ライフサイエンスコーパス: mitis

1 a protective function for the capsule in S. mitis.

2 aphylococcus and the other for Streptococcus mitis.

3 both platelet binding and aggregation by S. mitis.

4 of Selenomonas, Neisseria, and Streptococcus mitis.

5 the mitilysin gene from seven isolates of S. mitis.

6 els of bacteremia caused predominantly by S. mitis.

7 ver, hybridized to DNA from S. oralis and S. mitis.

8 China was caused by a toxigenic clone of S. mitis.

9 regions contribute to platelet binding by S. mitis.

10 cus gordonii Blackburn, 10558, Streptococcus mitis 10712, 903, Streptococcus oralis 10557, 9811, and

11 Streptococcus spp. were found, including S. mitis (25 strains, 50.0% of 50); currently unnamed Strep

12 Recently, Streptococcus mitis, a human commensal and a close relative of S. pneu

13 mutant showed that the capsule protected S. mitis against phagocytosis by RAW 264.7 macrophages.

14 antigens in certain strains of Streptococcus mitis and Eikenella corrodens.

15 No differences in the tox regions between mitis and gravis biotype strains were observed.

16 oxigenic Corynebacterium diphtheriae of both mitis and gravis biotypes, showing that the organism is

17 ups demonstrated increased proportions of S. mitis and S. oralis by day 1.

18 eumococci and the closely related species S. mitis and S. oralis, showing up to 10.4% nucleotide dive

19 han to most CSPs previously reported from S. mitis and S. oralis, suggesting that these particular or

20 ae, isolates phenotypically identified as S. mitis and S. oralis, which included isolates previously

21 l CSPs from disease-associated strains of S. mitis and S. oralis.

22 shared by eight species in the Streptococcus mitis and Streptococcus anginosus groups, is regulated b

23 s particularly problematic for Streptococcus mitis and Streptococcus oralis isolates.

24 ted oral streptococcal species Streptococcus mitis and Streptococcus oralis on the basis of three dif

25 Together, Streptococcus mitis and Streptococcus oralis were significantly more l

26 enetic exchange is known to occur between S. mitis and Streptococcus pneumoniae, this finding may hav

27 Eight species including Streptococcus mitis and Streptococcus species HOT070 were prevalent in

28 ns of S. gordonii, S. sanguis, S. mutans, S. mitis, and S. oralis but only weakly by S. salivarius.

29 characterized isolates of S. pneumoniae, S. mitis, and S. oralis.

30 killed Pseudomonas aeruginosa, Streptococcus mitis, and Streptococcus pneumoniae in a dose-dependent

31 S. mitis appear to release mitilysin extracellularly.

32 ncorrect identification (e.g., Streptococcus mitis), as did matrix-assisted laser desorption ionizati

33 The organism was identified as Streptococcus mitis based on biochemical and 16S rRNA sequence analyse

34 of multiple transposases in a Streptococcus mitis biofilm when the periodontopathogen P. gingivalis

35 , and dtxR type 1 was characteristic for the mitis biotype strains.

36 rains (62 of the gravis biotype and 4 of the mitis biotype) isolated during the Georgian diphtheria e

37 the pioneer oral streptococci Streptococcus mitis biovar 1 and Streptococcus oralis, the late oral c

38 from Streptococcus oralis and Streptococcus mitis biovar 1 strains but were cleaved to various degre

39 the components in unripe calamondin (Citrus mitis Blanco) peel were investigated by performing bioas

40 ds of mature and immature calamondin (Citrus mitis Blanco) peel were investigated.

41 d genetically are most closely related to S. mitis but which harbor genes encoding the virulence dete

42 we hypothesize that P. gingivalis induces S. mitis cell death by an unknown mechanism, shaping the or

43 In a multivariate analysis, S. aureus, S. mitis, Corynebacterium accolens, and bacilli were signif

44 demonstrating that P. gingivalis induces S. mitis death and DNA fragmentation in an in vitro biofilm

45 mmon among the strains of S. gordonii and S. mitis examined.

46 the toxic effect of E. corrodens extract S. mitis extracts contained a single, strongly reactive ant

47 ity was produced only by some members of the mitis group (Streptococcus mitis, Streptococcus oralis,

48 Eighty-four species verified mitis group isolates were subjected to our bile solubili

49 In general, Streptococcus mitis group organisms are resistant to more antimicrobia

50 ptococcus pneumoniae from nonpneumococcal S. mitis group species.

51 neumoniae strains but misidentified 13 other mitis group strains.

52 pseudopneumoniae but misidentified 16 other mitis group strains.

53 The mitis group streptococci (MGS) are widespread in the ora

54 ntiated S. pneumoniae from all but one other mitis group streptococci (one S. mitis isolate generated

55 ation of Streptococcus pneumoniae from other mitis group streptococci, including differentiation of S

56 communities rich in Candida are also rich in mitis group Streptococci,a community pattern associated

57 newly described member of the Streptococcus mitis group.

58 ecies Streptococcus mutans and Streptococcus mitis, however, lactoferrin containing Lys at position 2

59 l from the chamber compared to cells from S. mitis-infected chambers.

60 These results suggest that S. mitis is the most common cause of viridans streptococcal

61 t one other mitis group streptococci (one S. mitis isolate generated an OD-value above 2.1).

62 tes did fall into a well-separated group, S. mitis isolates did not cluster into a well-separated gro

63 itilysin, in a small number of Streptococcus mitis isolates.

64 cell wall polysaccharide from Streptococcus mitis J22 are correlated with individual glycosidic dihe

65 e show that while the polysaccharide from S. mitis J22 is flexible, requiring multiple conformations,

66 oral streptococci, including isolates of S. mitis known to possess pneumolysin and autolysin.

67 tralization assay results, one isolate of S. mitis may produce a further hemolytic toxin in addition

68 rom ten JEB patients (JEB gravis, n = 4; JEB mitis, n = 3; JEB plus pyloric atresia [JEB/PA], n = 3)

69 7.7 years) from an outbreak of Streptococcus mitis/oralis endophthalmitis after bevacizumab injection

70 S. mitis/oralis endophthalmitis is a devastating complicati

71 presence of a common strain of Streptococcus mitis/oralis in vitreous specimens and 7 unused syringes

72 vitreal bevacizumab injection, Streptococcus mitis/oralis was cultured from the majority of patients

73 ular testing confirmed a common strain of S. mitis/oralis.

74 affected patients also were positive for S. mitis/oralis.

75 10 patients were positive for Streptococcus mitis/oralis.

76 nts, and 13 of those cases were caused by S. mitis (P = 0.007).

77 bacterium acnes phage PA6, and Streptococcus mitis phage SM1.

78 The oral streptococcal group (mitis phylogenetic group) currently consists of nine rec

79 e were followed by a switch to Streptococcus mitis predominance after 3 months of age.

80 Under such conditions, S. mitis resistance to clearance could be enhanced by capsu

81 ogenic oral bacterial species, Streptococcus mitis, resulted in well-controlled infection, with bacte

82 Oral taxon 44, while Streptococcus mitis, Rothia mucilaginosa and Haemophilus parainfluenza

83 gainst challenge by the oral streptococci S. mitis, S. mutans, and S. salivarius.

84 ion observed for other species, including S. mitis, S. oralis, and S. pseudopneumoniae.

85 We found that S. mitis, S. oralis, and S. sanguis, as well as oral actino

86 SSA-3 hybridized to DNA from S. gordonii, S. mitis, S. oralis, S. parasanguinis, and S. vestibularis.

87 The LLY gene was identified in strains of S. mitis, S. pneumoniae, and Streptococcus pseudopneumoniae

88 icantly decreased included the Streptococcus mitis-S. pneumoniae-S. infantis group, Corynebacterium m

89 ks, and 6 mos, and were cultured on modified Mitis Salivarius agar for mutans streptococci and on blo

90 occi (MS) on mitis-salivarius-bacitracin and mitis-salivarius agar; (2) non-mutans streptococci (non-

91 gar; (2) non-mutans streptococci (non-MS) on mitis-salivarius agar; (3) organisms that were categoriz

92 lood agar or the predominant non-MS flora on mitis-salivarius agar; and (4) iodophilic polysaccharide

93 bjects were: (1) mutans streptococci (MS) on mitis-salivarius-bacitracin and mitis-salivarius agar; (

94 Platelet binding by Streptococcus mitis SF100 is mediated in part by a lysin encoded by th

95 atelet aggregation factor from Streptococcus mitis (Sm-hPAF) was characterized and shown to be a func

96 This effect was enhanced in the S. mitis strain expressing the S. pneumoniae capsule, which

97 Platelet binding by Streptococcus mitis strain SF100 (an endocarditis isolate) was recentl

98 d a Tn916deltaE-derived mutant library of S. mitis strain SF100 for reduced binding to human platelet

99 Platelet binding by Streptococcus mitis strain SF100 is mediated in part by two bacterioph

100 e prophage-encoded proteins of Streptococcus mitis strain SF100 that mediate binding to human platele

101 . mitis strains (28.0%, 7/25) and not non-S. mitis strains (0/25) (P = 0.004).

102 MIC, 4 to 12 mug/ml) was noted only among S. mitis strains (28.0%, 7/25) and not non-S. mitis strains

103 When the direct binding of S. mitis strains SF100 and PS344 to immobilized ganglioside

104 Significantly more S. mitis strains than non-S. mitis strains were resistant t

105 nificantly more S. mitis strains than non-S. mitis strains were resistant to fluoroquinolones and to

106 iae, Streptococcus oralis, and Streptococcus mitis strains.

107 lassification schemes within members of the "mitis" streptococcal group.

108 gher counts and proportions of Streptococcus mitis, Streptococcus oralis, and Streptococcus mutans, w

109 guinis, Abiotrophia defectiva, Streptococcus mitis, Streptococcus oralis, and Streptococcus sanguinis

110 me members of the mitis group (Streptococcus mitis, Streptococcus oralis, Streptococcus gordonii, Str

111 5 clinical blood cultures with Streptococcus mitis/Streptococcus oralis and 1/3 blood cultures spiked

112 S. mitis survival in horse blood or in a mouse model of bac

113 Since SM1 is the first prophage of S. mitis that has been identified and because of the possib

114 8) also inhibited the binding in vitro of S. mitis to human fibrinogen and platelets.

115 The direct binding of Streptococcus mitis to human platelets is mediated in part by two prot

116 life cycle, lysin mediates the binding of S. mitis to human platelets via its interaction with fibrin

117 pblA and pblB mediate the attachment of S. mitis to platelets and play a significant role in S. mit

118 lpA (5 to 100 microg/ml), from Streptococcus mitis, to induce the production of proinflammatory cytok

119 hen species (Evernia mesomorpha and Cladonia mitis), two vascular plant species (Rhododendron groenla

120 A recessively inherited variant, the mitis type of DEB (M-RDEB), is characterized by a mild p

121 showed higher rates of survival than the S. mitis type strain or the capsule-switching mutant, excep

122 In this study, the S. mitis type strain switched capsule by acquisition of the

123 ing in either a deficiency (in the nonlethal mitis variety) or a complete absence (in lethal Herlitz-

124 eslundii, Lactobacillus casei, Streptococcus mitis, Veillonella parvula, and Fusobacterium nucleatum)

125 platelets and play a significant role in S. mitis virulence in the endocardium, but have never previ

126 at mediate platelet binding by Streptococcus mitis, we screened a Tn916deltaE-derived mutant library

127 All 8 isolates of Streptococcus mitis were misidentified as being Streptococcus pneumoni

128 o HL60 cells, whereas similar extracts of S. mitis were nontoxic.

129 cells in chambers from mice infected with S. mitis were PI positive (apoptotic) or negative (live).

130 cterium diphtheriae strains; six were biovar mitis, which were associated with recent travel abroad.

131 ng oral streptococci including Streptococcus mitis (with the exception of 1 of 14 strains), Streptoco