ライフサイエンスコーパス: mitogen-activated protein kinase p38

1 nd that EGFR activation is downstream of p38 mitogen-activated protein kinase (p38).

2 m gene expression and activation of p38alpha mitogen-activated protein kinase (p38).

3 e gluconeogenesis in hepatocytes through p38 mitogen-activated protein kinase (p38).

4 c-jun NH2-terminal kinase (JNK), but not p38 mitogen-activated protein kinase (p38).

5 hyperalgesia that required activation of the mitogen-activated protein kinase p38.

6 se to HSV-1 infection and signaling from the mitogen-activated protein kinase p38.

7 t CD28 stimulation alone activates p38 alpha mitogen-activated protein kinase (p38 alpha).

8 The stress-regulated mitogen-activated protein kinase p38 also phosphorylates

9 ion of the transcription factor STAT1 by the mitogen-activated protein kinase p38 and decreased recru

10 se stimulates the phosphorylation of Sp1 via mitogen-activated protein kinase P38 and P44/42.

11 tion of nuclear factor kappaB as well as the mitogen-activated protein kinases p38 and extracellular

12 creased phosphorylation of stress-responsive mitogen-activated protein kinases p38 and JNK-1, 3) coun

13 ctivity was dependent upon activation of the mitogen-activated protein kinases p38 and JNK1 and prote

14 , which dephosphorylates and inactivates the mitogen-activated protein kinases p38 and Jun N-terminal

15 hutoff, viral gene expression, activation of mitogen-activated protein kinases p38 and Jun N-terminal

16 e differentiation of HL-60 cells allowed the mitogen-activated protein kinases p38 and p44/p42 to be

17 inase (ERK) and the common stress-associated mitogen-activated protein kinases p38 and SAPK/JNK.

18 is study Fas activated the stress-responsive mitogen-activated protein kinases, p38 and JNK, within 2

19 e-2, serum C-terminal telopeptide (CTX), p38 mitogen-activated protein kinase (p38), and receptor act

20 protein kinase (c-jun N-terminal Kinase and Mitogen-Activated Protein Kinase-p38), and chemokine (C-

21 ivated) phosphoinositide-dependent kinase 1, mitogen-activated protein kinase, p38, and HER2 (erbB2)

22 osphorylation of c-jun N-terminal Kinase and mitogen-activated protein kinase-p38, and eukaryotic tra

23 gement of CD40 and alpha5beta1 activates the mitogen-activated protein kinases, p38, and extracellula

24 ns and animal models, elevated levels of p38 mitogen-activated protein kinase (p38) are observed in s

25 In this study, we have identified p38 mitogen-activated protein kinase (p38) as a critical sig

26 bosomal S6 kinase, ribosomal protein S6, and mitogen activated protein kinase p38 at 2 and 8 d after

27 tes, specific activation of stress-activated mitogen-activated protein kinase, p38, by upstream activ

28 A novel inhibitor of p38 mitogen-activated protein kinase (p38), CMPD1, identifie

29 tein kinase 2, a downstream substrate of the mitogen-activated protein kinase p38, enhanced the assoc

30 n of nuclear factor kappa B (NF-kB/p65), p38 mitogen-activated protein kinase (p38), ERK, and JNK in

31 Effects of mitogen-activated protein kinases p38, ERK, and JNK were

32 Whereas NGF stimulated the mitogen-activated protein kinases p38, extracellular reg

33 protection from apoptosis and activation of mitogen-activated protein kinase, p38/HOG1, or p70S6 kin

34 ited IL-1beta-induced phosphorylation of the mitogen-activated protein kinase p38 in EL4 thymoma cell

35 nt5a, we observed a strong activation of the mitogen-activated protein kinase p38 in mouse F9 teratoc

36 sponse, demonstrating a critical role of the mitogen-activated protein kinase p38 in regulating the W

37 yl-3-oxide attenuated phosphorylation of the mitogen-activated protein kinase p38 in response to lipo

38 d the role of p53-dependent pathways and p38 mitogen-activated protein kinase (p38) in toxin A-induce

39 icity phosphatase 1, impairs the activity of mitogen-activated protein kinase p38, increases the acti

40 prolactin-induced incorporation was ERK and mitogen-activated protein kinase p38 independent but was

41 A stress-responsive, mitogen-activated protein kinase, p38, is activated by p

42 nhibitors of phosphodiesterase-4 (PDE4), p38 mitogen-activated protein kinase (p38), Janus kinases an

43 Inhibition of the 3 classical mitogen-activated protein kinases (p38 kinase, ERK, and

44 ivated protein kinases, specifically the two mitogen-activated protein kinases p38 kinase and c-Jun N

45 strong phosphorylation and activation of p38 mitogen-activated protein kinase (p38 MAP kinase) and re

46 IL-1beta acts in a p38 mitogen-activated protein kinase (p38 MAP kinase)-depend

47 ome changes correlate with activation of p38 mitogen activated protein kinase (p38 MAPK).

48 e it directly, as well as indirectly through mitogen activated protein kinase p38 MAPK.

49 s upregulated with aging, which enhances p38 mitogen-activated protein kinase (p38 MAPK) activation a

50 llular signal-regulated kinase (ERK) and p38 mitogen-activated protein kinase (p38 MAPK) activities,

51 for this were related to stimulation of p38 mitogen-activated protein kinase (p38 MAPK) and activati

52 s (TIMP-1 and TIMP-2), and activation of p38 mitogen-activated protein kinase (p38 MAPK) and extracel

53 also evaluated the potential role of the p38 mitogen-activated protein kinase (p38 MAPK) and Hsp27 as

54 ing that leads to the stimulation of the p38 mitogen-activated protein kinase (p38 MAPK) and the c-Ju

55 NH2 terminal protein kinases (JNKs) and p38 mitogen-activated protein kinase (p38 MAPK) as well as t

56 terone resulted in activation of JNK and p38 mitogen-activated protein kinase (p38 MAPK) in both cell

57 e we report a role of the stress-induced p38 mitogen-activated protein kinase (p38 MAPK) in the compo

58 Activation of p38 mitogen-activated protein kinase (p38 MAPK) is required

59 We found that the p38 mitogen-activated protein kinase (p38 MAPK) pathway medi

60 Here we show that p38 mitogen-activated protein kinase (p38 MAPK) phosphorylat

61 AICAR also stimulated p38 mitogen-activated protein kinase (p38 MAPK) phosphorylat

62 The noradrenergic and p38 mitogen-activated protein kinase (p38 MAPK) systems are

63 n of glucose levels induced PKCdelta and p38 mitogen-activated protein kinase (p38 MAPK) to increase

64 renergic receptor (beta(3)AR) stimulates p38 mitogen-activated protein kinase (p38 MAPK) via PKA in a

65 Pharmacological inhibition of p38 mitogen-activated protein kinase (p38 MAPK) with either

66 The stress-activated p38 mitogen-activated protein kinase (p38 MAPK), a member of

67 nase/c-Jun N-terminal kinase (SAPK/JNK), p38 mitogen-activated protein kinase (p38 MAPK), and activat

68 B in relation to mitoK(ATP) channels and p38 mitogen-activated protein kinase (p38 MAPK), and whether

69 tivated serine/threonine protein kinase, p38 mitogen-activated protein kinase (p38 MAPK), belongs to

70 Importantly, we show that the p38 mitogen-activated protein kinase (p38 MAPK), cAMP-respon

71 LPS induced phosphorylation of p38 mitogen-activated protein kinase (p38 MAPK), in part, vi

72 ell types certain stresses can stimulate p38 mitogen-activated protein kinase (p38 MAPK), leading to

73 A chemical inhibitor of p38 mitogen-activated protein kinase (p38 MAPK), SB202190, b

74 llular signal-regulated kinase (ERK) and p38 mitogen-activated protein kinase (p38 MAPK), two MAPK is

75 ides induce oxidative stress mediated by p38 mitogen-activated protein kinase (p38 MAPK).

76 sphatase calcineurin (CaN) or the kinase p38 mitogen-activated protein kinase (p38 MAPK).

77 a novel kinase modulator of h-channels, p38 mitogen-activated protein kinase (p38 MAPK).

78 , c-Jun NH(2)-terminal kinase (JNK), and p38 mitogen-activated protein kinase (p38 MAPK).

79 ress-activated protein kinase (SAPK) and p38 mitogen-activated protein kinase (p38 MAPK).

80 ERK), and SB 203580, an inhibitor of the p38 mitogen-activated protein kinase (p38 MAPK).

81 ar signal-regulated kinases (Erk1/2) and p38 mitogen-activated protein kinases (p38 MAPK).

82 explored the role of the osmoregulating, p38 mitogen-activated protein kinase (p38(MAPK)) pathway in

83 ylinositol 3-kinase but not by inhibitors of mitogen-activated protein kinases p38(MAPK) and p42/44(M

84 The p38 mitogen-activated protein kinase (p38-MAPK) pathway was

85 s appear to be mediated by activation of p38 mitogen-activated protein kinase (p38-MAPK), because act

86 olvement of Jun N-terminal kinase (JNK), p38-mitogen-activated protein kinase (p38-MAPK), Raf-1, and

87 The mitogen-activated protein kinase p38 mediates cellular r

88 of c-Jun N-terminal kinase (JNK), but not of mitogen-activated protein kinases p38 or p44.

89 Inhibition of either one of these mitogen-activated protein kinases, p38 or ERK1/2, induce

90 t on IkappaB kinase beta activity and on the mitogen-activated protein kinase p38 (p38).

91 TRAF2 regulates LKLF expression through the mitogen-activated protein kinase p38 pathway.

92 The p38 mitogen-activated protein kinase (p38) pathway is requir

93 DAR regulates one of these pathways, the p38 mitogen-activated protein kinase (p38) pathway, is curre

94 This effect was mediated by mitogen-activated protein kinase p38 phosphorylation and

95 and attenuated lipopolysaccharide-dependent mitogen-activated protein kinase p38 phosphorylation.

96 Mechanistically, we found that mitogen-activated protein kinase p38 played a key role f

97 The mitogen-activated protein kinase p38 plays a critical ro

98 ding p70 S6 kinase, ubiquitin ligase Cbl, or mitogen-activated protein kinase p38 was not observed.

99 In this study, we have shown that p38 mitogen-activated protein kinase (p38) was activated in

100 esponse to such ligands is the activation of mitogen-activated protein kinase p38, whereas double-str

101 ion triggers 'alternative' activation of the mitogen-activated protein kinase p38, whereby the Lck an

102 TACE activation requires the mitogen-activated protein kinase p38, which is activated