ライフサイエンスコーパス: mitral stenosis

1 matic valvular disease, or greater than mild mitral stenosis).

2 a further 1,262 (0.02%) were diagnosed with mitral stenosis.

3 treatment in selected patients with calcific mitral stenosis.

4 missurotomy (PMC) remain debated in calcific mitral stenosis.

5 rly, and those with more than mild aortic or mitral stenosis.

6 sis, and treatment options for patients with mitral stenosis.

7 ell, and no patient had clinical evidence of mitral stenosis.

8 actical technique for clinical evaluation of mitral stenosis.

9 nly used exercise test for the evaluation of mitral stenosis.

10 471 with aortic stenosis, and 193 with mild mitral stenosis.

11 illary basement membrane in diseases such as mitral stenosis.

12 ns in leaflet geometry seen in patients with mitral stenosis.

13 he application of balloon commissurotomy for mitral stenosis.

14 atients with a larger initial MVA and milder mitral stenosis (0.12 vs. 0.06 vs. 0.03 cm2/year for mil

15 t prolapse (17), chordal shortening (1), and mitral stenosis (1).

16 ated mitral valve replacement or evidence of mitral stenosis: A total of 504 (54%) had congenitally m

17 age, larger aortic root diameter, aortic and mitral stenosis and albuminuria.

18 Finally, mitral stenosis and mitral regurgitation studies evaluat

19 sing problem in elderly people, causing both mitral stenosis and regurgitation which are difficult to

20 tary effects of mitral balloon valvotomy for mitral stenosis and the development of a reasonable appr

21 alyzed long-term results of PMC for calcific mitral stenosis and the factors associated with late fun

22 Mitral stenosis and thromboembolism did not develop.

23 se with mitral regurgitation with or without mitral stenosis) and the multivalvular lesions (MVL) cat

24 creased after valvuloplasty in patients with mitral stenosis, and (2) whether the magnitude of the in

25 rable effect on the hemodynamic variables of mitral stenosis, and long-term follow-up data suggest th

26 rotid artery for aneurysm, John Abernethy on mitral stenosis, and Sir David Dundas on acute rheumatic

27 se with prosthetic heart valves, significant mitral stenosis, and valvular heart disease (VHD) requir

28 modynamics were measured in 57 patients with mitral stenosis before and 20 to 30 min after undergoing

29 use of OMC in 312 consecutive patients with mitral stenosis between 1983 and the present.

30 This effect has been recognized in mitral stenosis but assumed to be absent in aortic steno

31 Previously asymptomatic mitral stenosis can lead to remarkably sudden developmen

32 tral stenosis (DMS) is an important cause of mitral stenosis, developing secondary to severe mitral a

33 Degenerative mitral stenosis (DMS) is an important cause of mitral st

34 Because patients with mitral stenosis frequently exhibit greater improvements

35 e was no association between SBP and risk of mitral stenosis (HR per 20 mmHg higher SBP 1.03; CI 0.93

36 aortic regurgitation and without associated mitral stenosis) in adults in the Western world has been

37 Mitral stenosis is a common disease that causes substant

38 Balloon mitral valvuloplasty (BMV) for mitral stenosis is a procedure that has evolved signific

39 rience with congenital interatrial shunts in mitral stenosis, it has been hypothesized that the creat

40 sults of PMC are less satisfying in calcific mitral stenosis, long-term functional outcome depends on

41 from 1993 to 2005 that was unassociated with mitral stenosis, mitral valve replacement, or a previous

42 ies typical of the spectrum in patients with mitral stenosis: mobile doming, intermediate conical, an

43 igher prevalences of aortic stenosis (AS) or mitral stenosis (MS) (p<0.001).

44 mostly in patients with moderate and severe mitral stenosis (MS) and AS.

45 Severe congenital mitral stenosis (MS) is a rare anomaly that is frequentl

46 ant determinant of pulmonary hypertension in mitral stenosis (MS).

47 gitation (MR) and 16 with moderate to severe mitral stenosis (MS).

48 ients with significant mitral valve disease (mitral stenosis or > or = moderate mitral regurgitation)

49 Patients with moderate to severe mitral stenosis or mechanical heart valves were excluded

50 farin excluded patients with moderate/severe mitral stenosis or mechanical heart valves, but variably

51 mitral valve replacement provides relief of mitral stenosis or mitral regurgitation.

52 aortic regurgitation) and without associated mitral stenosis or mitral valve replacement strongly sug

53 history of atrial fibrillation, a pacemaker, mitral stenosis, or congenital heart disease.

54 utaneous approach is usually used to correct mitral stenosis, other valve lesions require surgical in

55 able on the echocardiographic progression of mitral stenosis, particularly on progressive changes in

56 , intensive care, right ventricular failure, mitral stenosis, prostacyclin, nitric oxide, sildenafil,

57 for mitral insufficiency, whereas congenital mitral stenosis remains extremely problematic in the you

58 In mild mitral stenosis, the half-time was approximately 100 mse

59 lidation n=121) of patients with symptomatic mitral stenosis undergoing PMV were studied.

60 Seventeen patients with mitral stenosis underwent echocardiography and CMR.

61 It also examines the following: 1) mitral stenosis versus mitral regurgitation and the pres

62 CBC for the treatment of mitral stenosis was performed in 132 patients from 1986

63 Thus, in selected patients with mitral stenosis who require an interventional procedure,

64 of 314 patients undergoing PMC for calcific mitral stenosis with 710 patients with noncalcified valv

65 mitral regurgitation might cause functional mitral stenosis, yet its clinical impact and underlying