ライフサイエンスコーパス: mitochondrial outer membrane

1 forming homo-oligomers that permeabilize the mitochondrial outer membrane.

2 their self-association to form pores in the mitochondrial outer membrane.

3 CBS localized to the cytosol, as well as the mitochondrial outer membrane.

4 1 protein located in discrete patches in the mitochondrial outer membrane.

5 he voltage-dependent anion channel (VDAC) of mitochondrial outer membrane.

6 omplex, which physically connects the ER and mitochondrial outer membrane.

7 K and BAX form large oligomeric pores in the mitochondrial outer membrane.

8 pro-apoptotic activity by permeabilizing the mitochondrial outer membrane.

9 psis (Arabidopsis thaliana) localized in the mitochondrial outer membrane.

10 s expressed in the heart and can bind to the mitochondrial outer membrane.

11 either the endoplasmic reticulum (ER) or the mitochondrial outer membrane.

12 h assembles its replication complexes on the mitochondrial outer membrane.

13 and translocates into and permeabilizes the mitochondrial outer membrane.

14 transfer of metabolites and ions across the mitochondrial outer membrane.

15 m, Sox10 is peripherally associated with the mitochondrial outer membrane.

16 t or assembly of beta-barrel proteins in the mitochondrial outer membrane.

17 on induction and localization of MAVS to the mitochondrial outer membrane.

18 RMCX3 is an integral membrane protein of the mitochondrial outer membrane.

19 argets a critical regulatory molecule at the mitochondrial outer membrane.

20 aling in maintenance of the integrity of the mitochondrial outer membrane.

21 (DeltaPsi(m)) but does not permeabilize the mitochondrial outer membrane.

22 Puf3p localizes to the cytosolic face of the mitochondrial outer membrane.

23 and reduction of metabolite flow across the mitochondrial outer membrane.

24 r primary pro- or antiapoptotic roles at the mitochondrial outer membrane.

25 Bcl-xL in regulating apoptotic events in the mitochondrial outer membrane.

26 lic fractions from rat brain, as well as the mitochondrial outer membrane.

27 family proteins regulate the permeability of mitochondrial outer membrane.

28 insertion of its N-terminal domain into the mitochondrial outer membrane.

29 n the cytoplasm but also associates with the mitochondrial outer membrane.

30 itochondria and integration of MUC1 into the mitochondrial outer membrane.

31 is associated with the outer leaflet of the mitochondrial outer membrane.

32 expansion of the matrix, and rupture of the mitochondrial outer membrane.

33 ndergo cell death is permeabilization of the mitochondrial outer membrane.

34 y preventing the integration of Bax into the mitochondrial outer membrane.

35 n may function as a molecular adaptor on the mitochondrial outer membrane.

36 Fzo1p, and Ugo1p physically interact in the mitochondrial outer membrane.

37 R associations occurred on extensions of the mitochondrial outer membrane.

38 translocase complex and forms a pore in the mitochondrial outer membrane.

39 actor of the translocase supercomplex of the mitochondrial outer membrane.

40 ing to Bax-dependent permeabilization of the mitochondrial outer membrane.

41 ess is localized on the outer surface of the mitochondrial outer membrane.

42 ammalian ortholog, Drp1, was undetectable in mitochondrial outer membranes.

43 s consistent with results obtained in native mitochondrial outer membranes.

44 ramide to form protein-permeable channels in mitochondrial outer membranes.

45 ransmembrane GTPase Fzo1p controls fusion of mitochondrial outer membranes.

46 or viral RNA replication complex assembly on mitochondrial outer membranes.

47 s too small to allow protein flux across the mitochondrial outer membrane, a process known to initiat

48 ltage-dependent anion channels (VDAC) in the mitochondrial outer membrane after ethanol exposure lead

49 e proapoptotic protein Bax permeabilizes the mitochondrial outer membrane, allowing the release of pr

50 uring apoptosis, the permeabilization of the mitochondrial outer membrane allows the release of cytoc

51 on, is routinely found to be associated with mitochondrial outer membranes, although the structure an

52 serine-threonine protein kinase PINK1 on the mitochondrial outer membrane and activates Parkin.

53 family proteins regulate permeability of the mitochondrial outer membrane and apoptosis.

54 TA preserves the functional integrity of the mitochondrial outer membrane and arrests the caspase dea

55 raction of nitrated Hsp90 was located on the mitochondrial outer membrane and down-regulated mitochon

56 membrane domain, targeted the protein to the mitochondrial outer membrane and exerted antiapoptotic f

57 hat MceA is specifically integrated into the mitochondrial outer membrane and forms a complex of appr

58 age dependent anion channel 1 (VDAC1) on the mitochondrial outer membrane and inhibited its opening.

59 member (known as NLR) that localizes to the mitochondrial outer membrane and interacts with MAVS.

60 e, we show that, in addition to being in the mitochondrial outer membrane and intermembrane space, SO

61 ulin interacts with integral proteins of the mitochondrial outer membrane and is important for the st

62 domain of Tom22 on the cytosolic side of the mitochondrial outer membrane and links TOM and SAM compl

63 We found that NSm integrates into the mitochondrial outer membrane and that the protein's N te

64 permeability pathway for metabolites in the mitochondrial outer membrane and therefore is a very att

65 They are embedded in the mitochondrial outer membrane and thought to fuse adjacen

66 ted X (Bax) protein, permeabilization of the mitochondrial outer membrane, and cell death.

67 passage of a variety of molecules across the mitochondrial outer membrane, and is central to mitochon

68 ainly distributed in the cytosol, not in the mitochondrial outer membrane as previously reported, sug

69 processed HtrA2 prevents the accumulation of mitochondrial-outer-membrane-associated activated Bax, a

70 fractionations indicated that tBid bound to mitochondrial outer membranes at both contact and noncon

71 In addition to their presence at the mitochondrial outer membrane, Bax and Bak can also local

72 Whether formed in liposomes or in mitochondrial outer membranes, Bax-induced pores exhibit

73 arly in mitochondria-mediated apoptosis, the mitochondrial outer membrane becomes permeable to protei

74 orting and assembly machinery/topogenesis of mitochondrial outer membrane beta-barrel proteins (SAM/T

75 membranes that have no consequences for the mitochondrial outer membrane but inhibit Bax membrane in

76 Mff and Fis1 are both tail anchored in the mitochondrial outer membrane, but other parts of these p

77 newly identified pathway is regulated at the mitochondrial outer membrane by a complex between the pr

78 poptosis by maintaining the integrity of the mitochondrial outer membrane by adopting both soluble an

79 oltage-dependent anion channel (VDAC) of the mitochondrial outer membrane by dimeric tubulin is being

80 Dislocation of polypeptides from the mitochondrial outer membrane by the p97/Cdc48-Ufd1-Npl4

81 nt levels, ceramides form stable channels in mitochondrial outer membranes capable of passing the lar

82 d also activates anti-apoptotic Bcl-2 in the mitochondrial outer membrane, changing it from a single-

83 All mitochondrial outer membrane channels known to date are

84 endent but independent of protease-sensitive mitochondrial outer membrane components or the host mito

85 and other respiratory substrates through the mitochondrial outer membrane, constituting a crucial poi

86 embranes with a lipid composition resembling mitochondrial outer membrane contact sites.

87 We found that mitochondrial outer membranes contained very little card

88 We conclude that the mitochondrial outer membrane contains a considerably lar

89 The mitochondrial outer membrane contains proteinaceous mach

90 eractions among BCL-2 family proteins at the mitochondrial outer membrane control the release of thes

91 interaction of Bcl-2 family proteins at the mitochondrial outer membrane controls membrane permeabil

92 This permeabilization of the mitochondrial outer membrane depends on activation and o

93 rthermore, the Mcl-1 expression level at the mitochondrial outer membrane determines the release effi

94 haracterized by Bax translocalization to the mitochondrial outer membrane, disruption of the mitochon

95 strictions suitable for Drp1 assembly on the mitochondrial outer membrane; Drp1 then further constric

96 and Bak mediate the permeabilization of the mitochondrial outer membrane during apoptosis.

97 ges to homooligomerize then permeabilize the mitochondrial outer membrane during apoptosis.

98 lieved to form large oligomeric pores in the mitochondrial outer membrane during apoptosis.

99 nd MceA is a complex-forming effector at the mitochondrial outer membrane during Coxiella infection.

100 MceA forms a homo-oligomeric species at the mitochondrial outer membrane during infection.

101 ssaying in which GRASP65 was targeted to the mitochondrial outer membrane either directly or via bind

102 n the endoplasmic reticulum membrane and the mitochondrial outer membrane facilitate efficient transf

103 ivation of a ubiquitin ligase complex at the mitochondrial outer membrane for temporally and spatiall

104 endent anion channel 1 (VDAC1), found in the mitochondrial outer membrane, forms the main interface b

105 VDAC, a major protein of the mitochondrial outer membrane, forms voltage-dependent, a

106 family of small pore-forming proteins of the mitochondrial outer membrane found in all eukaryotes.

107 in 2, which is an essential component of the mitochondrial outer membrane fusion apparatus, and the u

108 e structural insights into the mechanisms of mitochondrial outer membrane fusion by investigating the

109 Mitofusin (Mfn) 1 and 2 mediate mitochondrial outer membrane fusion, whereas Mfn2 but no

110 We show that the mitochondrial outer membrane guanosine triphosphatase mi

111 A adopts a polytopic conformation within the mitochondrial outer membrane, having both the N- and C-t

112 ctive protein [HTATIP], Vimentin, fission 1 (mitochondrial outer membrane) homolog [FIS1], and protei

113 peptides potently trigger disruption of the mitochondrial outer membrane in cells dependent on Bfl-1

114 timulator, La-related protein (Larp), to the mitochondrial outer membrane in ovaries.

115 ate, it is unclear how Drp1 assembles on the mitochondrial outer membrane in response to different li

116 change localization from the cytosol to the mitochondrial outer membrane in this regulation.

117 odeling protein that mediates fusion between mitochondrial outer membranes in animals and fungi.

118 nion channel (VDAC), isolated from rat liver mitochondrial outer membranes, including the transmembra

119 Targeting Larp to the mitochondrial outer membrane independently of MDI restor

120 e.g. Bcl-2, Bcl-X(L)) proteins that regulate mitochondrial outer membrane integrity, cytochrome c rel

121 eins that regulates apoptosis by controlling mitochondrial outer membrane integrity.

122 that contributes to apoptosis by disrupting mitochondrial outer membrane integrity.

123 In mammals, fusion of the mitochondrial outer membrane is controlled by two DRPs,

124 The mitochondrial outer membrane is essential for communicat

125 Association of tBid on the mitochondrial outer membrane is key to its biological fu

126 pro-apoptotic Bax protein permeabilizes the mitochondrial outer membrane is not fully understood.

127 During apoptosis, the mitochondrial outer membrane is permeabilized, leading t

128 Fusion of mitochondrial outer membranes is crucial for proper orga

129 HP1, or PTPN6) from Sab in the inside of the mitochondrial outer membrane, leading to its activation

130 Avicin channels in the mitochondrial outer membrane may favor apoptosis by alte

131 by inhibiting the insertion of Bax into the mitochondrial outer membrane, membrane insertion of Bax

132 ivation of large-conductance channels in the mitochondrial outer membrane, mitochondrial release of c

133 ix alpha9 of Bax protein can dimerize in the mitochondrial outer membrane (MOM) and lead to apoptotic

134 hway to apoptosis is permeabilization of the mitochondrial outer membrane (MOM) by oligomers of the B

135 se results demonstrate how lipid cues at the mitochondrial outer membrane (MOM) can alter Drp1 struct

136 nitine palmitoyltransferase 1a) in the liver mitochondrial outer membrane (MOM) catalyzes the primary

137 PINK1 promotes PARKIN recruitment to the mitochondrial outer membrane (MOM) for ubiquitylation of

138 ell lymphoma-2 (Bcl-2) protein family in the mitochondrial outer membrane (MOM) induce structural cha

139 Regulation of mitochondrial outer membrane (MOM) permeability has dual

140 Strikingly, the incorporation of isolated mitochondrial outer membrane (MOM) proteins into liposom

141 activity of Bax, but it is unclear how other mitochondrial outer membrane (MOM) proteins might facili

142 d insertion machinery has been uncovered for mitochondrial outer membrane (MOM) TA proteins.

143 Whereas PARKIN is recruited to the mitochondrial outer membrane (MOM) upon depolarization v

144 ernal surface of the mitochondria, i.e., the mitochondrial outer membrane (MOM), and modulate its per

145 d Bax changes conformation, inserts into the mitochondrial outer membrane (MOM), oligomerizes, and in

146 haemia by enhancing Drp1 partitioning to the mitochondrial outer membrane (MOM), which causes cytochr

147 ) to visualize Bax-induced pores in purified mitochondrial outer membranes (MOMs).

148 n of tBid via a conformational change at the mitochondrial outer membrane, Mtch2 accelerates tBid-med

149 almitoyltransferase-I (CPT-IL) isolated from mitochondrial outer membranes obtained in the presence o

150 form that is similar to the one found in the mitochondrial outer membrane of drug-treated cells.

151 Monoamine oxidase B, present in the mitochondrial outer membrane of glial cells, catalyzes t

152 ologue, disassemble ceramide channels in the mitochondrial outer membranes of isolated mitochondria f

153 The mitochondrial outer membrane (OM) contains single and mu

154 drion-associated membrane compartment to the mitochondrial outer membrane (OMM), where they inactivat

155 nery and dynamin-related protein 1 (Drp1) on mitochondrial outer membrane (OMM).

156 lin C, but not Cdk8, is required for loss of mitochondrial outer membrane permeability and apoptosis

157 f the mitochondrial lipids in control of the mitochondrial outer membrane permeability and hence mito

158 ng the long and variable delay that precedes mitochondrial outer membrane permeabilization (MOMP) and

159 Both mitochondrial outer membrane permeabilization (MOMP) and

160 uter mitochondrial membrane (OMM) to promote mitochondrial outer membrane permeabilization (MOMP) and

161 racting-domain death agonist, BID) to induce mitochondrial outer membrane permeabilization (MOMP) and

162 BCL-2 family proteins control mitochondrial outer membrane permeabilization (MOMP) by

163 Pro-apoptotic Bax induces mitochondrial outer membrane permeabilization (MOMP) by

164 inhibiting Bax oligomerization required for mitochondrial outer membrane permeabilization (MOMP) dur

165 Mitochondrial outer membrane permeabilization (MOMP) has

166 -dependent apoptosis execution subsequent to mitochondrial outer membrane permeabilization (MOMP) in

167 Most apoptotic stimuli require mitochondrial outer membrane permeabilization (MOMP) in

168 Bax/Bak-mediated mitochondrial outer membrane permeabilization (MOMP) is

169 Mitochondrial outer membrane permeabilization (MOMP) is

170 During apoptosis, mitochondrial outer membrane permeabilization (MOMP) is

171 Mitochondrial outer membrane permeabilization (MOMP) is

172 els of caspase activity triggered by limited mitochondrial outer membrane permeabilization (MOMP) pro

173 rface of apoptosis initiation and execution, mitochondrial outer membrane permeabilization (MOMP) rep

174 ntrinsic apoptotic pathway and the resultant mitochondrial outer membrane permeabilization (MOMP) via

175 The mechanism of Bax/Bak-dependent mitochondrial outer membrane permeabilization (MOMP), a

176 Bax induces mitochondrial outer membrane permeabilization (MOMP), a

177 Mitochondrial outer membrane permeabilization (MOMP), a

178 The defining event in apoptosis is mitochondrial outer membrane permeabilization (MOMP), al

179 oapoptotic members of the Bcl-2 family cause mitochondrial outer membrane permeabilization (MOMP), al

180 apoptosis, the BCL-2 protein family controls mitochondrial outer membrane permeabilization (MOMP), bu

181 (PIs) avert apoptosis in part by preventing mitochondrial outer membrane permeabilization (MOMP), bu

182 apoptosis involving regulation of Bax/Bcl-2, mitochondrial outer membrane permeabilization (MOMP), cy

183 The intrinsic pathway is characterized by mitochondrial outer membrane permeabilization (MOMP), cy

184 Thus, following Bax/Bak-mediated mitochondrial outer membrane permeabilization (MOMP), IM

185 n vertebrates is dependent on the process of mitochondrial outer membrane permeabilization (MOMP), wh

186 fficient apoptosis requires Bax/Bak-mediated mitochondrial outer membrane permeabilization (MOMP), wh

187 es apoptosis by activating Bax and eliciting mitochondrial outer membrane permeabilization (MOMP).

188 ing the apoptotic deficiency at the level of mitochondrial outer membrane permeabilization (MOMP).

189 a critical step in apoptosis referred to as mitochondrial outer membrane permeabilization (MOMP).

190 d BAX are required for apoptosis and trigger mitochondrial outer membrane permeabilization (MOMP).

191 sis, caspase activation is closely linked to mitochondrial outer membrane permeabilization (MOMP).

192 ondrial pathway of apoptosis is initiated by mitochondrial outer membrane permeabilization (MOMP).

193 pressing reporters of caspase activation and mitochondrial outer membrane permeabilization after expo

194 by producing G-CSF and GM-CSF, delaying the mitochondrial outer membrane permeabilization and caspas

195 Mitochondrial outer membrane permeabilization and cytoch

196 activates an apical protease that stimulates mitochondrial outer membrane permeabilization and proces

197 Mitochondrial outer membrane permeabilization and the re

198 Most importantly, mitochondrial outer membrane permeabilization becomes a

199 In the absence of active caspases, mitochondrial outer membrane permeabilization by Bax and

200 Mitochondrial outer membrane permeabilization by truncat

201 ttenuated H37Ra or the virulent H37Rv causes mitochondrial outer membrane permeabilization characteri

202 vents mitochondria division and Bax-mediated mitochondrial outer membrane permeabilization during apo

203 ily that plays a pivotal role in controlling mitochondrial outer membrane permeabilization during apo

204 ntial in mammals, in flies the importance of mitochondrial outer membrane permeabilization for apopto

205 most intrinsic apoptotic cues, provided that mitochondrial outer membrane permeabilization has occurr

206 of Bcl2-associated X protein activation and mitochondrial outer membrane permeabilization in vitro a

207 hondrial division dynamin directly regulates mitochondrial outer membrane permeabilization independen

208 Mitochondrial outer membrane permeabilization is transie

209 and signaling pathway(s) culminating in the mitochondrial outer membrane permeabilization that initi

210 oteins to oligomerization of BAX and BAK and mitochondrial outer membrane permeabilization, an intric

211 hain at the level of complex III, attenuated mitochondrial outer membrane permeabilization, and decre

212 rinsic pathway of apoptosis, as evidenced by mitochondrial outer membrane permeabilization, but downs

213 wn acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipati

214 ring the cell intrinsic apoptotic pathway is mitochondrial outer membrane permeabilization, leading t

215 This change contributed to mitochondrial outer membrane permeabilization, release o

216 ure of cells to prodeath stimuli, control of mitochondrial outer membrane permeabilization, switch-li

217 ax activation, Bax homo-oligomerization, and mitochondrial outer membrane permeabilization.

218 ests a process for controlling the degree of mitochondrial outer membrane permeabilization.

219 s, thereby triggering Bak/Bax activation and mitochondrial outer membrane permeabilization.

220 lls, mdivi-1 retards apoptosis by inhibiting mitochondrial outer membrane permeabilization.

221 apoptosis, either upstream or downstream of mitochondrial outer membrane permeabilization.

222 ss-activated caspases-4/12 were required for mitochondrial outer membrane permeabilization.

223 ember Bid, which, together with Bax, induces mitochondrial outer membrane permeabilization.

224 zmB-truncated Bid, and promotes GzmB-induced mitochondrial outer membrane permeabilization.

225 s that faithfully recapitulate BAX-dependent mitochondrial outer membrane permeabilization.

226 is known about how these interactions impact mitochondrial outer-membrane permeabilization (MOMP) and

227 Mitochondrial outer-membrane permeabilization by pro-apo

228 Mitochondrial outer-membrane permeabilization can lead t

229 at shock was RAIDD dependent and upstream of mitochondrial outer-membrane permeabilization.

230 Mitochondrial outer membrane permeabiliztaion (MOMP) was

231 (PC), the most abundant phospholipid of the mitochondrial outer membrane, play a role in the import

232 We identify a phospholipase of the mitochondrial outer membrane, PLD6, as the mediator of M

233 zation, but the molecular composition of the mitochondrial outer membrane pore is under debate.

234 ochondrial outer membrane, disruption of the mitochondrial outer membrane potential, and caspase acti

235 ts N-terminus and the targeting segment of a mitochondrial outer membrane protein (OMP25) at its C-te

236 The mechanism involves an integral mitochondrial outer membrane protein and general autopha

237 l mitochondria and additionally requires the mitochondrial outer membrane protein FIS1, the autophagy

238 Dnm1p recruitment depends on the mitochondrial outer membrane protein Fis1p.

239 the peripheral benzodiazepine receptor, is a mitochondrial outer membrane protein implicated as essen

240 MitoNEET (mNEET) is a dimeric mitochondrial outer membrane protein implicated in many

241 Here we show that the mitochondrial outer membrane protein Mcp1 functions in t

242 The human mitochondrial outer membrane protein mitoNEET is a novel

243 One of these is the novel mitochondrial outer membrane protein MOMA-1.

244 t in a complex with Mdm12p, another integral mitochondrial outer membrane protein required for mitoch

245 TOM36 is a trypanosomatid-specific essential mitochondrial outer membrane protein that has been impli

246 Fis1 is a mitochondrial outer membrane protein that recruits the d

247 The mitochondrial outer membrane protein Tom40 is the genera

248 mport of tRNAs requires four subunits of the mitochondrial outer membrane protein translocase but not

249 f PapC resemble that of the Tom40 complex, a mitochondrial outer membrane protein translocase.

250 hown that tubulin dimer interaction with the mitochondrial outer membrane protein voltage-dependent a

251 Ian4, a mitochondrial outer membrane protein with GTP-binding ac

252 The mitochondrial outer membrane protein, Mmm1p, is required

253 Here we identify a beta-barrel mitochondrial outer membrane protein, termed tripartite

254 mTOR is in a complex with the mitochondrial outer-membrane protein Bcl-xl and VDAC1.

255 In viable cells, we found BAK complexed with mitochondrial outer-membrane protein VDAC2, a VDAC isofo

256 s encoding actin cytoskeleton components and mitochondrial outer membrane proteins also cause accumul

257 These mitochondrial outer membrane proteins contain a GTPase d

258 ase parkin, which builds ubiquitin chains on mitochondrial outer membrane proteins, where they act to

259 Several mitochondrial outer membrane proteins-mitochondrial fiss

260 Both proteins are integral mitochondrial outer membrane proteins.

261 hese results indicate that remodeling of the mitochondrial outer membrane proteome is important for m

262 rone Hsp70/Hsp90 may function to prepare the mitochondrial outer membrane receptor Tom71 for preprote

263 tified mitochondrial fission factor (MFF), a mitochondrial outer-membrane receptor for DRP1, the cyto

264 tal signals, BAX and/or BAK permeabilize the mitochondrial outer membrane, resulting in cytochrome c

265 hosphorylated JNK (p-JNK) interacts with the mitochondrial outer membrane SH3 homology associated BTK

266 irst identified Fe-S protein anchored in the mitochondrial outer membrane, strictly depends on ISC ma

267 Agents that disrupt the mitochondrial outer membrane, such as digitonin, or main

268 We replaced the FHV protein A mitochondrial outer membrane-targeting sequence with the

269 consider that Bax and Bak form pores at the mitochondrial outer membrane that are responsible for th

270 ion channel (VDAC), the major channel of the mitochondrial outer membrane that controls most of the m

271 amide forms large and stable channels in the mitochondrial outer membrane that induce cell death thro

272 ccount for the increased permeability of the mitochondrial outer membrane that is responsible for thi

273 SAM complex is an essential component of the mitochondrial outer membrane that mediates the insertion

274 ff), a tail-anchored membrane protein of the mitochondrial outer membrane that recruits Drp1 to sites

275 t of beta-barrel precursor proteins into the mitochondrial outer membrane: The translocase of the out

276 ssibility that Mgm1p regulates fusion of the mitochondrial outer membrane through its interactions wi

277 substrates and adenine nucleotides cross the mitochondrial outer membrane through the voltage-depende

278 apoptosis relies on permeabilization of the mitochondrial outer membrane to factors such as cytochro

279 the mitochondria, where it inserts into the mitochondrial outer membrane to form a toxic pore.

280 poptotic proteins are believed to act at the mitochondrial outer membrane to form an apoptotic pore w

281 where it oligomerizes and permeabilizes the mitochondrial outer membrane to promote apoptosis.

282 nitiation of apoptosis by permeabilizing the mitochondrial outer membrane to small proteins, includin

283 ria, we show that PS is transferred from the mitochondrial outer membrane to the inner membrane indep

284 ssion of mitoNEET, a protein residing in the mitochondrial outer membrane, to probe its impact on mit

285 oteins to the peripheral and channel-forming mitochondrial outer membrane translocases (TOMs).

286 oteins to the peripheral and channel-forming mitochondrial outer membrane translocases (TOMs).

287 egin the importation process by crossing the mitochondrial outer membrane via a specialized protein i

288 metric dimers that coalesce to perforate the mitochondrial outer membrane via an unknown mechanism.

289 ls to form pores of unknown structure on the mitochondrial outer membrane via homooligomerization.

290 MitoNEET is anchored to the mitochondrial outer membrane via its N-terminal alpha he

291 Finally, isolated mitochondrial outer membrane was incubated with cytosol

292 Starting at 4 hours, the mitochondrial outer membrane was significantly permeabil

293 ol or mimicking the lipid composition of the mitochondrial outer membrane were tested, we did not det

294 des a 12-kDa protein, which localizes to the mitochondrial outer membrane when expressed in mammalian

295 amers, Drp1 is recruited by receptors on the mitochondrial outer membrane, where it further assembles

296 assemble into higher order structures on the mitochondrial outer membrane, where it is required for p

297 Fzo1, were required to promote the fusion of mitochondrial outer membranes, whereas electrical potent

298 egion (UTR) that confers localization to the mitochondrial outer membrane, which is postulated to aid

299 We have shown that UBP27 is embedded in the mitochondrial outer membrane with an Nin -Cout orientati

300 Ugo1p is anchored in the mitochondrial outer membrane with its N terminus facing