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2 ding rabbit antithymocyte globulin (rATG) to mixed lymphocyte co-cultures between human leukocyte ant
4 recipient alloreactivity was not observed in mixed lymphocyte co-cultures when donor-derived cells we
6 s were assayed for suppressor cell activity (mixed lymphocyte coculture assays), clonal deletion (T-c
9 we tested two of the regulators for in vitro mixed lymphocyte culture (MLC) and cytotoxic T lymphocyt
10 myeloid cells, isolated from sex-mismatched mixed lymphocyte culture (MLC) nonreactive siblings, hav
12 nalloreactive T lymphocytes, by performing a mixed lymphocyte culture (MLC) stimulation of donor cell
13 Immunological responses were monitored by mixed lymphocyte culture (MLC), CML, skin graft rejectio
14 duction, as measured by in vitro analysis of mixed lymphocyte culture (MLC), T cell cytotoxicity, T h
17 splant recipients 2 years postoperatively in mixed lymphocyte culture and cell-mediated lympholysis r
18 poresponsiveness of residual CD8+ T cells in mixed lymphocyte culture and cytotoxic T lymphocyte to L
19 cyte responsiveness was measured by in vitro mixed lymphocyte culture and cytotoxic T lymphocyte.
21 demonstrated nonspecific suppression of the mixed lymphocyte culture assays at 90 and 200 days and a
24 ion inhibited donor-specific and third-party mixed lymphocyte culture proliferation in a dose-depende
26 rvival was monitored daily from day 10, with mixed lymphocyte culture responses measured on day 14 or
27 Immunocompetence was assessed by one-way mixed lymphocyte culture using patients' peripheral bloo
28 absence of proliferation of CD4(+) T cells (mixed lymphocyte culture) and the description of an aber
30 were followed by inspection, serial biopsy, mixed lymphocyte culture, and alloantibody determination
31 responses to postnatal boosts) and in vitro (mixed lymphocyte culture, cytotoxicity, and cytokine rel
32 ning, using untreated bone marrow cells from mixed lymphocyte culture-nonreactive normal littermates.
41 cine had no effect on IL-2 production in the mixed lymphocyte culture; therefore, the effect of glyci
43 this stimulatory activity of DBMC, in vitro mixed lymphocyte cultures (MLC) and cell-mediated lympho
49 ion by human T-cell populations generated in mixed lymphocyte cultures or isolated from transplant ne
52 utatively tolerant animals was examined with mixed lymphocyte cultures, cell-mediated lympholysis ass
54 unophenotype profile, suppressive ability in mixed lymphocyte cultures, morphology, and ability to di
59 bset (26-76%) of CD8+ CTL generated in human mixed lymphocyte cultures; cell sorting experiments conf
60 not generate cytotoxic responses in primary mixed-lymphocyte cultures against MHC-disparate (allogen
61 ion on liver cell immunogenicity in vitro in mixed lymphocyte hepatocyte culture (MLHC), in vitro in
63 ocyte hepatocyte culture (MLHC), in vitro in mixed lymphocyte liver nonparenchymal cell culture (MLNP
66 emonstrated by significant depression of the mixed lymphocyte reaction (690+/-119 vs. 3850+/-148 cpm,
68 y T cell activity was assessed by autologous mixed lymphocyte reaction (AMLR), an in vitro assay for
69 ic responder cells taking part in allogeneic mixed lymphocyte reaction (MLR) and cell-mediated lympho
70 o antidonor responsiveness was determined by mixed lymphocyte reaction (MLR) and cell-mediated lympho
71 c analysis and functionally as modulators in mixed lymphocyte reaction (MLR) and cell-mediated lympho
72 e was assessed by skin grafting, and also by mixed lymphocyte reaction (MLR) and cell-mediated lympho
74 ty was assessed by in vitro assays including mixed lymphocyte reaction (MLR) and flow cytometry to de
75 cells stimulate naive T cells in the primary mixed lymphocyte reaction (MLR) and induce MC38 tumor-sp
76 ravenous immunoglobulin [IVIG]) inhibits the mixed lymphocyte reaction (MLR) and induces apoptosis pr
78 phocytic reactivity and cytokine analysis of mixed lymphocyte reaction (MLR) culture supernatants by
79 flow cytometric cell sorting approach after mixed lymphocyte reaction (MLR) culture, we have found t
80 proliferation in dendritic cell-driven allo-mixed lymphocyte reaction (MLR) cultures by more than 90
81 shows that blockade of B7/CD28 in anergizing mixed lymphocyte reaction (MLR) cultures of peripheral b
82 rowth factor (TGF)-beta treatment of primary mixed lymphocyte reaction (MLR) cultures resulted in sec
83 inst cell surface determinants, treatment of mixed lymphocyte reaction (MLR) cultures with interleuki
84 micro-cell-mediated lympholysis (m-CML) and mixed lymphocyte reaction (MLR) in recipients with quies
85 h irradiated allogenic stimulator cells in a mixed lymphocyte reaction (MLR) in the presence of solub
87 The demonstration that stimulation in the mixed lymphocyte reaction (MLR) mapped to the same regio
89 tors) in cell-mediated lympholysis (CML) and mixed lymphocyte reaction (MLR) responses of normal peri
91 noantibodies of these isotypes and displayed mixed lymphocyte reaction (MLR) tolerance to donor pig m
92 ctivated cell sorting analysis of cells from mixed lymphocyte reaction (MLR) treated with MEDI-507 re
93 dard skin grafting, flow cytometry (FC), and mixed lymphocyte reaction (MLR) were used to assess effi
95 , blocked binding of ICAM-1, and inhibited a mixed lymphocyte reaction (MLR) with potency significant
96 nisms of tolerance, we examined in vitro the mixed lymphocyte reaction (MLR), cell-mediated lymphocyt
97 APC function, assessed using an allogeneic mixed lymphocyte reaction (MLR), demonstrated equivalent
102 CD8+ cells were tested for veto activity by mixed lymphocyte reaction (MLR)-induced cell-mediated ly
108 ctor P3 (FOXP3) T-regulatory (Treg) cells in mixed lymphocyte reaction (Treg MLR) and now report addi
109 presenting cells able to induce the primary (mixed lymphocyte reaction [MLR]) and secondary (recall r
111 tudies, in addition to functional allogeneic mixed lymphocyte reaction and accessory-cell dependent m
113 s determined by various parameters including mixed lymphocyte reaction and cytotoxic T lymphocyte ass
115 iferation and interferon-gamma production in mixed lymphocyte reaction and enzyme-linked immunospot a
117 r allogeneic antigen stimulation in both the mixed lymphocyte reaction and in the skin explant assay.
119 with 381 also stimulated a higher autologous mixed lymphocyte reaction and induced a Th1-type respons
120 s showed enhanced functional activity in the mixed lymphocyte reaction and induced T cells to secrete
121 Donor-specific tolerance was assessed with mixed lymphocyte reaction and INF-gamma ELISPOT before (
123 T lymphocyte lytic activity were assessed by mixed lymphocyte reaction assay and 51 Chromium release
126 -cell costimulatory activity, as measured by mixed lymphocyte reaction assay, was lower in patients,
130 a role of iNKT cells in regulating aGVHD, in mixed lymphocyte reaction assays, CD4(-) iNKT cells effe
136 48 hr, however, the T cells from the primary mixed lymphocyte reaction began producing IFN-gamma, con
137 HLA-G, TGF-beta, and IL-10) were tested on a mixed lymphocyte reaction between antigen-presenting cel
139 unoconjugate pretreated donors inhibited the mixed lymphocyte reaction completely without the use of
140 In vitro investigation of baboon anti-pig mixed lymphocyte reaction confirmed that only the indire
141 % CD4/RT1a, 9.28% CD8/RT1a) transplants, and mixed lymphocyte reaction confirmed tolerance in recipie
142 duction and proliferation were assessed in a mixed lymphocyte reaction containing FK734, human T cell
143 CD40:CD40L costimulatory pathway in primary mixed lymphocyte reaction cultures resulted in profound
148 orphology, flow cytometry for phenotype, and mixed lymphocyte reaction for allostimulatory function.
151 y for at least 1 month, and then islets from mixed lymphocyte reaction mismatched primates were infus
152 n preventing T cell function using a one-way mixed lymphocyte reaction model for bone marrow transpla
153 d used either as stimulator cells in one-way mixed lymphocyte reaction or transplanted into naive Bal
154 te acute rejection had marked suppression of mixed lymphocyte reaction proliferation to intact donor
155 from the spleen of P5-primed rats had a high mixed lymphocyte reaction proliferative response to P5 p
156 ociated with a significant reduction in host mixed lymphocyte reaction reactivity and is correlated i
162 6 cells was induced by culture in allogeneic mixed lymphocyte reaction supernatant, an effect largely
165 ) demonstrated evidence of immune deviation; mixed lymphocyte reaction to ACI stimulator cells was vi
166 for more than 4 hr were able to inhibit the mixed lymphocyte reaction to almost background levels, b
167 The chimeric animal was less responsive by mixed lymphocyte reaction to pig-specific stimulators th
168 ory molecules both in vivo and in vitro, and mixed lymphocyte reaction using alloantigen-primed Mac-1
170 ever, evidence of sensitization in antidonor mixed lymphocyte reaction was observed in seven of eight
172 analysis of peripheral blood lymphocytes and mixed lymphocyte reaction were performed before transpla
174 otypes and clonality, cytokine production in mixed lymphocyte reaction, and characterization of intra
175 to stimulate naive T-cell proliferation in a mixed lymphocyte reaction, and increases the survival of
176 to mitogens and Ags, tolerance to donor in a mixed lymphocyte reaction, and normal Ab titers after te
177 profiling, cytokine assays, flow cytometry, mixed lymphocyte reaction, and T-cell proliferation assa
178 s controlling specific immune responses, the mixed lymphocyte reaction, and the structure of Ia antig
179 Cellular immune responses were monitored by mixed lymphocyte reaction, cytometric analysis of graft-
180 ed using fluorescence-activated cell sorter, mixed lymphocyte reaction, enzyme-linked immunospot, sig
181 fusion protein does not inhibit the one-way mixed lymphocyte reaction, immobilized RELT is capable o
182 owed donor-specific nonresponsiveness in the mixed lymphocyte reaction, lack of antidonor IgG antibod
183 fusion protein did not affect the allogeneic mixed lymphocyte reaction, our data indicate that TNFRSF
184 ed DC were capable of inducing an allogeneic mixed lymphocyte reaction, they did so to a significantl
185 ch combining a classical in vitro assay, the mixed lymphocyte reaction, with deep T cell receptor seq
186 he ability to suppress phytohemagglutinin or mixed lymphocyte reaction-induced splenocyte proliferati
188 nd bone were heterotopically transplanted to mixed lymphocyte reaction-mismatched Cynomolgus macaques
190 renal aortic segments were exchanged between mixed lymphocyte reaction-mismatched, blood group-compat
207 aluated ex vivo by enzyme-linked immunospot, mixed lymphocytes reaction, cytotoxic T lymphocyte, and
208 to the CD31 domain 6 and inhibits the human mixed-lymphocyte reaction (MLR) in a specific and dose-d
210 ro anti-donor responsiveness was assessed by mixed-lymphocyte reaction and cell-mediated lympholysis
215 f BEL on human regulatory T cells (Tregs) in mixed lymphocyte reactions (MLR) alone and in combinatio
219 ester-labeled CD4+CD25 high FOXP3+ cells in mixed lymphocyte reactions (MLRs) ("the Treg MLR"), with
220 with more than 95% suppression of allogeneic mixed lymphocyte reactions (MLRs) (29 of 30 donors).
221 cytokine production during donor-antipatient mixed lymphocyte reactions (MLRs) and acute graft-versus
224 ne costimulatory blockade was tested in xeno-mixed lymphocyte reactions (XMLRs) and in natural killer
225 ontrol levels of proliferation in allogeneic mixed lymphocyte reactions and a type 1 (IFN-gamma) cyto
226 ia cells are highly effective stimulators in mixed lymphocyte reactions and can induce generation of
227 T lymphocytes could be detected on day 2 of mixed lymphocyte reactions and continued to increase in
228 atch induces the expression of granzyme B in mixed lymphocyte reactions and in a model of graft-versu
230 tely 16 weeks posttransplant for analysis of mixed lymphocyte reactions and spectratyping of human CD
232 ng T cells in both autologous and allogeneic mixed lymphocyte reactions but less efficient at present
238 activity, skew T(H)1 responses in allogeneic mixed lymphocyte reactions through reduction of IL-4 and
239 mmunizing peptides, as well as having strong mixed lymphocyte reactions to donor cells prior to trans
243 Both pre- and postconversion sera inhibited mixed lymphocyte reactions, although only TAC sera suppr
244 tudies, including cell-mediated lympholysis, mixed lymphocyte reactions, and peptide proliferation as
245 Both agents effectively inhibited rhesus mixed lymphocyte reactions, but the combination was 100
247 ferent T-cell receptor (Tcr) ligand systems: mixed lymphocyte reactions, stimulation of Tcr transgeni
261 us postconversion sera on Treg generation in mixed lymphocyte reactions; (4) peripheral blood nonspec
272 for-mobilized T cells had similar phenotype, mixed lymphocyte reactivity, and Foxp3 gene expression l
275 class II MHC alpha chains inhibited the rat mixed lymphocyte response (MLR) in a dose-dependent mann
276 atment also depressed the secondary in vitro mixed lymphocyte response (MLR) of C57BL/6 splenocytes t
280 onor-recipient pairs with high pretransplant mixed lymphocyte response and cytotoxic T lymphocyte pre
281 n four of four animals tested, and the 5-day mixed lymphocyte response stimulation index and relative
282 ignificantly more IFN-gamma in an allogeneic mixed lymphocyte response than MDC matured without this
283 y reduction of host proliferative responses (mixed lymphocyte response) and depression of anti-donor
284 xic T lymphocyte precursor (CTLp) frequency, mixed lymphocyte response, and antidonor IgG response.
285 ated by cell-mediated lymphocytotoxicity and mixed lymphocyte response, and subsequent donor-matched
287 , while maintaining normal alloresponses, in mixed-lymphocyte-response assays performed after immunos
288 that bortezomib potently inhibited in vitro mixed lymphocyte responses and promoted the apoptosis of
291 ponses, as reflected in augmented anti-mouse mixed lymphocyte responses, cell-mediated cytotoxicity,
294 L), delayed-type hypersensitivity (DTH), and mixed-lymphocyte responses were evaluated in orally tole
297 were divided into blood-group compatible and mixed-lymphocyte, stimulation-mismatched, donor-recipien
299 imulate allogeneic T-cell proliferation in a mixed lymphocyte tumor reaction, and generate autologous
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