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1 te detachment and/or mitotic podocyte death (mitotic catastrophe).
2 empted, it may lead to aberrant mitosis (ie, mitotic catastrophe).
3 cells eliminates Ras-activated cells through mitotic catastrophe.
4 ycle checkpoints, resulting in cell death by mitotic catastrophe.
5 only transient G(2)-M arrest before death by mitotic catastrophe.
6 emperature-sensitive lethal phenotype termed mitotic catastrophe.
7 iant, multinucleated cells characteristic of mitotic catastrophe.
8 and avoidance of replication catastrophe and mitotic catastrophe.
9 er hand, forced expression of KLF14 leads to mitotic catastrophe.
10 ugh into mitosis, resulting in cell death by mitotic catastrophe.
11 ed mitotic arrest then led to cell death via mitotic catastrophe.
12 s into mitosis where they ultimately died by mitotic catastrophe.
13 required for cell cycle progression undergo mitotic catastrophe.
14 PS SMCs exhibit prolonged mitosis and die of mitotic catastrophe.
15 on after DNA replication, ultimately causing mitotic catastrophe.
16 n clusters of nuclear vesicles indicative of mitotic catastrophe.
17 nd apoptosis induced in vivo in part through mitotic catastrophe.
18 ssion through mitosis, marked aneuploidy and mitotic catastrophe.
19 sing cells from centrosome reduplication and mitotic catastrophe.
20 s (G2/M) checkpoint activation and resultant mitotic catastrophe.
21 reas p53 wild-type tumors arrested, avoiding mitotic catastrophe.
22 , have more than two centrosomes and undergo mitotic catastrophe.
23 increased the frequency of radiation-induced mitotic catastrophe.
24 postmitotic endoreduplication checkpoint and mitotic catastrophe.
25 oint, leading to premature mitotic entry and mitotic catastrophe.
26 mitotic transition and results in increased mitotic catastrophe.
27 cell death, both of which are indicative of mitotic catastrophe.
28 damage- and heat shock-induced death, and in mitotic catastrophe.
29 itions of RBM3 downregulation, cells undergo mitotic catastrophe.
30 lity characterized by gross polyploidism and mitotic catastrophe.
31 itochondrial membrane potential, followed by mitotic catastrophe.
32 rther differs from cellular death defined by mitotic catastrophe.
33 t; centrosome overduplication; and, finally, mitotic catastrophe.
34 h the emergence of disorganized anaphase and mitotic catastrophe.
35 nd a strong Plk1 hypomorph underwent serious mitotic catastrophe.
36 efects, delays cells in mitosis, and induces mitotic catastrophe.
37 nhanced, temozolomide-induced senescence and mitotic catastrophe.
38 agenicity by a combination of senescence and mitotic catastrophe.
39 cytokinesis and, thereby, is responsible for mitotic catastrophe.
40 cells bypassing G(2)-M arrest and undergoing mitotic catastrophe.
42 lt cardiac myocytes resulted in induction of mitotic catastrophe, a previously unreported event in ca
43 c progression with 4N DNA content leading to mitotic catastrophe after abrogation of the S or G2 chec
45 s the engagement of apoptosis, probably by a mitotic catastrophe, after 8 Gy of gamma-irradiation in
46 -M checkpoint function, leading to increased mitotic catastrophe and a modest increase in radiation s
47 ately resulting in centrosome reduplication, mitotic catastrophe and abrogation of cell proliferation
48 ough telomeric DNA end fusions, resulting in mitotic catastrophe and apoptosis; and 4) induction of c
52 g prolongation, multipolarity, misalignment, mitotic catastrophe and loss of spindle checkpoint, that
53 4 knockdown or overexpression coincided with mitotic catastrophe and multinucleation that are typical
54 icroscopy studies revealed that it induces a mitotic catastrophe and precocious mitotic exit even in
56 however, with increases in radiation-induced mitotic catastrophe and senescence and produced only a m
57 ion abnormalities lead to cell death through mitotic catastrophe and that cell death occurred also fr
58 drive abnormal mitotic spindle formation and mitotic catastrophe and that deregulated FOXM1 and KIF20
59 hat FoxM1 depletion causes cell death due to mitotic catastrophe and that inhibiting FoxM1 represents
60 ing both pathways simultaneously resulted in mitotic catastrophe and tumor apoptosis and markedly red
64 asing the percentage of cells that underwent mitotic catastrophe as a consequence of G(2) checkpoint
65 sels against repeated doses, and introducing mitotic catastrophe (as opposed to arbitrary delayed cel
67 of EAG2 expression results in G2 arrest and mitotic catastrophe associated with failure of premitoti
69 odocyte injury, MDM2 drives podocyte loss by mitotic catastrophe, but the function of MDM2 in resting
70 ide an appraisal of the in situ pathology of mitotic catastrophe compared with other proposed types o
71 utated but not in wild-type cells leading to mitotic catastrophe, defective cell division and apoptos
73 human homolog of Xenopus gene which prevents mitotic catastrophe), directly binds to the EpRE and int
74 y of the SLX4 complex is critical to prevent mitotic catastrophe following common fragile site expres
76 abditis elegans ATR) and rad-5/clk-2 prevent mitotic catastrophe, function in the S-phase checkpoint
79 ubject to inhibitory phosphorylation induced mitotic catastrophe in cells lacking p53, the p53-expres
80 actor 1; Xenopus laevis XPMC2, prevention of mitotic catastrophe in fission yeast; Drosophila melanog
82 ur studies show centrosome reduplication and mitotic catastrophe in osteosarcoma cells inducibly expr
83 dc2/cyclin B complexes, specifically induced mitotic catastrophe in pRb-deficient myocytes, consisten
84 y a high level of spontaneous DNA damage and mitotic catastrophe in the absence of the master checkpo
87 ion stabilizes APC substrates and results in mitotic catastrophe including centrosome overduplication
90 more than two centrosomes and, indicative of mitotic catastrophe, irregularly shaped nuclei or multip
91 lear whether the termination of mitosis in a mitotic catastrophe is regulated by DNA damage checkpoin
93 olyploidy are hallmarks of radiation-induced mitotic catastrophe (MC), a common phenomenon occurring
94 nuclear bridging, nuclear fragmentation, and mitotic catastrophe, mirroring the effects of HDACi on c
95 reated with nocodazole were found to undergo mitotic catastrophe more readily when endogenous caspase
96 tion during metaphase, ultimately leading to mitotic catastrophe, multinucleation, and the loss of st
97 d protein (XORC2) by its ability to rescue a mitotic-catastrophe mutant of the fission yeast Schizosa
99 ng triggers multipolar spindle formation and mitotic catastrophe, offering an attractive therapeutic
102 s because apoptosis-resistant cells die from mitotic catastrophe or undergo senescence-like terminal
104 s originally isolated as a suppressor of the mitotic catastrophe phenotype of a cdc2-3w wee1-50 doubl
105 dition, filamin-A-deficient M2 cells undergo mitotic catastrophe-related nuclear fragmentation after
106 new mechanistic model for the phenomenon of mitotic catastrophe, relying on a combination of unregul
107 tinucleated cells that then arrest or die by mitotic catastrophe, revealing a new role for Myc in the
109 cell death that resembles a necrotic form of mitotic catastrophe suggesting that CD95 protects cancer
110 proliferation, inhibition of apoptosis, and mitotic catastrophe, suggesting an important potential m
114 ving into an aberrant mitosis, indicative of mitotic catastrophe that may contribute to increased sen
115 attenuates mitotic transition and increases mitotic catastrophe, thereby inhibiting glioma developme
117 uction of a further mechanism of cell death, mitotic catastrophe, was observed in the more sensitive
118 arrested state, which displays hallmarks of mitotic catastrophe, was prevented by expression of eith
119 a screen designed to discover suppressors of mitotic catastrophe, we identified the Xenopus ortholog
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