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1 emory T-helper or T-cytotoxic cells in 16-hr mixed leukocyte reaction.
2 -cell stimulatory potential in an allogeneic mixed leukocyte reaction.
3 T-cell proliferation and differentiation in mixed leukocyte reaction.
4 ultured them as stimulators in an allogeneic mixed leukocyte reaction.
5 y suppress T cell proliferative responses by mixed leukocyte reaction.
6 proinflammatory cytokines, was determined in mixed leukocyte reaction.
7 ed to be strong stimulators for T cells in a mixed leukocyte reaction.
8 by proliferation and cytokine secretion in a mixed leukocyte reaction.
9 reated monocytes are better stimulators in a mixed leukocyte reaction.
10 FN)-gamma production, and stimulation of the mixed leukocyte reaction.
11 y, and the ability to stimulate T cells in a mixed leukocyte reaction.
12 ase in DC-mediated T cell proliferation in a mixed leukocyte reaction.
13 ed proliferative responses of donor cells in mixed leukocyte reaction.
14 eta (V beta) repertoire as they occur in the mixed leukocyte reaction.
15 al blood T cells and naive CD4(+) T cells in mixed leukocyte reactions.
16 D4 T cell response than the H2-O(+/+) DCs in mixed leukocyte reactions.
17 derwent three or more divisions from primary mixed leukocyte reactions.
18 hancing their immunostimulatory functions in mixed leukocyte reactions.
19 T helper (Th) 1 (mainly) and Th2 cells in 3d-mixed leukocyte reactions.
20 tometric analysis, respectively, after 3-day mixed leukocyte reactions.
21 ing function of DC was determined in one-way mixed leukocyte reactions.
22 ation and cytotoxic T lymphocytes in primary mixed leukocyte reactions.
23 e monoclonal antibodies (mAb) were tested in mixed leukocyte reactions.
24 most potent stimulatory cells in allogeneic mixed leukocyte reactions.
25 sence of NB cells was observed in allogeneic mixed leukocyte reaction (33,508 +/- 1,613 cpm for contr
26 r capacity to stimulate a primary allogeneic mixed leukocyte reaction (8-fold increase in stimulation
27 de of ex vivo psoriatic DCs in an allogeneic mixed leukocyte reaction also showed a decrease in IL-17
28 que, and for anti-donor reactivity using the mixed leukocyte reaction and an ELISA screen for anti-HL
29 nged to 138, 428, and 509 days, and in vitro mixed leukocyte reaction and cell-mediated lympholysis (
30 e toward Gal SLA-matched PBMC were tested by mixed leukocyte reaction and cell-mediated lympholysis a
31 splenocytes and sera were also obtained for mixed leukocyte reaction and complement-mediated microcy
32 ostimulatory function of DCs was assessed by mixed leukocyte reaction and cytotoxic activity in vitro
34 eart transplant) revealed minimal anti-donor mixed leukocyte reaction and cytotoxic T lymphocyte reac
35 vivo by skin transplantation and in vitro by mixed leukocyte reaction and enzyme-linked immunospot (E
36 T cells resulted in a >90% inhibition of the mixed leukocyte reaction and marked protection from leth
37 occupancy was correlated with inhibition of mixed leukocyte reactions and clinical validation was ob
38 nses to donor alloantigen were quantified by mixed leukocyte reaction, and cytotoxic T lymphocyte (CT
39 splenocytes and kidney samples for ELISPOT, mixed leukocyte reaction, and immunohistochemical studie
40 reactivity to donor stimulator cells in the mixed leukocyte reaction, and no detectable serum anti-H
41 Functional maturation of mdDC was tested in mixed leukocyte reactions, and the synthesis of proinfla
43 ral compounds in a standard 2-way allogeneic mixed leukocyte reaction assay, we identified a potent i
47 hibited MHC class I- and class II-restricted mixed leukocyte reactions between the parental strains b
48 d cell-mediated lympholysis responses in the mixed leukocyte reaction, but the fresh and 2-day cultur
50 endent DCs were more potent stimulators of a mixed leukocyte reaction, compared with control GTS cult
51 r CD14(low) and stimulated potent autologous mixed leukocyte reactions, consistent with differentiati
55 cell proliferation to host alloantigens in a mixed-leukocyte reaction does not predict freedom from G
56 ytometry, and their function was assessed by mixed leukocyte reaction, enzyme-linked immunoadsorbent
57 uman peripheral blood mononuclear cells in a mixed leukocyte reaction, HTSU-IgG inhibited proliferati
58 antigen 4/B7 blockade resulting in secondary mixed leukocyte reaction hyporesponsiveness and toleranc
62 and functional assays of one-way allogeneic mixed leukocyte reactions indicated that rAAV-transduced
65 dney transplantations were performed between mixed leukocyte reaction-mismatched, ABO blood group-mat
68 and despite their in vitro alloreactivity in mixed leukocyte reaction (MLR) assays did not cause acut
71 to expand alloantigen-specific Tregs in the mixed leukocyte reaction (MLR) that develops from polycl
72 had superior immunostimulatory capacity in a mixed leukocyte reaction (MLR) when compared with DCs ex
74 scularized heart transplantation, allogeneic mixed leukocyte reaction (MLR), and graft versus host di
76 develop potent stimulating activity for the mixed leukocyte reaction (MLR), and the DCs producing HI
77 itro evidence of donor-specific tolerance by mixed leukocyte reaction (MLR), cell-mediated lysis (CML
81 ated both anti-H-2(k) and anti-H-2(d) CTL in mixed leukocyte reactions, providing evidence that the g
82 expressing the mature phase marker CD83, and mixed leukocyte reaction-stimulatory function were lower
83 the presence of anti-donor reactivity in the mixed leukocyte reaction, suggesting that neither chimer
85 nd allospecific CD154 TcM in 16-hr live-cell mixed leukocyte reaction using multiparametric flow cyto
88 ion with conditioned media from mixed cells (mixed leukocyte reaction) was 16.6 +/- 1.2%, which was h
89 allogeneic CD4 T cells as responder cells in mixed leukocyte reactions, we find that B cells preferen
90 and generation of cytotoxic T lymphocytes in mixed leukocyte reaction were determined by [3H]thymidin
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