ライフサイエンスコーパス: middle cerebral artery occlusion

1 rotid artery occlusion and 360 with isolated middle cerebral artery occlusion).

2 acranial carotid, basilar, and M1 segment of middle cerebral artery occlusions).

3 ntly in neonatal and adult rats by transient middle cerebral artery occlusion.

4 infarct cavity for 7 d, beginning 7 d after middle cerebral artery occlusion.

5 splanted into rat brains 6 h after transient middle cerebral artery occlusion.

6 r regional infarcts such as those induced by middle cerebral artery occlusion.

7 ver a large peri-infarct cortex region after middle cerebral artery occlusion.

8 to ischemic brain damage caused by permanent middle cerebral artery occlusion.

9 ntravenously or intra-arterially after right middle cerebral artery occlusion.

10 i-infarct cortex before and after unilateral middle cerebral artery occlusion.

11 hromosome 11 for collateral remodeling after middle cerebral artery occlusion.

12 entricularly starting 3 days after 30 min of middle cerebral artery occlusion.

13 l outcome in aged rats following a transient middle cerebral artery occlusion.

14 n damage and oxidative stress in response to middle cerebral artery occlusion.

15 Adult rats were subjected to a 90-minutes middle cerebral artery occlusion.

16 stered to stroke rats from days 6 to 9 after middle cerebral artery occlusion.

17 fective when initiated both before and after middle cerebral artery occlusion.

18 is attenuated in CD36-null mice subjected to middle cerebral artery occlusion.

19 wild-type animals after a 90 min reversible middle cerebral artery occlusion.

20 schemic lesion volume induced by a transient middle cerebral artery occlusion.

21 monitoring and subjected to transient (2 h) middle cerebral artery occlusion.

22 s, and to affect brain damage from transient middle cerebral artery occlusion.

23 levels in the ischemic penumbra region after middle cerebral artery occlusion.

24 in an in vivo rat stroke model of transient middle cerebral artery occlusion.

25 We used the stroke model of temporary middle cerebral artery occlusion.

26 ischemic cortex of rats 7 days after distal middle cerebral artery occlusion.

27 rons rapidly upregulate STC2 after transient middle cerebral artery occlusion.

28 Rats were subjected to embolic middle cerebral artery occlusion.

29 ease in infarction volume was observed after middle cerebral artery occlusion.

30 rebral ischemia was induced by 30 minutes of middle cerebral artery occlusion.

31 Cerebral ischemia was induced by middle cerebral artery occlusion.

32 sponses in the brains of 2D2 mice 14 d after middle cerebral artery occlusion.

33 t mediates AhR activation in the brain after middle cerebral artery occlusion.

34 (M/M) littermates received sham or transient middle cerebral artery occlusion.

35 d at 4 weeks into the treatment by transient middle cerebral artery occlusion.

36 e penumbra using a mouse model of reversible middle cerebral artery occlusion.

37 e matter bundles from degeneration following middle cerebral artery occlusion.

38 o mouse brains 6 h or 7 days after transient middle cerebral artery occlusion.

39 ow and 85% reduction of infarct volume after middle cerebral artery occlusion; 54% rescue of low skel

40 ale littermates were treated with reversible middle cerebral artery occlusion (90 min; 22 hr reperfus

41 a less inflammatory cytokine profile in the middle cerebral artery occlusion-affected right brain he

42 r CR2-fH have improved outcomes after 60-min middle cerebral artery occlusion and 24-h reperfusion.

43 We used rodent models of middle cerebral artery occlusion and cell-culture models

44 0 mg/kg) of Niaspan, starting 24 hours after middle cerebral artery occlusion and daily for 14 days.

45 mice were subjected to 60 min of reversible middle cerebral artery occlusion and evaluated for infar

46 rylation status, is increased following both middle cerebral artery occlusion and oxygen-glucose depr

47 a mouse model of focal cerebral ischemia by middle cerebral artery occlusion and reperfusion (I/R) i

48 In vivo, transient middle cerebral artery occlusion and reperfusion in kina

49 reduction in brain infarct size after acute middle cerebral artery occlusion and reperfusion, strong

50 control or energy-restricted diets prior to middle cerebral artery occlusion and reperfusion.

51 ical deficit, and infarct size at 24 h after middle cerebral artery occlusion and reperfusion.

52 Ab-deficient Rag1(-/-) mice after 60 min of middle cerebral artery occlusion and reperfusion.

53 ) mice were subjected to 90 min of transient middle cerebral artery occlusion and treated with either

54 increased significantly in rats subjected to middle cerebral artery occlusion and treated with no int

55 Adult male rats were subjected to middle cerebral artery occlusion and were treated with o

56 star rats were subjected to right hemisphere middle-cerebral artery occlusion and reperfusion.

57 s induced in mice (by permanent or transient middle cerebral artery occlusion) and rats (by 3-vessel

58 of amyotrophic lateral sclerosis (SOD1G93A), middle cerebral artery occlusion, and multiple mini-stro

59 or 96 h after (late post-ischemia) 60 min of middle cerebral artery occlusion, and were killed 10 day

60 In rats with middle cerebral artery occlusion, both the recovery of m

61 logically monitored rats received 2 hours of middle cerebral artery occlusion by intraluminal suture,

62 the peri-infarct zone from 24 to 72 h after middle cerebral artery occlusion by means of immunohisto

63 l deficits and poststroke inflammation after middle cerebral artery occlusion by preventing microglia

64 (n=94) were subjected to 2 hrs of transient middle cerebral artery occlusion by the intraluminal occ

65 (1.0-1.5%) rats were subjected to permanent middle cerebral artery occlusion by the intraluminal occ

66 Following transient middle cerebral artery occlusion, ck2beta(-/-) mice disp

67 ke Scale scores at those times, and proximal middle cerebral artery occlusion demonstrated prior to t

68 urs was calculated in patients with proximal middle cerebral artery occlusion (derivation cohort) wit

69 -) mice of either sex subjected to transient middle cerebral artery occlusion developed dramatically

70 mice of either sex challenged with transient middle cerebral artery occlusion developed significantly

71 adult male mice underwent unilateral distal middle cerebral artery occlusion (dMCA) occlusion and we

72 h, 1, 3, 5 and 7 days after permanent distal middle cerebral artery occlusion (dMCAO) in mice compare

73 Ischaemia was induced by distal middle cerebral artery occlusion (dMCAO) in normoxic (30

74 ult male rats received a 90-min right distal middle cerebral artery occlusion (dMCAo).

75 or were initiated at 48 h after mouse distal middle cerebral artery occlusion (dMCAO).

76 nfarct volume of mice subjected to transient middle cerebral artery occlusion even up to 3 to 5 hours

77 infarct volumes in mice following transient middle cerebral artery occlusion, even when given 3 hour

78 nd infarct volume in a standardized model of middle cerebral artery occlusion, even when the agent is

79 APN-KO) and wild-type (WT) mice to 1 hour of middle cerebral artery occlusion followed by 23 hours of

80 an in vivo focal ischemic model, 2 h of left middle cerebral artery occlusion followed by 24 h of rep

81 subjected to 90-min ischemia induced by left middle cerebral artery occlusion followed by 72-h reperf

82 intravenously 10 minutes after initiation of middle cerebral artery occlusion followed by reperfusion

83 c rats were subjected to 30 min of transient middle cerebral artery occlusion, followed by 0, 0.5, 3,

84 normal and hyperlipidemic mice to transient middle cerebral artery occlusion, followed by measuremen

85 Male Swiss Webster mice underwent middle cerebral artery occlusion for 1 h followed by rep

86 We performed right middle cerebral artery occlusion for 3 hours, administer

87 Mice were subjected to middle cerebral artery occlusion for 40 min, followed by

88 ent cerebral ischemia was induced in mice by middle cerebral artery occlusion for 60 minutes and s-NS

89 and endothelin receptors following permanent middle cerebral artery occlusion for 7 days.

90 Male Sprague-Dawley rats were subjected to middle cerebral artery occlusion for 70 min followed by

91 al cerebral ischemia was produced in rats by middle cerebral artery occlusion for 90 min.

92 Animals were subjected to transient middle cerebral artery occlusion for 90 mins.

93 We produced focal cerebral ischemia by middle cerebral artery occlusion for 90 minutes in the a

94 A 60 minute middle cerebral artery occlusion in C57 mice resulted in

95 We induced permanent middle cerebral artery occlusion in female mice that pre

96 To define the role of AhR in stroke, we used middle cerebral artery occlusion in mice and oxygen-gluc

97 creased the severity of stroke in a model of middle cerebral artery occlusion in mice.

98 g-term spontaneous functional recovery after middle cerebral artery occlusion in mice.

99 c injury, and cerebral edema formation after middle cerebral artery occlusion in mice.

100 6% following acute focal ischemia induced by middle cerebral artery occlusion in normotensive rats.

101 ome-encapsulated clodronate before transient middle cerebral artery occlusion in postnatal day 7 rats

102 e, we investigated the effects of VEGF after middle cerebral artery occlusion in rats using a series

103 ting the ET(B) receptors following permanent middle cerebral artery occlusion in rats.

104 t volume, and clinical outcome in a model of middle cerebral artery occlusion in rats.

105 -3 in the ischemic brain following transient middle cerebral artery occlusion in rats.

106 n = 7), and 14 (n = 7) after 2 hr unilateral middle cerebral artery occlusion in rats.

107 ic penumbra when administered six hours post middle cerebral artery occlusion in rats.

108 onset and until completion of 15 min distal middle cerebral artery occlusion in spontaneously hypert

109 f mild retinal ischemia induced by temporary middle cerebral artery occlusion in the adult rat.

110 Middle cerebral artery occlusion in the rat brain result

111 After transient middle cerebral artery occlusion in the rat brain, eleva

112 cerebral ischaemia was induced by transient middle cerebral artery occlusion in the rat using the fi

113 We utilized two model systems of ischemia, middle cerebral artery occlusion in vivo and oxygen-gluc

114 human umbilical cord blood cells 48 h after middle cerebral artery occlusion increased Akt phosphory

115 pharmacologically induced excitotoxicity and middle cerebral artery occlusion-induced brain damage.

116 vivo against NMDA-induced excitotoxicity and middle cerebral artery occlusion-induced stroke in mice.

117 Infarct volume following middle cerebral artery occlusion is dramatically diminis

118 eased aggression; (3) larger infarcts in the middle cerebral artery occlusion ischemic stroke model;

119 ental cerebral ischemia induced by transient middle cerebral artery occlusion it selectively dilates

120 stasis in the ischemic brain after transient middle cerebral artery occlusion leading to increased in

121 and wild-type mice were subjected to 1 h of middle cerebral artery occlusion (MCAO) and 24-72 h of r

122 HDAC6 inhibitor, in a rat model of transient middle cerebral artery occlusion (MCAO) and an in vitro

123 l groups consist of rats subjected to 2 h of middle cerebral artery occlusion (MCAo) and at 24 h afte

124 equent severe ischemic injury, as induced by middle cerebral artery occlusion (MCAo) and if this prot

125 Rats (n=57) were subjected to permanent middle cerebral artery occlusion (MCAo) and injected int

126 d-brain barrier (BBB) permeability following middle cerebral artery occlusion (MCAo) and intrastriata

127 ar dysfunction and tissue injury response to middle cerebral artery occlusion (MCAO) and reperfusion.

128 ntraction are attenuated following transient middle cerebral artery occlusion (MCAO) and reperfusion.

129 eeks, the animals were subjected to a 2-hour middle cerebral artery occlusion (MCAO) and sacrificed a

130 nimal model encompasses a combination of the middle cerebral artery occlusion (MCAO) and spatial rest

131 dels of acute brain injury: stroke caused by middle cerebral artery occlusion (MCAO) and traumatic br

132 ve in preventing brain damage from transient middle cerebral artery occlusion (MCAO) as was estrone.

133 ale Wistar rats underwent unilateral embolic middle cerebral artery occlusion (MCAo) by a single fibr

134 nopause (reproductive senescence) shows that middle cerebral artery occlusion (MCAo) causes a larger

135 days of age, rat pups underwent a permanent middle cerebral artery occlusion (MCAo) coupled with a t

136 al model of permanent and transient (45 min) middle cerebral artery occlusion (MCAO) during the hyper

137 homing to the ischemic site after transient middle cerebral artery occlusion (MCAO) followed by repe

138 ic (non-Tg) littermates underwent reversible middle cerebral artery occlusion (MCAO) for 1 hour follo

139 te experiments, rats were subjected to right middle cerebral artery occlusion (MCAO) for 2 h under ke

140 present study, the model of reversible right middle cerebral artery occlusion (MCAO) for 2 h was used

141 Adult male C57BL/6 mice were subjected to middle cerebral artery occlusion (MCAO) for stroke induc

142 BF data for the 30-min, 60-min and permanent middle cerebral artery occlusion (MCAO) group, respectiv

143 We also tested H(2) in middle cerebral artery occlusion (MCAO) in adult rats (M

144 Following transient middle cerebral artery occlusion (MCAO) in adult rats, e

145 cally to the cerebral cortex before a 60-min middle cerebral artery occlusion (MCAo) in adult rats.

146 in ischemia and 2) infarct volumes 24h after Middle Cerebral Artery Occlusion (MCAO) in all 3 types o

147 delayed preconditioning following 90 min of middle cerebral artery occlusion (MCAO) in male Wistar r

148 eroxide dismutase mimetic, against 90 min of middle cerebral artery occlusion (MCAO) in male Wistar r

149 Since we previously demonstrated that middle cerebral artery occlusion (MCAO) in mice induces

150 We evaluated various outcomes following middle cerebral artery occlusion (MCAO) in ovariectomise

151 In addition, the functional recovery to middle cerebral artery occlusion (MCAO) in rats and hMCT

152 en (HBO) reduces cerebral infarct size after middle cerebral artery occlusion (MCAO) in rats through

153 and improved behavioral outcome after distal middle cerebral artery occlusion (MCAO) in rats.

154 s therapeutic effect in a model of permanent middle cerebral artery occlusion (MCAo) in rats.

155 cts in ischemic brain damage after permanent middle cerebral artery occlusion (MCAO) in SHR-SP rats a

156 1/2 expression increased following 30 min of middle cerebral artery occlusion (MCAO) in the mouse bra

157 chemia, followed by continuous decline after middle cerebral artery occlusion (MCAO) in the mouse bra

158 Ischemia induced either by embolic middle cerebral artery occlusion (MCAO) in vivo or by ox

159 Experimental middle cerebral artery occlusion (MCAO) increases tPA ac

160 isoflurane and subjected to 2 h of temporary middle cerebral artery occlusion (MCAo) induced by means

161 We found that middle cerebral artery occlusion (MCAO) induces microgli

162 Middle cerebral artery occlusion (MCAO) is a popular mod

163 n Sprague-Dawley rats using the intraluminal middle cerebral artery occlusion (MCAO) method.

164 and improve sensorimotor functions in a rat middle cerebral artery occlusion (MCAO) model after a si

165 V injection were investigated in a permanent middle cerebral artery occlusion (MCAO) model in the adu

166 We used transient rat middle cerebral artery occlusion (MCAO) model of brain i

167 he vehicle-treated groups in a 12h permanent middle cerebral artery occlusion (MCAO) model of focal i

168 a single intravenous (IV) injection in a rat middle cerebral artery occlusion (MCAO) model of ischemi

169 itis (EAE) model of multiple sclerosis and a middle cerebral artery occlusion (MCAO) model of stroke,

170 Using an in vivo middle cerebral artery occlusion (MCAO) model only the 5

171 we utilized a murine transient endovascular middle cerebral artery occlusion (MCAO) model to examine

172 t (1 h) focal cerebral ischemia in the mouse middle cerebral artery occlusion (MCAO) model.

173 d the impact of gemfibrozil in two permanent middle cerebral artery occlusion (MCAO) models in young

174 ed brain injury in mouse photothrombotic and middle cerebral artery occlusion (MCAo) models.

175 ed GM6 on ischemic stroke was studied in the middle cerebral artery occlusion (MCAo) mouse model.

176 ral ischemia was induced by permanent distal middle cerebral artery occlusion (MCAO) on day 14 of veh

177 A reversible intraluminar suture middle cerebral artery occlusion (MCAO) procedure was us

178 Male rats were subjected to a two-hour middle cerebral artery occlusion (MCAO) procedure.

179 espectively) that are neuroprotective in the middle cerebral artery occlusion (MCAO) rat stroke model

180 ale Sprague-Dawley rats undergoing permanent middle cerebral artery occlusion (MCAO) received three i

181 rly as late as 5 h after 60 min of transient middle cerebral artery occlusion (MCAO) reduced infarct

182 this study, rats were sacrificed 24 h after middle cerebral artery occlusion (MCAO) stroke and gene

183 rating immune cell populations in mice after middle cerebral artery occlusion (MCAO) strongly implica

184 preconditioning stimulus in a mouse model of middle cerebral artery occlusion (MCAO) to examine wheth

185 n (MOG) 35-55 peptide to C57BL/6 mice before middle cerebral artery occlusion (MCAO) to induce an ant

186 ed a modified nylon suture in a rat model of middle cerebral artery occlusion (MCAO) under two anesth

187 ugh 72 hr after both transient and permanent middle cerebral artery occlusion (MCAo) via continuous D

188 Two weeks later, permanent middle cerebral artery occlusion (MCAO) was induced by i

189 Infarct sizes 72 h after 60 min middle cerebral artery occlusion (MCAo) were on average

190 s were greater in diabetic animals following middle cerebral artery occlusion (MCAO) when compared to

191 PGRN(+/-) and PGRN(-/-) mice underwent middle cerebral artery occlusion (MCAO) with monitoring

192 Studies were conducted following middle cerebral artery occlusion (MCAO) with or without

193 cit despite smaller infarcts after transient middle cerebral artery occlusion (MCAO) with the suture

194 This study utilized middle cerebral artery occlusion (MCAO) with tissue plas

195 tradiol replacement were subjected to 2 h of middle cerebral artery occlusion (MCAO), and phosphoryla

196 One week later the animals received a middle cerebral artery occlusion (MCAO), and T(2)-weight

197 uction of stroke, via transient intraluminal middle cerebral artery occlusion (MCAO), or SHAM surgery

198 In a mouse model of middle cerebral artery occlusion (MCAO), p38 MAPK activa

199 reduction in stroke volume in rats with the middle cerebral artery occlusion (MCAO), provided the BD

200 ischemia induced by permanent and transient middle cerebral artery occlusion (MCAO), we observed an

201 logical levels of estradiol protects against middle cerebral artery occlusion (MCAO)-induced brain in

202 ons of estradiol protects the cortex against middle cerebral artery occlusion (MCAO)-induced cell dea

203 the interaction between tPA and LRP plays on middle cerebral artery occlusion (MCAO)-induced NF-kappa

204 subjected to permanent, 60-min and 30-min of middle cerebral artery occlusion (MCAO).

205 llowing a focal stroke caused by a transient middle cerebral artery occlusion (MCAO).

206 ebral ischemia produced in rats by permanent middle cerebral artery occlusion (MCAO).

207 se model of focal ischemic stroke induced by middle cerebral artery occlusion (MCAO).

208 ts of rats infused with HUCB cells 48h after middle cerebral artery occlusion (MCAO).

209 ularly with ATX or vehicle prior to a 60-min middle cerebral artery occlusion (MCAo).

210 5 minutes) was induced by intraluminal right middle cerebral artery occlusion (MCAO).

211 tusion injury (CCI) and another to transient middle cerebral artery occlusion (MCAO).

212 ricularly with HNG before being subjected to middle cerebral artery occlusion (MCAO).

213 of STAZN in the intraluminal suture model of middle cerebral artery occlusion (MCAo).

214 istar rats were subjected to permanent right middle cerebral artery occlusion (MCAo).

215 C57BL/6 mice were subjected to transient middle cerebral artery occlusion (MCAO).

216 schemic reperfusion injury in a rat model of middle cerebral artery occlusion (MCAO).

217 ection of the integrity of the NVU following middle cerebral artery occlusion (MCAO).

218 ubjected to behavioral tests after 90 min of middle cerebral artery occlusion (MCAO).

219 into two groups for permanent and temporary middle cerebral artery occlusion (MCAO).

220 ravenously injected into rats 24 hours after middle cerebral artery occlusion (MCAo).

221 Rats underwent 2 hr of middle cerebral artery occlusion (MCAO).

222 Rats were subjected to embolic middle cerebral artery occlusion (MCAO).

223 Rats in the model groups underwent middle cerebral artery occlusion (MCAO).

224 ischemia was induced by a transient (90 min) middle cerebral artery occlusion (MCAO).

225 n to rats with experimental stroke caused by middle cerebral artery occlusion (MCAO).

226 fusion proteins, following a 1-h reversible middle cerebral artery occlusion (MCAO).

227 type (WT) mice were subjected to a transient middle cerebral artery occlusion (MCAO).

228 (WT) littermate were subjected to 45 min of middle cerebral artery occlusion (MCAO).

229 Male 129/SV mice were subjected to 30-min middle cerebral artery occlusion (MCAo)/reperfusion and

230 yzed at four time points following permanent middle cerebral artery occlusion (MCAO): 1, 3, 7, and 14

231 In the present study, transient middle cerebral artery occlusions (MCAO) were induced fo

232 ogical outcome were assessed after permanent middle-cerebral artery occlusion (MCAO).

233 n protein were investigated with a permanent middle cerebral artery occlusion model in the rat.

234 nd decreases infarct volume in the transient middle cerebral artery occlusion model of stroke.

235 neonatal model of hypoxia/ischemia and in a middle cerebral artery occlusion model of transient foca

236 , and have improved cerebral blood flow in a middle cerebral artery occlusion model than mice express

237 A transient middle cerebral artery occlusion model was used to inves

238 In the middle cerebral artery occlusion model, the volume and f

239 d increase in stroke volume in the permanent middle cerebral artery occlusion model.

240 diated cerebrovascular protection in a mouse middle cerebral artery occlusion model.

241 in male C57BL/6 mice using the intraluminal middle cerebral artery occlusion model.

242 We then used the murine suture and embolic middle cerebral artery occlusion models of stroke to inv

243 stroke groups: 30-min, 60-min, and permanent middle cerebral-artery occlusion (n=12 rats for each gro

244 ith ischemic stroke and in mice subjected to middle cerebral artery occlusion, natural killer (NK) ce

245 troke in females, we evaluated the effect of middle cerebral artery occlusion on infarct size and per

246 e-Dawley rats (12 months old) with permanent middle cerebral artery occlusion or sham operations on m

247 g photothrombotic cortical injury, transient middle cerebral artery occlusion, or neonatal hypoxic-is

248 Animals were subjected to permanent middle cerebral artery occlusion (pMCAo) or a sham surgi

249 profiles in transient (tMCAO) and permanent middle cerebral artery occlusion (pMCAO) stroke models u

250 within 1 and in most cases 2 h of permanent middle cerebral artery occlusion (pMCAO), mild sensory s

251 Following transient middle cerebral artery occlusion, progesterone was admin

252 At 6 hrs following middle cerebral artery occlusion, rats were treated in a

253 At 6 hrs following middle cerebral artery occlusion, rats were treated in a

254 rotects against cerebral ischemia induced by middle cerebral artery occlusion, reducing percent hemis

255 In the middle cerebral artery occlusion-reperfusion model of tr

256 rfusion deficit in a hyperglycemic rat right middle cerebral artery occlusion/reperfusion (MCAO/R) mo

257 neurological impairment in mice subjected to middle cerebral artery occlusion/reperfusion.

258 , administration of PAN-811 i.c.v. 1 h after middle cerebral artery occlusion resulted in a 59% reduc

259 rescence analyses of rat brains subjected to middle cerebral artery occlusion revealed marked increas

260 of infarcted tissue at 72 h after reversible middle cerebral artery occlusion (rMCAo) in adult rats.

261 e and excitatory neurotoxicity in reversible middle cerebral artery occlusion (rMCAO) model in vivo.

262 Following right middle cerebral artery occlusion (rMCAo) using an intral

263 erebral infarction using a stroke model with middle cerebral artery occlusion (see figure).

264 ble Fn14-Fc decoy receptor immediately after middle cerebral artery occlusion significantly reduced i

265 tion of TAT-C1aB in mice following transient middle cerebral artery occlusion significantly reduced i

266 In the model of transient middle cerebral artery occlusion stroke Gna(i2)(fl/fl)/P

267 d improved functional outcome in a transient middle cerebral artery occlusion stroke model.

268 assessment of blood flow anomaly in a mouse middle cerebral artery occlusion stroke model.

269 in rats implanted with CTX-DP 4 weeks after middle cerebral artery occlusion stroke prompted investi

270 further evaluated in vivo using a transient middle cerebral artery occlusion (t-MCAO) model of strok

271 We investigated in a murine model of middle cerebral artery occlusion the effect of blocking

272 In a mouse model of thrombin-induced middle cerebral artery occlusion, the efficacy of the di

273 1 reduced neuronal death following transient middle cerebral artery occlusion (tMCAO) by up to 90% wi

274 Furthermore, by transient middle cerebral artery occlusion (tMCAO) in rats, the tr

275 Transient middle cerebral artery occlusion (tMCAO) in spontaneousl

276 A transient middle cerebral artery occlusion (tMCAO) model was used

277 e in chronic diaschisis by using a transient middle cerebral artery occlusion (tMCAO) rat model.

278 Transient middle cerebral artery occlusion (tMCAO) significantly i

279 nt arterial thrombosis models: the transient middle cerebral artery occlusion (tMCAO) stroke model an

280 c brain injury in a mouse model of transient middle cerebral artery occlusion (tMCAO).

281 sure of anxiety) in rats following transient middle cerebral artery occlusion (tMCAO).

282 ng, and corresponding controls, to transient middle cerebral artery occlusion (tMCAO).

283 was induced in adult rats by 90-minute right middle cerebral artery occlusion (tMCAO).

284 kg, i.p.) 15 min before undergoing transient middle cerebral artery occlusion (tMCAO; 2 h occlusion)

285 Following middle cerebral artery occlusion to induce stroke in mic

286 treatment with an ischemic lesion (permanent middle cerebral artery occlusion) to determine the effec

287 ive patients with internal carotid artery or middle cerebral artery occlusions transferred over an 11

288 cerebral vascular endothelial cell death and middle cerebral artery occlusion-triggered cerebrovascul

289 The infarct produced by middle cerebral artery occlusion was 49% smaller in CD36

290 hemisphere reperfusion after photothrombolic middle cerebral artery occlusion was increased in Klkb1(

291 Middle cerebral artery occlusion was induced for 1 hour

292 marrow (BM) chimeras subjected to transient middle cerebral artery occlusion, we found that CD36(-/-

293 Following middle cerebral artery occlusion, we observed a rapid in

294 However, using a mouse model of transient middle cerebral artery occlusion, we observed that cereb

295 Using two distinct models of acute middle cerebral artery occlusion, we show by next-genera

296 tion volumes following a 60-minute transient middle cerebral artery occlusion were determined in adul

297 Rats subjected to 2h of transient middle cerebral artery occlusion were studied temporally

298 Rats subjected to embolic middle cerebral artery occlusion were treated with atorv

299 ncrease in infarct volume was observed after middle cerebral artery occlusion with 4 days of reperfus

300 icits following experimental stroke, using a middle cerebral artery occlusion with reperfusion model.