ライフサイエンスコーパス: mitochondrial gene

1 mains, and coopted the promoter of a genuine mitochondrial gene.

2 fragments (amplicons <200 base pairs) of the mitochondrial gene.

3 dinucleotide dehydrogenase subunit IV (ND4) mitochondrial gene.

4 tem for the proper expression of nuclear and mitochondrial genes.

5 rial NADH kinase, is a mutator, specific for mitochondrial genes.

6 ere assigned to key metabolic and regulatory mitochondrial genes.

7 sed oxygen consumption and the expression of mitochondrial genes.

8 n of some stress-related and nuclear-encoded mitochondrial genes.

9 ete and seven partial sets of protein coding mitochondrial genes.

10 itochondrial DNA hinder the transcription of mitochondrial genes.

11 ypes in rare diseases caused by mutations in mitochondrial genes.

12 mino acid homology of 91-97% among the seven mitochondrial genes.

13 receptor-coactivator 1alpha (PGC-1alpha) and mitochondrial genes.

14 es not regulate the effects of PGC-1alpha on mitochondrial genes.

15 henotypes, reflecting depletion of essential mitochondrial genes.

16 -S and cms-T cytotypes is linked to chimeric mitochondrial genes.

17 FKBIA, for activated pathways and RICTOR for mitochondrial genes.

18 expression, whereas having little effect on mitochondrial genes.

19 lar molecule of 16,065 bp and encodes the 37 mitochondrial genes (13 protein-coding, 22 tRNAs, and tw

20 Mitochondrial genes accumulate substitutions more quickl

21 responses (and suppression of metabolism and mitochondrial genes) akin to those observed when mice ar

22 Finally, more variable mitochondrial genes also exhibit a lower identity with n

23 ation and aggregation, splicing changes of a mitochondrial gene and mitochondrial defects.

24 ng a platform for introduction of almost any mitochondrial gene and perhaps even allowing insertion o

25 lic acid cycle and oxidative phosphorylation mitochondrial genes and (b) reduced oxygen consumption.

26 All of them displayed 37 typical animal mitochondrial genes and an A+T-rich region.

27 receptor, the glucocorticoid receptor, p11, mitochondrial genes and cannabinoids are bringing new pe

28 order may include the immediate early genes, mitochondrial genes and epigenetic mechanisms, although

29 We explore the possibility of re-engineering mitochondrial genes and expressing them from the nucleus

30 tion was also critical for the expression of mitochondrial genes and for the processing of self-splic

31 red by TNF as evidenced by downregulation of mitochondrial genes and increased free radical productio

32 on the nucleotide sequences of COI and COII mitochondrial genes and ITS2 spacer of nuclear ribosomal

33 ism and exercise capacity by using different mitochondrial genes and mechanisms in a tissue-specific

34 ssimilation were up-regulated, while diverse mitochondrial genes and other metabolic enzymes were dow

35 Twenty-six nuclear-encoded mitochondrial genes and the functional activities of mit

36 Parallel transfer of mitochondrial genes and their functional replacement by

37 nts, and DNA methylation analysis identified mitochondrial genes and their transcriptional regulators

38 ytoplasmic genomes and recombination between mitochondrial genes and within atp1, implying transient

39 e barcode region in the cytochrome oxidase I mitochondrial gene, and analyzed representative BMSB sam

40 e mostly and abundantly been described using mitochondrial genes, and in particular cytochrome b (cyt

41 le, decreasing expression of nuclear-encoded mitochondrial genes, and increasing extracellular matrix

42 loroplast integrants exist apart from native mitochondrial genes, and only a few, involving chloropla

43 on gene expression of glucose transporter 4, mitochondrial genes, and PGC-1alpha itself are almost en

44 ore of the membrane arm, are translated from mitochondrial genes, and the remaining subunits, the pro

45 rsal GRR include ribosomal, translation, and mitochondrial genes, and those with negative GRR include

46 Given that six mitochondrial genes are located within 22q13, including

47 Because mitochondrial genes are transcribed by a dedicated RNA p

48 regulation, and that several nuclear-encoded mitochondrial genes are under strong translational contr

49 Considerable evidence supports mutations in mitochondrial genes as the cause of maternally inherited

50 tional mechanisms for expressing nuclear and mitochondrial genes as well as unusual subcellular local

51 ociated with energy metabolism, particularly mitochondrial genes, as well as alterations in the expre

52 acts to suppress the expression of abnormal mitochondrial genes associated with cytoplasmic male ste

53 revealed over a dozen previously overlooked mitochondrial genes at the level of eukaryotic supergrou

54 There was remarkably high variation in two mitochondrial genes (atp1, atp9) and additional variatio

55 single-nucleotide polymorphism (SNP) in the mitochondrial gene, atp1.

56 Here, the inheritance of the mitochondrial genes atpA and coxI was studied in 318 Sil

57 which lineage sorting has taken place in the mitochondrial genes but not yet in the nuclear alleles.

58 of recurrent conversion of short patches of mitochondrial genes by chloroplast homologs during angio

59 genus Encope, based on one nuclear and four mitochondrial genes, calibrated with fossils at multiple

60 Mutations in nDNA-encoded mitochondrial genes can also disrupt OXPHOS, alter mtDNA

61 P2, alpha-galactosidase A (GLA), and several mitochondrial genes can cause rare familial cardiomyopat

62 ranscriptional regulation of nuclear-encoded mitochondrial genes can result in experimental glomerula

63 tatively linking male sterility to orf293, a mitochondrial gene causing homeotic conversion of anther

64 ed the barriers to transfer of yeast COX2, a mitochondrial gene coding for a subunit of cytochrome c

65 In the present study, we use three mitochondrial genes (COI, ND1 and ND5) and nine microsat

66 We sequenced two mitochondrial genes, COII and COIII, in samples of Mormo

67 A large number of these mitochondrial genes contain binding sites for the transc

68 he altered dosage of one or several of these mitochondrial genes contributing to 22qDS etiology and/o

69 ve was to determine the relationship between mitochondrial gene copy number and transcript abundance

70 Mitochondrial gene copy number decreased from 3 to 8-DPA

71 ication of these sublimons allows individual mitochondrial gene copy numbers to vary independently be

72 most strains of Saccharomyces cerevisiae the mitochondrial gene COX1, for subunit 1 of cytochrome oxi

73 d processed to produce DNA barcodes from the mitochondrial gene cytochrome c oxidase I (COI).

74 ty (h) and nucleotide diversity (pi) for the mitochondrial gene cytochrome c oxidase subunit I (cox1)

75 bfamilies of Chinese skippers based on three mitochondrial genes (cytochrome b (Cytb), the NADH dehyd

76 species delimitation techniques based on two mitochondrial genes (cytochrome c oxidase I and 16S rRNA

77 We show that the barcoding region of the mitochondrial gene, cytochrome oxidase subunit I (COI),

78 mparative approach in which we sequenced two mitochondrial genes, cytochrome b (cob) and cytochrome o

79 lly, expression of PGC-1-and nuclear-encoded mitochondrial genes decreased after acipimox, and expres

80 of the remaining patients (2.2% overall) had mitochondrial gene deletions consistent with Pearson mar

81 Phylogenetic analysis of plastid and mitochondrial genes demonstrated that the three endosymb

82 Expression of yeast mitochondrial genes depends on specific translational ac

83 In plants, mitochondrial genes directly influence pollen developmen

84 Changes in expression of nuclear-encoded mitochondrial genes do not always correlate with changes

85 nduction is accompanied by the expression of mitochondrial genes (e.g., TFAM, MtCO1) and increased mi

86 Because mitochondrial genes encode proteins essential for aerobi

87 By mutating or deleting mitochondrial genes encoded in the mtDNA [NADH dehydroge

88 visual loss when caused by a mutation in the mitochondrial gene encoding NADH:ubiquinone oxidoreducta

89 forms using the nucleotide sequences of the mitochondrial gene encoding the cytochrome c oxidase sub

90 itochondrial targeting sequence to carry the mitochondrial gene encoding the human NADH ubiquinone ox

91 s display aberrant expression of nuclear and mitochondrial genes encoding subunits of complex V resul

92 mto2 mutants is the defective expression of mitochondrial genes, especially CYTB and COX1, but only

93 Knowledge of mitochondrial gene evolution in angiosperms has taken a

94 Some mitochondrial genes exhibit atypically high degrees of n

95 ly of mTERF proteins whose members influence mitochondrial gene expression and DNA replication.

96 ) gamma agonist, by systematically analyzing mitochondrial gene expression and function in two mouse

97 trated that the action of TORCs in promoting mitochondrial gene expression and function requires PGC-

98 and saturated fatty acids decrease PGC-1 and mitochondrial gene expression and function via p38 MAPK-

99 Mitochondrial gene expression and function were not indu

100 However, PPARdelta agonism does not induce mitochondrial gene expression and function.

101 are accompanied by correspondingly increased mitochondrial gene expression and higher expression of P

102 down of YY1 caused a significant decrease in mitochondrial gene expression and in respiration, and YY

103 o statistically significantly down-regulates mitochondrial gene expression and induces oxidative stre

104 equired for the full CR-induced increases in mitochondrial gene expression and mitochondrial density

105 s role in post-transcriptional regulation of mitochondrial gene expression and more recently because

106 Akt3, but not Akt1, results in a decrease in mitochondrial gene expression and mtDNA content.

107 piratory factors, resulting in a decrease in mitochondrial gene expression and oxygen consumption.

108 A new study reveals that decreased mitochondrial gene expression and reduced lipid biosynth

109 ard to the mechanism and regulation of human mitochondrial gene expression and the potential multi-fu

110 ding of the molecular processes that mediate mitochondrial gene expression and the structure-function

111 Defects in mitochondrial gene expression are associated with aging

112 Similar effects on PGC-1beta and mitochondrial gene expression are seen in vivo; fraction

113 encoding a variety of proteins required for mitochondrial gene expression at both the transcriptiona

114 translational regulation of nuclear-encoded mitochondrial gene expression by 4E-BP plays an importan

115 Taken together, ClpXP regulates mitochondrial gene expression by changing the protein le

116 ) reversal of palmitate effects on PGC-1 and mitochondrial gene expression by p38 MAPK inhibitors, an

117 nerated depends on the precise nature of the mitochondrial gene expression defect and initiates a dow

118 owever, mechanisms through which compromised mitochondrial gene expression elicits the reported varie

119 In addition, we observed 40% residual mitochondrial gene expression in FRDA patient blood.

120 oteins that interact with POLRMT to regulate mitochondrial gene expression in humans.

121 ated pattern of reduction in nuclear-encoded mitochondrial gene expression in insulin resistance.

122 natural competence will allow us to explore mitochondrial gene expression in organello and provides

123 vulgaris an excellent model for the study of mitochondrial gene expression in plants.

124 down of PPARdelta affects PGC-1alpha-induced mitochondrial gene expression in primary myotubes.

125 d cannot defend their ATP levels or increase mitochondrial gene expression in response to reduced oxi

126 ck cycles may provide switch-like control of mitochondrial gene expression in response to the metabol

127 plicing isoform NT-PGC-1alpha, which control mitochondrial gene expression in the nucleus.

128 Mitochondrial gene expression in Trypanosoma brucei invo

129 RNA editing are essential for regulation of mitochondrial gene expression in Trypanosoma brucei.

130 h the synthetic ligand, GW501516, on FAO and mitochondrial gene expression in vitro and in vivo.

131 uced inflammation and surprisingly increased mitochondrial gene expression in white adipose tissue al

132 ription-PCR was used to determine changes in mitochondrial gene expression induced by MMC treatment.

133 Mitochondrial gene expression is a fundamental process t

134 nder strong evolutionary constraint and that mitochondrial gene expression is highly conserved across

135 In ANT1-deficient skeletal muscle, mitochondrial gene expression is induced in association

136 The induction of mitochondrial gene expression is more dependent on DMF's

137 Mitochondrial gene expression is necessary for proper mi

138 limited knowledge concerning the details of mitochondrial gene expression is surprising.

139 Consequent abnormal mitochondrial gene expression may contribute to the deve

140 heir interactions with other elements of the mitochondrial gene expression pathway.

141 Outbreak strains share a unique mitochondrial gene expression profile and an increased a

142 s recently emerged as a central regulator of mitochondrial gene expression through the function of an

143 of the molecular processes mediating altered mitochondrial gene expression to dysfunction associated

144 Mitochondrial gene expression was increased in adipose t

145 Reduced mitochondrial gene expression was specific for neurons.

146 integrity, preserved redox potential-coupled mitochondrial gene expression, and improved respiratory

147 e analysis revealed that YY1 is required for mitochondrial gene expression, and ultrastructural analy

148 n complexes that regulate different steps in mitochondrial gene expression, at least one of which doe

149 ed many factors central to the mechanisms of mitochondrial gene expression, but being able to investi

150 African sleeping sickness, normally requires mitochondrial gene expression, despite the absence of ox

151 Post-transcriptional control of mitochondrial gene expression, including the processing

152 Concurrent with these changes in mitochondrial gene expression, lower O(2) consumption wa

153 increase is accompanied by downregulation of mitochondrial gene expression, similar to that observed

154 ction, parallel to other factors controlling mitochondrial gene expression, such as NRF1 and NRF2/GAB

155 cts are independent of the downregulation of mitochondrial gene expression, suggesting novel mechanis

156 ed as key post-transcriptional regulators of mitochondrial gene expression.

157 to the relevance of RNA pseudouridylation in mitochondrial gene expression.

158 proach is likely to be useful for studies of mitochondrial gene expression.

159 factor is an essential protein necessary for mitochondrial gene expression.

160 tribute to between-individual variability in mitochondrial gene expression.

161 s known about translational control of plant mitochondrial gene expression.

162 tics suggested that ABCC6 deficiency affects mitochondrial gene expression.

163 RNA polymerase, is essential for maintaining mitochondrial gene expression.

164 putative higher order structures coordinate mitochondrial gene expression.

165 f acetyl-CoA, and which in turn may regulate mitochondrial gene expression.

166 tion and/or ribosome biogenesis during human mitochondrial gene expression.

167 N, including higher STAT1 levels and reduced mitochondrial gene expression.

168 of mitochondrial biogenesis or induction of mitochondrial gene expression.

169 e-repeat proteins as key regulators of plant mitochondrial gene expression.

170 NA mutations would increase our knowledge of mitochondrial gene expression.

171 eutics by manipulating nuclear regulation of mitochondrial gene expression.

172 f post-transcriptional mRNA modification for mitochondrial gene expression.

173 al or nuclear genes able to cause defects in mitochondrial gene expression.

174 at Cyp-D could play a key role in regulating mitochondrial gene expression.

175 volved in post-transcriptional regulation of mitochondrial gene expression.

176 on, we examined whether Cyp-D could regulate mitochondrial gene expression.

177 Comparison of the mitochondrial gene flanking sequences from normal fertil

178 Conversely, mitochondrial gene flow is more extensive between non-Af

179 Mutations in the nDNA-encoded mitochondrial genes for fumarate hydratase and succinate

180 redicted genes, our data identified nine new mitochondrial genes, four of which encode proteins that

181 lus nasutus is due to interactions between a mitochondrial gene from M. guttatus and two tightly link

182 quence the full complement of protein coding mitochondrial genes from pooled samples using the 454/Ro

183 s from glass sponges and examined individual mitochondrial genes from three others.

184 NA targets in cells and the brain, including mitochondrial genes, genes involved in glutathione metab

185 n of both positive and negative selection on mitochondrial genes has been put forward, and the comple

186 pathogenic mutations in both mtDNA and nDNA mitochondrial genes have been identified in the past 21

187 Patterns of amino-acid polymorphism in human mitochondrial genes have been interpreted as evidence fo

188 ene content, yet it is not known why not all mitochondrial genes have been transferred to the nuclear

189 , and a few animals (where historically, the mitochondrial genes have dominated species studies).

190 dministration up-regulated the expression of mitochondrial genes in AML-12 hepatocytes, with or witho

191 The rate of evolution of mitochondrial genes in Encope has been ten times slower

192 es differ by 11%-15% from putative thylacine mitochondrial genes in GenBank, with one of our samples

193 ed a significant downregulation of essential mitochondrial genes in glomeruli from diabetic D2 mice,

194 sequencing revealed altered expression of 41 mitochondrial genes in Gq hearts, with normalization of

195 in reduced expression of the thermogenic and mitochondrial genes in mice housed at ambient temperatur

196 g to decreased expression of nuclear-encoded mitochondrial genes in muscle and adipose tissue.

197 s necessary for transcriptional induction of mitochondrial genes in muscle with both RSV and SRT trea

198 Nuclear-encoded mitochondrial genes in particular correlate with protein

199 ns show a diminished capacity to up-regulate mitochondrial genes in response to hypoosmotic stress.

200 that activate expression of nuclear-encoded mitochondrial genes in response to hypoxia, and by loss

201 st study to explore genetic variation in the mitochondrial genes in the context of AIWG.

202 GC-1alpha-dependent transcription of several mitochondrial genes in vivo.

203 target of rapamycin (mTOR) did not suppress mitochondrial genes in YY1mKO mice.

204 ber of TCA cycle enzymes and nuclear-encoded mitochondrial genes including genes involved in oxidativ

205 transcription factors encoded by nuclear and mitochondrial genes, including mitochondrial transcripti

206 A number of nuclear encoded mitochondrial genes, including those in Complex I and IV

207 l outgroup in future evolutionary studies of mitochondrial genes, including those that have transferr

208 d compensatory expression of nuclear-encoded mitochondrial genes, indicating mitochondrial biogenesis

209 n nuclear genes than in maternally inherited mitochondrial genes, indicating that dispersal by males

210 Linezolid AUC0-24 best explained the mitochondrial gene inhibition, with 50% inhibition at 94

211 The technology of introducing human mitochondrial genes into the mouse germ line has never b

212 f nucleic acids into the mitochondrion, nDNA-mitochondrial genes into the nucleus, and mtDNA-encoded

213 cts are particularly puzzling given that the mitochondrial genes involved are under strong evolutiona

214 Mutations in nDNA-encoded mitochondrial genes involved in energy metabolism, antio

215 transcriptional induction of thermogenic and mitochondrial genes involved in energy metabolism.

216 s, the expression levels and copy numbers of mitochondrial genes involved in energy production and re

217 set in filamentous fungi expanded to include mitochondrial genes involved in the tricarboxylic acid (

218 te leaves, an editing site within the ccb206 mitochondrial gene is more highly edited in Col than in

219 Of note, downregulation of mitochondrial genes is associated with the worst clinica

220 eart, regulation of fatty acid oxidation and mitochondrial genes is controlled by the PPARgamma coact

221 o acid replacement to silent substitution in mitochondrial genes is higher in asexual lineages than i

222 Consistently, suppression of mitochondrial genes is identified as a key metabolic sig

223 uclear genes, yet the impact of selection on mitochondrial genes is significantly reduced relative to

224 To date, two mitochondrial gene markers, Cytochrome Oxidase I (COI) a

225 Several mechanisms could be operating: mitochondrial genes, maternal genes, or fetal genes expr

226 on of in vitro evolution strategies to other mitochondrial genes might ultimately lead to yeast entir

227 howed association of NEIL2 and PNKP with the mitochondrial genes MT-CO2 and MT-CO3 (cytochrome c oxid

228 ntioxidant protection contribute to elevated mitochondrial gene mutagenesis in cells lacking the mito

229 ctional evidence for the role of a bona fide mitochondrial gene mutation in cancer.

230 However, the development of mice harboring mitochondrial gene mutations is permitting demonstration

231 One of these processes involves genomic and mitochondrial gene mutations, mitochondrial protein expr

232 e chain reaction (ddPCR) assay targeting the mitochondrial gene nad1 to diagnose schistosomiasis japo

233 Nicotiana sylvestris mutant, CMS, lacks the mitochondrial gene nad7 and functional complex I, and re

234 Two mitochondrial genes, namely, cytochrome b (CYT B) and NA

235 e mitochondrial genome in Tachycineta, three mitochondrial genes (ND2, ND5, and CYTB) contain regions

236 These changes in mitochondrial gene networks were validated by quantitati

237 Here, we analyse the cytochrome oxidase II mitochondrial gene of 250 Sabethes albiprivus B mosquito

238 reviously appreciated and creates functional mitochondrial genes of chimeric origin.

239 Phylogenetic analysis of mitochondrial genes of H. oligactis and other cnidarians

240 port the discovery, in two lineages of plant mitochondrial genes, of novel gene combinations that aro

241 ects on the transcription of nuclear-encoded mitochondrial genes or through direct effects of the mut

242 Mitochondrial gene order within Decemunciger was similar

243 stigated the hypothesis that nuclear-encoded mitochondrial genes, particularly those involved in oxid

244 The mitochondrial gene pool thus may contain signals of loca

245 We found that various mitochondrial genes possessed shorter and less structure

246 we identified mutations in a nuclear-encoded mitochondrial gene, ppr, a homolog of human LRPPRC.

247 te provides predictive RNA editors for plant mitochondrial genes (PREP-Mt), for chloroplast genes (PR

248 how that when there is a benefit to having a mitochondrial gene present in the nucleus, but absent in

249 .004) and within nuclear-encoded genes with mitochondrial gene products (odds ratio, 1.075; 95% CI,

250 Many mitochondrial gene products exhibit extraordinary levels

251 Translation of mitochondrial gene products in Saccharomyces cerevisiae

252 biogenesis in which Cox1p and the two other mitochondrial gene products, Cox2p and Cox3p, constitute

253 Unlike Atp9p, the other mitochondrial gene products, including ATPase subunits A

254 To further our understanding of mitochondrial gene regulation, we identified proteins th

255 complex that coordinates various aspects of mitochondrial gene regulation.

256 y rescues myocyte triglyceride accumulation, mitochondrial gene regulatory derangements, and contract

257 acity and macromolecular interactions of the mitochondrial gene regulatory protein, RBP16.

258 eby coordinate the expression of nuclear and mitochondrial genes relevant to mitochondrial biogenesis

259 Potential clinical applications include mitochondrial gene replacement therapy to prevent transm

260 Mitochondrial gene reprogramming induced by CaMKIIdelta

261 Here we introduced a mutant human mitochondrial gene responsible for Leber hereditary opti

262 Phylogenetic trees inferred from mitochondrial genes retrieve Xenacoelomorpha as an early

263 ived from autosomal, X-linked, Y-linked, and mitochondrial gene segments (22,789 base pairs) and 16 f

264 t elevation in expression of nuclear-encoded mitochondrial gene sets and a mitonuclear protein imbala

265 he hypothesis of resistance exercise-induced mitochondrial gene-shifting in muscle containing satelli

266 and second codon positions) across multiple mitochondrial genes strongly supports a radically differ

267 Both uncouplers increased the expression of mitochondrial genes such as Tfam and COXIV while inducin

268 e expression of a cluster of nuclear-encoded mitochondrial genes, suggesting a role for VEGF in the r

269 Up-regulated mitochondrial genes support in vitro evidence associatin

270 ative stress, unlike many other mutations in mitochondrial genes that are associated with elevated ox

271 iption factor (ETS) that binds and activates mitochondrial genes that are required for electron trans

272 intracellular domain (NICD1) to nuclear and mitochondrial genes that encode respiratory chain compon

273 highly enriched (1.2 x 10(-9)) with nuclear mitochondrial genes that play a role in oxidative phosph

274 e), G6P (glucose-6-phosphatase), and certain mitochondrial genes through PGC-1alpha.

275 (106 and 120-bp) and buffalo (90 and 138-bp) mitochondrial genes to discriminate beef and buffalo in

276 quenced the COII (507 BP) and cyt b (428 BP) mitochondrial genes to examine the intraspecific phyloge

277 A recent study marshals mitochondrial genes to place that root between the enigm

278 The transfer of mitochondrial genes to the nucleus is a recurrent and co

279 Functional transfer of mitochondrial genes to the nucleus is very common in som

280 al and self-fertilizing plants transfer more mitochondrial genes to their nuclei than do outcrossing

281 Using microarray technology, adipose mitochondrial gene transcription was studied in db/db, h

282 pression of stress response genes, exuberant mitochondrial gene transcription, and increased expressi

283 Nuclear-encoded mitochondrial gene transcripts are also found to decreas

284 mption and altered expression of many muscle mitochondrial gene transcripts.

285 urther characterize factors that control the mitochondrial gene transfer/retention process, we have i

286 lve existing differences between nuclear and mitochondrial gene trees.

287 Most modern eukaryotes transmit mitochondrial genes uniparentally, often without recombi

288 A survey of 362 C-to-U editing sites in 33 mitochondrial genes was conducted on RNA extracted from

289 he rate of evolution for the nuclear-encoded mitochondrial genes was greater in post-WGD species than

290 tor 1 (NRF-1), which induces nuclear-encoded mitochondrial genes, was repressed in expression and act

291 The effects of RSG on adipose mitochondrial genes were confirmed by quantitative RT-PC

292 n volumes (>/=100 mul) and PCR targeting the mitochondrial genes were demonstrated.

293 a set of accessory gland-specific genes and mitochondrial genes were downregulated in males with res

294 Surprisingly, we found that when mitochondrial genes were highly transcribed, they also h

295 Importantly, nuclear-encoded mitochondrial genes were identified as the major functio

296 f mitochondrial complexes I, II, and III and mitochondrial genes were observed in rats given TNF.

297 storage cardiomyopathy-causing genes, and 27 mitochondrial genes were sequenced in unrelated individu

298 92% of 5,471 exons from 524 nuclear-encoded mitochondrial genes were successfully amplified from gen

299 sgenic mice, we found elevated expression of mitochondrial genes, which we hypothesize represents a c

300 o sustain the transcription of metabolic and mitochondrial genes, whose levels are unchanged compared