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1 fect, but prevented stimulation by 10 microM minoxidil.
2 l, nicardipine, nifedipine, enalaprilat, and minoxidil.
3                                              Minoxidil (0.1-100 microM, p<0.001), NNC 55-0118 (1 mM,
4 w- to moderate-quality evidence that topical minoxidil (2% and 5%) was associated with improvements i
5                               Treatment with minoxidil, an inhibitor of hypoxia-induced sarcoma metas
6 ve potassium (K(ATP)) channel openers, e.g., minoxidil and diazoxide, can induce hair growth, their m
7        These K(+) currents were augmented by minoxidil and pinacidil and attenuated by glibenclamide
8  contain K(ATP) channels that can respond to minoxidil and that tolbutamide may suppress hair growth
9                                              Minoxidil and tolbutamide, a K(ATP) channel blocker, opp
10                      K(ATP) channel openers (minoxidil, cromakalim, and pinacidil) increased cellular
11 0.001) and abolished the effect of 10 microM minoxidil, diazoxide and NNC 55-0118; glibenclamide (10
12             Despite the long-standing use of minoxidil for balding, its mechanism is unclear; suggest
13 opecia may begin in adolescence, and topical minoxidil is effective at retarding further hair loss.
14 s well as mTOR-independent carbamazepine and minoxidil, markedly attenuated cyst formation and restor
15 ATP) channels, Kir6.2/SUR1 and Kir6.1/SUR2B; minoxidil only stimulates SUR2 channels.
16            Although an arterial vasodilator (minoxidil) resulted in significant pulmonary vasodilatio
17 avenous infusion of KCO912 (K(ATP) agonist), minoxidil sulfate (K(ATP) agonist) or NS1619 (K(Ca) agon
18       In a rat brain tumor model, we infused minoxidil sulfate (MS), a selective K(ATP) channel activ
19 ranscription quantitative PCR indicated that minoxidil sulfotransferase mRNA is also up-regulated in
20        Northern blot analysis indicated that minoxidil sulfotransferase mRNA is up-regulated in the f
21                 The gene encoding the enzyme minoxidil sulfotransferase, an enzyme that catalyzes the
22  openers (diazoxide, pinacidil, chromakalim, minoxidil, testosterone) of the putative mitoKATP were a
23                     The commonest treatment, minoxidil, was originally an antihypertensive drug that

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