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1 and after their osteogenic differentiation (mineralized nodules).
2 n and alkaline phosphatase, and formation of mineralized nodules.
3 al cells undergo osteoblastogenesis and form mineralized nodules.
4 ent pericytes that were downregulated in the mineralized nodules.
5 by confluent cells and was downregulated in mineralized nodules.
6 and osteocalcin and accelerated formation of mineralized nodules.
7 st cultures revealed a dramatic reduction in mineralized nodules, a significant reduction in Runx2, S
10 ov in ST-2 cells inhibited the appearance of mineralized nodules and decreased alkaline phosphatase a
14 from SRC-2 KO mice formed significantly more mineralized nodules (by 3-fold) in the presence of the P
15 antly reduced bone mineral density and fewer mineralized nodules, coincident with increased expressio
16 steopontin were analyzed along with in vitro mineralized nodule formation and calcium accumulation.
17 oted, whereas that of RCAS-Dlx5RH inhibited, mineralized nodule formation as compared with that in co
19 alcin, osteonectin/osteopontin, and in vitro mineralized nodule formation compared to stimulation wit
21 ial- and bone marrow-derived osteoblasts for mineralized nodule formation in vitro showed increased m
22 ressed bone formation parameters in vivo and mineralized nodule formation in vitro similarly to those
26 were significantly larger, and the onset of mineralized nodule formation was delayed when compared w
28 alkaline phosphatase, increased matrix, and mineralized nodule formation when compared with untreate
29 amine the effects of P. gingivalis lipids on mineralized nodule formation, cell viability, apoptosis,
33 nalysis techniques to micro-Raman spectra of mineralized nodules formed in vitro, we reveal cell-sour
34 he ability of the mutant osteoblasts to form mineralized nodules in culture was severely reduced.
35 Bs from TIEG(+/+) calvaria displayed several mineralized nodules in culture, whereas those from TIEG(
37 kout osteoblasts produced significantly more mineralized nodules in ex vivo cell cultures than did wi
39 olates were tested for their ability to form mineralized nodules in vitro and to express alterations
40 ocedure and screened for the ability to form mineralized nodules in vitro, a property of cells with o
41 e cells expressed osteoblastic genes, formed mineralized nodules in vitro, and formed bone in an in v
43 es in vitro, PDL cells from diabetics formed mineralized nodules more slowly than did the controls.
45 rface areas of the cell cultures occupied by mineralized nodules were measured using computerized ima
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