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1 h a wound-closure assay in the presence of a mitotic inhibitor.
2 B protein levels in response to colcemid, a mitotic inhibitor.
3 otypes suggest a possible role for Skb1 as a mitotic inhibitor.
4 ibed role for PAK-related protein kinases as mitotic inhibitors.
5 in response to treatment with epirubicin and mitotic inhibitors.
6 complex/cyclosome (APC/C), such as the early mitotic inhibitor 1 (Emi1) and spindle checkpoint protei
7 otic checkpoint complex and interphase early mitotic inhibitor 1 (Emi1) ensures the correct order and
10 rowing body of evidence indicates that early mitotic inhibitor 1 (Emi1) is essential for genomic stab
12 We identify the mutation as being in early mitotic inhibitor 1 (emi1), a negative regulator of the
16 t rather the relative abundance of the Swe1p mitotic inhibitor and the mitosis-promoting cyclins.
17 ompounds 8, 9a, 18 and 19a are highly potent mitotic inhibitors and selectively cytotoxic to cancer c
18 ization to other tyrosine kinase inhibitors, mitotic inhibitors, and platinum-based therapy, there is
19 AdBDNF, and intraventricular infusion of the mitotic inhibitor Ara-C completely blocked the performan
20 dicate that MCAK affects cell sensitivity to mitotic inhibitors by modulating the frequency of microt
21 can protect normal proliferating cells from mitotic inhibitors by preventing their entry into mitosi
25 s to evaluate the activity of ON 01910.Na, a mitotic inhibitor, in in vitro and in vivo models of pan
29 entry and exit and allow the suggestion that mitotic inhibitors may have selective effects in tumors
31 is lower than on hard even in the absence of mitotic inhibitors normally used to temper the astrocyte
32 kpoint proteins, formation of interphase and mitotic inhibitors of Cdc20, and correction of faulty mi
35 nt kinase (Cdk)-2 and -4, high levels of the mitotic inhibitors, p21Waf1 and p27Kipl, and decreased c
36 -like growth factor-I receptor (IGF-IR), and mitotic inhibitors, respectively, clustered with others
37 Finally, cells chronically treated with the mitotic inhibitor retinoic acid displayed a selective do
40 d size would cause G2 arrest enforced by the mitotic inhibitor Swe1p, explaining previous findings th
43 enabling them to survive in the presence of mitotic inhibitors, which is a characteristic of mature
44 ere discovered as proapoptotic molecules and mitotic inhibitors with potencies at low nanomolar conce
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