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1 (PDGF-BB), a potent fibroblast survival and mitogenic factor.
2 blast growth factor, a potent angiogenic and mitogenic factor.
3 prevented stimulation of the mTOR pathway by mitogenic factors.
4 illa cells to divide or to secrete autocrine mitogenic factors.
5 der serum-deprived conditions and to secrete mitogenic factors.
6 tive stressor, induced the prophage encoding mitogenic factor 4 (MF4), and there was a concomitant in
7 ng inducer of the myelinating phenotype, and mitogenic factors activating receptor tyrosine kinases (
8 ibited O-2A cell proliferation stimulated by mitogenic factors and prevented lineage progression by a
9 ascades lead to the downstream production of mitogenic factors and the proliferation of neighboring s
10 and removal, respectively, in the absence of mitogenic factors and without entering the cell cycle.
11 nonbalding scalp, produce soluble autocrine mitogenic factors and, if so, whether either cell type a
12 (more abundant in the CvfA(-) mutant), SpeB, mitogenic factor, and streptolysin S (less abundant).
13 tokinase), sagA (streptolysin S), and speMF (mitogenic factor) but did not affect transcription of sl
15 s strains have genes encoding IgA proteases, mitogenic factor deoxyribonucleases, nickel/cobalt uptak
18 r fibroblast growth factor (FGF) but not the mitogenic factor epidermal growth factor (EGF) in neuron
21 wth factor (EGF) is a potent chemotactic and mitogenic factor for epidermal keratinocytes, and these
24 fibroblast growth factor (bFGF) is a potent mitogenic factor for smooth muscle cells, myofibroblasts
30 monstrate, for the first time, that STAT5, a mitogenic factor in most cell types, including hematopoi
31 was undertaken first to identify fibroblast mitogenic factors in pulmonary edema fluid, and second t
32 moattractant protein-1 and hypoxia-inducible mitogenic factor, in the murine model of hypoxic pulmona
35 ved growth factor (rhPDGF-BB), a major serum mitogenic factor involved in subcutaneous wound healing.
36 sive molecules, procoagulant activities, and mitogenic factors, leading to development of thrombosis,
37 f fenestrations and expression of growth and mitogenic factors, leading to proliferative changes and
38 se (SPN), and a second secreted protein, the mitogenic factor (MF), routing the former into the host
39 that this growth factor, besides its role as mitogenic factor, plays a fundamental role during testic
40 Physiologic levels of testosterone increased mitogenic factor production by beard, but not scalp cell
43 rowth factors was demonstrated, in that only mitogenic factors that stimulated DNA synthesis 1) led t
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