to inves

190 In the middle cerebral artery occlusion model, the volume and f

191 diated cerebrovascular protection in a mouse middle cerebral artery occlusion model.

192 in male C57BL/6 mice using the intraluminal middle cerebral artery occlusion model.

193 We then used the murine suture and embolic middle cerebral artery occlusion models of stroke to inv

194 troke in females, we evaluated the effect of middle cerebral artery occlusion on infarct size and per

195 e-Dawley rats (12 months old) with permanent middle cerebral artery occlusion or sham operations on m

196 tion of TAT-C1aB in mice following transient middle cerebral artery occlusion significantly reduced i

197 In the model of transient middle cerebral artery occlusion stroke Gna(i2)(fl/fl)/P

198 assessment of blood flow anomaly in a mouse middle cerebral artery occlusion stroke model.

199 in rats implanted with CTX-DP 4 weeks after middle cerebral artery occlusion stroke prompted investi

200 We investigated in a murine model of middle cerebral artery occlusion the effect of blocking

201 Following middle cerebral artery occlusion to induce stroke in mic

202 hemisphere reperfusion after photothrombolic middle cerebral artery occlusion was increased in Klkb1(

203 tion volumes following a 60-minute transient middle cerebral artery occlusion were determined in adul

204 Rats subjected to 2h of transient middle cerebral artery occlusion were studied temporally

205 icits following experimental stroke, using a middle cerebral artery occlusion with reperfusion model.

206 s induced in mice (by permanent or transient middle cerebral artery occlusion) and rats (by 3-vessel

207 rotid artery occlusion and 360 with isolated middle cerebral artery occlusion).

208 of amyotrophic lateral sclerosis (SOD1G93A), middle cerebral artery occlusion, and multiple mini-stro

209 Following transient middle cerebral artery occlusion, ck2beta(-/-) mice disp

210 ith ischemic stroke and in mice subjected to middle cerebral artery occlusion, natural killer (NK) ce

211 g photothrombotic cortical injury, transient middle cerebral artery occlusion, or neonatal hypoxic-is

212 Following transient middle cerebral artery occlusion, progesterone was admin

213 rotects against cerebral ischemia induced by middle cerebral artery occlusion, reducing percent hemis

214 In a mouse model of thrombin-induced middle cerebral artery occlusion, the efficacy of the di

215 marrow (BM) chimeras subjected to transient middle cerebral artery occlusion, we found that CD36(-/-

216 Following middle cerebral artery occlusion, we observed a rapid in

217 However, using a mouse model of transient middle cerebral artery occlusion, we observed that cereb

218 Using two distinct models of acute middle cerebral artery occlusion, we show by next-genera

219 pharmacologically induced excitotoxicity and middle cerebral artery occlusion-induced brain damage.

220 vivo against NMDA-induced excitotoxicity and middle cerebral artery occlusion-induced stroke in mice.

221 cerebral vascular endothelial cell death and middle cerebral artery occlusion-triggered cerebrovascul

222 t mediates AhR activation in the brain after middle cerebral artery occlusion.

223 (M/M) littermates received sham or transient middle cerebral artery occlusion.

224 Cerebral ischemia was induced by middle cerebral artery occlusion.

225 d at 4 weeks into the treatment by transient middle cerebral artery occlusion.

226 e penumbra using a mouse model of reversible middle cerebral artery occlusion.

227 e matter bundles from degeneration following middle cerebral artery occlusion.

228 o mouse brains 6 h or 7 days after transient middle cerebral artery occlusion.

229 ntly in neonatal and adult rats by transient middle cerebral artery occlusion.

230 infarct cavity for 7 d, beginning 7 d after middle cerebral artery occlusion.

231 splanted into rat brains 6 h after transient middle cerebral artery occlusion.

232 r regional infarcts such as those induced by middle cerebral artery occlusion.

233 ver a large peri-infarct cortex region after middle cerebral artery occlusion.

234 to ischemic brain damage caused by permanent middle cerebral artery occlusion.

235 ntravenously or intra-arterially after right middle cerebral artery occlusion.

236 i-infarct cortex before and after unilateral middle cerebral artery occlusion.

237 hromosome 11 for collateral remodeling after middle cerebral artery occlusion.

238 entricularly starting 3 days after 30 min of middle cerebral artery occlusion.

239 l outcome in aged rats following a transient middle cerebral artery occlusion.

240 n damage and oxidative stress in response to middle cerebral artery occlusion.

241 rebral ischemia was induced by 30 minutes of middle cerebral artery occlusion.

242 sponses in the brains of 2D2 mice 14 d after middle cerebral artery occlusion.

243 ow and 85% reduction of infarct volume after middle cerebral artery occlusion; 54% rescue of low skel

244 stroke groups: 30-min, 60-min, and permanent middle cerebral-artery occlusion (n=12 rats for each gro

245 star rats were subjected to right hemisphere middle-cerebral artery occlusion and reperfusion.

246 ive patients with internal carotid artery or middle cerebral artery occlusions transferred over an 11

247 acranial carotid, basilar, and M1 segment of middle cerebral artery occlusions).

248 ly constant with a mean flow velocity in the middle cerebral artery of 71.5 (56.0-78.5) at 108 hrs (p

249 Under anaesthesia, the middle cerebral artery of adult rats was occluded for 60

250 ebral arteries, and (3) ipsilateral proximal middle cerebral artery of the circle of Willis.

251 areas of the internal carotid, basilar, and middle cerebral arteries on the first day at high altitu

252 cerebral artery sign is an appearance of the middle cerebral artery on non-contrast-enhanced computed

253 artery occlusion (internal carotid artery or middle cerebral artery) on outcomes.

254 between 2013 and 2014 for occlusions in the middle cerebral artery or carotid terminus by using a st

255 ve cohort study, we studied 72 patients with middle cerebral artery or terminal internal carotid arte

256 viscosity and the mean flow velocity in the middle cerebral artery (p = 0.0008).

257 ebral artery, anterior communicating artery, middle cerebral artery, persistent stapedial artery and

258 r cerebral artery-middle cerebral artery and middle cerebral artery-posterior cerebral artery watersh

259 no differences in mean flow velocity in the middle cerebral artery, pulsatility index, and jugular b

260 pon rewarming, the mean flow velocity in the middle cerebral artery remained relatively constant with

261 ) was undertaken using RNA isolated from rat middle cerebral arteries (RMCAs) and immunocytochemistry

262 g localized the clot in the internal carotid/middle cerebral artery segment of all rats.

263 Coexistence of hyperdense middle cerebral artery sign and pulmonary thromboembolis

264 r-old female patient who had both hyperdense middle cerebral artery sign and pulmonary thromboembolis

265 Hyperdense middle cerebral artery sign is an appearance of the midd

266 e at 22 to 36 hours of a baseline hyperdense middle cerebral artery sign was increased (63% [124 of 1

267 ood pressures and systolic velocities of the middle cerebral artery significantly decreased (p < 0.01

268 ital cortex of a subject who suffered a left middle cerebral artery stroke early in development.

269 onclusion In this study, patients with acute middle cerebral artery stroke with absence of cortical v

270 1 can prevent, as well as ameliorate, murine middle cerebral artery stroke, without thrombocytopenia

271 malignant infarction in the territory of the middle cerebral artery, TAT.ARC salvages brain tissue wh

272 children had low or uninterpretable baseline middle cerebral artery TCD velocities, which were associ

273 fected white matter tracts, and involved the middle cerebral artery territory for 112 patients (73%).

274 rast computed tomography (ie, over one-third middle cerebral artery territory hypodensity).

275 Three weeks after right middle cerebral artery territory injury, rats treated wi

276 urrounding a penumbra-like "area at risk" in middle cerebral artery territory of mouse somatosensory

277 33 subjects with chronic infarctions in the middle cerebral artery territory, and 109 age-matched no

278 odel was generated by occlusion of the right middle cerebral artery, then 90 min later, stroke rats w

279 As compared with aneurysms in the middle cerebral arteries, those in the posterior and ant

280 e cerebral artery was a ratio of flow in the middle cerebral artery to extracranial internal carotid

281 In mice with thrombotic occlusion of the middle cerebral artery, tPA administration increased bra

282 ilateral CBFV recordings from left and right middle cerebral arteries using 20 healthy subjects (10 f

283 al-to-contralateral ratio of the activity in middle cerebral artery-vascularized territories in each

284 The prevalence of middle cerebral artery vasospasm in children with modera

285 In non-injured patients, middle cerebral artery velocities were 18-26% below cont

286 cative of CBF and extra-cranial blood flow), middle cerebral artery velocity (MCA Vmean), arterial-ve

287 sodilatation, blood pressure, heart rate and middle cerebral artery velocity (MCAv) were measured dur

288 on (internal carotid artery vs M1 segment of middle cerebral artery vs M2 segment of middle cerebral

289 cranial Doppler ultrasound monitoring of the middle cerebral arteries was performed whenever possible

290 Mean duration of vasospasm in the middle cerebral artery was 2 days (+/- 2 d) and 1.5 days

291 eria required for vasospasm diagnosis in the middle cerebral artery was a ratio of flow in the middle

292 Mean flow velocity in the middle cerebral artery was low (26.5 (18.7-48.0) cm/sec)

293 Median mean flow velocity in the middle cerebral artery was low (27.0 cm/s [23.8-30.5 cm/

294 Mean flow velocity in the middle cerebral artery was measured by transcranial Dopp

295 bone marrow (BM) chimeric mice in which the middle cerebral artery was occluded and infarct volume w

296 Cerebral emboli entering the middle cerebral arteries were counted at baseline and ev

297 Bilateral middle cerebral arteries were insonated using transcrani

298 We examined the pial network of the middle cerebral artery, which distributes blood from the

299 acranial internal carotid artery or proximal middle cerebral artery who had last been known to be wel

300 ia was induced by temporary occlusion of the middle cerebral artery with a microfilament.