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1 SD), the electrophysiologic event underlying migraine aura.
2 the neurovascular unit in the development of migraine aura.
3 iac and extracardiac right-to-left shunts to migraine aura.
4 opagating slow depolarization that underlies migraine aura.
5 e of neuroglial depolarization implicated in migraine aura.
6 uronal activity, is the most likely cause of migraine aura.
7 g rCBF changes may help our understanding of migraine aura.
8  of the cerebral disturbances which subserve migraine aura.
9 ion (CSD) may be the underlying mechanism of migraine aura.
10 ents during spontaneous and visually induced migraine aura.
11 be similar to those in human patients during migraine aura.
12 l focal ischaemia, and may also underlie the migraine aura.
13 ain the predominant sensory phenomenology of migraine aura.
14  considered as the cellular correlate of the migraine aura.
15 ression (CSD), the putative mechanism of the migraine aura.
16             Here we establish a link between migraine aura and headache by demonstrating that cortica
17                        SD is associated with migraine aura and recently recognized as a novel mechani
18 ta help to explain the sensory nature of the migraine aura and reveal that sensory cortices are vulne
19 sion, the electrophysiological surrogate for migraine aura, and develop severe and prolonged motor de
20 ng depression, the experimental correlate of migraine aura, and further evaluated the response of spo
21 rlying the initiation and propagation of the migraine aura, and the visual percept that is produces,
22                        The visual percept of migraine aura changes depending on the region of the occ
23 preading depression, the neural correlate of migraine aura, closes the paravascular space and impairs
24 vents menstrually related migraine (MRM) and migraine aura frequency.
25 ish a mechanism linking microembolization to migraine aura in an experimental animal model.
26 (CSD) as the pathophysiological mechanism of migraine aura in human beings, whereas novel animal stud
27 e detailed drawings of his visual percept of migraine aura in real time during more than 1000 attacks
28 and temporal lobe involvement contributes to migraine aura in some instances.
29 ds were measured during spontaneous onset of migraine auras in 4 migraine patients, and compared with
30 reading depression (CSD), an animal model of migraine aura, induces a rapid and nearly complete closu
31                                              Migraine aura involves sensory percepts, suggesting that
32                                              Migraine aura is sparsely studied due to the highly chal
33 (CSD), the electrophysiological substrate of migraine aura, is enhanced in mice expressing a vascular
34       If translatable to humans, a subset of migraine auras may belong to a spectrum of hypoperfusion
35 ves in women with migraine in the absence of migraine aura or other risk factors.
36 ctivated in spontaneous and visually induced migraine aura patients.
37 ered various diagnoses, including persistent migraine aura, post-hallucinogen flashback, or psychogen
38 ing of the visual disturbance (36%), whereas migraine aura (seven patients) and consumption of illici
39 on-like neuroelectric event occurring during migraine aura that can arise spontaneously or be visuall
40  visual disturbance clinically distinct from migraine aura that can be disabling for patients.
41 ression (CSD), the proposed mechanism of the migraine aura, to shape the cortical activity that under
42 n real time during more than 1000 attacks of migraine aura without headache over 18 years.

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