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1 fect, but prevented stimulation by 10 microM minoxidil.
2 l, nicardipine, nifedipine, enalaprilat, and minoxidil.
4 w- to moderate-quality evidence that topical minoxidil (2% and 5%) was associated with improvements i
6 ve potassium (K(ATP)) channel openers, e.g., minoxidil and diazoxide, can induce hair growth, their m
8 contain K(ATP) channels that can respond to minoxidil and that tolbutamide may suppress hair growth
11 0.001) and abolished the effect of 10 microM minoxidil, diazoxide and NNC 55-0118; glibenclamide (10
13 opecia may begin in adolescence, and topical minoxidil is effective at retarding further hair loss.
14 s well as mTOR-independent carbamazepine and minoxidil, markedly attenuated cyst formation and restor
17 avenous infusion of KCO912 (K(ATP) agonist), minoxidil sulfate (K(ATP) agonist) or NS1619 (K(Ca) agon
19 ranscription quantitative PCR indicated that minoxidil sulfotransferase mRNA is also up-regulated in
22 openers (diazoxide, pinacidil, chromakalim, minoxidil, testosterone) of the putative mitoKATP were a
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