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1 ake possible cohesive vertical inhibition of minicolumns.
2 d pathological approaches involving cortical minicolumns.
3 tended to segregate into vertically oriented minicolumns 50-100 microm wide and at least 500 microm h
5 sly described, employs the use of disposable minicolumns and a single resin washing step using a weak
6 humans the prefrontal cortex possesses wider minicolumns and more neuropil space than other cortical
7 lts in white matter; underdeveloped cortical minicolumns; and reduced functional activation during so
8 ments, it was estimated that (i) directional minicolumns are approximately 30 mum in width, (ii) mini
10 tion of the anatomical basis of the cortical minicolumn, as well as its physiological properties, the
11 callosal white matter was also investigated; minicolumn asymmetry of both Heschl's gyrus and planum t
12 k neurons over scales of tens to hundreds of minicolumns, but on the scale of a few minicolumns, ense
13 the proper formation of prefrontal cortical minicolumns by regulating the expression of genes involv
15 ptures analyte species on a preconcentrating minicolumn containing a selective solid-phase sorbent.
16 ponding developmental disturbance (ie, large minicolumns) could account for the perceptual errors obs
18 ds of minicolumns, but on the scale of a few minicolumns, ensembles of neurons form complex subnetwor
19 ysiological properties, the potential of the minicolumn has not been exploited in fields such as comp
23 red to subcortical nuclei, the design of the minicolumn is a distinctive form of module that has evol
26 t determined through online leaching of rice minicolumns (maintained at 37 degrees C) sequentially wi
27 It is suggested that wider spacing of the minicolumns may relate to the enhanced discrimination se
29 ession of the MS signals for other lipids, a minicolumn of a silica gel cation exchanger was used to
30 The present study specifically assessed the minicolumns of cells that span the depth of the cortex i
31 pendent Ca(2+) transients are instructive to minicolumn organization because Crispr/Cas9-mediated mut
34 y 200 microm apart and were interleaved with minicolumns representing nearly orthogonal PDs; in addit
38 ccur in doublets or triplets, and (iii) such minicolumns tend to repeat every approximately 240 mum (
39 d planum temporale and the separation of the minicolumns that are held to be the structural units of
45 the minicolumnar arrangement of the cortex (minicolumn width, spacing and width of the associated ax
47 umns are approximately 30 mum in width, (ii) minicolumns with similar preferred directions tend to oc
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