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1 lence of self-reported migraine was 16% (13% migraine with aura).
2 1 (FHM1), a rare monogenic subtype of common migraine with aura.
3 miplegic migraine (FHM) is a rare subtype of migraine with aura.
4 hemiplegic migraine and in one patient with migraine with aura.
5 pression (CSD) is a key pathogenetic step in migraine with aura.
6 ma, and seizure phenotype in this variant of migraine with aura.
7 scular neurons that underlie the headache of migraine with aura.
8 cations such as cerebral emboli, stroke, and migraine with aura.
9 ising acute treatment for some patients with migraine with aura.
10 portable sTMS device for acute treatment of migraine with aura.
11 en ovale (PFO) is prevalent in patients with migraine with aura.
12 nce of right-to-left shunts in patients with migraine with aura.
13 between high oestrogen states and attacks of migraine with aura.
14 that cannot easily be controlled, including migraine with aura.
15 association between right-to-left shunts and migraine with aura.
16 ynamic changes during spontaneous attacks of migraine with aura.
17 bands, there appears to be an excess risk of migraine with aura.
20 assified into no history of migraine, active migraine with aura, active migraine without aura, and pa
21 tantial external noise-exclusion deficits in migraine with aura and a minor impairment of noise exclu
26 ith two diagnostic types of migraine, termed migraine with aura and migraine without aura, from the I
28 o difference in PFO prevalence in those with migraine with aura and those without (26.8% versus 26.1%
29 by seven individuals who had previously had migraine with aura and three who had previously had migr
30 ing headaches of intracranial origin such as migraine with aura and why this therapeutic approach may
31 e prospective cohort suggest that women with migraine with aura are at increased risk of experiencing
34 legic migraine type 1 (FHM1) is a subtype of migraine with aura caused by a gain-of-function mutation
36 Willis was significantly more common in the migraine with aura compared to control group (73% vs. 51
38 without aura (Group 1), 45 patients who had migraines with aura (Group 2), and 30 healthy participan
39 perience a TIA or stroke, women who reported migraine with aura had adjusted relative risk (95% confi
40 migraine history, women who reported active migraine with aura had multivariable-adjusted hazard rat
45 This is consistent with the hypothesis that migraine with aura in midlife is associated with late-li
56 ombined hormone contraceptive use in MRM and migraine with aura may decrease both headache frequency
59 ore per month (n = 3243), those with midlife migraine with aura (n = 361) had an increased risk of la
60 OR, 7.01 [95% CI, 4.43-11.09]; P < .001), or migraine with aura (n = 66; 69.7% vs 26.5%; OR, 5.73 [95
63 three SNP associations was preferential for migraine with aura or without aura, nor were any associa
66 atients, and compared with recordings from 8 migraine-with-aura patients and 6 normal controls during
69 of the burden of circle of Willis variants, migraine with aura subjects had a higher burden of varia
70 ncomplete circle of Willis is more common in migraine with aura subjects than controls, and is associ
72 1 (FHM1) is an autosomal dominant subtype of migraine with aura that is associated with hemiparesis.
73 contributes to the genetic susceptibility of migraine with aura that is distinct from the FHM locus.
76 hemiplegic migraine type 1 (FHM1), a severe migraine with aura variant, is caused by mutations in th
77 (prevalence of infarcts 23.0% for women with migraine with aura vs 14.5% for women not reporting head
78 a 19.3% prevalence of infarcts for men with migraine with aura vs 21.3% for men not reporting headac
79 s large, prospective cohort of women, active migraine with aura was associated with increased risk of
80 duals had to meet international criteria for migraine with aura, with visual aura preceding at least
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