コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 can happen in all cell cycle phases (except mitosis).
2 les to promote chromosome segregation during mitosis.
3 nd delay decondensation at the completion of mitosis.
4 or efficient chromosome biorientation during mitosis.
5 Aurora kinases are key effectors of mitosis.
6 s on the sperm's chromatin before entry into mitosis.
7 ltimately, the entry into the nucleus during mitosis.
8 NA damage, and reactivation of cardiomyocyte mitosis.
9 ated by Greatwall kinase inhibit PP2A during mitosis.
10 entosis in human epithelial cells, driven by mitosis.
11 romosome condensation and segregation during mitosis.
12 nderlie the processes of DNA replication and mitosis.
13 k1 at the plasma membrane precludes entry to mitosis.
14 compaction during chromosome condensation in mitosis.
15 ining the importance of MCPH1 turnover after mitosis.
16 aulty CENP-A loading, arresting cells before mitosis.
17 CDC20-MAD2 complex but is inactive later in mitosis.
18 to DSBs once cells have already committed to mitosis.
19 establishing proper KT-MT interaction during mitosis.
20 mosomes to spindle microtubules (MTs) during mitosis.
21 eus of a subset of KIT(+) progenitors during mitosis.
22 ications for local chromatin function during mitosis.
23 reased throughout the cell cycle, peaking in mitosis.
24 he protein it encodes in a basal crescent in mitosis.
25 aintenance and determination of cell fate in mitosis.
26 lear accumulation of Stu2 before cells enter mitosis.
27 passenger complex (CPC), a key regulator of mitosis.
28 iption that is necessary to drive cells into mitosis.
29 of genes whose transcript levels peak during mitosis.
30 basal body and the bilobe without affecting mitosis.
31 after adjusting for age, sex, thickness, and mitosis.
32 distortions in spindle MT remodeling during mitosis.
33 oncentration distributions in interphase and mitosis.
34 ased frequency of lagging chromosomes during mitosis.
35 P binding may relieve this inhibition during mitosis.
36 oordinates periodic entry into and exit from mitosis.
37 yotic virulence protein that interferes with mitosis.
38 recruitment to chromatin and microtubules in mitosis.
39 hful segregation of sister chromatids during mitosis.
40 ug-induced mitotic arrest and also in normal mitosis.
41 to the viral genome and to chromatin during mitosis.
42 most growth of a new daughter cell occurs in mitosis.
43 icrocephaly, regulates cell progression into mitosis.
44 which genome replication is completed before mitosis.
45 bility of cardiomyocytes to progress through mitosis.
46 air, transcription, chromatin regulation and mitosis.
47 ense mutations showed no apparent effects on mitosis.
48 nimising damage compounded by replication or mitosis.
49 spindle-based selection of cell fate during mitosis.
50 serine-threonine kinase previously linked to mitosis.
51 ined by repression in the mesoderm following mitosis.
52 at elevated levels from S phase until early mitosis.
53 ediated transcription to levels required for mitosis.
54 but not its centrosome localization, during mitosis.
55 s increase in size in G2 before they undergo mitosis.
56 ional activity at genes associated with cell mitosis.
57 ling axis regulates protein synthesis during mitosis.
58 entation and microtubule organization during mitosis.
59 cells and proper spindle orientation during mitosis.
60 gesting that they are functionally active in mitosis.
61 , drive the temporal ordering of S phase and mitosis.
62 inated by the deubiquitinase VCIP135 in late mitosis.
63 ociates with microtubules after the onset of mitosis.
64 synchronized HeLa cells in late G2 phase and mitosis.
65 the conserved centriole protein Sas-4 during mitosis.
66 ilium begins in quiescence and ends prior to mitosis.
67 re, we describe an unexpected role of ATR in mitosis.
68 hages, without evidence for DNA synthesis or mitosis.
69 over an unexpected role for Aurora A in late mitosis.
70 t together direct chromosome movement during mitosis.
71 uplicated DNA into two daughter cells during mitosis.
72 ression patterns, aneuploidy, and defects in mitosis.
73 ed tubulin led to inhibition of motility and mitosis.
74 phorylation is high in interphase but low in mitosis.
75 plexes that bind spindle microtubules during mitosis.
76 ganization transmissible through meiosis and mitosis.
77 cytoskeletal organization in interphase and mitosis.
78 r Complex (CPC) with well-known functions in mitosis.
79 ease of GCL from the nuclear envelope during mitosis.
80 ross cell generations and bookmarked through mitosis.
81 bodies (SPBs) from S phase until the end of mitosis.
82 s is replaced by fewer but larger domains in mitosis.
83 h their target cyclin B genes (CYCBs) during mitosis.
84 ins surrounding condensed chromosomes during mitosis.
85 crotubules, during metaphase and anaphase of mitosis.
86 d for accurate chromosome segregation during mitosis [1-6] and consists of two copies each of Ska1, S
87 AC protects mitotic accuracy by slowing down mitosis, 53BP1 and USP28 function in parallel to select
89 pathological chromatin bridges formed during mitosis, a trait of Topo IIalpha-deficient cells and a h
90 itical genes involving in pigment synthesis, mitosis, adherent junctions, but also to transactivate g
91 nd breaks (DSBs) prevent cells from entering mitosis allowing cells to repair their genomic damage.
92 neck interactions does not trigger exit from mitosis and 2) loss of these interactions does not prece
93 ibits defective cortical localization during mitosis and a greatly diminished ability to support cyto
94 Polo-like kinase 1 (Plk1) is a key player in mitosis and a target for drug development as it is upreg
96 duction of global CTD phosphorylation during mitosis and affects the expression of genes whose transc
98 xes compact and resolve sister chromatids in mitosis and by which cohesin generates topologically ass
101 ransition genes as well as genes involved in mitosis and cytokinesis such as CHFR, LARP1 and YWHAE.
102 kinase (CRIK), which has known functions in mitosis and cytokinesis, as induced in erythroblasts in
106 nated by the ubiquitin ligase HACE1 in early mitosis and deubiquitinated by the deubiquitinase VCIP13
107 led by ABRO1/BRCC36 and occurs as cells exit mitosis and enter G1, ensuring that telomere cohesion is
110 er-expressing cells partially rescues normal mitosis and impairs the tumorigenesis exerted by miR-26a
111 tubules of different cancer cells, inhibited mitosis and induced multipolar spindle formation in thes
112 s phosphorylation occurs specifically during mitosis and is required for the kinetochore localization
113 gulatory partner that coordinates entry into mitosis and is then destroyed in prometaphase within min
117 omponent of meiotic chromosomes, even though mitosis and meiosis share many processes, including the
124 nyluridine to pulse-label transcripts during mitosis and mitotic exit and found that many genes exhib
126 Using a fusion-based strategy to decouple mitosis and myotube formation, we demonstrate that the c
127 m expression during development could affect mitosis and ploidy in post-mitotic differentiated tissue
128 viously unknown, essential role of INPP5E in mitosis and prevention of aneuploidy, providing a new pe
129 c5 is a dynamic component of the SPBs during mitosis and provide new insight into its regulation duri
130 aration to occur, allowing cells to traverse mitosis and re-enter cycle with a normal complement of 2
131 omes, chromosomes, and kinetochores in early mitosis and shuttles to the midzone spindle at mitotic e
132 Our work highlights novel roles for Shot in mitosis and suggests a mechanism involving Dynein/Dynact
133 is transition do not require passage through mitosis and that conversion to primed pluripotency is li
134 ith inner centromere protein (INCENP) during mitosis and that INCENP is competent to drive accumulati
135 a subset of p150 localizes to the PCL during mitosis and that p150N is required for normal levels of
137 premature separation of sister chromatids in mitosis and thus ensures the fidelity of chromosome segr
138 d onto the intracytoplasmic axonemes late in mitosis and to accumulate near the ends of nascent axone
139 o activate compensatory DNA synthesis during mitosis and to resolve mitotic interlinks, thus facilita
140 ed with the incoming viral genome throughout mitosis and transiently resides in the nucleus until aft
141 r Bgcn is required for spermatogonia to stop mitosis and transition to meiotic prophase and the sperm
142 gene clusters associated with cell cycle and mitosis and with the presence or absence of immune cells
143 e can modulate its grip on microtubules over mitosis and yet retain its ability to couple to microtub
144 atic method, to identify novel regulators of mitosis, and in this study, we describe G2- and S phase-
145 aves a cell into two daughters at the end of mitosis, and must be spatially coordinated with chromoso
146 VprBP binding to CRL4 is reduced during mitosis, and our data suggest that VprBP activation of F
147 g mitosis, is asymmetrically inherited after mitosis, and relocates to the recently formed cytokineti
148 s for the cell to prevent premature entry to mitosis, and retaining Cyclin B1-Cdk1 at the plasma memb
149 phosphorylation at serine 365 occurs during mitosis, and that this phosphorylation is significantly
151 nd prevention of cell cycle progression into mitosis are required for LTR-mediated viral expression,
152 nsive to Mps1, whose activity peaks in early mitosis as a result of its T-loop autophosphorylation.
154 that many genes exhibit transcription during mitosis, as confirmed with fluorescein isothiocyanate-ur
157 ancer cells cluster extra centrosomes during mitosis, avoiding the detrimental effects of multipolar
159 propose that Ska3 phosphorylated by Cdk1 in mitosis binds to Ndc80C and recruits the Ska complex to
160 leavage retain their basal fibres throughout mitosis, both in primary tissue and in older organoids.
161 , is associated with closed chromatin during mitosis, but is increased at certain genes for transcrip
162 ted microcephaly results not from defects in mitosis, but rather the inability of neural progenitors
164 es and tumors, regulates MT stability during mitosis by inhibiting MCAK MT depolymerase activity.
165 s proper segregation of sister chromatids at mitosis by mediating the interaction between KTs and mic
166 up had more phospho-histone H3+ (a marker of mitosis) cardiomyocytes (p = 0.04), and noncardiomyocyte
169 e triggers an anti-oncogenic differentiation-mitosis checkpoint in human epidermal keratinocytes, res
171 e and the restoration of transcription after mitosis constitute an essential time window for AID-indu
173 ncogene-induced differentiation is caused by mitosis control and provide new insight into the mechani
176 ngly, formation of giant cells due to failed mitosis/cytokinesis is common in the blastomere stage of
180 r export of the anillin-like protein Mid1 at mitosis entry confirms or readjusts the division plane a
181 o complete DNA synthesis before the onset of mitosis, eukaryotic cells have evolved complex mechanism
182 bit sister-telomere loss and upon entry into mitosis, exhibit the formation of ultra-fine anaphase br
184 assemble in two phases-a burst at the end of mitosis, followed by steady increase during interphase t
185 we show that cell cycle progression through mitosis following double-stranded DNA breaks leads to th
186 , we identify Nek7, a member of the Never in Mitosis Gene A (NIMA) kinase family, as a regulator of t
187 Cells remain permanently attached following mitosis, giving rise to clonal clusters (staying togethe
188 onents in response to polarizing cues during mitosis, has roles in differentiation and development.
189 genes governing differentiation, rather than mitosis, have an impact on adult hippocampal volume and
190 by Aurora kinases plays an important role in mitosis; however, H3S10ph also marks regulatory regions
193 ckpoint, which inactivates APC/C, often exit mitosis in a process termed "mitotic slippage," which ge
195 esulted in ectopic microtubule asters during mitosis in C. elegans zygotes or HeLa cells, respectivel
196 um (ER) is asymmetrically partitioned during mitosis in epithelial cells just before delamination and
197 nal development by documenting the timing of mitosis in relation to multiple differentiation events f
198 but the contribution of DNA replication and mitosis in stem cell differentiation has not been extens
202 the membrane because fungi undergo a closed mitosis in which the nuclear envelope (NE) remains intac
206 increase in the number of cardiomyocytes in mitosis indicating that the majority of injury-induced c
207 these foci overlapped with telomeres during mitosis, indicating impairment of telomere replication a
208 diploid cells, haploid cells spend longer in mitosis, indicative of problems in chromosome segregatio
211 activation cyclin B1 stability in the first mitosis is also prolonged, leading to the unusual length
216 5, as well as Ser-2, during transcription in mitosis is in contrast to the phosphorylation marks put
217 is slowed by poor nutrients, the duration of mitosis is increased, which suggests that cells compensa
219 iation state of individual BC progenitors at mitosis is not arbitrary but matches the differentiation
221 progression from G1 through S phase and into mitosis is ordered by thresholds of increasing cyclin-de
222 ontrol of sister chromatid separation during mitosis is pivotal to maintaining genomic integrity.
223 The amount of growth required to complete mitosis is reduced in poor nutrients, leading to a large
224 The fidelity of chromosome segregation in mitosis is safeguarded by the precise regulation of kine
226 m unequal partitioning of chromosomes during mitosis, is a hallmark of many cancers, including those
227 in interphase, retains its structure during mitosis, is asymmetrically inherited after mitosis, and
228 reonine protein kinase normally expressed in mitosis, is frequently up-regulated in multiple types of
229 ral stem cells, experience massive errors in mitosis, leading to increased cell death, which reduces
231 known about the functions of the kinases in mitosis, less is known about how the three isoforms func
234 ic checkpoint, premature progression through mitosis, marked aneuploidy and mitotic catastrophe.
235 onal microvessels and provide evidence for a mitosis-mediated mechanism where tumor cells located alo
237 RT1 for degradation and thereby controls the mitosis-meiosis transition in mouse male germ cells.
239 ing history at cell-cycle checkpoints before mitosis, mother cells transmit DNA damage-induced p53 pr
241 exhibited abnormal cytokinesis with delayed mitosis, multipolar spindles, and increased apoptosis, r
242 ing binucleate cells, we show that exit from mitosis occurs when one SPB enters the bud despite the p
243 e identified Src-associated substrate during mitosis of 68 kDa (Sam68) as a novel signaling molecule
250 hanisms that control the extent of growth in mitosis play a major role in cell size control in buddin
251 synthesis (NPAT), DNA damage response (ATM), mitosis (PMF1, CENPN and MAD1L1) and apoptosis (TP53).
252 rst round of transcription immediately after mitosis primarily activates genes involved in the growth
254 erent types of stress, the gene encoding the mitosis-promoting phosphatase Cdc25C is transcriptionall
260 successively replicate their genomes without mitosis, resulting in an increase in nuclear DNA ploidy.
263 s unveil a role for organelle inheritance in mitosis, spindle alignment, and the choice of daughter p
267 hought to be transcriptionally silent during mitosis, technical limitations have prevented sensitive
271 ong the entire length of chromosomes, and in mitosis, these genomic regions come together linearly to
272 gate the episomes into daughter cells during mitosis, they are tethered to cellular chromatin by the
273 erin decreases cortical contractility during mitosis through a signaling cascade leading to multipola
274 he haploids die at or shortly after the last mitosis through activation of a p53-dependent cytotoxic
275 stress, RECQ5 associates with CFSs in early mitosis through its physical interaction with MUS81 and
277 Here we report an essential role of PTEN in mitosis through regulation of the mitotic kinesin motor
279 cycle and assembles a bipolar spindle during mitosis to capture and segregate sister chromatids.
280 Moreover, 53BP1 can transduce prolonged mitosis to cell cycle arrest independently of the spindl
281 pendent phosphorylation of substrates during mitosis to ensure efficient correction of k-MT attachmen
282 that beta-TrCP regulates the transition from mitosis to meiosis in male germ cells by targeting DMRT1
283 molog YTHDC2 facilitates a clean switch from mitosis to meiosis in mouse germ cells, revealing a cons
285 at common fragile sites (CFSs) during early mitosis to trigger DNA-repair synthesis that ensures fai
286 volume change (e.g., as occurs upon entry to mitosis) to regulate function by altering biomolecular c
289 understand the role of kinesin-8 proteins in mitosis, we studied the effects of deletion of the fissi
290 while also enabling cyclin E destruction in mitosis, when inappropriate cyclin E expression is genot
291 verride a131-induced growth arrest and enter mitosis where a131's ability to de-cluster supernumerary
292 ndle poles (MSPs) during the early phases of mitosis where it colocalizes with its paralog WDR62, whi
294 n is largely retained at a low level through mitosis, whereas the amplitude of transcription observed
295 of KT-MT structure and SAC regulation during mitosis which is essential for chromosomal stability.
296 vidence for a partially activated DDR during mitosis, which leads to ongoing chromosome segregation e
298 tion and periodic expression of genes during mitosis with entailing CTD phosphorylation and dephospho
300 es BRCA2-deficient cells to progress through mitosis with under-replicated DNA, which elicits severe
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。