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1 h optimal Ca(2+) transport into milk and for milk ejection.
2 yer that provides contractility required for milk ejection.
3 cessary to limit the explosive bursts during milk ejection.
4 us, because mutant mothers were deficient in milk ejection.
5 rturition and during suckling-induced reflex milk ejection.
6 ines the signaling processes responsible for milk ejection.
7 h-frequency bursting activity preceding each milk ejection.
8 cerebroventricularly reduced suckling-evoked milk ejections.
9 ely by Orai1 and are absolutely required for milk ejection and pup survival, an observation that rede
11 om the beginning of suckling up to the first milk-ejection burst, which occurred either within less t
12 ernal simulation of pups (e.g., grooming and milk ejection) consistently produced rapid, transient co
13 ion to uterine contractions during labor and milk ejection during nursing, oxytocin has been implicat
15 e mammary myoepithelial cells, which control milk ejection following oxytocin-induced contraction.
16 umulation up to eightfold, impaired maternal milk ejection, halved milk tetraiodothyronine (T4) conte
18 that OT mediation of suckling-evoked bursts/milk ejections is via interactions between pERK1/2 and a
19 ded from an OT-containing neurone during the milk ejection reflex was effective in supporting an exoc
20 ciated with pulsatile secretion of OT in the milk ejection reflex, and can be observed in slices from
23 fied the essential role of astrocytes in the milk-ejection reflex (MER) by disabling astrocytic funct
25 changes dramatically immediately before each milk ejection, when a brief burst of action potentials i
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