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1 nd largest subunit of RNA polymerase II, and minichromosome maintenance protein).
7 n identify colorectal cancer by detection of minichromosome maintenance protein 2 (MCM2) expression i
10 egulated loading of the replicative helicase minichromosome maintenance proteins 2-7 (MCM2-7) onto re
12 ereplication complex that contains a Mcm2-7 (minichromosome maintenance proteins 2-7) double hexamer.
13 lation sites in the carboxyl terminus of the minichromosome maintenance protein 3 (MCM3), a component
18 damage marker gammaH2AX is upregulated under minichromosome maintenance protein 7 (MCM7) knockdown in
19 We then used our mouse model to identify minichromosome maintenance protein 7 (MCM7), an E2F-indu
20 bers, whereas Double-parked protein/Cdt1 and minichromosome maintenance proteins are apparently essen
24 (origin recognition complex [ORC], Cdc6, and minichromosome maintenance proteins) causes a cell cycle
26 asmid DNA template by DNA polymerases (pol), minichromosome maintenance protein complex (Mcm), topois
28 s the Cdc19 (Mcm2) subunit of the six-member minichromosome maintenance protein complex purified from
30 etylation and RNR levels, maintains helicase minichromosome maintenance protein complexes (Mcm2-7) on
39 ession of E2F-5, centromere protein A and E, minichromosome maintenance proteins (MCM)-2, -3, -5, -6
40 ognition complex, and the elongation factors minichromosome maintenance proteins (MCM)2-7 and prolife
42 ication, involves the loading of six related minichromosome maintenance proteins (Mcm2-7) into prerep
45 ber of other replication proteins, including minichromosome maintenance proteins, the origin recognit
46 monstrate that uncontrolled DNA unwinding by minichromosome maintenance proteins upon Cdt1 overexpres
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