ライフサイエンスコーパス: midrib

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1 and for restricting auricle expansion at the midrib.

2 veins, with the majority localized near the midrib.

3 al signal propagating between both lobes and midrib.

4 ic reinitiation of blade primordia along the midrib.

5 ly from the margins of the leaves toward the midrib.

6 ion of two major CAD isoforms, SbCAD2 (Brown midrib 6 [bmr6]) and SbCAD4, in lignifying tissues of so

7 riven by embolism initiating in petioles and midribs across all species, and Kx vulnerability was str

8 rlier that the electrical stimulus between a midrib and a lobe closes the Venus flytrap upper leaf wi

9 es is to accumulate glucosinolates along the midrib and at the margin.

10 E plants developed wider leaves with thicker midrib and enlarged palisade parenchyma cells.

11 Mild leaf phenotypes have a reduced midrib and may be moderately narrow and furcated; severe

12 also displayed ectopic lignification in leaf midribs and elevated concentrations of soluble phenolic

13 Leaf midribs and stems are light green with sectors of dark g

14 city occurred acropetally, with the class I (midrib) and class II veins becoming functional in phloem

15 The wood phenotype resembles that of brown midrib (bm) mutants and some transgenic plants in which

16 The midribs of maize brown midrib (bm) mutants exhibit a reddish-brown color associ

17 The four brown midrib (bm) mutants of maize have a reduced content and

18 Brown-midrib (bm) mutants of maize have modified lignin of red

19 The brown midrib (bm) mutations of maize affect the biosynthesis o

20 ce showing that a set of three allelic brown midrib (bmr) lignin mutants contained mutations in this

21 Brown midrib (bmr) mutants in sorghum (Sorghum bicolor (L.) Mo

22 of the inner layer of the petals and in the midrib by providing a qualitatively different paradigm t

23 evelopment at the ligular region of the leaf midrib by transforming blade to sheath.

24 nsitivity of K(leaf) to damage; severing the midrib caused K(leaf) and gas exchange to decline, with

25 gle electrical charge between a lobe and the midrib causes closure of the trap and induces an electri

26 ity was strongly correlated with petiole and midrib conduit dimensions.

27 y abnormal patterns of cell expansion in the midrib cortex and in the epidermis of the elongation zon

28 sity of primary veins predicted tolerance of midrib damage.

29 djustment of endogenous leaf auxin levels on midrib elongation and final leaf size (fresh weight and

30 al tip leads to premature termination of the midrib into a knot and leaf bifurcation.

31 this assertion, we observe that maize brown midrib mutants affected in lignin biosynthesis are hyper

32 s, genetic and biochemical analyses of brown midrib mutants of maize, sorghum and related grasses hav

33 The levels of cad2 mRNA in the midribs of bm1-das1 and bm1-ref are reduced by 91 and 86

34 The midribs of maize brown midrib (bm) mutants exhibit a red

35 Support cells within the leaf midribs of mosses deposit cellulose-rich secondary cell

36 The cellulose-deficient midribs of ppcesa3/8 knockouts provided negative control

37 ered completely recessive, because the brown midrib phenotype is only apparent in plants homozygous f

38 uses a blade-to-sheath transformation at the midrib region of the maize (Zea mays L.) leaf.

39 e, auricle and ligule into sheath around the midrib region.

40 gion (UTR), conferring the spontaneous brown midrib trait and lignin reduction in the sorghum germpla

41 When the midrib was severed near the leaf base, the pinnately vei

42 frequency generation spectra collected from midribs were consistent with cellulose microfibril aggre

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