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1 e icosahedral T=1 capsids and virions of the minute virus of mice.
3 ing a truncated version of NS1, as seen with minute virus of mice and adeno-associated virus 2, was a
5 9 promoter was detected; however, similar to minute virus of mice and other members of the Parvovirus
6 s the interaction between the NS2 protein of minute virus of mice and the nuclear export factor Crm1
7 e parvovirus (ADV), canine parvovirus (CPV), minute virus of mice, and bovine parvovirus, also bind s
10 spectrometry to analyze the dynamics of the minute virus of mice capsid at increasing temperatures.
12 rated pre-mRNAs of the autonomous parvovirus minute virus of mice caused a decrease in the accumulate
14 parvovirus capsids, along with those of the minute virus of mice, have also advanced our understandi
15 rus (PRV), adeno-associated virus (AAV), and minute virus of mice i-strain (MVMi) and MVMp all infect
17 he P38 promoter of the autonomous parvovirus minute virus of mice is strongly transactivated by the n
18 rvovirus) and prototypic parvovirus species (minute virus of mice, Kilham's rat virus, and H-1) than
19 was constructed, based on a chimera between minute virus of mice (MVM) and LuIII, which expresses Bo
20 ng DNA replication, the hairpin telomeres of Minute Virus of Mice (MVM) are extended and copied to cr
21 e of the simplest virus particles known, the minute virus of mice (MVM) capsid, and experimentally an
23 o initiate DNA synthesis, the NS1 protein of minute virus of mice (MVM) first binds to a simple cogna
24 igin of DNA replication at the 3' end of the minute virus of mice (MVM) genome and functions as an es
26 ns surrounding the fivefold-symmetry axes in minute virus of mice (MVM) harbor central pores that pen
27 ion.IMPORTANCE Replication of the parvovirus minute virus of mice (MVM) induces a sustained cellular
29 The small nonstructural protein NS2 of the minute virus of mice (MVM) is required for efficient vir
30 The DNA damage response to infection with minute virus of mice (MVM) leads to activated p53; howev
36 d vectors based on the autonomous parvovirus minute virus of mice (MVM) that were designed to introdu
37 he Fab molecules inhibits the binding of the minute virus of mice (MVM) to permissive cells but can a
38 ral protein NS1 of the autonomous parvovirus minute virus of mice (MVM), a protein essential for vira
39 eningitis virus, an ambisense RNA virus, and minute virus of mice (MVM), a single-stranded DNA (ssDNA
42 cleotide NS2-specific exon of the parvovirus minute virus of mice (MVM), which is flanked by a large
49 rine fibroblasts, the lymphotropic strain of minute virus of mice (MVMi) does not efficiently produce
50 he lymphotropic, immunosuppressive strain of minute virus of mice (MVMi) in complex with the neutrali
51 l rearrangement that can occur in parvovirus minute virus of mice (MVMp) virions following prolonged
52 nity for CRM1 that is 100-fold less than the minute virus of mice NS2 protein, a high affinity cargo
53 ing enhancer within the NS2-specific exon of minute virus of mice P4 promoter-generated pre-mRNA caus
54 revealed that induced disassembly of single minute-virus-of-mice particles is frequently initiated b
55 ependovirus (AAV) and autonomous parvovirus (minute virus of mice) replication centers can colocalize
56 s to those observed in canine parvovirus and minute virus of mice, two members of the genus Parvoviru
58 ranscriptional attenuation of the parvovirus minute virus of mice, which is mediated by a stem-loop s
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