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1 tion at 1 year (6.0% of patients with severe mitral regurgitation).
2 art Association class, large QRS, and severe mitral regurgitation).
3 ely used in high surgical risk patients with mitral regurgitation.
4 mitral valve repair in patients with primary mitral regurgitation.
5 TR only occurred in those with no/trace/mild mitral regurgitation.
6 us in future repair algorithms for secondary mitral regurgitation.
7 opment of new repair techniques for ischemic mitral regurgitation.
8 l spectrum is associated with higher risk of mitral regurgitation.
9 , which was similar for FMR and degenerative mitral regurgitation.
10 ntricular size, that governs the severity of mitral regurgitation.
11 ce and severity of LV dysfunction in organic mitral regurgitation.
12 alve disease, particularly in the setting of mitral regurgitation.
13 e as predictors of prognosis in asymptomatic mitral regurgitation.
14 stic value in the management of asymptomatic mitral regurgitation.
15 rategy to improve the lives of patients with mitral regurgitation.
16 rformed in most elderly patients with severe mitral regurgitation.
17 n alternative to surgery for treating severe mitral regurgitation.
18 ejection fraction (EF) impairment in organic mitral regurgitation.
19 his large population of patients with severe mitral regurgitation.
20 pair and replacement in patients with native mitral regurgitation.
21 es of percutaneous repair versus surgery for mitral regurgitation.
22 nal (45%), degenerative (48%), or mixed (6%) mitral regurgitation.
23 s after mitral valve repair for degenerative mitral regurgitation.
24 ischemic mitral regurgitation and myxomatous mitral regurgitation.
25 egurgitation and 11 patients with myxomatous mitral regurgitation.
26 .38; 95% confidence interval, 1.27-4.45) for mitral regurgitation.
27 e distinction between organic and functional mitral regurgitation.
28 able to less severe and subclinical cases of mitral regurgitation.
29 ol may be of importance in the prevention of mitral regurgitation.
30 h an increased risk of primary and secondary mitral regurgitation.
31 ter mitral valve repair for the treatment of mitral regurgitation.
32 f the indication for surgery in degenerative mitral regurgitation.
33 reated in Yorkshire swine by inducing severe mitral regurgitation.
34 antly more recurrences of moderate or severe mitral regurgitation.
35 ef in such patients via reduction of SAM and mitral regurgitation.
36 ts with moderate-to-severe or severe organic mitral regurgitation.
37 of percutaneous interventions for functional mitral regurgitation.
38 strain intensity was higher in patients with mitral regurgitation (0.15+/-0.03) than in normals (0.11
39 re were 3950 patients with any VHD: 3101 had mitral regurgitation, 1179 with tricuspid regurgitation,
40 dence interval, 2.03-6.21) for aortic and/or mitral regurgitation, 19.8% versus 4.7% (odds ratio, 5.2
41 IPMD, mitral valve geometry, and severity of mitral regurgitation, 67 patients with ischemic heart di
42 ed in patients with moderate/severe ischemic mitral regurgitation (9.6 +/- 2.8 mm), but preserved in
45 absence of SAM and significant reduction in mitral regurgitation, although high systolic LVOT veloci
46 1 normal subjects, 11 patients with ischemic mitral regurgitation and 11 patients with myxomatous mit
47 p period, 28,655 (0.52%) were diagnosed with mitral regurgitation and a further 1,262 (0.02%) were di
48 in significantly reduced rates of recurrent mitral regurgitation and adverse cardiac events over tim
49 le to provide insights into the mechanism of mitral regurgitation and also to help discern the conseq
50 nular incisions resulted in mild to moderate mitral regurgitation and an increased vena contracta are
51 superior repair with decreased recurrence of mitral regurgitation and enhanced reversal of left ventr
52 ustained 1-year reduction of the severity of mitral regurgitation and improvement of clinical symptom
54 al annular dynamics are impaired in ischemic mitral regurgitation and myxomatous mitral regurgitation
57 1+/-12 years and 69% men) with >/=3+ primary mitral regurgitation and preserved left ventricular ejec
58 ymptomatic patients with significant primary mitral regurgitation and preserved left ventricular ejec
59 alvulopathy (>/= mild aortic or >/= moderate mitral regurgitation) and changes in regurgitant grade a
60 cardiac magnetic resonance and no or trivial mitral regurgitation, and 16 (6 female patients; median
61 et mobility, decreased severity of recurrent mitral regurgitation, and improved reverse remodeling wi
62 lmonale, left ventricular ejection fraction, mitral regurgitation, and inferior vena cava variability
63 controls and patients with aortic stenosis, mitral regurgitation, and left ventricular assist device
64 apy, implantable cardioverter defibrillator, mitral regurgitation, and mitral valve procedures; an in
65 New York Heart Association functional class, mitral regurgitation, and N-terminal pro-B-type natriure
66 ree survival was predicted by age, degree of mitral regurgitation, and postprocedural hemodynamic dat
67 k surgery, symptomatic severe AS, coexisting mitral regurgitation, and preexisting coronary disease w
68 etic resonance (CMR) can accurately quantify mitral regurgitation, and we examined whether this was a
69 ischemic mitral regurgitation and myxomatous mitral regurgitation, annular dynamics and anatomy are a
71 gitation (9.98 +/- 155 cm(2)) and myxomatous mitral regurgitation annuli (13.29 +/- 3.05 cm(2)) were
73 ography evidence of at least moderate aortic/mitral regurgitation, aortic stenosis, or prior valve su
74 ng severe or mild/moderate LV dysfunction in mitral regurgitation are doubted and poorly followed in
75 linical outcomes after surgical treatment of mitral regurgitation are worse if intervention occurs af
77 recommended in patients with severe primary mitral regurgitation as soon as ejection fraction (EF) <
78 owed a persistent reduction in the degree of mitral regurgitation at 1 year (6.0% of patients with se
79 The rate of moderate or severe recurrence of mitral regurgitation at 12 months was higher in the repa
81 06; P = .02), presence of moderate or severe mitral regurgitation at discharge (1.65; 95% CI, 1.21-2.
83 s) undergoing primary repair of degenerative mitral regurgitation between 2003 and 2011, 419 (65%) un
84 h a reduced prevalence of moderate or severe mitral regurgitation but an increased number of untoward
85 repair provided a more durable correction of mitral regurgitation but did not significantly improve s
86 h a reduced prevalence of moderate or severe mitral regurgitation, but patients had more adverse even
87 cement provided a more durable correction of mitral regurgitation, but there was no significant betwe
88 ventricular size, shape, and mass and reduce mitral regurgitation by reverse remodeling of the failin
89 a common condition that is a risk factor for mitral regurgitation, congestive heart failure, arrhythm
90 c, severe functional, degenerative, or mixed mitral regurgitation deemed at high risk or inoperable.
92 in follow-up intervals for TTE assessment of mitral regurgitation, despite risk-adjustment for patien
93 ndependent of age, end-systolic volume, sex, mitral regurgitation, diabetes mellitus, dyslipidemia, c
95 Clip implantation in noncentral degenerative mitral regurgitation (dMR) compared with central dMR.
97 tween EF and mortality in 1875 patients with mitral regurgitation due to flail leaflets in sinus rhyt
99 ; 64% male) with isolated moderate to severe mitral regurgitation (effective regurgitant orifice area
100 time, ejection time, total isovolumic time, mitral regurgitation, ejection fraction, and blood press
101 After mitral valve repair for degenerative mitral regurgitation, elevated mitral gradients is not u
105 and medical treatment options for functional mitral regurgitation (FMR) are limited and additional in
107 c left ventricular (LV) function, functional mitral regurgitation (FMR), and pulmonary hypertension (
109 quantified as risk factors for recurrence of mitral regurgitation following reduction annuloplasty.
110 py has emerged as an option for treatment of mitral regurgitation for selected, predominantly high-ri
111 tery disease and moderate or severe ischemic mitral regurgitation from 1990 to 2009, categorized by m
112 7 [1.43-2.20]), male sex (1.69 [1.32-2.16]), mitral regurgitation grade >/=III (1.61 [1.15-2.25]), hi
113 - 11 mm Hg to 20 +/- 8 mm Hg; p = 0.06), and mitral regurgitation grade (3.0 +/- 0 vs. 0.8 +/- 0.4; p
114 , defined as valve area >/=1.5 cm(2) without mitral regurgitation >2/4, were obtained in 912 patients
115 ts with echocardiographic moderate or severe mitral regurgitation had baseline CMR scans and were fol
117 SBP was associated with a 26% higher risk of mitral regurgitation (hazard ratio [HR] 1.26; CI 1.23, 1
118 athy of unknown etiology that predisposes to mitral regurgitation, heart failure and sudden death.
120 maller LA volumes, better function, and less mitral regurgitation, HFpEF patients had more atrial fib
121 dence interval [CI] 2.77-11.33), presence of mitral regurgitation (HR 8.13, 95% CI 4.09-12.16), lower
122 (HR, 10.2; 95% CI, 3.2-32.2; P < .001), and mitral regurgitation (HR, 4.78; 95% CI, 1.4-16.0; P = .0
127 (mean age, 61 years +/- 19; nine male) with mitral regurgitation in the 24 hours before mitral valve
129 eatment of asymptomatic patients with severe mitral regurgitation in valve reference centres, in whic
130 clinical risk factors, severity of ischemic mitral regurgitation, incomplete revascularization, and
131 cal risk factors, end-systolic volume index, mitral regurgitation, incomplete revascularization, and
134 s; median EF, 66% [60%-71%]) enrolled in the Mitral Regurgitation International Database (MIDA) regis
143 ographic (TTE) surveillance of patients with mitral regurgitation is indicated to avoid adverse ventr
145 e primary imaging modality for assessment of mitral regurgitation, it may be inadequate or suboptimal
147 ons onto the annulus, coaptation height, and mitral regurgitation jet height in 261 Framingham Offspr
148 iteria, first tested in a preliminary phase, mitral regurgitation jet length>/=2 cm or any aortic reg
149 s with MVP (17%) had moderate or more severe mitral regurgitation (jet height >/=5 mm) and 5 others (
151 diate results (valve area >/= 1.5 cm(2) with mitral regurgitation </= 2/4) were obtained in 251 patie
154 age and postprocedural variables, including mitral regurgitation, mean gradient, and pulmonary press
155 on the long-term association between SBP and mitral regurgitation (mediator-adjusted HR 1.22; CI 1.20
156 itral valve annuloplasty (RMA) for secondary mitral regurgitation might cause functional mitral steno
158 ViR group had more frequent post-procedural mitral regurgitation moderate or higher (19.4% vs. 6.8%;
159 lar ejection fraction, worse post-procedural mitral regurgitation, moderate or severe lung disease, d
160 lve (MV) disease is a common cause of severe mitral regurgitation (MR) and accounts for the majority
162 this study was to characterize patients with mitral regurgitation (MR) and atrial fibrillation (AF) t
164 uretic peptide (BNP) may predict outcomes of mitral regurgitation (MR) are plagued by small size, inc
165 ra, California) in patients with significant mitral regurgitation (MR) at high risk of surgical morta
166 l aggregation has been described for primary mitral regurgitation (MR) caused by mitral valve prolaps
167 alternative for patients with severe primary mitral regurgitation (MR) considered at high or prohibit
171 The risk for and consequences of recurrent mitral regurgitation (MR) following degenerative mitral
175 nal morphology of mitral valve and degree of mitral regurgitation (MR) in mitral valve prolapse.
177 This study aimed to evaluate treatment of mitral regurgitation (MR) in patients with severe DMR at
179 t subclavian artery, misdiagnosed as primary mitral regurgitation (MR) in transthoracic echocardiogra
186 Undersized ring annuloplasty for ischemic mitral regurgitation (MR) is associated with variable re
188 iseases of the mitral valve (MV) that causes mitral regurgitation (MR) is broad, and there is limited
191 ere aortic stenosis and concomitant relevant mitral regurgitation (MR) is present in 30% to 55% of th
198 of this study was to evaluate the impact of mitral regurgitation (MR) on outcomes after transcathete
199 e is known of the prognostic significance of mitral regurgitation (MR) on transcatheter aortic valve
203 nts with chronic severe primary degenerative mitral regurgitation (MR) remains controversial, and fur
206 tic patients make the accurate assessment of mitral regurgitation (MR) severity even more important.
209 stroke volume technique (patients) to assess mitral regurgitation (MR) severity using real-time volum
210 onsecutive patients with severe degenerative mitral regurgitation (MR) were treated with a mitral val
211 though surgery is indicated in patients with mitral regurgitation (MR) when left ventricular (LV) end
212 lve Edge-to-Edge Repair Study), treatment of mitral regurgitation (MR) with a novel percutaneous devi
213 ering by displaced papillary muscles induces mitral regurgitation (MR), which doubles mortality.
222 on fraction, 60% [45%-67%]; all </= moderate mitral regurgitation; n=6 with previous cardiac arrest a
223 sms and treatments of nonischemic functional mitral regurgitation (NIMR) are not fully established, i
224 l patients, resulting in procedural residual mitral regurgitation of grade 2+ or less in 22 (96%) pat
226 The rate of recurrence of moderate or severe mitral regurgitation over 2 years was higher in the repa
229 Mean age was 77+/-14 years; 71% male (n=29); mitral regurgitation pathogenesis was functional in 54%
230 2000 and December 2011 (excluding functional mitral regurgitation, prior valvular surgery, hypertroph
232 2014, with hospital mortality of 2% and with mitral regurgitation reduced to grade </=2 in 87% of pat
233 greater tubular ascending aorta, presence of mitral regurgitation, reduced left ventricular ejection
234 ographic core analysis after 6 months showed mitral regurgitation reduction in 50% of treated patient
235 +/-12 years, 138 males) with chronic organic mitral regurgitation referred to surgery underwent an ec
239 % versus 69%; P=0.003), and in patients with mitral regurgitation, reproducibility was improved with
240 months for severe, moderate, mild, and trace mitral regurgitation, respectively, with 20% of provider
241 alysis, exercise ejection fraction, exercise mitral regurgitation, rest and exercise tricuspid annula
242 spid valve, moderate aortic stenosis, severe mitral regurgitation, severe aortic regurgitation, or su
243 There was an interaction between TR and mitral regurgitation severity (P=0.04); the increased ha
244 le quantitation of cardiac remodeling and of mitral regurgitation severity and of annular and valvula
245 que is complex, and common methods to define mitral regurgitation severity based on 2-dimensional (2D
251 t ventricular (LV) dysfunction and secondary mitral regurgitation (SMR) are still controversial.
254 with outcomes, mixed data on SMR and primary mitral regurgitation, studies not clearly reporting the
256 AS) was significantly lower in patients with mitral regurgitation than in healthy control subjects (P
257 mitral prolapse is the most common cause of mitral regurgitation that requires surgical treatment.
258 High Risk Patients with Severe, Symptomatic Mitral Regurgitation - The Twelve Intrepid TMVR Pilot St
261 flow tract (LVOT) obstruction and associated mitral regurgitation, thereby leading to amelioration of
262 assigned 301 patients with moderate ischemic mitral regurgitation to CABG alone or CABG plus mitral-v
263 he role of EF in a large registry of organic mitral regurgitation to objectively establish thresholds
264 y assigned 251 patients with severe ischemic mitral regurgitation to undergo either mitral-valve repa
265 ay determine whether the lower prevalence of mitral regurgitation translates into a net clinical bene
266 o evaluate the relationship between ischemic mitral regurgitation treatment strategy and survival.
267 tery disease and moderate or severe ischemic mitral regurgitation undergoing CABG alone demonstrated
269 tients with grade III+ or greater myxomatous mitral regurgitation undergoing exercise echocardiograph
271 te-to-severe (grade 3+) or severe (grade 4+) mitral regurgitation using the Edwards PASCAL TMVr syste
272 to reduce systolic anterior motion (SAM) and mitral regurgitation using the transcatheter mitral clip
273 odynamic disorders can result from eccentric mitral regurgitation usually caused by chordae tendinae
275 ion II score and presence of sepsis, whereas mitral regurgitation was associated with longer hospital
277 h subpapillary infarction and impaired IPMD, mitral regurgitation was evident within 1 week, compared
278 e 2-year rate of moderate or severe residual mitral regurgitation was higher in the CABG-alone group
280 PERM) by these proximate causes of secondary mitral regurgitation was only 13% (CI 6.1%, 20%), and ac
281 replacement in patients with severe ischemic mitral regurgitation, we found no significant difference
282 ve repair in patients with moderate ischemic mitral regurgitation, we found no significant difference
283 ve repair or replacement for severe ischemic mitral regurgitation, we observed no significant between
284 d that patients with </= mild postprocedural mitral regurgitation were 4-fold more likely to experien
286 ariable analysis, the predictors of improved mitral regurgitation were the decrease of tenting area (
287 stolic volume was increased in patients with mitral regurgitation when compared with that in healthy
288 output and mechanical efficiency and reduced mitral regurgitation, whereas reduction in chamber volum
289 our primary outcome was incident reports of mitral regurgitation, which were identified from hospita
290 ucing good outcomes in patients with primary mitral regurgitation who are at high surgical risk.
291 % men) with grade III+ or greater myxomatous mitral regurgitation who underwent exercise echocardiogr
292 egistry enrolling patients with degenerative mitral regurgitation with a flail leaflet in 6 tertiary
295 SBP was continuously related to the risk of mitral regurgitation with no evidence of a nadir down to
296 of mitral valve prolapse and its associated mitral regurgitation with no or minimal requirements for
297 tus and factors influencing the reduction of mitral regurgitation with or without mitral leaflet teth
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