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1 eased by the antidepressants desipramine and mirtazapine.
2 n), (3) 1 mg/kg mirtazapine, or (4) 5 mg/ kg mirtazapine.
3 rence 1.17, 95% CI -0.23 to 2.58; p=0.10) or mirtazapine (0.01, -1.37 to 1.38; p=0.99), or between pa
6 ndent model of thermoregulatory dysfunction, mirtazapine (10 mg/kg, i.p.) induced an increase in tail
10 ive sertraline (target dose 150 mg per day), mirtazapine (45 mg), or placebo (control group), all wit
14 reated with the serotonin reuptake inhibitor mirtazapine and remains neurologically stable, with reso
15 .38; p=0.99), or between participants in the mirtazapine and sertraline groups (1.16, -0.25 to 2.57;
16 ) and atypical antidepressants (agomelatine, mirtazapine), and significant negative affective biases
17 maceuticals (efavirenz (EFV), acetaminophen, mirtazapine, and galantamine) prescribed for indications
22 tidepressants (eg, nefazodone hydrochloride, mirtazapine, bupropion hydrochloride, and venlafaxine hy
23 ovariectomized (OVX) rats has suggested that mirtazapine can alleviate thermoregulatory dysfunction b
24 significantly more among participants taking mirtazapine compared with those taking placebo (number o
29 , on day 11, rats received a pretreatment of mirtazapine, followed 30min later by a challenge injecti
30 ertraline group (46 of 107, 43%; p=0.010) or mirtazapine group (44 of 108, 41%; p=0.031), and fewer s
31 p=0.023), but not in a combined venlafaxine-mirtazapine group (n=140; accuracy 51.4%, p=0.53), sugge
32 treat analysis, participants assigned to the mirtazapine group had fewer methamphetamine-positive uri
34 up and the extended-release venlafaxine plus mirtazapine group, and the rates were not statistically
35 cific (5-HT1) serotonergic transmission, and mirtazapine has therefore been termed a noradrenergic an
36 atment with extended-release venlafaxine and mirtazapine in patients with treatment-resistant major d
37 ake inhibitors, nefazodone, venlafaxine, and mirtazapine) in participants younger than 19 years with
38 at the highest dose tested (10 mg/kg, i.p.), mirtazapine induced a small but significant decrease in
42 at functional blockade of these receptors by mirtazapine is not a likely mechanism for restoring ther
43 injection of: (1) saline, (2) 0.1 mg/kg +/- mirtazapine (labeled as Remeron), (3) 1 mg/kg mirtazapin
44 Collectively, these results indicate that mirtazapine may help to maintain abstinence in opioid de
45 Antidepressant therapy with 15 to 45 mg/d of mirtazapine (n = 124) or 20 to 40 mg/d of paroxetine (n
47 cific (non-SSRI) antidepressants (bupropion, mirtazapine, nefazodone, and venlafaxine), tricyclic ant
48 etine, escitalopram, fluoxetine, imipramine, mirtazapine, nefazodone, nortriptyline, paroxetine, sert
49 italopram oxalate, fluoxetine hydrochloride, mirtazapine, nortriptyline hydrochloride, paroxetine hyd
50 ive serotonin reuptake inhibitor [SSRI]) and mirtazapine (not an SSRI) in 246 elderly patients with m
51 esent study were to reexamine the effects of mirtazapine on temperature regulation in OVX rat models
54 of NREM sleep was unaffected by any dose of mirtazapine (p = 0.42), but NREM EEG delta power was inc
55 opram, fluoxetine, fluvoxamine, milnacipran, mirtazapine, paroxetine, reboxetine, sertraline, and ven
61 rictions, and ease of use of venlafaxine and mirtazapine suggest that this combination may be preferr
62 urrent investigation assessed the ability of mirtazapine to ameliorate morphine-induced behaviors.
65 study compared the efficacy of switching to mirtazapine to that of switching to a tricyclic antidepr
69 domly assigned to 14 weeks of treatment with mirtazapine (up to 60 mg/day) (N=114) or nortriptyline (
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