ライフサイエンスコーパス: mitochondrial respiration

1 ess so for a subset of genes associated with mitochondrial respiration.

2 perate to enhance ANT transport capacity and mitochondrial respiration.

3 eointimal growth, and evidence of higher SMC mitochondrial respiration.

4 the CO2 compensation point in the absence of mitochondrial respiration.

5 amaging both the redox properties of AIF and mitochondrial respiration.

6 and functional complex II, as well as normal mitochondrial respiration.

7 (HIF), promoting glycolysis and suppressing mitochondrial respiration.

8 roduction in M1 macrophages, while promoting mitochondrial respiration.

9 bility was largely mediated by its effect on mitochondrial respiration.

10 romised redox properties, causing diminished mitochondrial respiration.

11 n transport chain complex (ETC) activity and mitochondrial respiration.

12 ifferentiation, these cells fail to activate mitochondrial respiration.

13 m mice treated with Honokiol showed enhanced mitochondrial respiration.

14 ential, mitochondrial Ca(2+) hemostasis, and mitochondrial respiration.

15 restores differentiation through increasing mitochondrial respiration.

16 tion of reactive oxygen species and maintain mitochondrial respiration.

17 to the promoters of genes with a key role on mitochondrial respiration.

18 of oxidative folding rival those produced by mitochondrial respiration.

19 chondrial capacity consistent with increased mitochondrial respiration.

20 nhanced energy expenditure through uncoupled mitochondrial respiration.

21 other malignant diseases marked by increased mitochondrial respiration.

22 nied by a shift from anaerobic glycolysis to mitochondrial respiration.

23 myocardial contractile function and impairs mitochondrial respiration.

24 thanolamine, which is itself an inhibitor of mitochondrial respiration.

25 educed following inhibition of glycolysis or mitochondrial respiration.

26 ition of IDE increased mitAbeta and impaired mitochondrial respiration.

27 an over-the-counter drug, as an inhibitor of mitochondrial respiration.

28 l mitochondria ultrastructure, and defective mitochondrial respiration.

29 ctural abnormalities and displayed deficient mitochondrial respiration.

30 ion on lipid peroxidation, mtDNA damage, and mitochondrial respiration.

31 is being recognized as a potent regulator of mitochondrial respiration.

32 d lactate production, and partially restored mitochondrial respiration.

33 m-like cells, which is blocked by inhibiting mitochondrial respiration.

34 e is mirrored by a significant impairment in mitochondrial respiration.

35 chondrial membrane potential, and diminished mitochondrial respiration.

36 nergy through glycolysis rather than through mitochondrial respiration.

37 ation of glutamate enables cells to maintain mitochondrial respiration.

38 hosphorylation genes resulting in diminished mitochondrial respiration.

39 insight into the transcriptional control of mitochondrial respiration.

40 ells into brown adipose tissue and increased mitochondrial respiration.

41 I) hemes, most of which were associated with mitochondrial respiration.

42 ome clearance by the lysosome, and decreased mitochondrial respiration.

43 the host animal, leading to normalisation of mitochondrial respiration.

44 athological remodeling, and improved cardiac mitochondrial respiration.

45 Q (CoQ) biosynthesis-a pathway essential for mitochondrial respiration.

46 etabolic activity by reducing glycolysis and mitochondrial respiration.

47 s that FAK enhances glycolysis and decreases mitochondrial respiration.

48 ing that Mb could be a factor that regulates mitochondrial respiration.

49 be an unexpected treatment for disorders of mitochondrial respiration.

50 xhibited less proliferation, glycolysis, and mitochondrial respiration, accompanied by reduced mTOR k

51 d in mitochondrial supercomplex assembly and mitochondrial respiration activity.

52 Therefore, "spatiotemporal" mitochondrial respiration adaptively induced by PI3K the

53 nation of biochemical, live-cell imaging and mitochondrial respiration analysis, we found that brain

54 Nitrite exhibited an uncoupling of mitochondrial respiration and a decrease in ATP generati

55 t the arrested cells have a reduced level of mitochondrial respiration and a decrease in the expressi

56 me activity, as well as a strong reliance on mitochondrial respiration and a decreased dependence on

57 PDK1 inhibits pyruvate flux to mitochondrial respiration and a rescue of its expression

58 Mitochondrial respiration and aerobic capacity are thus

59 The functions of CoQ in mitochondrial respiration and as an antioxidant are esta

60 CKMT1 inhibition altered mitochondrial respiration and ATP production, an effect

61 miR33 therapy derepresses genes that enhance mitochondrial respiration and ATP production, which in c

62 lar species composition, and normal rates of mitochondrial respiration and ATP production.

63 Measurements of mitochondrial respiration and ATP synthesis revealed tha

64 in reduced abundance of proteins involved in mitochondrial respiration and beta-oxidation.

65 investigated ex vivo isometric contraction, mitochondrial respiration and calcium retention capacity

66 Pre-operative mitochondrial respiration and calcium retention capacity

67 nteracting protein (TXNIP) in the control of mitochondrial respiration and cell energy metabolism.

68 Accordingly, affected tissues had defects in mitochondrial respiration and complex I biogenesis that

69 cing mitochondrial PE accumulation preserves mitochondrial respiration and cristae formation.

70 loroacetate, an inhibitor of PDKs, increased mitochondrial respiration and decreased production of re

71 ild-type mice but accelerated the decline in mitochondrial respiration and development of macrocytic

72 upler that causes dose-dependent increase in mitochondrial respiration and dissipation of mitochondri

73 evealed high enrichment of genes involved in mitochondrial respiration and downstream targets of IL-6

74 s of MCJ in macrophages results in increased mitochondrial respiration and elevated basal levels of r

75 T) OGT isoforms is associated with increased mitochondrial respiration and elevated glycolysis, sugge

76 only EX improved cardiorespiratory fitness, mitochondrial respiration and enzyme activities, and car

77 TRAP1-deficiency promotes an increase in mitochondrial respiration and fatty acid oxidation, and

78 al trafficking during mitophagy and improves mitochondrial respiration and glucose-stimulated insulin

79 ral metabolic pathways including glycolysis, mitochondrial respiration and glutamine metabolism.

80 ific changes in ROS levels, increases either mitochondrial respiration and glycolysis rates.

81 red insulin signaling and insulin-stimulated mitochondrial respiration and glycolysis.

82 Overexpression of CLD1 leads to decreased mitochondrial respiration and growth and instability of

83 NADH and NADP(+), provides the link between mitochondrial respiration and H2O2 detoxification throug

84 l pathway linking methionine metabolism with mitochondrial respiration and have important implication

85 Two regulators of mitochondrial respiration and heat production in brown a

86 o identify a novel role of VCP in preserving mitochondrial respiration and in preventing the opening

87 ystemic metabolic defects, including reduced mitochondrial respiration and increased glycolysis, ener

88 nerated rho0 variants that lacked functional mitochondrial respiration and increased glycolytic metab

89 nd contractile weakness, but lacked impaired mitochondrial respiration and increased levels of oxidat

90 Decreased pre-operative mitochondrial respiration and increased sensitivity to c

91 n metabolism, DMOG treatment decreased iTreg mitochondrial respiration and increased their glycolytic

92 lpha, a key regulator of enzymes involved in mitochondrial respiration and insulin resistance, is pot

93 Mitochondrial respiration and intramyocellular triglycer

94 tudy aimed to evaluate the effect of RIPC on mitochondrial respiration and miR expression in human at

95 o parallel and synergistic induction of both mitochondrial respiration and nuclear transcription fact

96 ipocytes, which included positively enriched mitochondrial respiration and oxidation pathways.

97 amounts retained by ndufs4 did not increase mitochondrial respiration and oxidative phosphorylation

98 btained human specimens, we report here that mitochondrial respiration and oxidative phosphorylation

99 etion of PDK4 or activation of PDH increased mitochondrial respiration and oxidative stress in suspen

100 localized to the mitochondria and regulates mitochondrial respiration and oxidative stress resistanc

101 RIPC preserves mitochondrial respiration and prevents upregulation of m

102 s PGC-1alpha, PDK4, and SLC25A25 and boosted mitochondrial respiration and production of ATP.

103 hanistically, we show that caspase 1 reduces mitochondrial respiration and reactive oxygen species by

104 metabolites required for the maintenance of mitochondrial respiration and redox homeostasis.

105 However, the role of AIF in mitochondrial respiration and redox metabolism has remai

106 we show that loss of MFN2 leads to impaired mitochondrial respiration and reduced ATP production, an

107 are uncoupled, as characterized by increased mitochondrial respiration and reduced mitochondrial memb

108 tophagy, limiting substrate availability for mitochondrial respiration and reducing gluconeogenesis.

109 Interestingly, mutant CPCs are deficient in mitochondrial respiration and rely on glycolysis for ene

110 ly, these findings integrate HKL action with mitochondrial respiration and shape and substantiate a p

111 RT3 increased cardiolipin levels and rescued mitochondrial respiration and SOD2 expression in DOX-tre

112 ongs the quality attributes, affecting basic mitochondrial respiration and starch degradation rate.

113 Reactive oxygen species are byproducts of mitochondrial respiration and thus potential regulators

114 antiretroviral drug zalcitabine to suppress mitochondrial respiration and to force glycolysis in our

115 eases in glycolytic capacity, improvement of mitochondrial respiration, and concomitant attenuation o

116 onstrated a role for QIL1 in MICOS assembly, mitochondrial respiration, and cristae formation critica

117 ate secretion, decreased glutamine-dependent mitochondrial respiration, and decreased ATP transport a

118 llular processes including oxygen transport, mitochondrial respiration, and DNA synthesis.

119 ng enhanced cardiac autophagy, mitophagy and mitochondrial respiration, and it also improved the mech

120 phenomena including cell cycle progression, mitochondrial respiration, and learning and memory.

121 h a higher glycolytic rate at the expense of mitochondrial respiration, and led to lactate accumulati

122 eficient DCs exhibited increased glycolysis, mitochondrial respiration, and lipid synthesis that were

123 olic pathways, including aerobic glycolysis, mitochondrial respiration, and lipid synthesis.

124 gh regulation of genes in insulin signaling, mitochondrial respiration, and protein homeostasis.

125 w-fed mice compromises fatty acid oxidation, mitochondrial respiration, and the abundance of mitochon

126 tary-reducing equivalents by elevated muscle mitochondrial respiration appears to be the mechanism by

127 etabolism is reprogrammed with activation of mitochondrial respiration as in ESC.

128 r ATP production utilized via glycolysis and mitochondrial respiration as well as for glycosylation o

129 e effect (the glucose-mediated inhibition of mitochondrial respiration) as most in vitro experiments

130 o in vitro bioassays, namely the classic rat mitochondrial respiration assay, and a mitochondrial mem

131 ssimilation rate, mesophyll conductance, and mitochondrial respiration at darkness.

132 midpoint but suffer from a severe deficit in mitochondrial respiration at the clinical phase of disea

133 sor, which has emerged as a key regulator of mitochondrial respiration at the expense of glycolysis,

134 raction, followed by immediate impairment of mitochondrial respiration at the level of complex I of t

135 any described human disease, showed impaired mitochondrial respiration, being a potential cause of in

136 p53, a critical tumor suppressor, regulates mitochondrial respiration, but how a nuclear protein can

137 o fuel glycolysis and provide substrates for mitochondrial respiration, but neurons can also use alte

138 ubulin, a subunit of microtubules, regulates mitochondrial respiration by blocking the voltage-depend

139 here that HKL administration rapidly reduces mitochondrial respiration by broadly inhibiting ETC comp

140 e 2 and phosphofrucokinase, while decreasing mitochondrial respiration by downregulating respiratory

141 tin structure relieves glucose repression of mitochondrial respiration by inducing transcription of t

142 Hydrogen sulfide (H2S) impairs mitochondrial respiration by potently inhibiting the hem

143 rticularly evident in cancer where defective mitochondrial respiration can be restored and tumor-form

144 Reactive oxygen species, a byproduct of this mitochondrial respiration, can damage mitochondrial DNA

145 This was coupled to an enhanced mitochondrial respiration capacity and a preference for

146 A cycle block reflected by decreased maximal mitochondrial respiration) caused lethal fetal liver hem

147 om the pleiotropic effects of CO on cellular mitochondrial respiration, cellular energy utilization,

148 nal to the electron transfer activity of the mitochondrial respiration chain.

149 nduces a decrease in complex IV activity and mitochondrial respiration compared with the overexpressi

150 ression of Pgc1alpha and Sirt3, and improved mitochondrial respiration, compared to wild-type Treg ce

151 reperfusion-driven recovery of C-I-dependent mitochondrial respiration contributes not only to the ce

152 ved anti-helminth drug nitazoxanide (NTZ) on mitochondrial respiration could possess any therapeutic

153 ntricular cardiomyocytes initially activates mitochondrial respiration, coupled with increased mitoch

154 bserved in BMI1-depleted cells, for example, mitochondrial respiration, cytochrome c oxidase activity

155 d increased glycolytic activity but impaired mitochondrial respiration, decreased ATP production, and

156 that MIF-induced myocardial dysfunction and mitochondrial respiration deficit could be related to ca

157 Bexarotene also restored mitochondrial respiration deficits in P301L-Tau neurons.

158 iption factor A mitochondrial expression and mitochondrial respiration despite elevated RV pressure-o

159 , inflammation and coagulation patterns, and mitochondrial respiration did not differ between NE and

160 mycin reversed sesamol-induced apoptosis and mitochondrial respiration disorders.

161 ial to various cellular processes, including mitochondrial respiration, DNA repair, and iron homeosta

162 iency is associated with inhibited complex I mitochondrial respiration due to lack of NADH for the el

163 umor cells have been considered defective in mitochondrial respiration due to their dominant glycolyt

164 lations also unraveled the essential role of mitochondrial respiration during drought stress.

165 sults show that AOX is necessary to maintain mitochondrial respiration during moderate drought.

166 t ammonia results in reduced skeletal muscle mitochondrial respiration, electron transport chain comp

167 Efavirenz alters mitochondrial respiration, enhances reactive oxygen spec

168 normal fumarate levels but defective maximal mitochondrial respiration) failed to self-renew and disp

169 tance is associated with a transient rise in mitochondrial respiration followed by increased producti

170 eta3 activation, resulting in an increase of mitochondrial respiration for cell survival.

171 inhardtii (which use both photosynthesis and mitochondrial respiration for growth) are shifted to con

172 transition and, subsequently, to upregulate mitochondrial respiration for successful cell-cycle prog

173 xtreme host metabolic dependence and loss of mitochondrial respiration for which microsporidia are we

174 upling protein (UCP) 1 and thus can uncouple mitochondrial respiration from ATP synthesis.

175 ehydrogenase, two key enzymes that determine mitochondrial respiration, glutaminolysis and fatty acid

176 trates that the MOC1-dependent modulation of mitochondrial respiration helps control the stromal redo

177 that genetic or pharmacologic disruption of mitochondrial respiration improves cancer-free survival

178 n Control LCLs and resulted in a increase in mitochondrial respiration in AD-A LCLs.

179 exposure by itself resulted in a decline in mitochondrial respiration in all LCL groups.

180 ated conditional Cox10 mouse mutants lacking mitochondrial respiration in astrocytes, which forces th

181 Here, we show that EglN2 depletion decreases mitochondrial respiration in breast cancer under normoxi

182 J protein) acted as an endogenous break for mitochondrial respiration in CD8(+) T cells by interferi

183 x II substrate succinate increase ATP-linked mitochondrial respiration in CI-deficient human blood ce

184 Mitochondrial respiration in diabetic hearts was diverge

185 stem cells as opposed to the predominance of mitochondrial respiration in gametes.

186 tone H3 methylation patterns, and markers of mitochondrial respiration in gray matter from postmortem

187 Aif1m rescued glucose-induced disruption of mitochondrial respiration in human primary proximal tubu

188 t it is unknown whether manassantin inhibits mitochondrial respiration in intact mammalian cells and

189 role of Ca2+ signaling in the regulation of mitochondrial respiration in intact mouse cortical neuro

190 nvestigate the effect of Honokiol on cardiac mitochondrial respiration in mice subjected to Dox treat

191 ctivities showed decreased basal and maximal mitochondrial respiration in motor neurons from SMA mice

192 osis in patients' lymphocytes, a decrease in mitochondrial respiration in patient fibroblasts with a

193 eceptor gamma (ERRgamma) negatively controls mitochondrial respiration in prostate cancer cells.

194 ore required to understand the activation of mitochondrial respiration in skeletal muscle at the star

195 ite the elevated plasma NEFA levels, ex vivo mitochondrial respiration in skeletal muscle increased.

196 Moreover, mitochondrial respiration in skeletal muscle tissue tend

197 Thus, enhanced mitochondrial respiration in the absence of MCJ prevents

198 ory chain proteins may lead to impairment of mitochondrial respiration in the brain.IMPORTANCE Mitoch

199 luding controlled mechanical ventilation, on mitochondrial respiration in the human diaphragm.

200 vated CO2 during the daytime decreases plant mitochondrial respiration in the light and protein conce

201 esis in cancer cells, leading to decrease in mitochondrial respiration in these cells.

202 Here, we have studied astrocytes that lack mitochondrial respiration in vitro and in vivo A novel m

203 PON3 deficiency resulted in impaired mitochondrial respiration, increased mitochondrial super

204 Deregulated mitochondrial respiration induced by ClpXP targeting cau

205 riatric surgery-induced weight loss enhances mitochondrial respiration, induces cardiolipin remodelin

206 Following RNAi, defects in mitochondrial respiration, inner membrane potential and

207 modate hypoxia by diverting carbon away from mitochondrial respiration into alternative pathways that

208 Mitochondrial respiration is a crucial component of cell

209 These data suggest that targeting mitochondrial respiration is a promising novel avenue fo

210 Here, we report that mitochondrial respiration is active in hypoxia (1% oxyge

211 ouse models have revealed that impairment of mitochondrial respiration is an early event in the patho

212 fatty acid synthesis during hypoxia or when mitochondrial respiration is impaired.

213 Mitochondrial respiration is important for cell prolifer

214 Here, we demonstrate that muscle mitochondrial respiration is improved by Mb overexpressi

215 We find that normal mitochondrial respiration is perturbed in TORC2-Ypk1-def

216 Mitochondrial respiration is recognized as an indicator

217 Mitochondrial respiration is regulated according to the

218 Mitochondrial respiration is regulated by multiple elabo

219 Mitochondrial respiration is regulated in CD8(+) T cells

220 of energy, and so iron, a critical player in mitochondrial respiration, is an important component of

221 Here we show that PMI promotes mitochondrial respiration, leading to a superoxide-depen

222 ticancer activity is associated with reduced mitochondrial respiration, leading to bioenergetic catas

223 ological decrease in PO2 inhibits astroglial mitochondrial respiration, leading to mitochondrial depo

224 esence of oxygen, suggesting that defects in mitochondrial respiration may be the underlying cause of

225 ateral inhibition of oncogenic signaling and mitochondrial respiration may help enhance the therapeut

226 ent complex V activity, leading to decreased mitochondrial respiration, membrane hyperpolarization, a

227 Mitochondrial respiration, membrane potential (Deltapsim

228 Mitochondrial respiration, morphology and motility in th

229 of fission and fusion can cause deficits in mitochondrial respiration, morphology and motility.

230 mRNA expression in both macrophage subtypes, mitochondrial respiration negatively affected IL6, IL1B,

231 In this context, mitochondrial respiration, network dynamics, TCA cycle f

232 muscle mitochondrial VO2 max (Mito VO2 max , mitochondrial respiration of fibres biopsied from vastus

233 on, dynamically switching from glycolysis to mitochondrial respiration on demand.

234 catalase levels to normal, without changing mitochondrial respiration or expression of oxidative str

235 calcium handling without adversely impacting mitochondrial respiration or membrane potential.

236 Mitochondrial respiration partially relieved the reducta

237 lysis provides free fatty acids to support a mitochondrial respiration pathway essential to neutrophi

238 nslation of mRNAs for ribosomal proteins and mitochondrial respiration peaked at night.

239 Mitochondrial respiration plays a crucial role in determ

240 Robust mitochondrial respiration provides energy to support phy

241 Rather, mitochondrial respiration provides sufficient energy for

242 ability (blue native gel assay), decrease in mitochondrial respiration rate and reduction of mitochon

243 ucose oxidation by increasing glycolysis and mitochondrial respiration rate in C2C12 muscle cells.

244 NAL displayed a higher baseline mitochondrial respiration rate than SAL.

245 State III mitochondrial respiration rates (pmol O2/s/mg wet weight

246 g the effect of intermittent contractions on mitochondrial respiration rates in the human diaphragm f

247 State IV mitochondrial respiration rates were 3.59 +/- 1.25 and 2

248 intracellular potassium levels, and reduced mitochondrial respiration rates.

249 n of mitochondrial membrane proteins reduced mitochondrial respiration, reactive oxygen species forma

250 ffects and mechanisms of Honokiol on cardiac mitochondrial respiration remain unclear.

251 dministration of fatty acids or pyruvate for mitochondrial respiration rescued differentiation in aut

252 h normalized fumarate levels but not maximal mitochondrial respiration) rescued these phenotypes, ind

253 l the ENs studied induced an increase in the mitochondrial respiration resulting in uncoupling of the

254 -induced ROS accumulation was independent of mitochondrial respiration (rotenone-insensitive) but was

255 sed anti-diabetes drug, is the inhibition of mitochondrial respiration, specifically at complex I.

256 The mitochondrial respiration stay decreased in the ischemic

257 Trabeculae were then permeabilized for mitochondrial respiration studies using high-resolution

258 x-vivo working heart perfusions and isolated mitochondrial respiration studies.

259 mical inhibition of beta-secretase decreased mitochondrial respiration, suggesting that non-amyloidog

260 Adenosine diphosphate-stimulated mitochondrial respiration supported by palmitoyl-l-carni

261 of the resulting ENs was determined using a mitochondrial respiration test.

262 The HCL population had lower mitochondrial respiration, than did the control populati

263 Ps) oppose this phenotype by inducing futile mitochondrial respiration that is uncoupled from ATP syn

264 juncture, we uncovered daily oscillations in mitochondrial respiration that peak during different tim

265 Fat1 cadherin acts as a molecular 'brake' on mitochondrial respiration that regulates vascular smooth

266 er cells import lactate through MCT1 to fuel mitochondrial respiration, the role of MCT1 in glycolysi

267 pendent mitochondrial complex IV and reduces mitochondrial respiration, thereby possibly increasing o

268 Produced during mitochondrial respiration, this reactive oxygen species,

269 esis, adenosine triphosphate production, and mitochondrial respiration through modulation of mitochon

270 tes, microRNA-125b (miR-125b) attenuates the mitochondrial respiration through the silencing of the B

271 f combined targeting of the KRAS pathway and mitochondrial respiration to manage pancreatic cancer.

272 l of NTZ from incubation medium restored the mitochondrial respiration to that of controls.

273 o correlate with a metabolic shift away from mitochondrial respiration towards glycolysis, resulting

274 Hepatic mitochondrial respiration transiently increased and decl

275 epletion reduces mitochondrial integrity and mitochondrial respiration under conditions of cellular s

276 morigenic cells revealed their dependency on mitochondrial respiration versus glycolysis, suggesting

277 light, which was accompanied by an increased mitochondrial respiration via the alternative oxidase pa

278 Although light stress stimulated mitochondrial respiration via the energy-conserving cyto

279 the effects on adenine nucleotides, maximal mitochondrial respiration was approximately 30% lower in

280 Notably, the diurnal regulation of mitochondrial respiration was blunted in mice lacking PE

281 Liver mitochondrial respiration was higher in L rats when usin

282 Moreover, mitochondrial respiration was increased in cells lacking

283 The Dox-induced impairment of mitochondrial respiration was less pronounced in honokio

284 Mitochondrial respiration was measured in permeabilized

285 Mitochondrial respiration was measured in situ and miR a

286 Mitochondrial respiration was measured, alongside levels

287 Mitochondrial respiration was monitored in real time usi

288 nterestingly, a 3.1-fold decrease in maximal mitochondrial respiration was observed in cardiac mitoch

289 Maximal mitochondrial respiration was preserved throughout surge

290 Moreover, mitochondrial respiration was up-regulated in myoblasts

291 Secretion of all hormones and mitochondrial respiration were lowered when FFAR1 or fat

292 studies demonstrated that H3K4me3 levels and mitochondrial respiration were reduced in SH-SY5Y cells

293 , respective measures of glycolytic flux and mitochondrial respiration, were assayed in real time for

294 f toxic lipids by Schwann cells deficient in mitochondrial respiration, which are capable of destroyi

295 ese cells can be suppressed by inhibition of mitochondrial respiration, which suggest that a Fat1-med

296 activity and light metabolism but depends on mitochondrial respiration, which was increased at higher

297 We show that PGC-1beta triggers mitochondrial respiration while protecting enterocytes f

298 echanisms underlying the coordination of the mitochondrial respiration with cell-cycle progression, e

299 tochondria-targeted cyclin B1/Cdk1 increases mitochondrial respiration with enhanced oxygen consumpti

300 we sought to determine the mechanism linking mitochondrial respiration with H2O2 catabolism in brain