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1 dle assembly, pole splitting, and a delay in mitotic progression.
2 essary for locating NIMA back to SPBs during mitotic progression.
3 80 complex formation, and adversely affected mitotic progression.
4 eostat to fine-tune Aurora B activity during mitotic progression.
5 dissolution of sister chromatid cohesion and mitotic progression.
6 f stable KT-MT fibers (K-fibers), disrupting mitotic progression.
7 me, and the outer nuclear membrane to direct mitotic progression.
8 plicated in playing an important role during mitotic progression.
9 mporal regulation of miR159 by APC/C ensures mitotic progression.
10 et), a Mad2-binding protein, is required for mitotic progression.
11 f early mitotic spindle poles and the timely mitotic progression.
12 e regulation of PP2A localization for proper mitotic progression.
13 ylation of KIBRA on Ser(539) plays a role in mitotic progression.
14 omplish normal bipolar spindle formation and mitotic progression.
15 hestrated regulation is important for normal mitotic progression.
16 ation of these pathways results in defective mitotic progression.
17 res, defects in spindle assembly and blocked mitotic progression.
18 on, neither trap had a discernable effect on mitotic progression.
19 to be required in smaller amounts for normal mitotic progression.
20 he genes essential for G(1)/S transition and mitotic progression.
21 re NIMA-related kinases essential for proper mitotic progression.
22 t prolong interphase but instead interrupted mitotic progression.
23 is degraded during G(2) and mitosis to allow mitotic progression.
24 dissolution of sister chromatid cohesion and mitotic progression.
25 ation of Aurora kinase B, a key regulator of mitotic progression.
26 e exosome-independent role in cell cycle and mitotic progression.
27 ssion of the G(2)/M phase genes and impaired mitotic progression.
28 ulatory role for Chk1 phosphorylation during mitotic progression.
29 and another as a factor required for proper mitotic progression.
30 nt translation that is required for accurate mitotic progression.
31 e requirement of S326 phosphorylation during mitotic progression.
32 Both proteins are required for normal mitotic progression.
33 vates the spindle checkpoint, and delays the mitotic progression.
34 oes delay inactivation of Mad2 signaling and mitotic progression.
35 or of spindle dynamics that is essential for mitotic progression.
36 basic cellular machinery required for robust mitotic progression.
37 rkinetochore tension at metaphase and alters mitotic progression.
38 abrogates the requirement for tankyrase 1 in mitotic progression.
39 hreonine protein kinase essential for normal mitotic progression.
40 AP2 protein level is critical for the normal mitotic progression.
41 is critical for spindle assembly and normal mitotic progression.
42 atwall have previously been shown to disrupt mitotic progression.
43 me (APC/C), a ubiquitin ligase that controls mitotic progression.
44 r spindle assembly, chromosome alignment, or mitotic progression.
45 tant component of this system that regulates mitotic progression.
46 demonstrated that NHK-1 is also required for mitotic progression.
47 me (APC/C), a ubiquitin ligase essential for mitotic progression.
48 ical for G(1)/S transition and essential for mitotic progression.
49 vertebrate Plks, is essential for successful mitotic progression.
50 s on Cdc55 and Igo2 did not cause defects in mitotic progression.
51 ulans, dynein is not apparently required for mitotic progression.
52 me (APC/C), a ubiquitin ligase that controls mitotic progression.
53 regulatory proteins to the spindle poles for mitotic progression.
54 tion of hepatocellular metaphase-to-anaphase mitotic progression.
55 , whose activity appears to be essential for mitotic progression.
56 h we show is important for TPX2 function and mitotic progression.
57 regation relies on the precise regulation of mitotic progression.
58 is an important regulator of hepatocellular mitotic progression.
59 cilitate evasion from apoptosis and aberrant mitotic progression.
60 tion whose degradation is a prerequisite for mitotic progression.
61 hospholigand binding is necessary for proper mitotic progression.
62 , chromosome congression defects and delayed mitotic progression.
63 that is responsive to DNA damage as well as mitotic progression.
64 ses whose combined function is important for mitotic progression.
65 esent a new signaling pathway that regulates mitotic progression.
66 tants show that both pathways are needed for mitotic progression.
67 organization, as well as in other aspects of mitotic progression.
68 chanisms that regulate Aur-A activity during mitotic progression.
69 elomere elongation, and specifically affects mitotic progression.
70 ted that overexpression of Pin2/TRF1 affects mitotic progression.
71 h chromatin compaction and the regulation of mitotic progression.
72 is known about how Pin2/TRF1 is involved in mitotic progression.
73 multisubunit ubiquitin ligase that mediates mitotic progression.
74 iferative activity due to a potent arrest of mitotic progression.
75 hown to play an important role in regulating mitotic progression.
76 mines whether it also plays a role in normal mitotic progression.
77 at MAP kinases might be important for normal mitotic progression.
78 versatile ubiquitin code is read out during mitotic progression.
79 cate that Plx1 may participate in control of mitotic progression.
80 liated cells to promote ciliogenesis without mitotic progression.
81 bule attachment, a condition known to arrest mitotic progression.
82 Delta plays a direct role in the control of mitotic progression.
83 misattached kinetochores to prevent further mitotic progression.
84 hugoshins is a fundamental step in mammalian mitotic progression.
85 SET8 reaccumulation is important for normal mitotic progression.
86 tochore in activating APC/C-Cdc20 for normal mitotic progression.
87 mitotic blockers and agents that accelerate mitotic progression.
88 oss of centromeric cohesion disrupts orderly mitotic progression.
89 ase of the cell cycle, with stable arrest of mitotic progression.
90 atic cells, where DNA damage fails to affect mitotic progression.
91 ers defects in spindle assembly and prevents mitotic progression.
92 otubule spindle function, thereby inhibiting mitotic progression.
93 ial role in genome integrity maintenance and mitotic progression.
94 R signaling in a pathway to promote faithful mitotic progression.
95 accelerated entry into mitosis, but delayed mitotic progression.
96 cells disturbs neither spindle formation nor mitotic progression.
97 r Mad2 and promotes silencing of the SAC and mitotic progression.
98 chestrates accurate chromosome alignment and mitotic progression.
99 SUN proteins in bipolar spindle assembly and mitotic progression.
100 nal activity of SRSF1 is required for normal mitotic progression.
101 d is crucial for proper spindle assembly and mitotic progression.
102 e loading of Bub3, chromosome alignment, and mitotic progression.
103 APC(Cdc20)) plays pivotal roles in governing mitotic progression.
104 mpletely eliminated Nuf2 binding and blocked mitotic progression.
105 mposed by TIF1gamma knockdown and allows for mitotic progression.
107 the yeast Ndc80 tail is required for timely mitotic progression and accurate chromosome segregation.
108 Spindle integrity is critical for efficient mitotic progression and accurate chromosome segregation.
109 -Cyclin A, Cyclin B, and Cyclin B3-influence mitotic progression and are degraded in a stereotyped se
111 applies sophisticated live imaging to assess mitotic progression and cell cycle control in these cell
118 ts by CLASP1, astrin and Kif2b is central to mitotic progression and chromosome segregation fidelity.
122 mitotic apparatus, An-Mlp1 may help monitor mitotic progression and coordinate efficient mitotic exi
123 previously underappreciated role for Ras in mitotic progression and demonstrate a pharmacologically
124 cal link between two important regulators of mitotic progression and demonstrate the critical role of
125 ssential ubiquitin protein ligase, regulates mitotic progression and exit by enhancing degradation of
128 tion mediated by NDR1 kinase is required for mitotic progression and for Sgo1 binding to mitotic cent
130 tify a common function for NudE and NudEL in mitotic progression and identify an alternative mechanis
132 aging, we show that BET inhibition prolonged mitotic progression and induced mitotic cell death, both
133 a, is a novel anticancer agent that inhibits mitotic progression and induces apoptosis in most cancer
134 oforms of cyclin E (LMW-E) overexpression on mitotic progression and its link to genomic instability
135 TM and DNA damage and is required for proper mitotic progression and maintenance of genomic stability
136 escribed mechanism for interrupting faithful mitotic progression and may ultimately inform the design
137 kinase that plays important roles in normal mitotic progression and mitotic checkpoint signaling, co
138 hat Pak1 is required for cell proliferation, mitotic progression and Plk1 activity in HeLa cells.
139 nd approximately 100 candidate regulators of mitotic progression and proliferation; the availability
140 have shown that pRB inactivation also slows mitotic progression and promotes aneuploidy, but reasons
141 central switch at kinetochores that defines mitotic progression and promotes fidelity by temporally
143 etion of Borealin by RNA interference delays mitotic progression and results in kinetochore-spindle m
145 ion of oncogenic KRAS in regulating accurate mitotic progression and suggest new avenues to therapeut
146 sults emphasize the importance of CENP-C for mitotic progression and suggest that Vmw110 may be inter
147 H2 terminus of human CENP-A is essential for mitotic progression and that localization of CENP-C, ano
148 ight the potential role of EDD in regulating mitotic progression and the cellular response to perturb
149 he fly ATR orthologue), the abnormalities of mitotic progression and the cyclin B protein level were
150 define an essential role for IKK2 in normal mitotic progression and the maintenance of spindle bipol
151 s1 is a protein kinase that regulates normal mitotic progression and the spindle checkpoint in respon
152 evel of Plk1 depletion caused a 2-h delay of mitotic progression, and a high degree of Plk1 depletion
154 localization of Bub3 was required for normal mitotic progression, and Bub3 and Cdc20 colocalized at t
155 and dynamic regulation of mitotic spindles, mitotic progression, and chromosome segregation fidelity
156 role for Pak in regulating Plk1 activity and mitotic progression, and connect Pak to the complex prot
157 -like kinase 1 (Plk1) is critical for proper mitotic progression, and its association with the centro
158 hat Wdr62 interacts with Aurora A to control mitotic progression, and loss of these interactions lead
159 entrosomal Aurora-A kinase (AURKA) regulates mitotic progression, and overexpression and hyperactivat
160 the importance of ROS scavenging for normal mitotic progression, and provide a reference for judicio
161 ession of many late cell cycle genes, delays mitotic progression, and reduces fitness in budding yeas
163 Additionally, hBUBR1 is essential for normal mitotic progression as it prevents cells from prematurel
165 activity of Bora is also required for normal mitotic progression, as knockdown of Bora activates the
166 trate that Golgi disassembly is required for mitotic progression because failure to vesiculate the Go
167 Calmodulin activation was essential for mitotic progression: both entry into mitosis and exit fr
168 tivating enzyme are zygotically required for mitotic progression but are dispensable for cell viabili
169 cle and have been specifically implicated in mitotic progression, but little is known about the signa
170 g of Golgi cisternae, vesicle tethering, and mitotic progression, but their specific functions are un
171 ears to be essential for the coordination of mitotic progression, but which also plays an unexpected
172 reticulum-localized protein FAM134A impairs mitotic progression by affecting metaphase plate alignme
174 is the primary upstream kinase that directs mitotic progression by phosphorylation of a large number
175 g protein that controls spindle dynamics and mitotic progression by regulating MT depolymerases.
176 In this study, we show that USP2a mediates mitotic progression by regulating the stability of Auror
177 ome pathway ensures the unidirectionality of mitotic progression by removing cell-cycle regulators re
178 hat this physical interaction is crucial for mitotic progression by targeting polo kinase activity to
179 ured E4F(-/-) blastocysts exhibit defects in mitotic progression, chromosomal missegregation, and inc
180 These defects are associated with impaired mitotic progression coupled with abnormal mitotic spindl
181 or CaM-dependent activation cause defects in mitotic progression, cytokinesis, and ciliary resorption
182 well as wild-type siblings, indicating that mitotic progression delays alone do not alter overall gr
183 g spindle assembly renders Eg5 essential for mitotic progression, demonstrated by the lethal effects
184 ese cells initiated anaphase after a delayed mitotic progression due to the presence of unaligned chr
186 us, our data implicate mnm as a regulator of mitotic progression during the proliferative phase of ey
187 ase Cdc28 is a well established regulator of mitotic progression, evidence for a direct role in mitot
188 1 microtubule-binding domain perturbs normal mitotic progression, explaining the critical role of the
189 est for extended periods, moderate delays in mitotic progression have significant effects on the resu
190 Although these defects transiently delayed mitotic progression, HeLa cells initiated anaphase witho
191 nstrate that several fundamental features of mitotic progression in adult stem and progenitor cells a
196 result in defective chromosome alignment and mitotic progression in cells using a CRISPR/Cas9-based r
197 enes are required maternally for egg laying, mitotic progression in early embryos, and embryonic surv
198 that changes in organismal physiology affect mitotic progression in germline stem and progenitor cell
200 ntent imaging to identify genes required for mitotic progression in human cancer cells and applied it
201 largely dispensable for rounding and timely mitotic progression in isolated cells, it is needed to d
202 Survivin is induced by OX40 independent of mitotic progression in late G1, and blocking Survivin su
206 nk between 3R and 4R-Tau isoform expression, mitotic progression in neuronal progenitors and post-mit
207 e conserved checkpoint kinase Chk1 regulates mitotic progression in response to DNA damage and replic
208 conserved checkpoint kinase, Chk1, regulates mitotic progression in response to DNA damage by blockin
209 y as well as targeting proteins that mediate mitotic progression in response to kinetochore attachmen
211 anges, such as dietary intake or age, affect mitotic progression in stem and/or progenitor cells is l
215 hful inheritance of chromosomes by arresting mitotic progression in the presence of kinetochores that
216 ein phosphatases, on different parameters of mitotic progression in the presence or absence of stathm
217 to 14-3-3 may partially modulate hepatocyte mitotic progression, in association with nuclear redistr
218 c kinase responsible for multiple aspects of mitotic progression, including assembly of the outer kin
219 ation of key cell-cycle proteins involved in mitotic progression, including cyclin-dependent kinase 1
222 f responding cells and the normal pattern of mitotic progression, indicating that the other CD28-indu
223 re linked at least in part to alterations in mitotic progression induced by defective ARF6 cycling.
224 forces ( approximately 5 nN) that accelerate mitotic progression, intermediate forces where cells res
226 dy illustrates that such an expert system of mitotic progression is able to highlight the complexity
235 s, Lte1 has dual functions for regulation of mitotic progression: it both induces mitotic exit and pr
236 elling might have important consequences for mitotic progression: it might contribute to produce stro
237 ent, blocks cell proliferation by inhibiting mitotic progression leading to mitotic and postmitotic a
238 s, uncoupling of replication initiation from mitotic progression led to altered genome ploidy in the
239 addition to the anaphase promoting complex, mitotic progression may involve another E3 ubiquitin lig
240 migration; however, more dramatic defects on mitotic progression, mitotic orientation, and mitotic ch
243 leucine zipper kinase (MELK) is critical for mitotic progression of cancer cells; however, its mechan
245 croscopy, we analyzed the dynamic process of mitotic progression of HeLa(H2B)-GFP cells lacking CENP-
249 whether MAP kinase function is required for mitotic progression or the spindle assembly checkpoint i
251 kinase activity of proteins associated with mitotic progression, particularly the cyclin B1-p34cdc2
253 (-/-) mammary mouse tumours, suggesting that mitotic progression promotes PARP-inhibitor-induced cell
254 centrations that are insufficient to inhibit mitotic progression, PTX induced both p53 and p21 causin
259 ulation, rs11556924 alters the regulation of mitotic progression resulting in an extended mitosis.
260 and loss of IRC15 function leads to delayed mitotic progression, resulting from failure to establish
261 of these cells and emphasized the timing of mitotic progression, spindle structure, and chromosome b
262 ive effect of PDGF in SMCs without affecting mitotic progression, suppressed neointimal formation in
263 We found that two genes responsible for mitotic progression, SWI5 and CLB2, exhibit a mitosis-de
264 fiber assembly, ch-TOG/TACC3 recruitment and mitotic progression that also resulted from Nek6 depleti
265 an aberrant accumulation of PTTG1 disrupting mitotic progression that could contribute to thyroid car
266 ke kinases is important in the regulation of mitotic progression; this work keys on one member, namel
267 role for EGR-1 in regulating hepatocellular mitotic progression through the spindle assembly checkpo
269 hese results suggest that moderate delays in mitotic progression trigger the initiation of centriole
270 ir response to external forces are linked to mitotic progression under conditions of mechanical confi
271 ce revealed that the pathway is required for mitotic progression under normal growth conditions.
272 The spindle assembly checkpoint (SAC) delays mitotic progression until all sister chromatid pairs ach
273 served role of these checkpoints is to block mitotic progression until DNA replication and repair are
274 The spindle assembly checkpoint arrests mitotic progression until each kinetochore secures a sta
279 The spindle assembly checkpoint (SAC) delays mitotic progression when chromosomes are not properly at
280 Bub1 (KBB) pathway is required during normal mitotic progression when kinetochores are misaligned but
281 erase inhibitors by Chk1 siRNA, which showed mitotic progression with 4N DNA content leading to mitot
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