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1 rogeneity) and reinfection (which results in mixed infections).
2 .3% of the strains had more than one G type (mixed infections).
3 f pandemic H1N1/2009, strongly suggestive of mixed infection.
4 ification of bone loss in the non-capsulated mixed infection.
5  North America, at least 11% of patients had mixed infection.
6 (16%) specimens as short RNA patterns, and 1 mixed infection.
7  the combination of serotypes present during mixed infection.
8  same strain or with a different strain, and mixed infection.
9 the drug-susceptible strain in patients with mixed infection.
10 ibuted 10 cases to reinfection and 1 case to mixed infection.
11 r new mutation, were found after 2 months of mixed infection.
12 ism that might be operative in a scenario of mixed infection.
13 cific killing of bystander bacteria during a mixed infection.
14 amydia trachomatis serovars and detection of mixed infection.
15 ely outcompeted by the wild-type strain in a mixed infection.
16  identification of serial isolates in murine mixed infection.
17 wer in male-only worm infection than that of mixed infection.
18 nt is less competitive than the wild type in mixed infection.
19 gocytosis, compared with that of heat-killed mixed infection.
20 t of bacterial viability on the virulence of mixed infection.
21 tosis of P. gingivalis was reduced following mixed infection.
22 ctor-alpha (TNF-alpha) levels induced by the mixed infection.
23  numbers of AZ(2) than those of SP(6) in the mixed infection.
24 d against one another in vivo in a series of mixed infections.
25  technique can quantify uncommon variants in mixed infections.
26 tory strategies to aid in the recognition of mixed infections.
27 nct strains, are well suited to detection of mixed infections.
28 out-competed the tir tyrosine mutants during mixed infections.
29 colonization of BALB/c-ByJ mice in single or mixed infections.
30 or more derepressed mutants may occur during mixed infections.
31 tional 40 viral infections, of which 11 were mixed infections.
32 arum infections, and 4% were predicted to be mixed infections.
33 , we demonstrate several dramatic effects of mixed infections.
34 exhibits the essential patterns of long-term mixed infections.
35 t commensals and pathogens in sequential and mixed infections.
36                              No cheetahs had mixed infections.
37  there is competition between viruses during mixed infections.
38               We also looked for evidence of mixed infections.
39 ciently compete with the wild-type strain in mixed infections.
40 s and is outcompeted by wild-type strains in mixed infections.
41 lity to compete with the wild-type strain in mixed infections.
42 ffers an exceptional advantage for detecting mixed infections.
43 y associated with diarrhoea in children with mixed infections.
44 es identification and higher sensitivity for mixed infections.
45  to these lineages and 12 (1.4%) represented mixed infections.
46 ections, but different between the clones in mixed infections.
47 y explain why F. nucleatum is often found in mixed infections.
48                        Two patients (4%) had mixed infection (1 + 3, 1 + 4).
49 nding risks were 5.4 (95% CI, 5.02-5.80) for mixed infections, 3.73 (95% CI, 3.51-3.97) for Plasmodiu
50 flanking genetic markers from the progeny of mixed infections, a novel selective advantage imparted b
51 enetic markers predominate in the progeny of mixed infections, a phenomenon termed marker exclusion.
52 -only infections, (2) gram-positive-only and mixed infections-adequate GNC, and (3) gram-negative-onl
53                     There was no evidence of mixed infections among those classified as reinfections.
54 netic and phenotypic diversity, both through mixed infection and de novo mutation.
55 n our inability to exclude the occurrence of mixed infection and template switching during amplificat
56 o attenuation of bone loss in the capsulated mixed infection and to intensification of bone loss in t
57              Ten samples (28.6%) exhibited a mixed infection and were positive for both Haemophilus a
58                                              Mixed infections and 'unknown' organisms may be importan
59 ing our understanding of the pathogenesis of mixed infections and in identifying ways to use this bet
60 ram-negative-only infections (n = 9) or with mixed infections and inadequate GNC (n = 4) and all had
61 SP1(33) genotype infection was 3D7, with few mixed infections and no sole FVO infections.
62              Interactions of retroviruses in mixed infections and their effects on disease induction
63  induced by a P. gingivalis and F. nucleatum mixed infection, and also on the local host response usi
64 btypes coinfect the same cells, generating a mixed infection, and then reassort.
65 nt) were due to P. ovale, 3 (3 percent) were mixed infections, and 2 (2 percent) were due to unidenti
66 method identified four previously undetected mixed infections, and a previously undetected transmissi
67 ntly in children, mostly in association with mixed infections, and are recovered mixed with mouth and
68 rgence through local evolution, host shifts, mixed infections, and global spread.
69 tibility and relative fitness in settings of mixed infections, and raise important considerations abo
70                                              Mixed infections appear to be rare in our setting and ar
71               In hyperendemic settings where mixed infections are common, the Xpert results might nee
72                                              Mixed infections are infrequently reported, potentially
73                                              Mixed infections are the rule rather than the exception
74                                    Moreover, mixed infections are underestimated by routine microscop
75 This study aimed to describe reinfection and mixed infection as underlying mechanisms of changing dru
76 selection resulting from reassortment during mixed infections, better regulation of gene expression,
77                                           In mixed infection, both IL-1beta and IL-10 levels were aug
78 ial neoplasia was found more frequently with mixed infection by moderate-risk human papillomavirus ty
79                    Samples were screened for mixed infections by denaturant gel gradient electrophore
80                    Four samples diagnosed as mixed infections by microscopy and single infection by t
81               We propose that these types of mixed infections, by increasing the ability of low fitne
82               These results demonstrate that mixed infections can be detected without additional sequ
83  comparison to the single-species infection, mixed infection caused significantly increased inflammat
84  B. pertussis heme system was assessed using mixed infection competition experiments in a mouse model
85                                     Previous mixed infection competition studies established the impo
86                         In this study, using mixed-infection competition experiments in a mouse respi
87 rotavirus strains and the high prevalence of mixed infections confirm trends reported earlier and hel
88                                              Mixed infections containing Fn and non-capsulated or cap
89 ammation and dilatation resulting from these mixed infections correlated directly with the amount of
90 a has used a PCR approach and suggested that mixed infection could be common.
91                   The lower response to live mixed infection could stem from a mechanism enabling the
92 0 (53.4%) had cellulitis, and 82 (15.6%) had mixed infection, defined as at least one abscess lesion
93                                          The mixed infections dramatically altered the behavior of th
94 luble PT produced by the wild-type strain in mixed infections enhanced respiratory tract colonization
95 vels than those with a pure gram-positive or mixed infection, especially in the late shock group (142
96 of our algorithm with in silico and in vitro mixed infection experiments, and apply it to transmissio
97 hanisms underlying these late-phase effects, mixed-infection experiments with mutant and wild-type vi
98                                           In mixed-infection experiments, the DeltaCYA strain was sig
99                                       During mixed-infection experiments, the S. typhimurium invA lpf
100  Interactions among the retroviruses of this mixed infection facilitate disease induction.
101                                In the latter mixed infection, Fn evaded phagocytosis, whereas in the
102                             The relevance of mixed infections for the patient, clinician, and control
103 tes collected in a population to distinguish mixed infections from clonal heterogeneity.
104 ss-reactivity; the inability to discriminate mixed infections from nonspecific cross-reactivity is on
105 rial sequence data, direct reconstruction of mixed infection haplotypes from current short-read seque
106 olation of bacteria in pure bacterial and/or mixed infection; however, their role in isolating fungus
107 n analysis in 9 of the 11 cases demonstrated mixed infection in 5 cases, reinfection in 3 cases, and
108 tum, response to treatment, and detection of mixed infection in clinical specimens.
109 heterodiploids over wild-type cells during a mixed infection in the mouse abscess model.
110  the FilmArray and Luminex panels identified mixed infections in 21.1% and 13.0% of positive prospect
111  1 in other mammals and the low frequency of mixed infections in humans is the inability of type 1 to
112 ever, it is limited in its ability to detect mixed infections in multiplex assay platforms without th
113                              We investigated mixed infections in northern Malawi using two lineage-sp
114  resistant C. trachomatis that originated in mixed infections in our experiments and demonstrate gene
115 influenza patients we reveal a high level of mixed infection, including diverse lineages of the same
116                          However, undetected mixed infections-infection with >/=2 unrelated strains o
117 is OG1RF and TX5179 strains were tested in a mixed-infection (inoculum, approximately 1:1) mouse urin
118 the pathogenic bacteria are often not known, mixed infections involving both Gram-negative and Gram-p
119                                  Analysis of mixed infections involving single and double mutant stra
120         Most malaria-endemic regions feature mixed infections involving two or more of these species.
121            The inflammatory response to live mixed infection is attenuated with reduced phagocytosis,
122                               In cases where mixed infection is confirmed, the dominant and minor hap
123 hod to distinguish clonal heterogeneity from mixed infections is based on a rather simple rule.
124                     A priori, the outcome of mixed infections is hard to predict.
125 tion of each serotype to pathogenesis during mixed infections is not known.
126                                Despite this, mixed infections lead to pseudotyping, the association o
127 hat, in some patients with MDR tuberculosis, mixed infection may be responsible for observations attr
128 ize direct clinical samples and can identify mixed infections may contribute significantly to studies
129 ocandin-resistant C. albicans in competitive mixed infection models.
130 = 27.3%, 2a = 7.4%, 2b = 8.3%, 3a = 14%, and mixed infection (more than one subtype) = 3.3%.
131                            After enrollment, mixed infections (multiplicity of infection, >1) were on
132                                           In mixed infections mutant ICP0 is stable, whereas the wild
133                               After removing mixed infections (n = 17), 83% of H. pylori strains had
134 robiological analyses, it was found that, in mixed infection, NF1 selectively disseminated to mouse p
135                                           In mixed infection, none of the cases required liquid media
136 ability to compete with wild-type virus in a mixed infection of 3-day-old ducklings.
137 ion-specific retinal ganglion cells, because mixed infection of a gE deletion mutant and a gI deletio
138                                              Mixed infection of a gE deletion mutant and a gI deletio
139 trains of Helicobacter pylori during natural mixed infection of a patient in Lithuania.
140 pose that the recombinant virus arose during mixed infection of a single individual in northern China
141                                              Mixed infection of LLC-PK1 cells with wt RPV and wt CPV
142 r a strain that made only the Tf receptor in mixed infection of male volunteers.
143 CM was developed to identify both single and mixed infection of P. falciparum and Plasmodium vivax.
144                                   However, a mixed infection of TYLCV with cucumber mosaic virus comp
145                          From animals with a mixed infection of viruses that were resistant and sensi
146           We validated this prediction using mixed infections of a wild-type Trpv1 expression vector
147 methicillin-susceptible S. aureus (MSSA), or mixed infections of CoNS and MRSA, CoNS and MSSA, or non
148 les were probably derived from patients with mixed infections of Enterocytozoon bieneusi and E. intes
149 ave investigated protective immunity against mixed infections of genetically distinct cloned "strains
150                            The occurrence of mixed infections of Mycobacterium tuberculosis is no lon
151 l mixing of individuals and the incidence of mixed infections of N. gonorrhoeae.
152                              We show that in mixed infections of the bacterium Pseudomonas aeruginosa
153 sortment of BTV gene segments during natural mixed infections of vertebrate and invertebrate hosts.
154  a sufficient frequency in vivo to result in mixed infections on the mucosal surface of human hosts,
155 ble cause of deleterious immune responses to mixed infection or immunization and call for a greater u
156 re a consequence of species diversity due to mixed infections or minor contaminants as measured by UB
157  infections caused by single strains than in mixed infections (P < 0.05), whereas serotype G1 strains
158                                              Mixed infections (P. falciparum and P. vivax) were ident
159 aded phagocytosis, whereas in the capsulated mixed infection Pg displayed a greater capacity for inva
160 ations based on quantification, including in mixed infections, providing rich and unprecedented measu
161  phagocytosis pattern of F. nucleatum in the mixed infection remained similar to that upon monoinfect
162  malariae/Plasmodium knowlesi infection, and mixed infection, respectively.
163 2 during coinfection, 3D spatial analysis of mixed infections revealed that DspB is required for Aa t
164                                 In contrast, mixed infections showed a significantly reduced in vitro
165     When the two variants were combined in a mixed infection, SP6 outcompeted AZ2 in the lower genita
166 otein synthesis in the Us9 null mutants, and mixed-infection studies of rats, we conclude that the re
167 serotype G1 strains were found more often in mixed infections than in infections caused by single str
168 ore total viruses either alone or as part of mixed infections than the xTAG RVP, as well as an additi
169  Peri-implantitis represents a heterogeneous mixed infection that includes periodontopathic microorga
170 ositive and gram-negative pathogens or both (mixed infections), the adequacy of gram-negative coverag
171                                           In mixed infection, the capsule also participates in coaggr
172  in competition with wild-type Yersinia in a mixed infection, the yopJ mutant strain is deficient for
173                                           In mixed infections, the competitive advantage of a yopM(+)
174                           Similar to in vivo mixed infections, the polytropic MuLV genome was extensi
175 void suppression by more virulent species in mixed infections, thereby increasing transmission opport
176  cultured M. tuberculosis isolates and found mixed infections to be rare.
177                                           In mixed infections transcripts for both types were detecte
178                         From MHV-A59/MHV-JHM mixed infection, variant viruses (MHV-H1 and MHV-H2) whi
179                                           In mixed infections, virus synergism is proposed to result
180                                Prevalence of mixed infection was assessed by random amplified polymor
181                                              Mixed infection was detected in 3.7% of specimens, and 4
182                       A higher proportion of mixed infection was found when biopsies were used.
183                               Synergy in the mixed infections was defined as a statistically signific
184                                 Moreover, in mixed infections we predict there to be an interaction b
185        Six cases of relapse and six cases of mixed infection were classified differently by whole-gen
186                  Multiple species suggesting mixed infection were isolated from 27 cultures.
187               Progeny virions recovered from mixed infection were passaged in the presence and absenc
188 r likely transmission events associated with mixed infection were selected.
189  six recognized species were identified, and mixed infections were commonly found in this group of pa
190                                 In addition, mixed infections were detected in three subjects, with c
191         To date, the analyses of plant virus mixed infections were limited to reports of emerging sym
192                            Four samples with mixed infections were misdiagnosed by microscopy as sing
193 thermore, some discrepancies in detection of mixed infections were observed and were addressed by rev
194                                              Mixed infections were present but gave mixed nodules les
195  consisting of different malaria species and mixed infections were used to test the utility of the no
196 ublineages, as well as distinct strains from mixed infections, were differentially distributed throug
197 ad protective effects against LT(+) ETEC and mixed infections when using a toxin-based enteric vaccin
198 llows reassortment of genome segments during mixed infections, which is the major distinguishing feat
199                             The frequency of mixed infection with both genotypes in humans is relativ
200 cription of bovine group B rotaviruses (in a mixed infection with bovine coronavirus or singly in fec
201                                              Mixed infection with capsulated Pg augmented alveolar bo
202 d and were found to be capable of persistent mixed infection with certain H. pylori strains.
203 ts diversification of a founding strain or a mixed infection with distinct strain populations is not
204                                              Mixed infection with epidermodysplasia verruciformis, cu
205 und to serve as a unique virulence factor in mixed infection with Fn.
206 esponsible for the severity of disease after mixed infection with Fn.
207 apsule as a virulence factor in coaggregated mixed infection with Fusobacterium nucleatum (Fn).
208 1.2 %), genotype A in 55 samples (7.7 %) and mixed infection with genotypes A and D in 124 samples (1
209  and passage of both types was achieved as a mixed infection with HPV-40 predominating.
210      Determining the nature and frequency of mixed infection with influenza virus is therefore centra
211 urprisingly, there was no evidence that in a mixed infection with its parent, the ompU-defective stra
212 ted alveolar bone loss compared with that of mixed infection with non-capsulated Pg.
213 yzable material [NDM]) on the virulence of a mixed infection with Porphyromonas gingivalis and Fusoba
214 se is an important factor in the dynamics of mixed infection with strains that display different path
215 ate the virB mutant from the spleen during a mixed infection with the B. abortus wild type.
216 ial cells and inhibited IP-10 secretion in a mixed infection with the otherwise stimulatory Fusobacte
217 uction in a murine abscess model elicited by mixed infection with the periodontopathogens Fusobacteri
218          This study is the first to report a mixed infection with the same ST.
219 oreover, in a murine model of periodontitis, mixed infection with the two species induces synergistic
220                We have previously shown that mixed infection with two avirulent HSV-1 strains (OD4 an
221  days apart as the same CDI, 7% of cases had mixed infections with >1 genotype.
222  survives (3D7), the coinfection resulted in mixed infections with both live and encapsulated parasit
223 tal), while uncommon types represented 5.7%, mixed infections with common types represented 14.8%, an
224                                        Using mixed infections with green fluorescent protein (GFP) an
225 C, and (3) gram-negative-only infections and mixed infections with inadequate GNC.
226 gle tests and was shown to be able to detect mixed infections with no loss in sensitivity.
227 s associating respiratory tract biofilms and mixed infections with pneumococcal serotypes of lower vi
228                                              Mixed infections with seasonal influenza A virus strains
229 2 are partially excluded from the progeny of mixed infections with the related phage T4 (general, or
230 ation of the bladder or kidneys in single or mixed infections with the wild type and CP9Delta hlyA(1)
231 ther isolates, suggesting the possibility of mixed infections with two divergent isolates.
232                                  Analysis of mixed infections with two WSMV isolates suggests that ab
233 f experimental periodontitis compared with a mixed infection without NDM treatment.
234 mputationally efficient method for detecting mixed infection without the need for resource-intensive

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