ライフサイエンスコーパス: mineralization

1 phatidylcholine is broken down in MVs during mineralization.

2 lage thickness, increased TRAP activity, and mineralization.

3 ly hosting economically important base metal mineralization.

4 is D) causes osteomalacia and poor long bone mineralization.

5 interactions salient to methanogenic alkane mineralization.

6 ence of nanoconfinement on calcium carbonate mineralization.

7 e cells enhanced osteoblastic maturation and mineralization.

8 terial calcification but did not affect bone mineralization.

9 he combination of Wnt3a and PEDF potentiated mineralization.

10 f As, Hg and Pb that obviate dissolution and mineralization.

11 ogenitors, alkaline phosphatase activity and mineralization.

12 74-3p induced osteoblast differentiation and mineralization.

13 ved structures that often arise in accretive mineralization.

14 ely on matrix vesicle-mediated initiation of mineralization.

15 community in SFs showing intrinsic 2,6-DCBA mineralization.

16 al role of noncollagenous proteins in matrix mineralization.

17 ified systems provided the highest degree of mineralization.

18 ine phosphatase (ALP) activity, and enhanced mineralization.

19 me of the bone vessels were found to undergo mineralization.

20 ghts of this intracellular protein in matrix mineralization.

21 P-1 binds to Type-I collagen and can promote mineralization.

22 ations of the collagen prior to the onset of mineralization.

23 only the PCL+FA scaffold could sustain cell mineralization.

24 ew mechanism to the pathogenesis of vascular mineralization.

25 ain the elevated contribution of lignin to C mineralization.

26 osteoblast differentiation and reduced bone mineralization.

27 mobilized acidic macromolecules in directing mineralization.

28 ts, but does not block, parbendazole-induced mineralization.

29 ded to reduce microbial biomass and nitrogen mineralization.

30 ering ion transporters, in modulating enamel mineralization.

31 o maintain pH homeostasis and support enamel mineralization.

32 lops postnatally, concurrent with epiphyseal mineralization.

33 teraction are responsible for intrafibrillar mineralization.

34 imbalanced metabolism, leading to defective mineralization.

35 for polyelectrolyte-directed intrafibrillar mineralization.

36 specific roles in the initiation of skeletal mineralization.

37 in extracellular matrix (ECM) formation and mineralization.

38 sm that causes defective skeletal and dental mineralization.

39 ensional model of extracellular matrix (ECM) mineralization.

40 e in trabecular rods, and EDX confirmed less mineralization.

41 ntrast with known patterns of post-mortem Fe mineralization.

42 the matrix vesicle membrane during skeletal mineralization.

43 pha heterodimer (VDRRXRalpha) regulates bone mineralization.

44 , consequently, completely prevented ectopic mineralization.

45 or of pth4-expressing neurons in larval bone mineralization.

46 deformity, presence of fractures and delayed mineralization.

47 ential mechanisms involved in intrafibrillar mineralization.

48 dental mesenchymal cell differentiation and mineralization.

49 sms involved in polyester biodegradation and mineralization.

50 erwent progressive deep-zone hypertrophy and mineralization.

51 recruited to the affected tissue and induce mineralization.

52 tive marsh vegetation), and deep SOM-derived mineralization (40-80 cm, below active root zone of nati

53 Aiming to enhance the mineralization, a sequential combination of EC with elec

54 ct in the proliferation, differentiation and mineralization abilities of patDp/+ osteoblasts while os

55 The structural and mineralization abnormality led to slight reduction in ap

56 P2 demonstrated significantly higher ALP and mineralization activity (P <0.05).

57 microarchitecture, mineral distributions or mineralization activity of bone-forming cells, represent

58 out to better understand microbial (i) early mineralization and (ii) potential for further fossilisat

59 content but with two indirect parameters of mineralization and aromaticity, suggesting that soil org

60 can expose soil organic carbon and result in mineralization and carbon dioxide (CO2) release.

61 able after 6 months although the kinetics of mineralization and cell-mineral interactions depended on

62 tion increased although it displayed delayed mineralization and cementoid.

63 2 sequestration (GCS) can be affected by CO2 mineralization and changes in the permeability of geolog

64 ting in significantly reduced fibrocartilage mineralization and decreased biomechanical function.

65 s by illuminating the evolution of immunity, mineralization and development (mediated, for example, b

66 -cell conditioned medium impaired osteoblast mineralization and enhanced osteoclast-progenitor surviv

67 igated the impact of physiological status on mineralization and long-term fossilisation by exposing c

68 ganic N pools, gross proteolytic rate, net N mineralization and microbial extracellular enzyme activi

69 ent the results of a study investigating the mineralization and microbial uptake of surface-functiona

70 ilitates the role of the endothelium in bone mineralization and morphogenesis.

71 ed increases in resource availability (via N mineralization and N2 fixation) and (ii) elevated resour

72 pecies composition affects the dynamics of N mineralization and nitrification.

73 d performance in increasing soil DOC, DON, N mineralization and plant biodiversity, we recommend that

74 distributions, we show that root-accelerated mineralization and priming can account for up to one-thi

75 the human disease, including diminished bone mineralization and propensity to fracture.

76 mple formulations of processes affecting the mineralization and storage of detrital OM.

77 ignals from the GH/IGF-1 to enhance skeletal mineralization and strength during pubertal growth.

78 rentiation by enhancing extracellular matrix mineralization and the expression of osteoblastic marker

79 The results suggest that mineralization and vascularization are coupled by locali

80 CO2 (as indicator of microbial activity and mineralization) and microbial growth can be used to esti

81 f Tnap expression, phosphate production, and mineralization, and decreased pyrophosphate concentratio

82 se new criteria based on sequences of dental mineralization, and the presence of vestigial teeth, to

83 ppression of odontogenic differentiation and mineralization as demonstrated by alkaline phosphatase (

84 the alkaline phosphatase (ALPL) activity and mineralization as well as gene expression of Alpl and ot

85 CM demonstrated that they exhibited enhanced mineralization, as determined by alizarin red staining a

86 ce revealed a developmental delay in condyle mineralization, as measured by micro-computed tomography

87 implications, was confirmed in a cell-based mineralization assay in vitro.

88 e osteogenic potential was evaluated through mineralization assay.

89 the samples (in situ degradation estimates, mineralization assays, culturable bacteria, catabolic ge

90 ons during late pregnancy and offspring bone mineralization assessed at birth with the use of dual-en

91 bon quality temperature hypothesis to soil N mineralization at a global scale.

92 We show that in response to reduced skeletal mineralization at lower pH, corals increase their skelet

93 Induction of biomimetic mineralization at near physiological conditions reveals

94 of complex structure particles, bioreaction/mineralization at the two-phase interface, and biosepara

95 es commonly associated with ossification and mineralization but also genes important for general prot

96 rew in size over time and exhibited regional mineralization by 12 mo.

97 -containing integrin, resulting in increased mineralization by and differentiation of osteoblasts.

98 blasts that functions in skeletal and dentin mineralization by initiating deposition of hydroxyapatit

99 4 plays a pivotal role in fine tuning enamel mineralization by modulating SLC4A4 and CFTR to maintain

100 used as a scaffold to induce template-guided mineralization by osteoblasts.

101 orage of anthropogenic CO2 emissions through mineralization can be far faster than previously postula

102 emic factors regulating extracellular matrix mineralization can be possible therapeutic strategies to

103 AM mineralization was rare, whereas 2,6-DCBA mineralization capacity appeared widespread, with high m

104 sociated with the sucrose transformation and mineralization caused pronounced, temporary shifts in th

105 O mice were shorter and had a lower level of mineralization compared to the normal mice.

106 Inhibitors of bone mineralization completely prevented ectopic cardiac calc

107 e and anaerobiosis initially suppress soil C mineralization, consistent with theory.

108 naling and greatly reduced capacity for bone mineralization, contributing to profound skeletal defici

109 t in both SF and soil samples, high 2,6-DCBA mineralization correlated with high organic carbon conte

110 osteogenic differentiation attenuated matrix mineralization, cytoskeletal rearrangement, mitochondria

111 Theory and models posit that C mineralization declines under elevated moisture and asso

112 tified a novel mechanism contributing to the mineralization defect in Hyp mice.

113 decreased serum phosphate level, which cause mineralization defects in the skeleton and teeth (osteom

114 is suggests that the Dspp-independent dentin mineralization defects in Trps1-Tg mice are a result of

115 Despite alveolar bone mineralization defects, periodontal attachment and funct

116 the co-existence of carbonate and phosphate mineralization demonstrates a biomineralization system t

117 ication of infancy is an intractable ectopic mineralization disorder caused by mutations in the ENPP1

118 seudoxanthoma elasticum, a heritable ectopic mineralization disorder, is caused by mutations in the A

119 Inactivation of these factors results in mineralization disorders affecting teeth and their suppo

120 Heritable ectopic mineralization disorders represent a phenotypically dive

121 n pseudoxanthoma elasticum and other ectopic mineralization disorders, as presented in the symposium,

122 icum (PXE), a prototype of heritable ectopic mineralization disorders, is caused in most cases by ina

123 lasticum is a prototype of heritable ectopic mineralization disorders, with phenotypic overlap with g

124 lue for the short-term enhancement of soil C mineralization due to soil disruption and displacement/t

125 sitive period in relation to bone growth and mineralization during childhood.We examined whether 25-h

126 MC3T3-E1 cells resulted in increased matrix mineralization during differentiation and knockdown resu

127 or microspheres, the combined osteogenic and mineralization effect of PRP and BMP2 on MSCs was studie

128 enty-first century, in-reservoir burial plus mineralization eliminated 4.0+/-0.9 Tmol per year (48+/-

129 f the turkey tendon that are associated with mineralization exhibit distinct and observable chemical

130 anced analysis of structural, mechanical and mineralization features.

131 bcc6(-/-) mice expressing human ENPP1, small mineralization foci were still evident despite increased

132 zed invertebrates, adopted calcium carbonate mineralization for bulk skeleton reinforcement.

133 ased histological analyses revealed that the mineralization had started from the outside, then procee

134 which PHOSPHO1 substrates are formed before mineralization have not been determined.

135 New evidence on the mechanisms of mineralization identified calcification-competent extrac

136 ro, and for long-term in vivo monitoring the mineralization in 3D scaffolds subcutaneously implanted

137 nt in the respired CO2, with higher fullerol mineralization in an organic, clay-rich soil versus a si

138 Acetate was a key intermediate for carbon mineralization in both zones.

139 neralization while correcting decreased bone mineralization in generalized arterial calcification of

140 f of the CO2 produced came from plant tissue mineralization in invasive and native communities; the r

141 ular stress and indeed 4PBA ameliorated bone mineralization in larvae and skeletal deformities in adu

142 ), it could provide a target for controlling mineralization in metabolic bone disease.

143 thetic systems, biominerals, and patterns of mineralization in natural environments.

144 unds showed slow equilibration and the least mineralization in OECD 308, whereas the modified systems

145 increased alkaline phosphatase activity and mineralization in osteoblast cultures.

146 f a single enzyme capable of both catalyzing mineralization in otherwise unreactive solution and of t

147 reased bone formation and substandard matrix mineralization in patients.

148 f molecular correction to counteract ectopic mineralization in pseudoxanthoma elasticum.

149 st that PPi is the major mediator of ectopic mineralization in PXE, but there might be an alternative

150 that for the period 1970-2030, global carbon mineralization in reservoirs exceeds carbon fixation (P<

151 ce increased oxidative stress and changes in mineralization in response to ocean acidification and wa

152 phosphonate treatments significantly reduced mineralization in skin and aorta.

153 showed defects in chondrocyte maturation and mineralization in the absence of Ddr2.

154 tic processes mediate BPA transformation and mineralization in the absence of oxygen, indicating that

155 otein thought to act as a promoter of matrix mineralization in the final stage of enamel development,

156 We find that SOC mineralization in the presence of roots is 23% higher (P

157 f pseudoxanthoma elasticum depicting ectopic mineralization in the skin, eyes, and the arterial blood

158 ratio is the cause of soft connective tissue mineralization in these disorders.

159 rolyzable PPi analogs, in preventing ectopic mineralization in these mice.

160 er to account for a large fraction of carbon mineralization in this system.

161 net effect on microbial biomass or nitrogen mineralization in those ecosystems.

162 PTHrP1-17 promotes osteoblast migration and mineralization in vitro, and systemic administration of

163 tly and potently induced differentiation and mineralization in vitro.

164 wed enhanced odontogenic differentiation and mineralization in vivo.

165 Temperature sensitivity of anaerobic carbon mineralization in wetlands remains poorly represented in

166 ining for mineral showed significantly lower mineralization in XLH cell-seeded scaffolds, using nonpa

167 hydroxyapatite and directing intrafibrillar mineralization in-vitro.

168 The study of their sequence of mineralization indicates that the lower and upper canini

169 teeth, liver, and kidney that hydrolyzes the mineralization inhibitor inorganic pyrophosphate.

170 ion and subsequent gene up-regulation of the mineralization inhibitors matrix Gla protein and osteopo

171 (HA) crystals in vitro and on the known role mineralization inhibitors, like PPi, play in the regulat

172 Mineralization is a key process in the formation of bone

173 ion and (ii) the first metabolic step in BAM mineralization is rare in microbial communities, rather

174 Cementum and bone mineralization is regulated by factors including enzymes

175 ing effect (PE) on soil organic matter (SOM) mineralization is still under debate.

176 This study demonstrates that (i) 2,6-DCBA mineralization is widely established in SFs of DWTPs, al

177 alization that supplements existing collagen mineralization mechanisms.

178 lt petrogenesis, and precious metal geology, mineralization, mining, and processing.

179 of lesion (lytic/sclerotic or mixed), matrix mineralization, multiplicity and involvement of other si

180 Soil net nitrogen (N) mineralization (Nmin ) is a pivotal process in the globa

181 Whereas BAM mineralization occurred rarely and only in SFs exposed t

182 5-fold lower rates, and CO2 evolution (i.e., mineralization) occurred.

183 ly established in SFs of DWTPs, allowing the mineralization of 2,6-DCBA produced during BAM degradati

184 The artificial mineralization of a polyresistant bacterial strain isola

185 his study highlights the role of PHOSPHO1 in mineralization of alveolar bone and cellular cementum, f

186 gly inhibited osteogenic differentiation and mineralization of ASCs in the 3-dimensional model.

187 Mineralization of bones and teeth is tightly regulated b

188 However, the internal production and mineralization of carbon in freshwaters remain to be qua

189 es governing the fixation, partitioning, and mineralization of carbon in soils are under increasing s

190 l attachment, migration, differentiation and mineralization of dental mesenchymal cells.

191 Mineralization of fibrillar collagen with biomimetic pro

192 mino acid on a 34-mer PEDF peptide increased mineralization of hMSC cultures compared with the native

193 Continuous Wnt3a exposure impeded mineralization of hMSCs, whereas the combination of Wnt3

194 was capable of inducing differentiation and mineralization of human dental pulp stem cells.

195 l enhance our understanding of the microbial mineralization of IPU and provide insights into the evol

196 iron storage system, where the oxidation and mineralization of iron are distributed between two prote

197 the role of ATX-lysophosphatidic acid in the mineralization of isolated valve interstitial cells and

198 Stable C isotopes show that mineralization of older C3-derived C released following

199 abolic interaction to accomplish cooperative mineralization of organic compounds to CH4 and CO2 .

200 Photodegradation, the photochemical mineralization of organic matter, has been recently iden

201 In return, the PSB enhanced mineralization of organic P, increasing P availability f

202 Aerobic mineralization of PCBs, which are toxic and persistent o

203 The degree of mineralization of permanent tooth germs in dental age as

204 film formation and dramatically enhanced the mineralization of phenanthrene, up to 30 times greater t

205 c concentrations induced differentiation and mineralization of preosteoblastic MC3T3-E1 cells and pre

206 show the feasibility for in vivo monitoring mineralization of scaffold implanted subcutaneously by d

207 e silaffin peptide R5 is instrumental to the mineralization of silica cell walls of diatom organisms.

208 mical reactions that result in bleaching and mineralization of tDOM.

209 ophosphatidic acid promotes inflammation and mineralization of the aortic valve and thus could repres

210 out to document the impact of lncRNA on the mineralization of the aortic valve.

211 alve disease is characterized by an abnormal mineralization of the aortic valve.

212 The impaired mineralization of the cranial bone correlated well with

213 the rate-limiting step to dehalogenation and mineralization of the lampricide.

214 roliferation, proteoglycan distribution, and mineralization of the MCC.

215 ate level was sufficient to prevent abnormal mineralization of the nasal septum in Mgp(-/-);Hyp compo

216 esized that the enzyme would regulate proper mineralization of the periodontal apparatus.

217 Although we observed a mild increase in mineralization of the spheno-occipital synchondrosis, it

218 T as a sole carbon source resulted in the re-mineralization of these nitrogen-rich compounds into amm

219 h intracellular domain prevented H19-induced mineralization of valve interstitial cells.

220 sils from the Araripe Sedimentary Basin: the mineralization of zinc sulfide interfacing to hydroxyapa

221 sure conditions control rock deformation and mineralization on geological faults, and hence the distr

222 cient circulating phosphate availability for mineralization or also by a direct, local intrinsic effe

223 llustrate their probable involvement in iron mineralization, oxidative and nitrosative stress resista

224 We include a discussion of the different mineralization pathways available as organic matter move

225 addition of FGF2 during the differentiation/mineralization phase of the in vitro growth of pulp cult

226 netics with experimental data for growth and mineralization, physiology and metabolomics, we demonstr

227 zed for initial Fe(2+) oxidation but not for mineralization, pointing to a role for this protein in b

228 ment with Phen was sufficient to enhance the mineralization potential of DPSCs in vitro.

229 s; the rest of the CO2 was produced from SOM mineralization (priming).

230 ect winter precipitation related to nitrogen mineralization prior to the growing season.

231 characterized model system for studying the mineralization process of bone.

232 implementation including the need to develop mineralization process routes for permanent carbon stora

233 6 activities may represent a key step in the mineralization process.

234 can be prevented by inhibiting the activated mineralization process.

235 nt is impacting our understanding of natural mineralization processes and holds promise for novel mat

236 t implications for our understanding of S(0) mineralization processes and sulfur interactions with or

237 s a maximum because uptake can be revered by mineralization processes.

238 magnetite (NAu2) were detected as secondary mineralization products upon reaction of the nontronites

239 ulation, primary productivity (P) and carbon mineralization (R) along the river continuum.

240 ads to a 10-fold increase in volume-specific mineralization rate, illustrating the sensitivity of ana

241 and light as a function of CO2 and nitrogen mineralization rate.

242 Substrate mineralization rates and shifts in relative abundance of

243 Estimates of RDX mineralization rates based on the production and gas tra

244 eriment, with higher N (+175%) and P (+211%) mineralization rates compared to ambient rings, although

245 tion capacity appeared widespread, with high mineralization rates found especially in forest soils.

246 e greatest increases in soil inorganic N and mineralization rates had a much greater litter N content

247 The annual net N mineralization rates of 10-mixed and 30-mixed plantation

248 biomass, and increasing FGR strongly reduced mineralization rates, because of lower root N concentrat

249 ntaminated water showed the highest 2,6-DCBA mineralization rates.

250 omposition also significantly affected net N mineralization rates.

251 did not alter root N concentrations or net N mineralization rates.

252 We define the two groups of genes as mineralization related versus protein anabolism signatur

253 Smad4 is necessary for the activation of the mineralization-related genes, it is dispensable for BMP2

254 onse functions for soil microbial carbon (C) mineralization remain a critical uncertainty for predict

255 he Smpd3 gene causes poor bone and cartilage mineralization resulting in severe congenital skeletal d

256 gnificantly increased osteoblastogenesis and mineralization, reversed bone loss caused by ovariectomy

257 soil fauna, microbial biomass, and nitrogen mineralization shifted from neutral to negative with inc

258 mice featured disturbances in alveolar bone mineralization, shown by accumulation of unmineralized o

259 at is induced upon injury and regulates bone mineralization, significantly attenuated cardiac calcifi

260 od has potential use in the assessment of de-mineralization states in humans, such as caries of teeth

261 lankton development, physiology and skeletal mineralization status, potentially reducing their defens

262 trates the widespread occurrence of the dual mineralization strategy in the Malacostraca, suggesting

263 l as markers for chondrocyte hypertrophy and mineralization such as Col10, osterix, alkaline phosphat

264 an equilibrium in a polyelectrolyte-directed mineralization system establishes a new model for collag

265 more important determinants of short-term C mineralization than current soil moisture content in the

266 ed higher cell attachment, proliferation and mineralization than the HCCS group in vitro.

267 n variable alterations of bone formation and mineralization that are caused by mutations in the ALPL

268 shes a new model for collagen intrafibrillar mineralization that supplements existing collagen minera

269 (FGFR3) signaling, leading to inhibition of mineralization through accumulation of the TNAP substrat

270 to feedbacks of leaf traits on soil nitrogen mineralization through litter quality.

271 ted molecules were observed as a function of mineralization time and matrix.

272 ratio is critical for prevention of ectopic mineralization under homeostatic conditions.

273 dent of PPi, by which ABCC6 prevents ectopic mineralization under physiologic conditions.

274 structure of intramembranous bone and dentin mineralization using 3 different age groups of DSPP-null

275 l x((13)C)DIC allowing to quantify microbial mineralization using mass-balance calculations.

276 ve, and simple approach to monitor microbial mineralization using reverse stable isotope labeling ana

277 pha heterodimer (VDRRXRalpha) regulates bone mineralization via transcriptional control of osteocalci

278 itiated at 4 weeks of age, and the degree of mineralization was assessed at 12 weeks of age by quanti

279 ely and only in SFs exposed to BAM, 2,6-DCBA mineralization was common in SFs, including those treati

280 Mineralization was conducted in mineral matrixes commonl

281 The highest rate of net N mineralization was found in A. crassicarpa monoculture,

282 However, when mineralization was induced via oxidative stress, DRP1 in

283 In contrast, although significantly reduced mineralization was noted in Abcc6(-/-) mice expressing h

284 While apatitic mineralization was observed along collagen fibrils by el

285 As in SF samples, BAM mineralization was rare, whereas 2,6-DCBA mineralization

286 rmore, the activation energy (Ea ) of soil N mineralization was significantly and negative correlated

287 erobic conditions, and cumulative methoxyl-C mineralization was statistically equivalent under static

288 ionic electrolyte is used to direct collagen mineralization, we argue that additional types of long-r

289 arches, and their varying modes of skeletal mineralization, we can better appreciate the detailed me

290 ation- and polyanion-directed intrafibrillar mineralization, we challenge the popular paradigm that e

291 ole of miR-138 in the OC-mediated control of mineralization, we demonstrated that the LPS-induced dow

292 For comparison, 2,6-DCBA and BAM mineralization were determined in various topsoil sample

293 asma levels of PPi and the degree of ectopic mineralization were determined.

294 Raman spectroscopy features associated with mineralization were identified by probing (on the microm

295 (-/-) mice, acellular cementum formation and mineralization were unaffected, whereas cellular cementu

296 ophosphate serves as a powerful inhibitor of mineralization, whereas inorganic phosphate is a promine

297 oblast differentiation and produce extensive mineralization, which is in marked contrast to the behav

298 es in controlling the rate and extent of CO2 mineralization, which will likely occur in diffusive zon

299 al effect for preventing ectopic soft tissue mineralization while correcting decreased bone mineraliz

300 elation between surface science and confined mineralization, with implications for diverse areas of i