ライフサイエンスコーパス: mitochondrial membrane depolarization

1 ong, and Bcl-x(L) that were accompanied with mitochondrial membrane depolarization.

2 levels of the death receptor DR5 and caused mitochondrial membrane depolarization.

3 MAPK), and caspase-3 and was associated with mitochondrial membrane depolarization.

4 lease H(2)O(2), which can cause apoptosis by mitochondrial membrane depolarization.

5 disrupted plasma and acrosome membranes, and mitochondrial membrane depolarization.

6 sol is not a consequence of events requiring mitochondrial membrane depolarization.

7 spholipid asymmetry, caspase activation, and mitochondrial membrane depolarization.

8 to the cytosol followed by subsequent inner mitochondrial membrane depolarization.

9 e induced by PA-LTx was followed by a strong mitochondrial membrane depolarization.

10 of Bak activation, cytochrome c release, and mitochondrial membrane depolarization.

11 ficient Drosophila Schneider cells exhibited mitochondrial membrane depolarization, a 60% decrease in

12 cytochrome c and apoptosis-inducing factor, mitochondrial membrane depolarization, activation of a s

13 terized by concentration- and time-dependent mitochondrial membrane depolarization, activation of cas

14 Simvastatin attenuates mitochondrial membrane depolarization after exposure to

15 elect lymphoma cell lines and induced potent mitochondrial membrane depolarization and apoptosis when

16 n treatment of cells led to a tumor-specific mitochondrial membrane depolarization and ATP depletion

17 Because mitochondrial membrane depolarization and calcium are kn

18 posure to K5 ultimately led to apoptosis via mitochondrial membrane depolarization and caspase activa

19 th was caspase-dependent and associated with mitochondrial membrane depolarization and cytochrome c r

20 to proceed via a classical pathway involving mitochondrial membrane depolarization and cytochrome c r

21 Mitochondrial membrane depolarization and cytochrome c r

22 A interference, or genetic deletion prevents mitochondrial membrane depolarization and cytotoxicity i

23 -rosamine and Indo-1 revealed FimH-dependent mitochondrial membrane depolarization and elevated [Ca(2

24 DA receptor activation, leading to increased mitochondrial membrane depolarization and excitotoxic ce

25 The mechanism involved mitochondrial membrane depolarization and inhibition of

26 membrane depolarization in synaptosomes, and mitochondrial membrane depolarization and nuclear apopto

27 Other factors that regulated the mitochondrial membrane depolarization and subsequent los

28 HSC death conferred by NO occurred through mitochondrial membrane depolarization and through a casp

29 ecreased AKT phosphorylation at Ser-473, (2) mitochondrial membrane depolarization, and (3) activated

30 optotic events such as cytochrome c release, mitochondrial membrane depolarization, and activation of

31 cells showed extensive cytochrome c release, mitochondrial membrane depolarization, and caspase activ

32 cell death was preceded by NAD(+) depletion, mitochondrial membrane depolarization, and MPT.

33 ic NMDA exposure triggers AIF translocation, mitochondrial membrane depolarization, and phosphatidyl

34 ase in the level of reactive oxygen species, mitochondrial membrane depolarization, and premature sen

35 phosphorylation of RyR2, SR Ca(2+) leak and mitochondrial membrane depolarization are critically inv

36 Ca(2+) overload, as a consequence of partial mitochondrial membrane depolarization by mitoK(ATP) chan

37 Low MOI reduced mitochondrial membrane depolarization, caspase-3 and cas

38 the intracellular level of ROS and prevented mitochondrial membrane depolarization, correlating with

39 o significantly reduced H(2)O(2) generation, mitochondrial membrane depolarization, cytochrome c rele

40 following staurosporine treatment results in mitochondrial-membrane depolarization, cytochrome c rele

41 AT-101 also induced potent mitochondrial membrane depolarization (Delta Psi m) and

42 cytes, HBx activation of NF-kappaB prevented mitochondrial membrane depolarization; however, when NF-

43 to Abeta resulted in caspase activation and mitochondrial membrane depolarization in dendrites and c

44 itochondrial respiratory chain and prevented mitochondrial membrane depolarization in response to a p

45 ndent neurotrophic support may contribute to mitochondrial membrane depolarization in sensory neurons

46 A caspase inhibitor prevented Abeta-induced mitochondrial membrane depolarization in synaptosomes, a

47 FRD produced mitochondrial membrane depolarization in WT mice but not

48 sis (defined by annexin V staining) prior to mitochondrial membrane depolarization, in contrast to cy

49 a(2+) levels and inhibited beta-Lap-mediated mitochondrial membrane depolarization, intracellular ATP

50 occurrence of shared early events, including mitochondrial membrane depolarization, permeability tran

51 s, including phosphatidylserine exposure and mitochondrial membrane depolarization, PMN-SA had sustai

52 apoptosis to a non-apoptotic form, caused by mitochondrial membrane depolarization, probably initiate

53 with activation of c-Jun-N-terminal kinase, mitochondrial membrane depolarization, release of cytoch

54 All antineoplastic agents tested caused mitochondrial membrane depolarization that was inhibited

55 Mitochondrial membrane depolarization was detected in fl

56 However, mitochondrial membrane depolarization was not detected i

57 nitiation of protein kinase C signaling, and mitochondrial membrane depolarization with resultant apo