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1 an ameliorate the disease phenotype in a SCD model animal.
2 ing enables precise genetic modifications in model animals.
3 or dual color, whole body imaging studies in model animals.
4 encephalographic (EEG) methods in humans and model animals.
5 ng a mouse model of PTSD in wild-type and AD model animals.
6 ge synchrony is evaluated in both humans and model animals.
7 igenesis, embryogenesis, and inflammation in model animals.
8 s are inadequate for MHC typing of these key model animals.
9 of earthworms compared to the well-annotated model animals.
10 DNA do not always correlate with lifespan in model animals.
11 to prolong lifespan in various experimental model animals.
16 Although CNV has been reported in several model animal and plant species, the presence of CNV and
17 creatic islets of several different diabetic model animals and is possibly involved in suppression of
18 luate these hypotheses using cichlid fish as model animals, and although differences in attributes pl
21 red that, on the basis of recent findings in model animals, are expected to be polygenic and regulato
22 s has begun to shed light on this problem by modelling animals as random walkers with scent-mediated
24 l symptoms, 2) a lack of equivalency between model animal behavior and human psychiatric symptoms, an
25 riteria, FDA also used in vitro, ex vivo and model animal data to ensure there was no increased immun
30 logenetic analyses of hundreds of genes from model animals have placed flies closer to vertebrates th
31 wever, recent data, primarily obtained using model animal herpesviruses, suggest that viral miRNAs, w
32 Direct assessment of the vascular lesions of model animals in vivo is important for the development o
33 egulate neural development in all bilaterian model animals indicating that they represent a component
35 gical response of cardiovascular function on model animals is important especially in the early stage
36 C-1alpha) expression were decreased in ADPKD model animal kidneys, with PGC-1alpha expression inverse
39 of spermatogenesis occurring in two pivotal model animals - mouse and Caenorhabditis elegans - and c
46 ndependent of the number of generations, for model animal resources such as the Collaborative Cross (
47 onsiderable evidence in humans and mammalian model animals shows that steroid hormones, which are rel
48 stigated the genome-wide diversity of 76 non-model animal species by sequencing the transcriptome of
50 rformed in simpler organisms or in mammalian model animals supports the feasibility of such multidime
51 targeted disruption of STAT1 were used as a model animal system and infected with the viruses it was
53 we take a conceptually different approach to modelling animal telemetry data for making RSF inference
55 s, and sebaceous glands) for wound repair in model animals, the present study was designed to assess
56 nsity and duration of infection in these two models, animals were given anti-gamma interferon monoclo
59 mice, which we previously characterized as a model animal with construct, face, and predictive validi
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